¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"squamous cell hyperplasia"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 3
¿µ¹® glia cell ÇÑ±Û ¾Æ±³¼¼Æ÷
¼³¸í   
  ½Å°æ¼¼Æ÷ »çÀÌ¿¡¼­ ±×¹°±¸Á¶¸¦ ÀÌ·ç¸ç À̸¦ ÁöÁöÇϴ Á¶Á÷. ½Å°æ¾Æ±³¼¼Æ÷´Â ½Å°æ¸ð¼¼Æ÷¿Í °¥¶óÁø ¾Æ±³¸ð¼¼Æ÷°¡ ´Ù½Ã ¿©·¯ ÇüÅ·ΠºÐÈ­-¼ºÀåÇÑ °ÍÀÌ´Ù. ³ú½ÇÀ̳ª Ã´¼öÁ߽ɰüÀÇ º®À» µ¤°í ¿øÁÖ»ó ¶Ç´Â ÀÔ¹æÇüÀ̸ç, Ãʱ⿡´Â À¯¸®¸é¿¡ ¼¶¸ð°¡ ÀÖ´Ù. ´ëÇü¼¼Æ÷´Â º°³ú½Ç¸·¼¼Æ÷´Â ¾Æ±³¼¼Æ÷¶ó°í Çϸç, ½Å°æ¼¼Æ÷³ª ½Å°æ¼¶À¯ »çÀÌ¿¡ »êÀçÇÑ´Ù. ±× ¿Ü¿¡ Èñ¼Òµ¹±â¾Æ±³¼¼Æ÷µµ Æ÷ÇԵȴÙ.
¿µ¹® reserve cell ÇÑ±Û ¿¹ºñ¼¼Æ÷
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  ÀϹÝÀûÀ¸·Î »óÇÇÁ¶Á÷¿¡¼­ À̹̠ÀÖ´ø »óÇǼ¼Æ÷°¡ ¼Õ»óÀ» ¹Þ¾Æ »ç¸êÇϸ頸ŲãÁö´Â ±× ¹Ø¿¡ Àִ ¹ÌºÐÈ­¼¼Æ÷ ¿¹¸¦ µé¸é, ±â°üÁö ³»Ç¥¸éÀ» µ¤´Â ÁßÃþ ¿øÁÖ »óÇÇÀÇ ±âÀú¿¡ Àִ ÀÛÀº ¹ÌºÐÈ­ »óÇÇ ¼¼Æ÷.
¿µ¹® stem cell ÇÑ±Û Áٱ⼼Æ÷, °£¼¼Æ÷
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  Àڱ⠺¹Á¦¸¦ ÇÏ¿© ÀÚ½ÅÀ» Á¸¼Ó½ÃŰ¸é¼­ ÇÑÆíÀ¸·Î´Â Áõ½Ä°ú ºÐÈ­¸¦ ÇÏ¿© »õ·Î¿î ¼¼Æ÷¸¦ Çü¼ºÇϴ ¼¼Æ÷·Î¼­ Á¶Ç÷Áٱ⼼Æ÷°¡ ´ëÇ¥ÀûÀÌ´Ù. Á¶Ç÷Áٱ⼼Æ÷´Â °ñ¼ö¿¡ Àִ ¼¼Æ÷·Î¼­ ¸ðµç Ç÷±¸¼¼Æ÷°¡ ¿©±â¿¡¼­ ºÐÈ­µÇ¾î ¹ß»ýÇÑ´Ù.
