¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"tumor suppressor gene"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • renin-secreting juxtaglomerular tumor
    ·¹´ÑºÐºñÅ丮°çÁ¾¾ç, ·¹´ÑºÐºñ»ç±¸Ã¼¿·Á¾¾ç
  • retroperitoneal tumor
    Èĺ¹¸·Á¾¾ç, ¹è¸·µÚÁ¾¾ç
  • solid tumor
    °íÇüÁ¾¾ç
  • solid-cystic tumor
    °íÇü³¶¼ºÁ¾¾ç
  • spinal cord tumor
    ô¼öÁ¾¾ç
  • steroid cell tumor
    ½ºÅ×·ÎÀ̵弼Æ÷Á¾¾ç
  • stromal tumor
    °£ÁúÁ¾¾ç, ¹öÆÀÁúÁ¾¾ç
  • submucosal tumor
    Á¡¸·¹ØÁ¾¾ç, Á¡¸·ÇÏÁ¾¾ç
  • sebaceous tumor
    ÇÇÁö»ùÁ¾¾ç, ±â¸§»ùÁ¾¾ç
  • Sertoli-Leydig cell tumor
    ¼¼¸£Å縮¶óÀ̵ðÈ÷Á¾¾ç
  • sex cord-stromal tumor
    ¼º²ö±âÁúÁ¾¾ç, ¼º»è±âÁúÁ¾¾ç
  • smooth muscle tumor
    ÆòȰ±ÙÁ¾¾ç
  • virilizing tumor
    ³²¼ºÈ­Á¾¾ç
  • Wilms¡¯ tumor
    ºô¸§½ºÁ¾¾ç
  • autosomal gene
    º¸Åë¿°»öüÀ¯ÀüÀÚ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • major histocompatibility gene
    ÁÖÁ¶Á÷ÀûÇÕ¼ºÀ¯ÀüÀÚ
  • marker gene
    Ç¥ÁöÀ¯ÀüÀÚ
  • modulator gene
    ÀÛµ¿À¯ÀüÀÚ, ¸Å°³À¯ÀüÀÚ
  • molecular check point gene
    ºÐÀڰ˹®À¯ÀüÀÚ
  • mutant gene
    µ¹¿¬º¯ÀÌÀ¯ÀüÀÚ
  • mutator gene
    º¯ÀÌÀ¯¹ßÀ¯ÀüÀÚ
  • nucleolar gene
    ÇÙ¼ÒüÀ¯ÀüÀÚ
  • operator gene
    ÀÛµ¿À¯ÀüÀÚ
  • recessive gene
    ¿­¼ºÀ¯ÀüÀÚ
  • regulator gene
    Á¶ÀýÀ¯ÀüÀÚ
  • reporter gene
    Á¤º¸Á¦°øÀ¯ÀüÀÚ
  • repressor gene
    ¾ïÁ¦À¯ÀüÀÚ
  • retinoblastoma gene
    ¸Á¸·¸ð¼¼Æ÷Á¾À¯ÀüÀÚ
  • structural gene
    ±¸Á¶À¯ÀüÀÚ
  • suicide gene
    ÀÚ»ìÀ¯ÀüÀÚ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • rb gene
    Rb À¯ÀüÀÚ(¡­ë¶îîí­)
  • recessive autosomal gene
    ¿­¼ºº¸Åë¿°»öüÀ¯ÀüÀÚ
  • recessive gene
    ¿­¼ºÀ¯ÀüÀÚ(¡­ë¶îîí­).
  • recessive gene
    ¿­¼ºÀ¯ÀüÀÚ
  • recessive gonosomal gene
    ¿­¼º¼º¿°»öüÀ¯ÀüÀÚ
  • recombination, gene
    À¯ÀüÀÚÀçÁ¶ÇÕ
  • regulator gene
    Á¶ÀýÀ¯ÀüÀÚ(ðàï½ë¶îîí­).
