| EIA | 1) Exercise Induced Asthma; 운동 유발성 천식 = EIB 2) Enzyme Immu... |
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| ELISA | Enzyme-Linked Immuno-Sorbent Assay; 효소 면역법 |
| IFA | Immuno-Fluorescence Antibody Test(= Assay) |
| IRMA | 1) Intra-Retinal Microvascular Abnormalities 2) Immuno-Radio-Metric Assay... |
| MHA-TP | Micro-Hemagglutination Assay for antibodies to Treponema Pallidum |
| factor x assay | A test used to measure the activity of a blood clotting factor X. This test may be used to evaluate excessive bleeding. Abnormally low factor X assays may be seen in the following conditions: congenital deficiency of factor X, fat malabsorption, heparin administration, cirrhosis, vitamin K deficiency and warfarin administration. (27 Sep 1997) |
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| factor xii assay | A test used to measure the activity of a blood clotting factor XII. This test may be used to evaluate excessive bleeding. Low factor XII may be seen in cases of congenital deficiency of factor XII, heparin administration and liver disease. (27 Sep 1997) |
| Farr type assay | <investigation> Method of radioimmunoassay in which free antigen remains soluble and antibody antigen complexes are precipitated. (18 Nov 1997) |
| 17-ketogenic steroid assay test | A colourimetric test, based on the Zimmermann reaction, which indicates metabolites or adrenal and testicular steroids excreted as 17-ketones in the urine; increased values are most striking in adrenocortical tumours, decreased values in Addison's disease or in panhypopituitarism. Synonym: ketogenic corticoids test. (05 Mar 2000) |
| focus-forming assay | <investigation> A lab technique used to find out if a particular piece of DNA contains oncogenes (genes which are associated with cancer). This is done by putting the DNA into animal cells which normally show contact inhibition, or which stop growing when they come into physical contact with other cells or reach a certain density in the culture. If the cells lose contact inhibition and form areas of densely-packed cells (called foci) after receiving the DNA, it means that the DNA did contain oncogenes. (05 Jan 1997) |
| leukocyte adherence assay test | A test to detect the ability of leukocytes to adhere to bacteria, performed in vitro using nylon fibres to measure adherence. (05 Mar 2000) |
| leukocyte bactericidal assay test | A test of leukocytes to determine their ability to kill a culture of live bacteria. (05 Mar 2000) |
| LH assay | A blood test which measures the amount of luteinising hormone (LH). LH is a protein hormone that is secreted by the anterior pituitary gland. In women, a surge in LH at midcycle causes ovulation. For the next 7-10 days LH maintains the corpus luteum which synthesises progesterone. The corpus luteum disintegrates after about 10 days if fertilization does not occur. In men, LH stimulates production of testosterone by the Leydig cells in the testes. This test may be used to evaluate anovulatory bleeding, infertility, ovarian cysts and multiple endocrine neoplasia (MEN) I. Normal values in males: 7 to 24 U/L. Normal values in females: greater than 6-30 U/L. Greater than normal values can indicate: anorchia, hypogonadism, Klinefelter syndrome, menopause, ovarian failure, polycystic ovary disease, precocious puberty and Turner's syndrome. Lower than normal values can indicate hypopituitarism. (27 Sep 1997) |
| Lowry-Folin assay | A method for determining protein concentrations using the Folin-Ciocalteu reagent. Synonym: Lowry-Folin assay. (05 Mar 2000) |
| Lowry protein assay | A method for determining protein concentrations using the Folin-Ciocalteu reagent. Synonym: Lowry-Folin assay. (05 Mar 2000) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| amino acid receptor | <biochemistry> Ligand gated ion channels with specific receptors for amino acid transmitters. An extended protein superfamily that also includes subunits of the nicotinic acetylcholine receptor. (18 Nov 1997) |
| AMPA receptor | <cell biology> Glutamate operated ion channel. See: excitatory amino acid receptor channels. (05 Feb 1998) |
| ANP receptor | <molecular biology> Family of 3 receptors for atrial natriuretic peptide. ANP A and ANP B have intracellular guanylate cyclase and protein kinase like domains. ANP C, shares the extracellular ligand binding and transmembrane domains, but lacks the functional intracellular domains and is not thought to be involved in signal transduction. (18 Nov 1997) |
| asialoglycoprotein receptor | A surface receptor found in hepatocytes that binds galactose-terminal glycoproteins; thus, this receptor removes those proteins from circulation and they are in turn acted upon by hepatocyte lysosomes. (05 Mar 2000) |