¿µ¹® renal cell carcinoma ÇÑ±Û ÄáÆÏ¼¼Æ÷¾ÏÁ¾
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  ÄáÆÏ¿¡ »ý±ä ¿ø½ÃÄáÆÏÁ¶Á÷¿¡¼­ ¹ß»ýÇÑ ¾Ï. ÁַΠ¿ø½Ã¼¼´¢°üÁ¶Á÷¿¡¼­ ¹ß»ýÇÑ´Ù. ´ëÇ¥ÀûÀΠ¼¼Æ÷Á¶Á÷ÇüÀº ¿°»ö½Ã ¼¼Æ÷ÁúÀÌ ¸¼°Ô ºñ¾îº¸À̴ ¸¼Àº¼¼Æ÷¾ÏÁ¾ÀÌ´Ù. Ä¡·á´Â ¼ö¼ú°ú Ç×¾ÏÈ­Çпä¹ýÀ̸砾ÆÁÖ µå¹°Áö¸¸ ÀúÀý·Î ³´´Â °æ¿ìµµ Àִ °ÍÀ¸·Î º¸°íµÇ¾î ÀÖ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • follicular hyperplasia
    ÅÐÁý°ú´ÙÇü¼º
  • gingival hyperplasia
    ÀÕ¸öÁõ½Ä
  • glandular hyperplasia
    »ùÁ¾Áõ½Ä
  • hyperplasia
    °ú´ÙÇü¼º, °úÇü¼º, Áõ½Ä(Áõ)
  • intravascular papillary endothelial hyperplasia
    Ç÷°ü³»À¯µÎ¸ð¾ç³»ÇǼ¼Æ÷Áõ½Ä
  • intraductal hyperplasia
    °ü³»Áõ½Ä
  • lentiginous melanocytic hyperplasia
    Èæ»öÁ¡¸á¶ó´Ñ¼¼Æ÷Áõ½ÄÁõ
  • lipoid adrenal hyperplasia
    ÁöÁúºÎ½Å°ú´ÙÇü¼º
  • lymphoid hyperplasia
    ¸²ÇÁ±¸Áõ½Ä
  • nodular hyperplasia
    °áÀý°ú´ÙÇü¼º
  • oral focal hyperplasia
    ÀԾȱ¹¼Ò»óÇÇÁõ½Ä
  • papillary endothelial hyperplasia
    À¯µÎ¸ð¾ç³»ÇǼ¼Æ÷Áõ½Ä
  • pseudocarcinomatous hyperplasia
    °ÅÁþ¾ÏÁ¾Áõ½Ä
  • pseudoepitheliomatous hyperplasia
    °ÅÁþ»óÇÇÁ¾Áõ½Ä
  • reactive nodular hyperplasia
    ¹ÝÀÀ¼º°áÀýÁõ½Ä
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • goblet cell
    ¼úÀܼ¼Æ÷
  • hair cell
    Åм¼Æ÷
  • inflammatory cell
    ¿°Áõ¼¼Æ÷
  • killer cell
    »ìÇØ¼¼Æ÷
  • Kupffer's cell
    º°Å«Æ÷½Ä¼¼Æ÷, ÄíÆÛ¼¼Æ÷
  • mast cell
    ºñ¸¸¼¼Æ÷
  • mesenchymal cell
    Áß°£¿±¼¼Æ÷
  • mesothelial cell
    ÁßÇǼ¼Æ÷
  • mother cell
    ¸ð¼¼Æ÷, ¾î¹Ì¼¼Æ÷
  • neuroendocrine cell
    ½Å°æ³»ºÐºñ¼¼Æ÷
  • packed red blood cell
    ³óÃàÀûÇ÷±¸
  • parietal cell
    º®¼¼Æ÷
  • perivascular cell
    Ç÷°üÁÖÀ§¼¼Æ÷
  • plasma cell
    ÇüÁú¼¼Æ÷
  • polynucleated cell
    ¹µÇÙ¼¼Æ÷
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • hyperplasia
    °ú´ÙÇü¼º, Áõ½Ä(Áõ)
  • intravascular papillary endothelial hyperplasia
    Ç÷°ü³»À¯µÎ¸ð¾ç³»ÇǼ¼Æ÷Áõ½Ä
  • lentiginous melanocytic hyperplasia
    Èæ»öÁ¡¸á¶ó´Ñ¼¼Æ÷Áõ½Ä
  • lipoid adrenal hyperplasia
    ÁöÁúºÎ½Å°ú´ÙÇü¼º
