¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"cell deficiency"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 2 ÆäÀÌÁö: 3
¿µ¹® renal cell carcinoma ÇÑ±Û ÄáÆÏ¼¼Æ÷¾ÏÁ¾
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  ÄáÆÏ¿¡ »ý±ä ¿ø½ÃÄáÆÏÁ¶Á÷¿¡¼­ ¹ß»ýÇÑ ¾Ï. ÁַΠ¿ø½Ã¼¼´¢°üÁ¶Á÷¿¡¼­ ¹ß»ýÇÑ´Ù. ´ëÇ¥ÀûÀΠ¼¼Æ÷Á¶Á÷ÇüÀº ¿°»ö½Ã ¼¼Æ÷ÁúÀÌ ¸¼°Ô ºñ¾îº¸À̴ ¸¼Àº¼¼Æ÷¾ÏÁ¾ÀÌ´Ù. Ä¡·á´Â ¼ö¼ú°ú Ç×¾ÏÈ­Çпä¹ýÀ̸砾ÆÁÖ µå¹°Áö¸¸ ÀúÀý·Î ³´´Â °æ¿ìµµ Àִ °ÍÀ¸·Î º¸°íµÇ¾î ÀÖ´Ù.
¿µ¹® squamous cell carcinoma ÇÑ±Û ÆíÆò¼¼Æ÷¾ÏÁ¾
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  ÆíÆò¼¼Æ÷ ±â¿øÀÇ ¾ÏÀ¸·Î¼­, ÆíÆò¼¼Æ÷°¡ Àִ ¾î¶² °÷¿¡¼­µç ¹ß»ý°¡´ÉÇÔ. µû¶ó¼­ ½Äµµ¾Ï, ÇǺξÏ, Æó¾Ï, ÀڱþϠµîÀÌ ¿©±â¿¡ ÇØ´çµÈ´Ù. Æ¯È÷ ÇǺξÏÀº ¸¹Àº Àڿܼ±Á¶»ç¿¡ ÀÇÇØ »ý±â´Â ±¤¼±°¢È­Áõ¿¡¼­ ¹ß»ý°¡´ÉÇÏ´Ù. º´¸®Á¶Á÷ÇÐÀû Æ¯¼ºÀ¸·Î¼­ °¢ÁúÀ» »ý¼ºÇÑ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • acinic cell carcinoma
    »ù²Ê¸®¼¼Æ÷¾ÏÁ¾, ¼¼¿±¼¼Æ÷¾ÏÁ¾
  • adult T-cell leukemia/lymphoma
    ¼ºÀÎT¼¼Æ÷¹éÇ÷º´/¸²ÇÁÁ¾
  • amacrine cell
    ¹«Ãà»è¼¼Æ÷
  • ameboid cell
    ¾Æ¸Þ¹Ù¸ð¾ç¼¼Æ÷
  • anaplastic large cell lymphoma
    ¿ªÇü¼ºÅ«¼¼Æ÷¸²ÇÁÁ¾
  • basal cell
    ¹Ù´Ú¼¼Æ÷, ±âÀú¼¼Æ÷
  • basal cell adenoma
    ¹Ù´Ú¼¼Æ÷»ùÁ¾, ±âÀú¼¼Æ÷¼±Á¾
  • basal cell carcinoma
    ¹Ù´Ú¼¼Æ÷¾ÏÁ¾, ±âÀú¼¼Æ÷¾ÏÁ¾
  • basal cell epithelioma
    ¹Ù´Ú¼¼Æ÷»óÇÇÁ¾, ±âÀú¼¼Æ÷»óÇÇÁ¾
  • basal cell nevus
    ¹Ù´Ú¼¼Æ÷¸ð¹Ý, ±âÀú¼¼Æ÷¸ð¹Ý
  • basal cell nevus syndrome
    ¹Ù´Ú¼¼Æ÷¸ð¹ÝÁõÈıº, ±âÀú¼¼Æ÷¸ð¹ÝÁõÈıº
  • basket cell
    ¹Ù±¸´Ï¼¼Æ÷
  • basophilic cell
    È£¿°±â¼¼Æ÷
  • basosquamous cell acanthoma
    ¹Ù´ÚÆíÆò¼¼Æ÷°¡½Ã¼¼Æ÷Á¾, ±âÀúÆíÆò±Ø¼¼Æ÷Á¾
  • basosquamous cell carcinoma
    ¹Ù´ÚÆíÆò¼¼Æ÷¾ÏÁ¾, ±âÀúÆíÆò¼¼Æ÷¾ÏÁ¾
