¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"Tumor Necrosis Factors"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • fecal tumor
    ´ëº¯µ¢ÀÌ, ºÐÁ¾(ÝÐðþ)
  • gastrointestinal autonomic nerve tumor
    À§Ã¢ÀÚÀÚÀ²½Å°æÁ¾¾ç, À§Àå°üÀÚÀ²½Å°æÁ¾¾ç
  • gastrointestinal stromal tumor
    À§Ã¢ÀÚ±âÁúÁ¾¾ç, À§Àå°ü±âÁúÁ¾¾ç
  • giant cell tumor
    °Å´ë¼¼Æ÷Á¾¾ç
  • Gleason tumor grade
    ±Û¸®½¼Á¾¾çµî±Þ
  • glomus tumor
    Å丮Á¾¾ç, »ç±¸Á¾¾ç
  • granular cell tumor
    °ú¸³¼¼Æ÷Á¾¾ç
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç
  • granulosa-theca cell tumor
    °ú¸³³­Æ÷¸·¼¼Æ÷Á¾¾ç
  • homologous tumor
    µ¿Á¾Á¾¾ç
  • juxtaglomerular cell tumor
    Å丮°ç¼¼Æ÷Á¾¾ç, »ç±¸Ã¼¿·¼¼Æ÷Á¾¾ç
  • Krukenberg¡¯s tumor
    Å©·çÄ˹ö±×Á¾¾ç
  • Leydig cell tumor
    ¶óÀ̵ðÈ÷¼¼Æ÷Á¾¾ç
  • mixed germ cell tumor
    È¥ÇÕÁ¾ÀÚ¼¼Æ÷Á¾¾ç
  • mixed tumor
    È¥ÇÕÁ¾¾ç
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 7 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • smooth muscle tumor
    ÆòȰ±ÙÁ¾¾ç
  • solid tumor
    °íÇüÁ¾¾ç
  • stromal tumor
    ¹öÆÀÁúÁ¾¾ç, ±âÁúÁ¾¾ç
  • subareolar location of tumor
    Á¥²ÉÆÇ¹ØÁ¾¾ç
  • submucosal tumor
    Á¡¸·¹ØÁ¾¾ç
  • trophoblastic tumor
    ¿µ¾ç¸·Á¾¾ç
  • Wilms' tumor
    Àª¸§ÁîÁ¾¾ç
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • fecal tumor
    (¢¡stercoroma) ´ëº¯µ¢ÀÌ
  • tumor angiogenesis factor
    Á¾¾çÇ÷°üÇü¼ºÀÎÀÚ
  • genitourinary tumor
    ºñ´¢»ý½ÄÁ¾¾ç
  • glomus tumor
    Å丮Á¾¾ç, »ç±¸Á¾¾ç
  • granular cell tumor
    °ú¸³¼¼Æ÷Á¾¾ç
  • gross tumor volume
    ¸Ç´«Á¾¾çüÀû
  • tumor suppressor gene
    Á¾¾ç¾ïÁ¦À¯ÀüÀÚ
  • heterogenous tumor
    ÀÌÁúÁ¾¾ç
  • homologous tumor
    µ¿Á¾Á¾¾ç
  • hormone dependent tumor
    È£¸£¸óÀÇÁ¸Á¾¾ç
  • infiltrating tumor
    ħÀ±Á¾¾ç
  • malignant tumor
    ¾Ç¼ºÁ¾¾ç
  • melatonic neuroectodermal tumor
    ¸á¶ó´Ñ½Å°æ¿Ü¹è¿±Á¾¾ç
  • mesenchymal tumor
    Áß°£¿±Á¾¾ç
  • mesodermal tumor
    Á߹迱Á¾¾ç
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • antigen, tumor-specific transplantation
    Á¾¾çƯÀÌ À̽ÄÇ׿ø
  • antigen,fetal tumor-associated
    žÆÁ¾¾ç °ü·Ã¼º(÷Ãä®ðþåË Î¼Ö¤àõ)
  • antigen,tumor-specific transplantation
    Á¾¾ç ƯÀÌÀ̽Ä(ðþåË ÷åì¶ì¹ãÕ)
  • appendigeal tumor
    ºÎ¼Ó±â Á¾¾ç(ðþåË)
  • genitourinary tumor
    ºñ´¢»ý½Ä±â Á¾¾ç
  • germ cell tumor
    »ý½Ä¼¼Æ÷Á¾(ßæãÖá¬øàðþ)
  • giant cell tumor
    °Å¼¼Æ÷Á¾¾ç.