  • lymphoid hyperplasia
    ¸²ÇÁ±¸Áõ½Ä
  • nodular hyperplasia
    °áÀý°ú´ÙÇü¼º
  • oral focal v hyperplasia
    ÀԾȱ¹¼Ò»óÇÇÁõ½Ä
  • papillary endothelial hyperplasia
    À¯µÎ¸ð¾ç³»ÇǼ¼Æ÷Áõ½Ä
  • pseudocarcinomatous hyperplasia
    °ÅÁþ¾ÏÁ¾Áõ½Ä
  • pseudoepitheliomatous hyperplasia
    °ÅÁþ»óÇÇÁ¾Áõ½Ä
  • reactive nodular hyperplasia
    ¹ÝÀÀ°áÀýÁõ½Ä
  • sebaceous hyperplasia
    ÇÇÁö»ùÁõ½Ä
  • syringolymphoid hyperplasia
    ¶¡»ù°ü¸²ÇÁ¸ð¾çÁõ½Ä
  • verrucous hyperplasia
    »ç¸¶±Í¸ð¾çÁõ½Ä
  • acantholytic cell
    °¡½Ã¼¼Æ÷ÇØ¸®¼¼Æ÷
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • B cell stimulating factor (BSF)
    B¼¼Æ÷ ÀÚ±ØÀÎÀÚ
  • B cell study
    B ¼¼Æ÷°Ë»ç
  • B cell/lymphocyte
    B ¼¼Æ÷/¸²ÇÁ±¸
  • B-cell
    ºñ ¼¼Æ÷
  • C-cell
    C ¼¼Æ÷
  • CD4+ cell
    CD4+ ¼¼Æ÷
  • Cell adhesion molecules
    ¼¼Æ÷À¯ÂøºÐÀÚ
  • Chinese hamster ovary tumor cell
    Áß±¹ÇÔ½ºÅÍ ³­¼Ò¼¼Æ÷, CHO¼¼Æ÷
  • Graham cell
    ±×¶óÇÔ¼¼Æ÷
  • Granulosa cell
    °ú¸³¸·¼¼Æ÷(Î¨Ø£Ø¯á¬øà)
  • Granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç(Î¨Ø£Ø¯á¬øàðþåË)
  • H-9 cell line
    H-9 ¼¼Æ÷°è
  • HeLa cell
    Çï¶ó¼¼Æ÷.
  • HeLa cell
    Çï¶ó¼¼Æ÷
  • Heidenhain s cell
    ÇÏÀ̵§ÇÏÀμ¼Æ÷.
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • squamous epithelitis
    ÆíÆò»óÇÇ¿° (¡­ß¾ù«æú)
  • squamous epithelium
    ÆíÆò»óÇÇ(ø·øÁß¾ù«).
  • squamous eruption
    Àμ³Çü ¹ßÁø(ìçàÚû¡Û¡òÖ)
  • squamous intraepithelial lesions
    ÆíÆò¼¼Æ÷ »óÇdz»º´º¯
  • squamous margin
    ºñ´Ã¸ð¼­¸®, Àο¬(ìçæÞ).
  • squamous margin of parietal bone
    µÎÁ¤°ñºñ´Ã¸ð¼­¸®, µÎÁ¤°ñÀο¬ºÎ.
  • squamous margin of sphenoid bone
    Á¢»ó°ñÀο¬ºÎ.
  • squamous metaplasia
    ÆíÆò»óÇÇÈ­»ý
  • squamous metaplasia
    ÆíÆò»óÇÇÈ­»ý(¡­ûùßæ)
  • squamous papilloma
    ÆíÆò¼¼Æ÷À¯µÎÁ¾(¡­)
  • squamous part
    ºñ´ÃºÎºÐ, ÀκÎ(ìçÝ»).
  • squamous part of occipital bone
    ÈĵΰñÀκÎ.
  • squamous part of temporal bone
    ÃøµÎ°ñÀκÎ.
  • squamous suture
    ºñ´ÃºÀÇÕ
  • stratified squamous epithelium
    ÁßÃþÆíÆò»óÇÇ(ñìöµø·øÁß¾ù«).