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • plasma cell
    ÇüÁú¼¼Æ÷
  • polynucleated cell
    ¹µÇÙ¼¼Æ÷
  • prickle cell
    °¡½Ã¼¼Æ÷
  • principal cell
    ÁÖ¼¼Æ÷, À¸¶ä¼¼Æ÷
  • Purkinje cell
    1. ½ÉÀåÀüµµ±ÙÀ°¼¼Æ÷, 2. Á¶·Õ¹Ú¼¼Æ÷
  • pyramidal cell
    ÇǶó¹Ìµå¼¼Æ÷
  • red blood cell
    ÀûÇ÷±¸
  • reserve cell
    ¿¹ºñ¼¼Æ÷
  • resting cell
    ÈÞÁö±â¼¼Æ÷, Á¤Áö¼¼Æ÷
  • reticuloendothelial cell
    ±×¹°³»ÇǼ¼Æ÷, ¼¼¸Á³»ÇǼ¼Æ÷
  • rod cell
    ¸·´ë¼¼Æ÷
  • round cell
    ¿øÇü¼¼Æ÷
  • satellite cell
    À§¼º¼¼Æ÷
  • scavenger cell
    û¼Ò¼¼Æ÷
  • Schwann's cell
    ½Å°æÁý¼¼Æ÷
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • acidophilic cell
    È£»ê¼º¼¼Æ÷
  • acinar cell
    »ù²Ê¸®¼¼Æ÷
  • acinic cell carcinoma
    »ù²Ê¸®¼¼Æ÷¾ÏÁ¾, ¼¼¿±¼¼Æ÷¾ÏÁ¾
  • amacrine cell
    ¹«Ãà»è¼¼Æ÷
  • ameboid cell
    ¾Æ¸Þ¹Ù¸ð¾ç¼¼Æ÷
  • annular elastotic giant cell granuloma
    °í¸®Åº·Â¼¶À¯°Å´ë¼¼Æ÷À°¾ÆÁ¾, ȯ»óź·Â¼¶À¯°Å´ë¼¼Æ÷À°¾ÆÁ¾
  • antibody-dependent cell-mediated cytotoxicity
    Ç×üÀÇÁ¸¼¼Æ÷¸Å°³¼¼Æ÷µ¶¼º
  • antibody-producing cell
    Ç×ü»ý»ê¼¼Æ÷
  • antibody-screening cell
    Ç×ü¼±º°Ç÷±¸
  • antigen-presenting cell
    Ç׿øÀü´Þ¼¼Æ÷
  • antigen-reactive cell
    Ç׿ø¹ÝÀÀ¼¼Æ÷
  • antigen-recognizing cell
    Ç׿øÀÎÁö¼¼Æ÷
  • argentaffin cell
    ģũ·ÒÀº¼¼Æ÷
  • argyrophilic cell
    ÀºÄ£È­¼¼Æ÷
  • asexual cell
    ¹«¼º¼¼Æ÷
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • glucose 6 phosphatase deficiency hepa
    ±Û·çÄÚ¿À½º-6 -ÀÎ»ê °áÇ̰£½ÅÇü´ç
  • glucose-6-phosphate dehydrogenase deficiency
    ±Û·çÄÚ½º-6-ÀλêµðÇÏÀÌ µå·ÎÀú³×À̽º °áÇÌ(Áõ)
  • glucuronidase deficiency disease
    ±Û·çÅ¥·Î´Ïµ¥À̽º °áÇÌÁõ
  • glutathione reductase deficiency
    ±Û·çŸƼ¿Â ȯ¿øÈ¿¼Ò °áÇÌÁõ.