  • giant cell tumor
    °Å´ë¼¼Æ÷Á¾¾ç.(¡­ðþåË)
  • glomus jugulare tumor
    °æÁ¤¸Æ±¸Á¾¾ç
  • glomus tumor<³ª>
    »ç±¸Á¾¾ç, ±Û·Î¹«½ºÁ¾¾ç(¡­ðþåË)
  • glomus tumor<³ª>
    »ç±¸Á¾, ±Û·Î¹«½ºÁ¾¾ç(¡­ðþåË)
  • gonadal stromal tumor
    ¼º¼±°£ÁúÁ¾¾ç
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç.
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • fibrinoid necrosis
    ¼¶À¯¼Ò¾ç±«»ç
  • focal necrosis
    ±¹¼Ò±«»ç
  • gangrenous necrosis
    ±«Àú¼º±«»ç
  • gangrenous necrosis
    ±«Àú¼º ±«»ç(¡­ÎÕÞÝ)
  • hyaline necrosis
    À¯¸®¾ç ±«»ç
  • ischemic cortical necrosis
    ÇãÇ÷¼º ÇÇÁú±«»ç(¡­ù«òõÎÕÞÝ).
  • kidney,diffuse cortical necrosis
    ¹Ì¸¸¼ºÇÇÁú±«»ç( Ø·àõù«òõ ÎÕÞÝ)
  • kidney,papillary necrosis
    À¯µÎ±«»ç(êáÔéÎÕÞÝ)
  • kidney,tubular necrosis
    ¼¼´¢°ü ±«»ç(á¬Òãη ÎÕÞÝ)
  • lipolytic necrosis
    Áö¹æ À¶Çؼº ±«»ç
  • liquefaction,necrosis
    ¾×È­±«»ç
  • liquefactive necrosis
    ¾×È­±«»ç
  • liver cell necrosis
    °£¼¼Æ÷ ±«»ç(¡­ÎÕÞÝ)
  • liver necrosis
    °£±«»ç.
  • liver,central hemorrhagic necrosis
    Á᫐ ÃâÇ÷¼º ±«»ç(ñéãý õóúìàõ ¡­)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
  • tumor vessel
    Á¾¾çÇ÷°ü
  • vanishing tumor
    Àϰú¼ºÁ¾¾ç»óÀ½¿µ, Àϰú¼º¿±°£Èä¼öÀú·ù(»ó)
  • Warthin's tumor
    ¿Í¸£Æ¾Á¾¾ç
  • Wilms' tumor
    Àª¸§Á¾¾ç
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 4
TNFBR tumor necrosis factor beta receptor
TNS total nuclear score; transcutaneous nerve stimulation; tumor necrosis serum
ANF Atrial Natriuretic Factors
ECF   1) Eosinophilic Chemotatic Factors
  2) Extra-Cellular Fluid; ¼¼Æ÷ ¿Ü¾×
NCF Neutrophilic Chemotatic Factors
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 4
TNF-R2 Tumor necrosis factor receptor 2
TNF beta Tumor necrosis factor beta
TNF Tumor necrosis factor/cachectin
nHuTNF-alpha natural human tumor necrosis factor alpha
rTNF-alpha recombinant human tumor necrosis factor alpha
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • cartilaginous tumor
    ¿¬°ñ¼º Á¾¾ç
    Á¶ÀýÇÒ ¼ö ¾øÀ» Á¤µµ·Î °è¼Ó ÁøÇàµÇ´Â ¼¼Æ÷ºÐ¿­¿¡ ÀÇÇÑ ¿¬°ñ Á¶Á÷ÀÇ »õ·Î¿î Áõ½Ä ¹× Áõ´ë.
