| SBE | Subacute Bacterial Endocarditis; ¾Æ±Þ¼º ¼¼±Õ¼º ½É³»¸·¿° |
|---|---|
| SBP | Spontaneous Bacterial Peritonitis |
| ABCDES | abnormal alignment, bones-periarticular osteoporosis, cartilage-joint space loss, deformities, margi... |
| ABE | acute bacterial endocarditis; American Board of Endodontics; botulism equine trivalent antitoxin |
| BA | Bachelor of Arts; backache; bacterial agglutination; basilar artery; basion; benzyladenine; best amp... |
| sexually transmitted diseases, bacterial | Bacterial diseases transmitted or propagated by sexual conduct. (12 Dec 1998) |
|---|---|
| skin diseases, bacterial | Skin diseases caused by bacteria. (12 Dec 1998) |
| spores, bacterial | Heat and stain resistant, metabolically inactive bodies formed within the vegetative cells of bacteria of the genera bacillus and clostridium. (12 Dec 1998) |
| subacute bacterial endocarditis | Subacute bacterial endocarditis is usually due to Streptococcus viridans or S. Fecalis. (05 Mar 2000) |
| DNA, bacterial | Deoxyribonucleic acid that makes up the genetic material of bacteria. (12 Dec 1998) |
| endocarditis, subacute bacterial | Infection of the endocardium caused by species of streptococcus. This condition does not produce metastatic foci and if untreated may take up to a year to be fatal. (12 Dec 1998) |
| transformation, bacterial | The heritable modification of the properties of a competent bacterium by DNA from another bacterial strain. (12 Dec 1998) |
| eye infections, bacterial | Infections in the inner or external eye caused by microorganisms belonging to several families of bacteria. Some of the more common genera found are haemophilus, neisseria, staphylococcus, streptococcus, and chlamydia. (12 Dec 1998) |
| fibrous bacterial viruses | Deoxyribonucleoproteins that "infect" and replicate in Gram-negative bacteria having sex pili and that, unlike bacteriophage, are released from infected bacteria without damage to the cell; they seem to be of two kinds, one of which has a specificity for F pili and the other for I pili. Synonym: fibrous bacterial viruses. (05 Mar 2000) |
| filamentous bacterial viruses | Deoxyribonucleoproteins that "infect" and replicate in Gram-negative bacteria having sex pili and that, unlike bacteriophage, are released from infected bacteria without damage to the cell; they seem to be of two kinds, one of which has a specificity for F pili and the other for I pili. Synonym: fibrous bacterial viruses. (05 Mar 2000) |
| fimbriae, bacterial | Thin, hairlike appendages, 1 to 20 microns in length and often occurring in large numbers, present on the cells of gram-negative bacteria, particularly enterobacteriaceae and neisseria. Unlike flagella, they do not possess motility, but being protein (pilin) in nature, they possess antigenic and haemagglutinating properties. They are of medical importance because some fimbriae mediate the attachment of bacteria to cells via adhesins (adhesins, bacterial). Bacterial fimbriae refer to common pili, to be distinguished from the preferred use of "pili", which is confined to sex pili (pili, sex). (12 Dec 1998) |
| luminescence, bacterial | The emission of light by bacteria. It is applied to studies on bacterial viability, metabolism, genetics, adhesiveness, and other physiological properties. (12 Dec 1998) |
| acetoacetyl-acyl carrier protein synthase | <enzyme> E coli enzyme, that catalyses condensation of malonyl-acyl carrier protein plus acetyl-acyl carrier protein; not inhibited by cerulenin Registry number: EC 2.3.1.- Synonym: acetoacetyl-acp synthase (26 Jun 1999) |
| acid soluble spore protein | <molecular biology> A DNA binding protein in the spores of some bacteria, thought to stabilise the DNA in an A configuration, so protecting it from cleavage by enzymes or UV light. (18 Nov 1997) |
| acute-phase protein | <haematology> These plasma proteins (in addition to fibrinogen) increase 25% or more in response to inflammation and injury are under direct control of interleukin-6 (IL-6) (hepatocyte-stimulating factor). Other proteins which increase are ceruloplasmin, C3 and C4 which increase 50% or more; alpha-1 acid glycoprotein, alpha-1 antitrypsin, haptoglobin and fibrinogen (the major determinant of viscosity 1 ) which increase two- to fourfold; C-reactive protein (CRP) and serum amyloid A which increase several hundred-fold. Despite long-held clinical opinion to the contrary, available data indicate that neither ESR nor measurement of specific acute-phase reactants are useful in excluding underlying infection or inflammation regardless of the pretest probability. These proteins are secreted into the blood in increased or decreased quantities by hepatocytes in response to trauma, inflammation, or disease. They can serve as inhibitors or mediators of the inflammatory processes. Certain acute-phase proteins have been used to diagnose and follow the course of diseases or as tumour markers. See also: amyloid, c-reactive protein, erythrocyte sedimentation rate, viscosity. (25 Jun 1999) |
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