  • glycosidase deficiency
    ±Û¸®ÄڽôپÆÁ¦°áÇÌ(Áõ).
  • gonadal deficiency
    »ý½Ä¼±ºÎÀü(ßæãÖàÍÝÕîï).
  • gonadal deficiency
    »ý½Ä¼±ºÎÀü(ßæãÖàÍÝÕîï).
  • hepatophosphorylase deficiency
    °£Æ÷½ºÆ÷¸±¶ó¾ÆÁ¦°áÇÌÁõ.
  • hexokinase deficiency
    Çí¼ÒÄ«À̳×À̽º°áÇÌ.
  • hexokinase deficiency
    Çí¼ÒŰ³ªÁ¦°áÇÌ.
  • hexosaminidase a deficiency
    Çí¼Ò»ç¹Ì´Ïµ¥À̽º A °áÇÌ(Áõ)
  • iduronate sulfatase deficiency
    Iduronate sulfatase deficiency
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
  • immune deficiency disease
    ¸é¿ª°áÇÌÁúȯ<º´>.
  • immunoglobulin A deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ(Áõ)
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • cellular deficiency (acallosal cerebrum)
    ¼¼Æ÷°áÇÌ (³úµéº¸°á¿©³ú)
  • chromosomal deficiency
    ¿°»öü°áÇÌ
  • coagulation factor deficiency
    ÀÀ°íÀÎÀÚ°áÇÌ
  • cobalamin deficiency
    Äڹ߶ó¹Î°áÇÌ
  • color vision deficiency
    »ö°¢ÀÌ»ó
  • combined immunity deficiency syndrome
    º¹Çո鿪°áÇÌÁõÈıº.
  • combined immunity deficiency synsdrome
    º¹Çո鿪°áÇÌÁõÈıº.
  • complement deficiency
    º¸Ã¼°áÇÌ
  • congenital deficiency of glucuronyl transfe ra se
    ¼±Ãµ¼º ±Û·çÄí·Ð»ê Àü À§È¿¼Ò°áÇÌÁõ(¡­ï®êÈý£áÈÌÀù¹ñø).
  • copper deficiency
    ±¸¸®°áÇÌÁõ(¡­ÌÀù¹ñø)
  • cytochrome C oxidase deficiency
    ½ÃƮũ·Ò C ¿Á½Ã´ÙÁ¦(»êÈ­È¿¼Ò)°áÇÌ
  • cytochrome b5 reductase deficiency
    ½ÃÅäÅ©·Ò b5 ȯ¿øÈ¿¼Ò °áÇÌ
  • cytogenetic deficiency
    ¼¼Æ÷¹ß»ý°áÇÌ
  • deficiency
    °áÇÌ(Áõ)(ÌÀù¹ñø)
  • deficiency
    °áÇÌ
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • Alpha cell [Glucagon cell]
    ¾ËÆÄ¼¼Æ÷ [±Û·çÄ«°ï¼¼Æ÷]
    [¿¾ ¿ë¾î] ¾ËÆÄ¼¼Æ÷
  • Dark cell [Norepinephrine cell]
    ¾îµÎ¿î¼¼Æ÷ [³ë¸£¿¡Çdz×ÇÁ¸°¼¼Æ÷]
    [¿¾ ¿ë¾î] ¾Ï¼¼Æ÷(³ë¸£¿¡Çdz×ÇÁ¸°ºÐºñ¼¼Æ÷)
  • Chief cell [Type I glomus cell]
    °ú¸³¼¼Æ÷
    [¿¾ ¿ë¾î] ÁÖ¼¼Æ÷
  • Supporting cell [Sertoli cell]