  • cavernous tumor
    ÇØ¸é»ó Á¾¾ç, ÇØ¸éÁ¾
    µ¿ÀǾî=cavenoma.
  • central giant cell tumor
    Á߽ɼº °Å´ë ¼¼Æ÷ ¾ÏÁ¾
  • cerebellopontine tumor
    ¼Ò³ú ±³°¢ Á¾¾ç
  • children tumor
    ¼Ò¾Æ ¾Ï
    ¼Ò¾Æ¿¡°Ô »ý±â´Â ¾Ç¼º Á¾¾ç. ¼Ò¾Æ¿¡°Ô´Â À§¾Ï, Æó¾Ï, Àڱà ¾Ï µîÀº °ÅÀÇ º¼ ¼ö ¾øÁö¸¸, ±Þ¼º ¹éÇ÷º´, ¾Ç¼º ¸²ÇÁÁ¾, ³úÁ¾¾ç, °íȯ žƼº ¾Ï, ½Å°æ¾Æ¼¼Æ÷Á¾, °£¾Ï, °ñ À°Á¾, ºñ·ç½º¼º Á¾¾ç µîÀÌ ¸¹´Ù.
  • congenital intracranial tumor
    ¼±Ãµ¼º µÎ°³³» Á¾¾ç
  • delta cell tumor
    µ¨Å¸ ¼¼Æ÷ Á¾¾ç
    ¼Ò¸¶Å佺ŸƾÀ» ºÐºñÇÏ´Â Á¾¾çÀ¸·Î ¼Ò¸¶Å佺ŸƾÁ¾
  • dermoid tumor
    À¯ÇÇÁ¾
  • dysembryoplastic neuroepithelial tumor
    ÀÌÇü¼º ¹è¾Æ¼º ½Å°æ»óÇÇÁ¾
    ¾à¹° Ä¡·á¿¡ ¹ÝÀÀÇÏÁö ¾ÊÀº ºÎºÐ º¹ÇÕÇü °£ÁúÀ» Ư¡ÀûÀ¸·Î µ¿¹ÝÇÏ´Â Á¾¾çÀ¸·Î¼­ 20¼¼ ¹Ì¸¸ÀÇ Ã»¼Ò³â±â¿¡ Áõ»óÀÌ ½ÃÀ۵ȴÙ. ÃøµÎ¿±¿¡ È£¹ßÇϸç ÀüµÎ¿±À̳ª µÎÁ¤¿±¿¡¼­ ¹ß»ýµÇ±âµµ ÇÑ´Ù. ´Ù°áÀý¼º Á¾±«·Î¼­ ÇÌÁö±³Á¾¼º ¼¼Æ÷µéÀÌ Á¡¾×¼º ±âÁú°ú ÇÔ²² ³ªÅ¸³­´Ù. ÀÌ·¯ÇÑ ÇÌÁö±³Á¾¼º ¼¼Æ÷µéÀº ¸é¿ªÁ¶Á÷È­ÇÐÀû ¶Ç´Â ÀüÀÚÇö¹Ì°æÀû °üÂû»ó ¼º»ó ¼¼Æ÷, ÇÌÁö±³ ¼¼Æ÷, ½Å°æ¿ø ¼¼Æ÷µéÀÌ ¼¯¿©Á® ÀÖÀ½ÀÌ Áõ¸íµÇ¾ú´Ù. ¾ç¼º Á¾¾çÀ̸ç ÁÖÀ§¿¡ ´ë³ú ÀÌÇü¼ºÁõÀ» ÈçÈ÷ µ¿¹ÝÇÑ´Ù.