  • Chinese hamster ovary tumor cell
    Áß±¹ÇÔ½ºÅÍ ³­¼Ò¼¼Æ÷, CHO¼¼Æ÷
  • Dermoid tumor
    À¯ÇÇÁ¾
  • Granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç(Î¨Ø£Ø¯á¬øàðþåË)
  • Leydig cell tumor
    ·¹À̵ðÈ÷¼¼Æ÷Á¾¾ç
  • Schwann cell tumor
    ½´¹Ý¼¼Æ÷Á¾¾ç
  • TNF => tumor necrosis factor
    Á¾¾ç±«»çÀÎÀÚ
  • Warthins tumor
    ¿Í¸£Æ¾ Á¾¾ç
  • Warthins tumor => adenolymphoma
    ¼±¸²ÇÁÁ¾
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • dominant autosomal gene
    ¿ì¼ºº¸Åë¿°»öüÀ¯ÀüÀÚ
  • dominant gene
    ¿ì¼ºÀ¯ÀüÀÚ
  • dominant gene
    ¿ì¼ºÀ¯ÀüÀÚ(¡­ë¶îîí­).
  • dominant gene
    ¿ì¼ºÀ¯ÀüÀÚ(éÐàõë¶îîí­).
  • dq3.2 gene
    DQ3.2 À¯ÀüÀÚ(¡­ë¶îîí­)
  • duplicate gene
    º¹»çÀ¯ÀüÀÚ.
  • exaggeration gene
    °­Á¶À¯ÀüÀÚ(¡­ë¶îîí­).
  • familial colon carcinoma gene
    °¡Á·¼º ´ëÀå¾ÏÁ¾ À¯ÀüÀÚ
  • fmr-1 gene
    FMR-1 À¯ÀüÀÚ
  • gene
    À¯ÀüÀÚ(ë¶îîí­)
  • gene
    À¯Àü(ÀÎ)ÀÚ(ë¶îîì×í­).
  • gene
    À¯ÀüÀÚ
  • gene amplification
    À¯ÀüÀÚÁõÆø
  • gene amplification
    À¯ÀüÀÚ ÁõÆø
  • gene analyses
    À¯ÀüÀںм®
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • gene duplication
    À¯ÀüÀÚ º¹»ç(ë¶îîí­ÜÜÞÐ)
  • gene expression
    À¯ÀüÀÚ ¹ßÇö(ë¶îîí­Û¡úÞ)
  • gene family
    À¯ÀüÀÚ Á·(ë¶îîí­ðé)
  • gene frequence
    À¯ÀüÀÚ ºóµµ(ë¶îîí­ÞºÓø)
  • gene fusion
    À¯ÀüÀÚ À¶ÇÕ(ë¶îîí­ë×ùê)
  • gene hypothesis
    À¯ÀüÀÚ ¼³(ë¶îîí­àã)
  • gene insertion
    À¯ÀüÀÚ »ðÀÔ(ë¶îîí­ßºìý)
  • gene library
    À¯ÀüÀÚ(ë¶îîí­) ¶óÀ̺귯¸®
  • gene linkage
    À¯ÀüÀÚ ¿¬°ü(ë¶îîí­Ö¤Î¼)
  • gene locus
    À¯ÀüÀÚ ºÎÀ§(ë¶îîí­Ý»êÈ)
  • gene mapping
    À¯ÀüÀÚ ÀÛµµ(ë¶îîí­íÂÓñ)
  • gene pair
    À¯ÀüÀÚ ½Ö(ë¶îîí­äª)
  • gene pool
