| IDA | 1) Imino-Diacetic Acid 2) Iron Deficiency Anemia &nb... |
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| IGHD | Idiopathic Growth Hormone Deficiency |
| MEDAC Syndrome | Multiple-Endocrine Deficiency Autoimmune-Candidiasis |
| MR | 1) Mitral Regurgitation = MI 2) Minor Response... |
| PKD | Pyruvate Kinase Deficiency |
| immunologic and biological factors | A collective grouping for biologically active substances that play a role in the functioning of the immune system and those that show biological or physiological activity. (12 Dec 1998) |
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| immunologic capping | The process by which lymphoid cell surface immunoglobulin receptors, when exposed to bivalent anti-ig antibodies, collect in patches and form a cap at one pole of the cell. The caps may then be endocytosed or shed into the environment in the form of antigen-antibody complexes. Capping has also been induced by lectins and antigens. (12 Dec 1998) |
| immunologic diseases | Disorders caused by abnormal or absent immunologic mechanisms, whether humoral, cell-mediated or both. (12 Dec 1998) |
| immunologic factors | Biologically active substances whose activities affect or play a role in the functioning of the immune system. (12 Dec 1998) |
| immunologic high dose tolerance | Induction of tolerance by exposure to large amounts of protein antigens. (05 Mar 2000) |
| immunologic memory | <immunology> The ability to rapidly produce large quantities of specific immune cells after subsequent exposure to a previously encountered antigen. (09 Oct 1997) |
| immunologic pregnancy test | A general term for test's for detection of increased human chorionic gonadotropin in plasma or urine by immunologic techniques including latex particle agglutination, haemagglutination inhibition, radioimmunoassay, and radioreceptor assays. (05 Mar 2000) |
| immunologic surveillance | The theory that T-cells monitor cell surfaces and detect structural changes in the plasma membrane and/or surface antigens of virally or neoplastically transformed cells. (12 Dec 1998) |
| immunologic tests | Diagnostic techniques involving the demonstration or measurement of an immune response, including antibody production or assay, antigen-antibody reactions, serologic cross-reactivity, delayed hypersensitivity reactions, or heterogenetic responses. (12 Dec 1998) |
| immunologic tolerance | Lack of immune response to antigen. Theories of tolerance induction include clonal deletion and clonal anergy. In clonal deletion, the actual clone of cells is eliminated whereas in clonal anergy the cells are present but nonfunctional. Synonym: immunological tolerance, immunotolerance, nonresponder tolerance. (05 Mar 2000) |
| overgrowth syndromes | Conditions with multiple abnormalities including excessive growth. Early overgrowth syndromes that affecting children include the fragile x and beckwith-wiedemannn syndromes. Overactivity of the pituitary gland with overproductiuon of growth hormone causes overgrowth before adolescence and a distinctive pattern of overgrowth called acromegaly. (12 Dec 1998) |
| euthyroid sick syndromes | Abnormalities in thyroid hormone and thyroid-stimulating hormone levels, often simulating hypothyroidism, in euthyroid patients suffering some other illness, such as diabetes mellitus or liver cirrhosis. (12 Dec 1998) |
| abdominal muscle deficiency syndrome | <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear. (05 Mar 2000) |
| adult lactase deficiency | Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults. (05 Mar 2000) |
| alpha-1 antitrypsin deficiency | <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues. The lack of this protein leads to damage of various organs, but mainly to the lung and liver. symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant (12 Dec 1998) |
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