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • Cortical endocrine cell
    °ÑÁú³»ºÐºñ¼¼Æ÷
    [¿¾ ¿ë¾î] ÇÇÁú³»ºÐºñ¼¼Æ÷
  • Nodal cell
    °áÀý¼¼Æ÷
    [¿¾ ¿ë¾î] °áÀý¼¼Æ÷
  • Granule cell
    °ú¸³¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³¼¼Æ÷
  • Granular lutein cell
    °ú¸³ÃþȲ(»ö)ü¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³ÃþȲü¼¼Æ÷
  • Granulosa lutein cell
    °ú¸³ÃþȲ(»ö)ü¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³ÃþȲü¼¼Æ÷
  • Myoepithelial cell
    ±ÙÀ°»óÇǼ¼Æ÷
    [¿¾ ¿ë¾î] ±Ù»óÇǼ¼Æ÷
  • Myoid cell layer
    ±ÙÀ°¼¶À¯¸ð¼¼Æ÷Ãþ
    [¿¾ ¿ë¾î] ±Ù¼¶À¯¾Æ¼¼Æ÷Ãþ
  • Satellite cell of skeletal muscle
    ±ÙÀ°À§¼º¼¼Æ÷
    [¿¾ ¿ë¾î] ±ÙÀ§¼º¼¼Æ÷
  • Sebaceous cell
    ±â¸§»ù¼¼Æ÷
    [¿¾ ¿ë¾î] ÇÇÁö¼¼Æ÷
  • Centroacinar cell
    ²Ê¸®Á߽ɼ¼Æ÷
    [¿¾ ¿ë¾î] ¼±Æ÷Á߽ɼ¼Æ÷
  • Thecal cell
    ³­Æ÷¸·¼¼Æ÷
    [¿¾ ¿ë¾î] ³­Æ÷¸·¼¼Æ÷
  • Theca lutein cell
    ³­Æ÷¸·È²(»ö)ü¼¼Æ÷
    [¿¾ ¿ë¾î] ³­Æ÷¸·È²Ã¼¼¼Æ÷
  • Follicular cell
    ³­Æ÷¼¼Æ÷
    [¿¾ ¿ë¾î] ³­Æ÷¼¼Æ÷
  • Endothelial cell
    ³»ÇǼ¼Æ÷
    [¿¾ ¿ë¾î] ³»ÇǼ¼Æ÷
  • Ependymal cell
    ³ú½Ç¸·¼¼Æ÷
    [¿¾ ¿ë¾î] »óÀǼ¼Æ÷
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • centrifuge cell
    ¿ø½ÉºÐ¸®½Ç(êÀãýÝÂ×îãø)
  • competent cell
    Àû°Ý¼¼Æ÷(îêÌ«á¬øà)
  • constitutive secretory cell
    ±¸¼º¼º ºÐºñ¼¼Æ÷(ϰà÷àõÝÂÝôá¬øà)
  • continuous cell line
    "Áö¼Ó¼¼Æ÷ÁÖ(ò¥áÙá¬øàñ»), (ÔÒ) established cell line"
  • COS cell
    COS ¼¼Æ÷(á¬øà)
  • cytotoxic T cell
    ¼¼Æ÷µ¶¼º(á¬øàÔ¸àõ) T ¼¼Æ÷(á¬øà)
  • double-sector cell
    ÀÌÁß±¸È¹½Ç(ì£ñìÏ¡üñãø)
  • effector cell
    È¿°ú±â ¼¼Æ÷(üùÍýÐïá¬øà)
  • enucleated cell
    Á¦ÇÙ ¼¼Æ÷ (ð¶ú·á¬øà)
  • established cell line
    ¼ö¸³ ¼¼Æ÷ÁÖ (â§Ø¡á¬øàñ»)
  • feeder cell
    °ø±ÞÀÚ¼¼Æ÷ (ÍêÐåíºá¬øà)
  • flow cell
    È帧 ½Ç(ãø)
  • founder cell
    ½ÃÁ¶ ¼¼Æ÷(ã·ðÓá¬øà)
  • germ cell
    ¹è¼¼Æ÷(ÛÏá¬øà)