  • endoaermal sinus tumor
    ³»¹è¿±µ¿ Á¾¾ç
    ¹ß»ý ºóµµ°¡ ³·Áö¸¸ ³­¼ÒÀÇ ¾Ç¼º »ý½Ä ¼¼Æ÷Á¾ Áß¿¡ µÎ ¹øÂ°·Î ÈçÇϰí Àüü ³­¼Ò Á¾¾çÀÇ 1% Á¤µµ¸¦ Â÷ÁöÇÑ´Ù. Á¶Á÷ÇÐÀûÀ¸·Î ³­È² ³¶ÀÇ ³»¹è¿± µ¿À» ´à¾Ò´Ù ÇÏ¿© ³­È² ³­ Á¾¾ç ¶Ç´Â ³»¹è¿±µ¿ Á¾¾çÀ̶ó°í ÇÑ´Ù. ÀÌ Á¾¾çÀº ÁÖ·Î ¾î¸°ÀÌ¿Í ÀþÀº ¿©¼º¿¡ È£¹ßÇÏ°í Æò±Õ ¿¬·ÉÀº 19¼¼ÀÌ´Ù. Çö¹Ì°æÀûÀ¸·Î ÀÛÀº ´Ù¹æ¼º ³¶ ±¸Á¶¸¦ °®´Â °íÇü¼º Á¾¾çÀÌ°í ´ë°³ ÀÏÃø¼ºÀÌ´Ù. ¾û¼ºÇÑ ¸Á»óÇü ¾ç»ó, Schiller-¿·¹Ì ¼Òü¸¦ Çü¼ºÇÏ´Â À¯µÎ ¾ç»ó, ´Ù¼Ò ³¶Æ÷¼º ³­È² ¾ç»ó, °íÇü¼º ¾ç»ó µîÀÌ È¥ÇÕµÇ¾î ³ª¿À°í ±¸¼º ¼¼Æ÷´Â ¹Ì¼º¼÷ ¹è ¼¼Æ÷ÀÇ Æ¯Â¡À» °®´Â´Ù. ¼¼Æ÷Áú ¾ÈÀ̳ª ¼¼Æ÷ ¹Û¿¡ À¯¸®Áú ±¸»óüµéÀÌ °üÂûµÇ´Âµ¥ À̵éÀº ¸é¿ª È¿¼Ò ¿°»ö¿¡¼­ ¾ËÆÄ ÅÂ¾Æ ´Ü¹é
  • endometrioid tumor
    Àڱà ³»¸·¾ç Á¾¾ç
  • exophytic mucosal tumor
    ¿ÜÇ⼺ Á¡¸· Á¾¾ç
  • exophytic tumor
    ¿ÜÇ⼺ Á¾¾ç
  • fibroid tumor
    ¼¶À¯¾ç Á¾¾ç
  • fibroma-theca cell tumor
    ¼¶À¯Á¾-³­Æ÷¸· ¼¼Æ÷Á¾
    ¼¶À¯¾Æ¼¼Æ÷³ª ³­Æ÷¸· ¼¼Æ÷·Î ±¸¼ºµÈ´Ù. ±¸¼º ¼¼Æ÷°¡ ´ëºÎºÐ ³­Æ÷¸· ¼¼Æ÷ÀÎ ÀÌ Á¾¾çÀº È£¸£¸óÀ» »ý»êÇÒ ¼ö ÀÕ´Ù. ±×·¯³ª ¼ø¼öÇÑ ³­Æ÷¸· ¼¼Æ÷Á¾Àº µå¹°°í ´ëºÎºÐ Á¾¾çÀº ÁÖ·Î ¼¶À¯¸ð¼¼Æ÷·Î ±¸¼ºµÇ¾î ÀÖÀ¸¸ç È£¸£¸óÀ» »ý»êÇÏÁö ¾Ê´Â´Ù. 90%¿¡¼­ ÇÑÂÊ ³­¼Ò¿¡¼­¸¸ ¹ß»ýÇÑ´Ù. Á¾¾çÀº ȸ¹é»öÀÌ¸ç °íÇüÀÌ°í ±¸ÇüÀÌ¸ç ´Ü´ÜÇÏ´Ù. Á¶Á÷ÇÐÀûÀ¸·Î ¼¶À¯¸ð¼¼Æ÷¿Í ÄݶóÁ¨ °áü Á¶Á÷À¸·Î ±¸¼ºµÇ¾î ÀÖÀ¸¸ç ³­Æ÷¸· ¼¼Æ÷°¡ È¥ÀçÇÒ ¼ö ÀÖ´Ù. ȯÀÚ´Â °ñ¹ÝÅë°ú °ñ¹Ý Á¾±« µîÀÇ ºñƯÀÌÀû Áõ»óÀ» È£¼ÒÇϰųª º¹¼ö°¡ ³ªÅ¸³¯ ¼ö ÀÖ´Ù. ³­Æ÷¸· ¼¼Æ÷Á¾Àº ¾Ç¼ºÀÌ ¾ø´Ù.