    À¯ÀüÀÚ(ë¶îîí­) Ç®
  • gene product
    À¯ÀüÀÚ(ë¶îîí­) »ê¹°
  • gene reduncdancy
    À¯ÀüÀÚ Áߺ¹(ë¶îîí­ñìÜÜ)
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 4
SSF soluble suppressor factor; supplemental sensory feedback
SUS Saybolt Universal Seconds; solitary ulcer syndrome; stained urinary sediment; suppressor sensitive
Ts T-cell suppressor
TVSS transient voltage surge suppressor
CPA tumor Cerebello-Pontine Angle(¼Ò³ú±³°¢ºÎ) tumor
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 4
Su(H) Suppressor of Hairless
Su(Hw) Suppressor of Hairy wing
SOCS3 suppressor of cytokine signalling 3
Su(f) Suppressor of forked
Spm Suppressor-mutator
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • cerebellopontine tumor
    ¼Ò³ú ±³°¢ Á¾¾ç
  • children tumor
    ¼Ò¾Æ ¾Ï
    ¼Ò¾Æ¿¡°Ô »ý±â´Â ¾Ç¼º Á¾¾ç. ¼Ò¾Æ¿¡°Ô´Â À§¾Ï, Æó¾Ï, Àڱà ¾Ï µîÀº °ÅÀÇ º¼ ¼ö ¾øÁö¸¸, ±Þ¼º ¹éÇ÷º´, ¾Ç¼º ¸²ÇÁÁ¾, ³úÁ¾¾ç, °íȯ žƼº ¾Ï, ½Å°æ¾Æ¼¼Æ÷Á¾, °£¾Ï, °ñ À°Á¾, ºñ·ç½º¼º Á¾¾ç µîÀÌ ¸¹´Ù.
  • congenital intracranial tumor
    ¼±Ãµ¼º µÎ°³³» Á¾¾ç
  • delta cell tumor
    µ¨Å¸ ¼¼Æ÷ Á¾¾ç
    ¼Ò¸¶Å佺ŸƾÀ» ºÐºñÇÏ´Â Á¾¾çÀ¸·Î ¼Ò¸¶Å佺ŸƾÁ¾
  • dermoid tumor
    À¯ÇÇÁ¾
  • dysembryoplastic neuroepithelial tumor
    ÀÌÇü¼º ¹è¾Æ¼º ½Å°æ»óÇÇÁ¾
    ¾à¹° Ä¡·á¿¡ ¹ÝÀÀÇÏÁö ¾ÊÀº ºÎºÐ º¹ÇÕÇü °£ÁúÀ» Ư¡ÀûÀ¸·Î µ¿¹ÝÇÏ´Â Á¾¾çÀ¸·Î¼­ 20¼¼ ¹Ì¸¸ÀÇ Ã»¼Ò³â±â¿¡ Áõ»óÀÌ ½ÃÀ۵ȴÙ. ÃøµÎ¿±¿¡ È£¹ßÇϸç ÀüµÎ¿±À̳ª µÎÁ¤¿±¿¡¼­ ¹ß»ýµÇ±âµµ ÇÑ´Ù. ´Ù°áÀý¼º Á¾±«·Î¼­ ÇÌÁö±³Á¾¼º ¼¼Æ÷µéÀÌ Á¡¾×¼º ±âÁú°ú ÇÔ²² ³ªÅ¸³­´Ù. ÀÌ·¯ÇÑ ÇÌÁö±³Á¾¼º ¼¼Æ÷µéÀº ¸é¿ªÁ¶Á÷È­ÇÐÀû ¶Ç´Â ÀüÀÚÇö¹Ì°æÀû °üÂû»ó ¼º»ó ¼¼Æ÷, ÇÌÁö±³ ¼¼Æ÷, ½Å°æ¿ø ¼¼Æ÷µéÀÌ ¼¯¿©Á® ÀÖÀ½ÀÌ Áõ¸íµÇ¾ú´Ù. ¾ç¼º Á¾¾çÀ̸ç ÁÖÀ§¿¡ ´ë³ú ÀÌÇü¼ºÁõÀ» ÈçÈ÷ µ¿¹ÝÇÑ´Ù.