    ¹öÆÀ¼¼Æ÷
    [¿¾ ¿ë¾î] ÁöÁÖ¼¼Æ÷
  • Supporting cell [Type II glomus cell]
    ¹öÆÀ¼¼Æ÷
    [¿¾ ¿ë¾î] ÁöÁö¼¼Æ÷
  • Supporting cell [Type II glomus cell]
    ¹öÆÀ¼¼Æ÷
    [¿¾ ¿ë¾î] ÁöÁö¼¼Æ÷(Á¦2Çü»ç±¸¼¼Æ÷)
  • Striated muscle cell
    °¡·Î¹«´Ì±ÙÀ°¼¼Æ÷
    [¿¾ ¿ë¾î] Ⱦ¹®±Ù¼¼Æ÷
  • Sensory epithelial cell
    °¨°¢»óÇǼ¼Æ÷
    [¿¾ ¿ë¾î] °¨°¢»óÇǼ¼Æ÷
  • Cortical endocrine cell
    °ÑÁú³»ºÐºñ¼¼Æ÷
    [¿¾ ¿ë¾î] ÇÇÁú³»ºÐºñ¼¼Æ÷
  • Nodal cell
    °áÀý¼¼Æ÷
    [¿¾ ¿ë¾î] °áÀý¼¼Æ÷
  • Granule cell
    °ú¸³¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³¼¼Æ÷
  • Granular lutein cell
    °ú¸³ÃþȲ(»ö)ü¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³ÃþȲü¼¼Æ÷
  • Granulosa lutein cell
    °ú¸³ÃþȲ(»ö)ü¼¼Æ÷
    [¿¾ ¿ë¾î] °ú¸³ÃþȲü¼¼Æ÷
  • Myoepithelial cell
    ±ÙÀ°»óÇǼ¼Æ÷
    [¿¾ ¿ë¾î] ±Ù»óÇǼ¼Æ÷
  • Myoid cell layer
    ±ÙÀ°¼¶À¯¸ð¼¼Æ÷Ãþ
    [¿¾ ¿ë¾î] ±Ù¼¶À¯¾Æ¼¼Æ÷Ãþ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • cell fractionation
    ¼¼Æ÷ºÐȹȭ(á¬øàÝÂüñûù)
  • cell-free amino acid incorporating system
    ¹«¼¼Æ÷(Ùíá¬øà) ¾Æ¹Ì³ë»ê ÆíÀÔ(øºìý)¾¾½ºÅÛ
  • cell-free extract
    ¹«¼¼Æ÷ÃßÃâ¹°(Ùíá¬øàõÎõóÚª)
  • cell-free protein synthesis
    ¹«¼¼Æ÷´Ü¹éÁúÇÕ¼º(Ùíá¬øàÓ±ÛÜòõùêà÷)
  • cell-free system
    ¹«¼¼Æ÷(Ùíá¬øà)½Ã½ºÅÛ
  • cell fusion
    ¼¼Æ÷À¶ÇÕ(á¬øàë×ùê)
  • cell hybridization
    ¼¼Æ÷(á¬øà) Æ¢±âÇü¼º(û¡à÷)
  • cell line
    ¼¼Æ÷ÁÖ(á¬øàñ»)
  • cell-mediated immunity
    ¼¼Æ÷¸Å°³¸é¿ª(á¬øàØÚË¿Øóæ¹)
  • cell membrane
    ¼¼Æ÷¸· (á¬øàØ¯)
  • cell strain
    ¼¼Æ÷ÁÖ(á¬øàñ»)
  • cell wall
    ¼¼Æ÷º®(á¬øàÛú)
  • centrifuge cell
    ¿ø½ÉºÐ¸®½Ç(êÀãýÝÂ×îãø)
  • competent cell
    Àû°Ý¼¼Æ÷(îêÌ«á¬øà)
  • constitutive secretory cell
    ±¸¼º¼º ºÐºñ¼¼Æ÷(ϰà÷àõÝÂÝôá¬øà)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
  • granulosa cell
    °ú¸³¸·¼¼Æ÷
  • granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç
  • islet cell
    µµ¼¼Æ÷