  • germinal cell
    ¹è¾Æ¼¼Æ÷(ÛÏä´á¬øà)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • granulosa cell
    °ú¸³¸·¼¼Æ÷
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç
  • islet cell
    µµ¼¼Æ÷
  • islet cell adenoma
    Ãéµµ¼¼Æ÷¼±Á¾
  • islet cell carcinoma
    µµ¼¼Æ÷¾ÏÁ¾
  • Langerhans' cell
    ¶û°Ô¸£Çѽº¼¼Æ÷
  • Langhans' glant cell
    ¶û±×Çѽº°Å¼¼Æ÷
  • large cell
    ´ë¼¼Æ÷
  • lymphoid cell
    ¸²ÇÁ¾ç¼¼Æ÷, ¸²ÇÁ°è¼¼Æ÷, ¸²ÇÁ±¸¾ç¼¼Æ÷
  • mast cell
    ºñ¸¸¼¼Æ÷
  • mastoid air cell
    À¯µ¹ºÀ¼Ò
  • mastoid cell
    À¯(¾ç)µ¹(±â)ºÀ¼Ò
  • nerve cell
    ½Å°æ¼¼Æ÷
  • neuroepithelial cell
    ½Å°æ»óÇǼ¼Æ÷
  • neuroglial cell
    ½Å°æ¾Æ±³¼¼Æ÷, ½Å°æ±³¼¼Æ÷
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
SCCC squamous cell cervical carcinoma
SCCHN squamous cell carcinoma of the head and neck
SCV sensory nerve conduction velocity; smooth, capsulated, virulent; subclavian vein; squamous-cell carc...
BCH basal cell hyperplasia
CCH C-cell hyperplasia; chronic chloride hemagglutination; chronic cholestatic hepatitis
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
SCC Squamous Cell Carcinoma Antigen
SCC Squamous carcinoma cell
SCCA Squamous cell carcinoma
SQCC Squamous cell carcinoma
SCCA Squamous cell carcinoma antigen
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • inflammatory hyperplasia
    ¿°Áõ¼º °úÇü¼º, ¿°Áõ¼º Áõ½Ä
    ¿°Áõ¿¡ ÀÇÇÏ¿© »ý±â´Â °úÇü¼º.
  • lentiginous melanocytic hyperplasia(ÈæÀÚ¼º ¸á¶ó´Ñ ¼¼Æ÷ Áõ½Ä)

    lentiglobus

    °ø ¸ð¾ç ¼öÁ¤Ã¼, ±¸Çü ¼öÁ¤Ã¼
    ¼öÁ¤Ã¼ Àü¸éÀÌ ±¸ÇüÀ¸·Î µÇ¾î¼­, ¼öÁ¤Ã¼ÀÇ °î¸éÀÌ °úÀåµÇ¾î º¸ÀÌ´Â °Í.
  • papillary hyperplasia
    À¯µÎ»ó Áõ½Ä, À¯µÎ Áõ½Ä
    1. ±¹¼ÒÀû ÀÚ±Ø, ºÒ·®ÇÑ ±¸°­ À§»ý, °¨¿°¿¡ ÀÇÇØ ¹ß»ýµÇ´Â »óÇÇÀÇ ´Ù¹ß¼º À¯µÎ»ó µ¹Ãâ. 2. ±¸°³ À¯µÎÁ¾ÁõÀ̶ó°íµµ ÇÑ´Ù. ÀÇÄ¡ ÇϹæ Á¡¸·¿¡ »ý±ä Ä­µð´ÙÁõÀº °æ±¸°³°¡ ÀÌ·¯ÇÑ Áõ½Ä¼º Á¶Á÷ ¹ÝÀÀÀ» ÀÏÀ¸Å°´Â ÀڱؿøÀÌ µÈ´Ù.
