| ¿µ¹® | congenital heart disease | ÇÑ±Û | ¼±Ãµ½ÉÀ庴 |
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| ¼³¸í | ¼±ÃµÀûÀ¸·Î ½ÉÀåÀÇ ±¸Á¶¿¡ ÀÌ»óÀÌ ÀÖ´Â º´. |
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| ¿µ¹® | venereal disease, sexually transmitted diseases | ÇÑ±Û | ¼ºº´ |
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| ¼³¸í | º¸ÆíÀûÀ¸·Î ¼º±³ ¶Ç´Â ¼º±âÁ¢ÃË¿¡ ÀÇÇØ °É¸®´Â Á¢ÃË Àü¿°º´À¸·Î ¸Åµ¶, ÀÓÁú, ¹«¸¥±Ë¾ç, »ô±¼À°¾ÆÁ¾ µîÀ» ¸»ÇÑ´Ù. Ä¡·á´Â ¿øÀαտ¡ µû¸¥ ÀûÀýÇÑ Ç×»ý¿ä¹ýÀÌ´Ù. |
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| ¿µ¹® | VDRL(venereal disease research laboratory) | ÇÑ±Û | ¼ºº´ ¿¬±¸½ÇÇè½Ç |
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| ¼³¸í | ¸Åµ¶±ÕÀÇ reagin¿¡ ´ëÇÑ Ç×ü. ¸Åµ¶ÀÇ ¼±º°°Ë»ç·Î »ç¿ëµÇ¸ç ¸Åµ¶ Ä¡·á¿¡ ´ëÇÑ ¹ÝÀÀ ¿©ºÎ¿Í Ä¡·áÈ¿°ú ÆÇÁ¤¿¡µµ »ç¿ëµÈ´Ù. ¸Åµ¶¿Ü¿¡µµ Àü½Å¼ºÈ«¹Ý¼º³¶Ã¢, ·ù¸¶Æ¼½º°üÀý¿° µî¿¡¼µµ ¾ç¼ºÀ¸·Î ³ªÅ¸³¯ ¼ö ÀÖ´Ù. |
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| ¿µ¹® | hemolytic disease of newborn | ÇÑ±Û | ½Å»ý¾Æ¿ëÇ÷º´ |
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| ¼³¸í | ½Å»ý¾Æ¿¡¼ ÀûÇ÷±¸°¡ ºñÁ¤»óÀûÀ¸·Î ¸¹ÀÌ ÆÄ±«µÇ´Â º´À¸·Î žÆÀû¸ð±¸Áõ(erythroblastosis fetalis)¿Í °°Àº ¶æÀ¸·Î ¾²ÀδÙ. À̰ÍÀº ¾î¸Ó´Ï¿¡°Ô¼ »ý»êµÈ ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸¿¡ ´ëÇÑ Ç×ü°¡ ŹÝÀ» °Ç³Ê¿Í¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÇÏ¿©¼ »ý±â´Â ¿ëÇ÷¼ººóÇ÷À» À̸£´Â ¸». Áï ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸ÀÇ Ç×ü°¡ ¾î¸Ó´ÏÀÇ ¸ö¿¡¼ »ý»êÀÌ µÇ°í À̰ÍÀÌ Å¹ÝÀ» ÅëÇØ¼ žƿ¡°Ô ³Ñ¾î°¡¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÀ» Çϰí ÀÌ Ç×ü¿Í °áÇÕÇÑ ÀûÇ÷±¸´Â ÆÄ±«°¡ µÇ¾î¼ ºóÇ÷ÀÌ »ý±ä °ÍÀ» žÆÀû¸ð±¸ÁõÀ̶ó°í ÇÑ´Ù. À̰ÍÀº Rh Àû¸ð±¸Áõ(Rh erythroblastosis)¿Í ABO Àû¸ð±¸Áõ(ABO erythroblastosis)·Î ³ª´ ¼ö°¡ ÀÖ´Ù. |
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| ¿µ¹® | coronary artery disease | ÇÑ±Û | ½ÉÀ嵿¸Æº´ |
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| ¼³¸í | ½ÉÀ嵿¸ÆÀ̶õ ½ÉÀå¿¡ Ç÷¾×À» °ø±ÞÇÏ´Â µ¿¸ÆÀ» ¸»ÇÑ´Ù. ½ÉÀåµµ ´Ù¸¥ ±ÙÀ°°ú ¸¶Âù°¡Áö·Î Ç÷¾×À» °ø±Þ¹Þ¾Æ¾ß ¼öÃàÀ» ÇÒ ¼ö°¡ ÀÖ´Â ±â°üÀ¸·Î ½ÉÀåÀÇ Ç÷¾×À» °ø±ÞÇÏ´Â µ¿¸ÆÀ» ½ÉÀ嵿¸ÆÀ̶ó°í ÇÑ´Ù. ½ÉÀ嵿¸Æº´À̶õ ½ÉÀ嵿¸ÆÀÇ ³»°æÀÌ Á¼¾ÆÁ®¼ »ý±â´Â ÁúȯÀ¸·Î ÇãÇ÷½ÉÀ庴(ischemic heart disease)À̶ó°íµµ ºÒ¸°´Ù. °ü»óµ¿¸ÆÀÌ Á¼¾ÆÁú °æ¿ì¿¡´Â ½ÉÀåÀ¸·Î °¡´Â Ç÷¾×ÀÇ ¾çÀÌ Àû¾îÁ®¼ ½ÉÀå¿¡ ÃæºÐÇÑ Ç÷¾×ÀÌ °ø±ÞÀÌ µÇÁö ¸øÇϹǷΠ¿©·¯ °¡Áö º´ÀûÀÎ Çö»óÀÌ »ý±ä´Ù. ½ÉÀ嵿¸ÆÀÌ Á¼¾ÆÁö´Â µ¥¿¡´Â ¿©·¯ °¡Áö ¿øÀÎÀÌ ÀÖÀ» ¼ö°¡ ÀÖÀ¸³ª ÁÖ·Î ½ÉÀ嵿¸ÆÀÇ µ¿¸Æ°æÈÁõ¿¡ ÀÇÇÑ´Ù. µ¿¸Æ°æÈÁõÀ̶õ µ¿¸ÆÀÇ ³»Ãþ¿¡ Áö¹æ°ú ÄÝ·¹½ºÅ×·Ñ·Î ÀÌ·ç¾îÁø Á×Á¾(atheroma)°¡ »ý±â´Â ÁúȯÀ¸·Î Á×Á¾ÀÌ »ý±ä µ¿¸ÆÀº Á×Á¾ÀÌ Ç÷°üÀÇ ³»ºÎ·Î µ¹ÃâÇÏ°Ô µÇ¾î¼ Ç÷°üÀÇ ³»°æÀÌ ÀÛ¾ÆÁö°Ô µÈ´Ù. ½ÉÀ嵿¸Æº´Àº ±× Á¤µµ¿¡ µû¶ó¼ Çù½ÉÁõ(angina pectoris)¿Í ½É±Ù°æ»öÁõ(myocardial infarction)À¸·Î ³ª´«´Ù. Çù½ÉÁõÀº ½ÉÀ嵿¸ÆÀÇ ºÎºÐÀû Æó¼â¿¡ ÀÇÇØ¼ Æò»ó½Ã¿¡´Â Áõ»óÀÌ ¾øÁö¸¸ ½ÉÀåÀÌ ¸¹Àº Ȱµ¿À» ÇÒ °æ¿ì¿¡ ½ÉÀå¿¡ Çǰ¡ ÃæºÐÇÑ ¸¸Å °ø±ÞÀÌ µÇÁö ¾Ê¾Æ¼ »ý±â´Â Áúº´À¸·Î Æò»ó½Ã¿¡ ½¯ °æ¿ì¿¡´Â ¾Æ¹« Áõ»óÀÌ ¾øÁö¸¸ ¿îµ¿À̳ª °ú½Ä µîÀÇ ¿øÀÎÀ¸·Î ½ÉÀåÀÌ ¸¹Àº ¿îµ¿À» ÇÒ °æ¿ì¿¡ ½ÉÀå¿¡ °ø±ÞµÇ´Â Ç÷¾×ÀÇ ¾çÀÌ ¸ðÀÚ¶ó¼ Áõ»ó(´ë°³ °¡½¿ºÎÀ§¿¡ Áã¾îÂ¥´Â µíÇÑ ÅëÁõ)ÀÌ »ý±ä´Ù. ½É±Ù°æ»öÁõÀ̶õ ½ÉÀ嵿¸ÆÀÇ ¿ÏÀüÆó¼â¿¡ ÀÇÇØ¼ ½ÉÀåÀÇ ±ÙÀ°ÀÌ Ç÷¾×À» ÀüÇô °ø±Þ¹ÞÁö ¸øÇؼ ½ÉÀåÀÇ ±ÙÀ°ÀÌ ½â´Â °æ¿ì¸¦ ¸»ÇÑ´Ù. |
