| IAA | imidazoleacetic acid; indoleacetic acid; infectious agent, arthritis; insulin autoantibody; Internat... |
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| IB | idiopathic blepharospasm; immune body; inclusion body; index of body build; infectious bronchitis; I... |
| IBK | infectious bovine keratoconjunctivitis |
| IBR | infectious bovine rhinotracheitis |
| IBRV | infectious bovine rhinotracheitis virus |
| infectious synovitis | A disease of chickens and turkeys caused by the bacterium Mycoplasma synoviae and characterised by lameness with swollen hocks and foot pads. (05 Mar 2000) |
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| infectious warts | A keratotic papilloma of the epidermis which occurs most frequently in young persons as a result of localised infection by human papilloma virus, usually types 2 and 4; the lesions are of variable duration, eventually undergoing spontaneous regression, and are both exophytic and endophytic, with hyperkeratosis, parakeratosis, hypergranulosis, koilocytosis, and papillomatosis. Synonym: common wart, infectious warts, verruca simplex, viral wart. (05 Mar 2000) |
| ectromelia, infectious | A viral infection of mice, causing oedema and necrosis followed by limb loss. (12 Dec 1998) |
| tissue culture infectious dose | The quantity of a cytopathogenic agent, such as a virus, that will produce a cytopathic effect in 50% of the cultures inoculated. (05 Mar 2000) |
| keratoconjunctivitis, infectious | Infectious diseases of cattle, sheep, and goats, characterised by blepharospasm, lacrimation, conjunctivitis, and varying degrees of corneal opacity and ulceration. In cattle the causative agent is moraxella (moraxella) bovis; in sheep, mycoplasma, rickettsia, chlamydia, or acholeplasma; in goats, rickettsia. (12 Dec 1998) |
| feline infectious enteritis | A highly contagious and fatal disease of cats, particularly young cats, caused by feline panleukopenia virus, a member of the family Parvoviridae, and manifested by severe leukopenia, prostration, fever, vomiting and diarrhoea. Synonym: distemper, feline agranulocytosis, feline distemper, feline infectious enteritis. (05 Mar 2000) |
| feline infectious peritonitis | Common coronavirus infection of cats caused by the feline infectious peritonitis virus (infectious peritonitis virus, feline). The disease is characterised by a long incubation period, fever, depression, loss of appetite, wasting, and progressive abdominal enlargement. Infection of cells of the monocyte-macrophage lineage appears to be essential in fip pathogenesis. (12 Dec 1998) |
| achlorhydric anaemia | A form of chronic hypochromic microcytic anaemia associated with achlorhydria or achylia gastrica; observed most frequently in women in the third to fifth decades. Synonym: Faber's anaemia, Faber's syndrome. (05 Mar 2000) |
| achrestic anaemia | A form of chronic progressive macrocytic anaemia that can be fatal in which the changes in bone marrow and circulating blood closely resemble those of pernicious anaemia, but in which there is only transient or no response to therapy with vitamin B12; glossitis, gastrointestinal disturbances, central nervous system disease, and pyrexia are not observed, and there is only little bleeding or haemolysis. Origin: G. A-priv. + chresis, a using (05 Mar 2000) |
| acquired haemolytic anaemia | Nonhereditary acute or chronic anaemia associated with or caused by extracorpuscular factors, e.g., certain infectious agents, chemicals (including autoantibodies or therapeutic agents), burns, toxic materials from higher plant and animal forms (including snake venoms). (05 Mar 2000) |
| addisonian anaemia | <haematology> A form of anaemia (low red blood cell counts) that results when the bone marrow fails to produce adequate numbers of red blood cells due to a deficiency in vitamin B12. Intrinsic factor, necessary for normal B12 absorption, may be the underlying cause for B12 deficiency if is not produced in the gastric glands (in the stomach). Origin: Gr. Haima = blood (27 Sep 1997) |
| Addison's anaemia | <haematology> A form of anaemia (low red blood cell counts) that results when the bone marrow fails to produce adequate numbers of red blood cells due to a deficiency in vitamin B12. Intrinsic factor, necessary for normal B12 absorption, may be the underlying cause for B12 deficiency if is not produced in the gastric glands (in the stomach). Origin: Gr. Haima = blood (27 Sep 1997) |
| anaemia | <haematology> Too few red blood cells in the bloodstream, resulting in insufficient oxygen to tissues and organs. Origin: Gr. Haima = blood (16 Dec 1997) |
| anaemia, aplastic | A form of anaemia in which the bone marrow fails to produce adequate numbers of peripheral blood elements. (12 Dec 1998) |
| anaemia, Cooley's | Better known today as thalassaemia (or as beta thalassaemia or thalassaemia major).The clinical picture of this important type of anaemia was first described in 1925 by the paediatrician Thomas Benton Cooley. Another name for the disease is Mediterranean anaemia. The name thalassaemia was coined by the Nobel Prise winning pathologist George Whipple and the professor of paediatrics Wm Bradford at Univ. Of Rochester because thalassa in Greek means the sea (like the Mediterrranean Sea) + -aemia means in the blood so thalassaemia means sea in the blood. Thalassaemia is not just one disease. It is a complex contingent of genetic (inherited) disorders all of which involve underproduction of haemoglobin, the indispensable molecule in red blood cells that carries oxygen. The globin part of normal adult haemoglobin is made up of 2 alpha and 2 beta polypeptide chains. In beta thalassaemia, there is a mutation (change) in both beta globin chains leading to underproduction (or absence) of beta chains, underproduction of haemoglobin, and profound anaemia. The gene for beta thalassaemia is relatively frequent in people of Mediterranean origin (for example, from Italy and Greece). Children with this disease inherit one gene for it from each parent. The parents are carriers (heterozygotes) with just one thalassaemia gene, are said to have thalassaemia minor, and are essentially normal. Their children affected with beta thalassaemia seem entirely normal at birth because at birth we still have predominantly foetal haemoglobin which does not contain beta chains. The anaemia surfaces in the first few months after birth and becomes progressively more severe leading to pallor and easy fatiguability, failure to thrive (grow), bouts of fever (due to infections) and diarrhoea. Treatment based on blood transfusions is helpful but not curative. Gene therapy will, it is hoped, be applicable to this disease. (12 Dec 1998) |