  • paracortical lymphoid hyperplasia
    ºÎÇÇÁú¼º ¸²ÇÁ¾ç ¼¼Æ÷ Áõ½Ä
  • perforating hyperplasia
    õ°ø¼º Áõ½Ä
  • pseudocarcinomatous hyperplasia
    °¡¼º ¾ÏÁ¾¼º Áõ½Ä
  • salt-losing adrenal hyperplasia
    ¿°·ù ¼Ò½Ç¼º ºÎ½Å Áõ½ÄÁõ
  • total hyperplasia
    Àüü °ú´Ù Çü¼º
  • clear cell basal cell carcinoma
    Åõ¸í ¼¼Æ÷ ±âÀú¼¼Æ÷¾Ï
  • Abbe-Zeiss counting cell
    ¾Ðº£-ÀÚÀ̽º Ç÷±¸ °è»ê
  • abnormality of cell interaction
    ¼¼Æ÷ »óÈ£ÀÛ¿ë ÀÌ»ó
    ¼¼Æ÷ »çÀÌ¿¡ ÀϾ´Â ÀÛ¿ëÀÌ ºñÁ¤»óÀûÀÎ °Í.
  • absolute cell increase
    Àý´ë ¼¼Æ÷ ¼ö Áõ°¡
  • absorptive cell
    Èí¼ö ¼¼Æ÷
  • accessory cell
    ºÎ¼¼Æ÷
    °ñÀú¼± Áß¿¡¼­ ÁÖ¼¼Æ÷, ¹æ¼¼Æ÷¿¡ ¼¯¿©¼­ Á¸ÀçÇÑ´Ù. ÀÔ¹æÇüÀ̸ç Á¡¾×¼ºÀÇ ¹°ÁúÀ» °£Á÷ÇÑ´Ù. ÇÙÀº ¼¼Æ÷Àú¿¡ Ä¡¿ìÃÄ ÀÖ¾î ÆíÆò¿¡ °¡±õ´Ù.
  • acinic cell carcinoma
    ¼±¹æ ¼¼Æ÷ ¾ÏÁ¾
    1. ¼±¹æ ¼¼Æ÷, ±Ù»óÇǼ¼Æ÷°¡ Áõ½ÄÇÏ¿© Çü¼ºµÇ°í ³·Àº ¾Ç¼ºµµ¸¦ º¸ÀδÙ. 2. Ÿ¾×¼± ¾à¼º Á¾¾ç Áß 5¹øÂ°ÀÇ ¹ß»ý ºñÀ²À» °®´Â Á¾¾çÀ¸·Î 90%¿¡¼­ ÀÌÇϼ±¿¡¼­ ¹ß»ýÇÏ¸ç ¾ÇÇϼ±°ú ¼ÒŸ¾×¼±¿¡¼­µµ µå¹°°Ô ¹ß»ýÇÑ´Ù. ¿©¼º¿¡¼­ ´Ù¼Ò È£¹ßÇϰí, ¾î´À ¿¬·É¿¡¼­³ª ¹ß»ýÇϳª ÁÖ·Î 30-70´ë¿¡ °ñ°í·ç ¹ß»ýÇÑ´Ù. ¿¹Àü¿¡´Â ¾ç¼ºÀ¸·Î »ý°¢ÇÏ¿© ¼±¹æ¼¼Æ÷Á¾À̶ó ºÎ¸¥ ÀûÀÌ ÀÖÀ¸³ª ºÐ¸íÇÑ ¾Ç¼ºÀ¸·Î ¼±¾ÏÁ¾À¸·Î ºÎ¸¥´Ù. Á¾¾ç ¼¼Æ÷´Â Àå¾×¼º ¼±¹æ¼¼Æ÷¿Í À¯»çÇÏÁö¸¸ ¿©·¯ °¡Áö ´Ù¸¥ ¼¼Æ÷µéÀÌ ³ªÅ¸³ª¸ç, °³Á¦°ü ¿¹ºñ¼¼Æ÷¿¡¼­ ±â¿øÇÑ´Ù°í º»´Ù.
