| ROAD | reversible obstructive airways disease |
|---|---|
| RSC | rat spleen cell; rested state contraction; reversible sickle-cell; right subclavian |
| AVPU | alert, verbal, painful, unresponsive [neurologic test] |
| NA | Avogadro constant or number; nalidixic acid; Narcotics Anonymous; network administrator; neuraminida... |
| NC | nasal cannula; nasal clearance; neck complaint; neonatal cholestasis; neural crest; neurologic check... |
| ischemic optic neuropathy | Optic nerve neuropathy secondary to hypoperfusion of the low pressure posterior ciliary arteries supplying the optic nerve head (nonarteritic) or to temporal arteritis (arteritic). (05 Mar 2000) |
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| ischemic preconditioning | A technique in which tissue is rendered resistant to the deleterious effects of prolonged ischemia and reperfusion by prior exposure to brief, repeated periods of vascular occlusion. (am j physiol 1995 may;268(5 pt 2):h2063-7, abstract) (12 Dec 1998) |
| ischemic preconditioning, myocardial | Exposure of myocardial tissue to brief, repeated periods of vascular occlusion in order to render the myocardium resistant to the deleterious effects of ischemia or reperfusion. The period of pre-exposure and the number of times the tissue is exposed to ischemia and reperfusion vary, the average being 3 to 5 minutes. (12 Dec 1998) |
| optic neuropathy, ischemic | A severely blinding disease resulting from loss of the arterial blood supply to the optic nerve as a result of occlusive disorders of the nutrient arteries. Optic neuropathy is divided into anterior, which causes a pale oedema of the optic disk, and posterior, in which the optic disk is not swollen and the abnormality occurs between the globe and the optic chiasm. Ischemic anterior optic neuropathy usually causes a loss of vision that may be sudden or occur over several days; ischemic posterior optic neuropathy is uncommon and the diagnosis depends largely upon exclusion of other causes, chiefly stroke and brain tumour. (12 Dec 1998) |
| transient ischemic attack | A sudden focal loss of neurological function with complete recovery usually within 24 hours; caused by a brief period of inadequate perfusion in a portion of the territory of the carotid or vertebral basilar arteries. (05 Mar 2000) |