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| PD | Doctor of Pharmacy; Dublin Pharmacopoeia; interpupillary distance; Paget disease; pancreatic duct; p... |
|---|---|
| RD | radial deviation; radiology department; rate difference; Raynaud disease; reaction of degeneration; ... |
| ALL | Acute Lymphocytic Leukemia ÇüÅÂÇÐÀû ºÐ·ù L1; Small, Homogenous(... |
| MEN | Multiple Endocrine Neoplasia ; AD Trait 1. MEN Type I(= Wermer Syndro... |
| CHD | Chediak-Higashi disease; childhood disease; chronic hemodialysis; congenital or congestive heart dis... |
| storage protein deamidase | <enzyme> Deamidates glutaminyl residues; isolated from germinating wheat grains Registry number: EC 3.5.1.- Synonym: seed storage protein deamidase (26 Jun 1999) |
|---|---|
| information storage and retrieval | A branch of computer or library science relating to the storage, locating, searching, and selecting, upon demand, relevant data on a given subject. (12 Dec 1998) |
| iron-storage protein | <chemical> Soluble iron storage protein from e coli; different from ferritin and haemosiderin (05 Dec 1998) |
| optical storage devices | A computer disk read by a laser beam, containing data prerecorded by a vendor. The buyer cannot enter or modify data in any way but the advantages lie in the speed of accessibility, relative immunity to damage, and relatively low cost of purchase. (12 Dec 1998) |
| lead storage battery | <chemistry> A battery (used in cars) in which the anode is lead, the cathode is lead coated with lead dioxide, and the electrolyte is a sulfuric acid solution. (09 Jan 1998) |
| lipid storage diseases | A series of disorders due to inborn errors in lipid metabolism resulting in the abnormal accumulation of lipids in the wrong places (examples include gaucher, fabry and niemann-pick diseases and metachromatic leukodystrophy). (12 Dec 1998) |
| lysosomal storage diseases | Inborn errors of metabolism characterised by defects in specific lysosomal hydrolases and resulting in intracellular accumulation of unmetabolised substrates. (12 Dec 1998) |
| gaucher's disease, type 1 | A progressive genetic disease caused by a defect in an enzyme. The enzyme, called glucocerebrosidase, is needed to break down the chemical glucocerebroside. The enzyme defect in persons with Gaucher's disease (GD) leads to the accumulation of glucocerebroside in the spleen, liver, and lymph nodes. The most common early sign is enlargement of the spleen (located in the upper left abdomen). Other signs include low red blood cell counts (anaemia), a decrease in blood