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
angiolymphoid hyperplasia with eosinophilia Solitary or multiple benign cutaneous nodules comprised of immature and mature vascular structures intermingled with endothelial cells and a varied infiltrate of eosinophils, histiocytes, lymphocytes, and mast cells.
(12 Dec 1998)
atypical melanocytic hyperplasia Proliferation of melanocytes showing nuclear atypicality, especially as scattered single cells high in the epidermis; interpreted by some pathologists as malignant melanoma in situ.
(05 Mar 2000)
benign giant lymph node hyperplasia Solitary masses of lymphoid tissue containing concentric perivascular aggregates of lymphocytes, occurring usually in the mediastinum or hilar region of young adults; similar changes have been reported outside the mediastinum and, if associated with interfollicular sheets of plasma cells, may progress to lymphoma or plasmacytoma.
Synonym: angiofollicular mediastinal lymph node hyperplasia, Castleman's disease.
(05 Mar 2000)
benign prostatic hyperplasia <urology> A benign enlargement of the prostate gland begins normally after age 50 years probably secondary to the effects of male hormones. If significant enlargement occurs, it may pinch off te urethra making urination difficult or impossible.
See: urinary retention.
Origin: Gr. Plassein = to form
(06 Aug 1998)
verrucous hyperplasia A non-invasive precursor of verrucous or squamous carcinoma of the oral mucosa, occurring in the elderly, characterised by sharp or blunt upward papillary projections of squamous epithelium.
(05 Mar 2000)
giant lymph node hyperplasia Large benign, hyperplastic lymph nodes. The more common hyaline vascular subtype is characterised by small hyaline vascular follicles and interfollicular capillary proliferations. Plasma cells are often present and represent another subtype with the plasma cells containing IgM and IgA.
(12 Dec 1998)
gingival hyperplasia Non-inflammatory enlargement of the gingivae produced by factors other than local irritation. It is characteristically due to an increase in the number of cells.
(12 Dec 1998)
cementum hyperplasia A regressive change of teeth characterised by excessive development of secondary cementum on the tooth surface. It may occur on any part of the root, but the apical two-thirds are most commonly affected.
(12 Dec 1998)
congenital adrenal hyperplasia <endocrinology> A genetic disorder present at birth characterised by a deficiency of the hormones aldosterone and cortisol and an overproduction of male sex hormones (androgens). In males this may manifest as enlarged penis, small testes and early development of masculine characteristics. In females features include ambiguous genitalia, failure to menstruate, deep voice and excessive hair.
Origin: Gr. Plassein = to form
(27 Sep 1997)
congenital sebaceous hyperplasia Misnomer for nevus sebaceus.
(05 Mar 2000)
congenital virilizing adrenal hyperplasia A series of inherited inborn errors of metabolism with hyperplasia of the adrenal cortex and overproduction of virilizing hormones. Most common forms are due to partial or complete 21-hydroxylase deficiency, leading to increased ACTH production by the pituitary, stimulating adrenal growth and function. Severe form is characterised by salt-losing state.
(05 Mar 2000)
cystic hyperplasia Formation of multiple retention cysts from obstruction of ducts or glands by hyperplasia of the lining epithelium, as in fibrocystic disease of the breast and metropathia haemorrhagica.
(05 Mar 2000)
cystic hyperplasia of the breast A benign disease common in women of the third, fourth, and fifth decades characterised by formation, in one or both breasts, of small cysts containing fluid which may appear as blue dome cysts; associated with stromal fibrosis and with variable degrees of intraductal epithelial hyperplasia and sclerosing adenosis.
Synonym: cystic hyperplasia of the breast.
(05 Mar 2000)
prostatic hyperplasia Enlargement or overgrowth of the prostate gland as a result of an increase in the number of its constituent cells.
(12 Dec 1998)
pseudoepitheliomatous hyperplasia Pseudocarcinomatous hyperplasia, a benign marked increase and downgrowth of epidermal cells, observed in chronic inflammatory dermatoses; microscopically, it resembles well-differentiated squamous cell carcinoma.
(05 Mar 2000)
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