  • endoaermal sinus tumor
    ³»¹è¿±µ¿ Á¾¾ç
    ¹ß»ý ºóµµ°¡ ³·Áö¸¸ ³­¼ÒÀÇ ¾Ç¼º »ý½Ä ¼¼Æ÷Á¾ Áß¿¡ µÎ ¹øÂ°·Î ÈçÇϰí Àüü ³­¼Ò Á¾¾çÀÇ 1% Á¤µµ¸¦ Â÷ÁöÇÑ´Ù. Á¶Á÷ÇÐÀûÀ¸·Î ³­È² ³¶ÀÇ ³»¹è¿± µ¿À» ´à¾Ò´Ù ÇÏ¿© ³­È² ³­ Á¾¾ç ¶Ç´Â ³»¹è¿±µ¿ Á¾¾çÀ̶ó°í ÇÑ´Ù. ÀÌ Á¾¾çÀº ÁÖ·Î ¾î¸°ÀÌ¿Í ÀþÀº ¿©¼º¿¡ È£¹ßÇÏ°í Æò±Õ ¿¬·ÉÀº 19¼¼ÀÌ´Ù. Çö¹Ì°æÀûÀ¸·Î ÀÛÀº ´Ù¹æ¼º ³¶ ±¸Á¶¸¦ °®´Â °íÇü¼º Á¾¾çÀÌ°í ´ë°³ ÀÏÃø¼ºÀÌ´Ù. ¾û¼ºÇÑ ¸Á»óÇü ¾ç»ó, Schiller-¿·¹Ì ¼Òü¸¦ Çü¼ºÇÏ´Â À¯µÎ ¾ç»ó, ´Ù¼Ò ³¶Æ÷¼º ³­È² ¾ç»ó, °íÇü¼º ¾ç»ó µîÀÌ È¥ÇÕµÇ¾î ³ª¿À°í ±¸¼º ¼¼Æ÷´Â ¹Ì¼º¼÷ ¹è ¼¼Æ÷ÀÇ Æ¯Â¡À» °®´Â´Ù. ¼¼Æ÷Áú ¾ÈÀ̳ª ¼¼Æ÷ ¹Û¿¡ À¯¸®Áú ±¸»óüµéÀÌ °üÂûµÇ´Âµ¥ À̵éÀº ¸é¿ª È¿¼Ò ¿°»ö¿¡¼­ ¾ËÆÄ ÅÂ¾Æ ´Ü¹é
  • endometrioid tumor
    Àڱà ³»¸·¾ç Á¾¾ç
  • exophytic mucosal tumor
    ¿ÜÇ⼺ Á¡¸· Á¾¾ç
  • exophytic tumor
    ¿ÜÇ⼺ Á¾¾ç
  • fibroid tumor
    ¼¶À¯¾ç Á¾¾ç
  • fibroma-theca cell tumor
    ¼¶À¯Á¾-³­Æ÷¸· ¼¼Æ÷Á¾
    ¼¶À¯¾Æ¼¼Æ÷³ª ³­Æ÷¸· ¼¼Æ÷·Î ±¸¼ºµÈ´Ù. ±¸¼º ¼¼Æ÷°¡ ´ëºÎºÐ ³­Æ÷¸· ¼¼Æ÷ÀÎ ÀÌ Á¾¾çÀº È£¸£¸óÀ» »ý»êÇÒ ¼ö ÀÕ´Ù. ±×·¯³ª ¼ø¼öÇÑ ³­Æ÷¸· ¼¼Æ÷Á¾Àº µå¹°°í ´ëºÎºÐ Á¾¾çÀº ÁÖ·Î ¼¶À¯¸ð¼¼Æ÷·Î ±¸¼ºµÇ¾î ÀÖÀ¸¸ç È£¸£¸óÀ» »ý»êÇÏÁö ¾Ê´Â´Ù. 90%¿¡¼­ ÇÑÂÊ ³­¼Ò¿¡¼­¸¸ ¹ß»ýÇÑ´Ù. Á¾¾çÀº ȸ¹é»öÀÌ¸ç °íÇüÀÌ°í ±¸ÇüÀÌ¸ç ´Ü´ÜÇÏ´Ù. Á¶Á÷ÇÐÀûÀ¸·Î ¼¶À¯¸ð¼¼Æ÷¿Í ÄݶóÁ¨ °áü Á¶Á÷À¸·Î ±¸¼ºµÇ¾î ÀÖÀ¸¸ç ³­Æ÷¸· ¼¼Æ÷°¡ È¥ÀçÇÒ ¼ö ÀÖ´Ù. ȯÀÚ´Â °ñ¹ÝÅë°ú °ñ¹Ý Á¾±« µîÀÇ ºñƯÀÌÀû Áõ»óÀ» È£¼ÒÇϰųª º¹¼ö°¡ ³ªÅ¸³¯ ¼ö ÀÖ´Ù. ³­Æ÷¸· ¼¼Æ÷Á¾Àº ¾Ç¼ºÀÌ ¾ø´Ù.