  • islet cell adenoma
    Ãéµµ¼¼Æ÷¼±Á¾
  • islet cell carcinoma
    µµ¼¼Æ÷¾ÏÁ¾
  • Langerhans' cell
    ¶û°Ô¸£Çѽº¼¼Æ÷
  • Langhans' glant cell
    ¶û±×Çѽº°Å¼¼Æ÷
  • large cell
    ´ë¼¼Æ÷
  • lymphoid cell
    ¸²ÇÁ¾ç¼¼Æ÷, ¸²ÇÁ°è¼¼Æ÷, ¸²ÇÁ±¸¾ç¼¼Æ÷
  • mast cell
    ºñ¸¸¼¼Æ÷
  • mastoid air cell
    À¯µ¹ºÀ¼Ò
  • mastoid cell
    À¯(¾ç)µ¹(±â)ºÀ¼Ò
  • nerve cell
    ½Å°æ¼¼Æ÷
  • neuroepithelial cell
    ½Å°æ»óÇǼ¼Æ÷
  • neuroglial cell
    ½Å°æ¾Æ±³¼¼Æ÷, ½Å°æ±³¼¼Æ÷
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
HCG, hCG Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó
  1. Placental Glycoprotein Hormone
&nbs...
Th cell helper T cell(= T4 cell)
Ts cell suppressor T cell(= T8 cell)
BC Bachelor of Surgery [Lat. Baccal-aureus Chirurgiae]; back care; bactericidal concentration; basal ce...
DRBC denaturated red blood cell; dog red blood cell; donkey red blood cell
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
GH-D GH deficiency
G6PD Glucose-6-Phosphate dehydrogenase deficiency
GKD Glycerol kinase deficiency
GHD Growth Hormone Deficiency
IgA-D IgA deficiency
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • mineral deficiency
    ¹«±âÁú °áÇÌ, ¹«±âÁú °áÇÌÁõ
  • myeloperoxidase deficiency
    ¸¶ÀÌ¿¤·Î ÆÛ·Ï½Ãµ¥À̽º °áÇÌÁõ
    »ó¿°»öü¼º ¿­¼º ÇüÁú·Î À¯ÀüµÇ¸ç, Áß¼º±¸¿Í ´Ü±¸ÀÇ È£¾ÆÁÖ¸£¼º °ú¸³ ³»¿¡ MPO°¡ ÀüÇô ¾ø´Ù.
  • myoadenylate deaminase deficiency
    ¸¶ÀÌ¿À ¾Æµ¥´Ò·¹ÀÌÆ® µð¾Æ¹Ì³×À̽º °áÇÌÁõ
  • nutrition deficiency
    ¿µ¾ç °áÇÌÁõ
  • nutritional deficiency
    ¿µ¾ç °áÇÌ
  • plasma thromboplastin antecedent deficiency
    PTA °áÇÌÁõ
  • pyridoxine deficiency
    ÇǸ®µ¶½Å °áÇÌ
  • salt deficiency
    ¿°·ù °áÇÌ
  • sulfatase deficiency
    ¼³ÆÄÅ×À̽º °áÇÌ
  • vitamin B2 deficiency
    ºñŸ¹Î B2 °áÇÌÁõ
  • vitamin D deficiency
    ºñŸ¹Î D °áÇÌ, ºñŸ¹Î D °áÇÌÁõ
    ±¸·çº´-°ñ¿¬È­ÁõÀ¸·Î µî»À³ª °¡½¿»À µûÀ§°¡ ±Á´Â º´. °ö»çº´.
