| MODY | Maturity-Onset Diabetes of the Young(in Youth) |
|---|---|
| MRDM | Malnutrition Related Diabetes Mellitus |
| NIDDM | Non-Insulin Dependent Diabetes Mellitus = Type II DM |
| ADA | adenosine deaminase; American Dental Association; American Dermatological Association; American Diab... |
| AOD | Academy of Operative Dentistry; Academy of Oral Dynamics; adult onset diabetes; anesthesiologist-on-... |
| diabetes, gestational | A diabetic condition that appears during pregnancy (gestation) and may go away after the birth of the baby. (12 Dec 1998) |
|---|---|
| diabetes innocens | The recurring or persistent excretion of glucose in the urine, in association with blood glucose levels that are in the normal range; results from the failure of proximal renal tubules to reabsorb glucose at a normal rate from the glomerular filtrate (low renal threshold); defect in the glucose carrier in the nephron. Synonym: diabetes innocens, normoglycaemic glycosuria, renal diabetes. (05 Mar 2000) |
| diabetes insipidus | <disease, endocrinology> Rare form of diabetes in which the kidney tubules do not reabsorb sufficient water. This can be because (a) either the renal tubules have defective receptors for antidiuretic hormone (ADH, vasopressin) or (b) a class of aquaporin water channel in the collecting duct is defective or (c) there is inadequate ADH production by the pituitary, leading to the excessive production of dilute urine. (18 Nov 1997) |
| diabetes insipidus, nephrogenic | A rare congenital and familial form of diabetes insipidus, resulting from failure of the renal tubules to absorb water. There is excessive production of antidiuretic hormones but the tubules fail to respond to it. (12 Dec 1998) |
| diabetes intermittens | Diabetes mellitus in which there are periods of relatively normal carbohydrate metabolism followed by relapses to the previous diabetic state. (05 Mar 2000) |
| diabetes mellitus | <disease> Relative or absolute lack of insulin leading to uncontrolled carbohydrate metabolism. In juvenile onset diabetes (that may be an autoimmune response to pancreatic _ cells) the insulin deficiency tends to be almost total, whereas in adult onset diabetes there seems to be no immunological component but an association with obesity. (18 Nov 1997) |
| diabetes mellitus, experimental | Diabetes mellitus induced experimentally by administration of various diabetogenic agents or by pancreatectomy. (12 Dec 1998) |
| diabetes mellitus, insulin-dependent | Diabetes mellitus characterised by insulin deficiency, sudden onset, severe hyperglycaemia, rapid progression to ketoacidosis, and death unless treated with insulin. The disease may occur at any age, but is most common in childhood or adolescence. (12 Dec 1998) |
| diabetes mellitus, lipoatrophic | A disorder characterised by complete absence of subcutaneous adipose tissue, insulin-resistant diabetes with little tendency to ketoacidosis, hyperlipidemia with subcutaneous xanthomatosis, hepatomegaly, and an elevated basal metabolic rate. (12 Dec 1998) |
| diabetes mellitus, non-insulin-dependent | Diabetes characterised by the ability to survive without ketoacidosis in the absence of insulin therapy. It is usually of slow onset and patients exhibit a tendency to obesity. (12 Dec 1998) |
| diabetes related peptide | <hormone, protein> Peptide of 37 amino acids that selectively inhibits insulin stimulated glucose uptake in muscle. Structurally related to calcitonin gene-related peptide. (15 Oct 1997) |
| diabetes, type 1 | Insulin dependent diabetes or juvenile diabetes. (12 Dec 1998) |
| diabetes, type 2 | Non-insulin dependent diabetes, adult-onset diabetes or insulin-resistant diabetes. (12 Dec 1998) |
| insulin dependent diabetes | A form of diabetes that requires the daily injection of the hormone insulin to maintain normal body chemistry. Avoidance of insulin in these patients will result in a severe metabolic derangement known as diabetic ketoacidosis. (27 Sep 1997) |
| insulin-dependent diabetes mellitus | A chronic condition in which the pancreas makes little or no insulin because the beta cells have been destroyed. The body is then not able to use the glucose (blood sugar) for energy. IDDM usually comes on abruptly, although the damage to the beta cells may begin much earlier. The signs of IDDM are a great thirst, hunger, a need to urinate often, and loss of weight. To treat the disease, the person must inject insulin, follow a diet plan, exercise daily, and test blood glucose several times a day. IDDM usually occurs in children and adults who are under age 30. This type of diabetes used to be known as juvenile diabetes, juvenile-onset diabetes, and ketosis-prone diabetes. (09 Oct 1997) |