| LIBC | latent iron-binding capacity |
|---|---|
| LMP | large multifunctional protease; last menstrual period; latent membrane potential; left mentoposterio... |
| LP | labile peptide; labile protein; laboratory procedure; lactic peroxidase; lamina propria; laryngophar... |
| MLN | manifest latent nystagmus; membranous lupus nephropathy; mesenteric lymph node; motilin |
| SL | systolic wave, latent |
| anhepatogenous jaundice | Jaundice due to haemolysis, with normal function of the liver and biliary tract. Synonym: anhepatogenous jaundice. (05 Mar 2000) |
|---|---|
| painless jaundice | Jaundice not associated with abdominal pain; usually used for obstructive jaundice resulting from obstruction of the common bile duct at the head of the pancreas by a tumour or impaction of a stone. (05 Mar 2000) |
| malignant jaundice | Jaundice associated with high fever and delirium; seen in severe hepatitis and other diseases of the liver with severe functional failure. Synonym: malignant jaundice. (05 Mar 2000) |
| catarrhal jaundice | An obsolete term for viral hepatitis type A. (05 Mar 2000) |
| regurgitation jaundice | Jaundice due to biliary obstruction, the bile pigment having been conjugated and secreted by the hepatic cells and then reabsorbed into the bloodstream. (05 Mar 2000) |
| mechanical jaundice | Jaundice resulting from obstruction to the flow of bile into the duodenum, whether intra-or extrahepatic. Synonym: mechanical jaundice. (05 Mar 2000) |
| retention jaundice | Jaundice due to insufficiency of liver function or to an excess of bile pigment production; the bilirubin is unconjugated because it has not passed through the liver cells; van den Bergh test is indirect. (05 Mar 2000) |
| choleric jaundice | Jaundice with the presence of biliary derivatives in the urine; occurs in regurgitation hyperbilirubinaemia. (05 Mar 2000) |
| cholestatic jaundice | Jaundice produced by inspissated bile or bile plugs in small biliary passages in the liver. (05 Mar 2000) |
| chronic acholuric jaundice | <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane. This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged. Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal. (27 Sep 1997) |
| chronic familial jaundice | <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane. This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged. Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal. (27 Sep 1997) |
| chronic idiopathic jaundice | <syndrome> An inherited disorder (autosomal recessive) that is characterised by long-standing mild jaundice. This occurs secondary to an abnormality in the transport of bilirubin from the liver to the biliary system. This leads to an accumulation of bilirubin in the liver. Avoidance of alcohol and medications which can affect the liver is important. Inheritance: autosomal recessive. (27 Sep 1997) |
| physiologic jaundice | Icterus which can be accentuated by many factors including excessive haemolysis, sepsis, neonatal hepatitis or congenital atresia of the biliary system. Synonym: physiologic icterus, jaundice of the newborn, neonatal jaundice, physiologic jaundice. (05 Mar 2000) |
| congenital haemolytic jaundice | <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane. This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged. Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal. (27 Sep 1997) |
| postarsphenamine jaundice | Liver toxicity, causing jaundice, in a patient who has received arsphenamine. (05 Mar 2000) |