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 4
femur head necrosis Aseptic or avascular necrosis of the femoral head. The major types are idiopathic (primary), as a complication of fractures or dislocations, and legg-perthes disease.
(12 Dec 1998)
fibrinoid necrosis Necrosis in which the necrotic tissue has some staining reactions resembling fibrin and becomes deeply eosinophilic, homogenous, and refractile.
(05 Mar 2000)
focal necrosis Occurrence of numerous, relatively small or tiny, fairly well-circumscribed, usually spheroidal portions of tissue that manifest coagulative, caseous, or gummatous necrosis and are characteristically associated with agents that are haematogenously disseminated; frequently observed only in histologic sections, but the foci may be as large as 1 to 3 mm and macroscopically visible; arbitrarily, foci larger than that are usually not termed focal necrosis.
(05 Mar 2000)
laminar cortical necrosis The breaking down of a definite cell layer in the cerebral cortex, encountered typically after temporary cardiac arrest or perinatal hypoxia.
(05 Mar 2000)
liquefactive necrosis A type of necrosis characterised by a fairly well-circumscribed, microscopically or macroscopically visible lesion that consists of the dull, opaque or turbid, gray-white to yellow-gray, soft or boggy, partly or completely fluid remains of tissue that became necrotic and was digested by enzymes, especially proteolytic enzymes liberated from disintegrating leukocytes; it is classically observed in abscesses, and frequently in infarcts of the brain.
(05 Mar 2000)
age factors Age as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or the effect of a circumstance. It is used with human or animal concepts but should be differentiated from aging, a physiological process, and time factors which refers only to the passage of time.
(12 Dec 1998)
bacteriocin factors Bacterial plasmid's responsible for the elaboration of bacteriocins.
Synonym: bacteriocin factors, bacteriocinogens.
(05 Mar 2000)
B-cell differentiation/growth factors Various substances, usually obtained from the supernatant of T-cell cultures, such as interleukin 4, 5, and 6. These substances are necessary for B-cell growth, maturation, and differentiation into plasma cells or B memory cells.
(05 Mar 2000)
biological factors Compounds made by living organisms. They have biological or physiological activities.
(12 Dec 1998)
biotic factors Environmental factor's or influences resulting from the activities of living organisms, as contrasted to those resulting from climatic, geological, or other factor's.
(05 Mar 2000)
blood coagulation factors Endogenous substances, usually proteins, that participate in the blood coagulation process.
(12 Dec 1998)
macrophage migration-inhibitory factors Proteins released by sensitised lymphocytes and possibly other cells that inhibit the migration of macrophages away from the release site. The structure and chemical properties may vary with the species and type of releasing cell.
(12 Dec 1998)
recognition factors Factors which effect "recognition" of target antigens by polymorphonuclear leukocytes; apparently the Fc portion of antibody molecules and the activated third component of complement (C3), for both of which phagocytes have receptor sites.
(05 Mar 2000)
glaucoma, risk factors If you belong to a high-risk group for glaucoma, have your eyes examined through dilated pupils every 2 years by an eye care professional. High-risk groups include everyone with a family history of glaucoma, everyone over the age of 60 and any Black over the age of 40. (Among Blacks, studies show that glaucoma is: 5 times more likely to occur in Blacks than in Whites and about 4 times more likely to cause blindness in Blacks than in Whites).
(12 Dec 1998)
releasing factors Hormones produced by the hypothalamus whichstimulate the release of other hormones in the pituitary gland.
(09 Oct 1997)
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
KMLE ¾àǰ/ÀǾàǰ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 4
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