  • hormone dependent tumor
    È£¸£¸ó ÀÇÁ¸¼º Á¾¾ç
  • induced tumor
    Àΰø ¾Ï
    µ¿¹°¿¡°Ô ÀΰøÀûÀÎ ¾Ï¿ø¼º ÀÚ±ØÀ» ÁÖ¾î Çü¼º½ÃŲ ¾Ï. µ¿¹°ÀÇ ÀÚ¿¬¹ß»ý ¾Ï¿¡ ´ëÀÀÇÏ´Â ¸»ÀÌ´Ù.
  • intraosseous salivary gland tumor
    °ñ³» Ÿ¾×¼± Á¾¾ç
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
gene dosage compensation The putative mechanism that adjusts the X-linked phenotypes of males and females to compensate for the haploid state in males and the diploid state in females. It is now largely ascribed to lyonization which compensates the mean of the dose but not its variance, which is greater in females.
(05 Mar 2000)
gene dosage effect In codominant alleles, the more or less linear relationship between the phenotypic value and the number of genes of one type substituted by another type.
(05 Mar 2000)
gene duplication <molecular biology> A class of DNA rearrangement that generates a supernumerary copy of a gene in the genome. This would allow each gene to evolve independently to produce distinct functions. Such a set of evolutionarily related genes can be called a gene family.
(18 Nov 1997)
gene, evolutionarily conserved A gene that has remained essentially unchanged throughout evolution. Conservation of a gene indicates that it is unique and essential. There is not an extra copy of that gene with which evolution can tinker. And changes in the gene are likely to be lethal.
(12 Dec 1998)
gene expression <molecular biology> The full use of the information in a gene via transcription and translation leading to production of a protein and hence the appearance of the phenotype determined by that gene. Gene expression is assumed to be controlled at various points in the sequence leading to protein synthesis and this control is thought to be the major determinant of cellular differentiation in eukaryotes.
(18 Nov 1997)
gene expression regulation Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action at the level of transcription or translation. These processes include gene activation and genetic induction.
(12 Dec 1998)
gene expression regulation, archaeal Any of the processes by which cytoplasmic or intercellular factors influence the differential control of gene action in archaea.
(12 Dec 1998)
gene expression regulation, bacterial Any of the processes by which cytoplasmic or intercellular factors influence the differential control of gene action in bacteria.
(12 Dec 1998)
gene expression regulation, developmental Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action during the developmental stages of an organism.
(12 Dec 1998)
gene expression regulation, enzymologic Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action in enzyme synthesis.
(12 Dec 1998)
gene expression regulation, fungal Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action in fungi.
(12 Dec 1998)
gene expression regulation, leukaemic Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action in leukaemia.
(12 Dec 1998)
gene expression regulation, neoplastic Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action in neoplastic tissue.
(12 Dec 1998)
gene expression regulation, plant Any of the processes by which nuclear, cytoplasmic, or intercellular factors influence the differential control of gene action in plants.
(12 Dec 1998)
gene expression regulation, viral Any of the processes by which cytoplasmic factors influence the differential control of gene action in viruses.
(12 Dec 1998)
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
KMLE ¾àǰ/ÀǾàǰ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 4
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
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KMLE ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 4
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 4
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