clotting cells (platelets), increased pigmentation of the skin, and a yellow fatty spot on the white of the eye (a pinguecula). Severe bone involvement can lead to pain and collapse of the bone of the hips, shoulders, and spine. The GD gene is on chromosome 1. The disease is a recessive trait. Both parents carry a GD gene and transmit it for their child with the disease. The parents' risk of a child with the disease is 1 in 4 with each pregnancy. This type of Gaucher's disease (noncerebral juvenile Gaucher's disease) is most common in Ashkenazi Jews (of European origin) and is the most common genetic disease among Jews in the United States. (12 Dec 1998) |
| Recklinghausen's disease type I | type 2 neurofibromatosis |
| central Recklinghausen's disease type II | type 1 neurofibromatosis |
| disease, gaucher's type 1 | A progressive genetic disease caused by a defect in an enzyme. The enzyme, called glucocerebrosidase, is needed to break down the chemical glucocerebroside. The enzyme defect in persons with Gaucher's disease (GD) leads to the accumulation of glucocerebroside in the spleen, liver, and lymph nodes. The most common early sign is enlargement of the spleen (located in the upper left abdomen). Other signs include low red blood cell counts (anaemia), a decrease in blood clotting cells (platelets), increased pigmentation of the skin, and a yellow fatty spot on the white of the eye (a pinguecula). Severe bone involvement can lead to pain and collapse of the bone of the hips, shoulders, and spine. The GD gene is on chromosome 1. The disease is a recessive trait. Both parents carry a GD gene and transmit it for their child with the disease. The parents' risk of a child with the disease is 1 in 4 with each pregnancy. This type of Gaucher's disease (noncerebral juvenile Gaucher's disease) is most common in Ashkenazi Jews (of European origin) and is the most common genetic disease among Jews in the United States. (12 Dec 1998) |
| acrocephalosyndactyly type 1 | <paediatrics> An inherited disease (autosomal dominant) or a spontaneously occurring disease characterised by a peaked head and unusual facial appearance, due to the premature closure of the cranial sutures. A skull X-ray can confirm the diagnosis and treatment is surgical. Inheritance: autosomal dominant. (27 Sep 1997) |
| Alzheimer type I astrocyte | Enlarged frequently multinucleated astrocytes, seen in progressive multifocal leukoencephalopathy. (05 Mar 2000) |
| Alzheimer type II astrocyte | Enlarged astrocytes with vesicular nuclei and one or more small basophilic nucleoli, seen in hepatocerebral disease and Wilson's disease. (05 Mar 2000) |
| American Type Culture Collection | <cell culture> A key resource for cultured cells, located in Rockville, USA. (12 Dec 1998) |
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