  • vitamin deficiency
    ºñŸ¹Î °áÇÌ, ºñŸ¹Î °áÇÌÁõ
    ¸é Á¾·ùÀÇ ½ÄǰÀ» ¼¯Àº º¸ÅëÀÇ ½Ä»ç¸¦ º¸Åë Á¶¸®¹ý¿¡ µû¶ó ¸ÔÀ» °æ¿ì´Â ºñŸ¹Î °áÇÌÁõÀÌ ³ªÅ¸³ªÁö ¾Ê´Â´Ù. ±×·¯³ª ¿¹¿Ü·Î½á ºñŸ¹Î D°áÇÌÁõÀº ÀÚÁÖ ³ªÅ¸³­´Ù. ½Äǰ Áß¿¡´Â ¿©·¯ Á¾·ùÀÇ ºñŸ¹ÎÀÌ °øÁ¸Çϰí Àֱ⠶§¹®¿¡ 1 Á¾·ù¸¸ÀÇ ºñŸ¹Î °áÇÌÁõº¸´Ùµµ ¿©·¯ Á¾·ùÀÇ ºñŸ¹Î °áÇÌÁõÀÌ ¸¹´Ù. ¼ÒÈ­±â Áúȯ¿¡ ¼ö¹ÝµÇ´Â Èí¼öÀå¾Ö, °¢Á¾ ¾àÀçÀÇ º¹¿ë¿¡ ÀÇÇÑ Àå³» ¼¼±ÕÃþÀÇ º¯È­, ü³»¿¡¼­ÀÇ ºñŸ¹Î Ȱ¼ºÈ­ÀÇ ÀúÇØ, »ý¸®Àû º¯È­¿¡ ´ëÀÀÇÑ ¿ä±¸·®ÀÇ Áõ°¡, °¨¿° µî¿¡ ÀÇÇØ °áÇÌÁõÀÌ ¹ß°ßµÇ´Â °æ¿ì°¡ ÀÖ´Ù. ±×·¯³ª ¾àÇÑ Á¤µµÀÇ °áÇÌÀÎ °æ¿ì¿¡´Â ÀüÇüÀû Áõ»óÀº º¸ÀÌÁö ¾ÊÀ¸¸ç, ÀÌ ¶§ÀÇ Áõ»óÀ» ºñŸ¹Î °¨¼ÒÁõÀ̶ó°í ÇÑ´Ù.
  • vitamin deficiency symptom
    ºñŸ¹Î °áÇÌÁõ
    ºñŸ¹ÎÀÇ ºÎÁ·À¸·Î ÀϾ´Â »ý¸® ±â´É Àå¾Ö. ¾ß¸ÍÁõ, °¢±âº´ µûÀ§°¡ ÀÖ´Ù.
  • vitamin I deficiency
    ºñŸ¹Î °áÇÌ, ºñŸ¹Î °áÇÌÁõ
  • vitamin K deficiency
    ºñŸ¹Î K °áÇÌ, ºñŸ¹Î K °áÇÌÁõ
    Ç÷¾×ÀÇ ÀÀ°í ½Ã°£ÀÌ ±æ¾îÁø´Ù.
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
glucose-6-dehydrogenase deficiency <biochemistry> An inherited condition that results in a deficiency in glucose-6-phosphate dehydrogenase. Particular drugs (sulphonamides) can exacerbate this problem. The result is haemolytic anaemia.
(27 Sep 1997)
cellular immunity deficiency syndrome <syndrome> A syndrome marked by increased susceptibility to infection, especially to viral infection, associated with defective functioning of the mechanism responsible for acquired immunity of the cell-mediated kind.
See: immunodeficiency.
(05 Mar 2000)
glucose-6-phosphate dehydrogenase deficiency A deficiency of glucose-6-phosphate dehydrogenase, an enzyme important for maintaining cellular concentrations of reduced nucleotides.
Deficiency of this enzyme is the commonest disease-causing enzyme defect in humans affecting an estimated 400 million people.
The gene for this enzyme is on the X chromosome and there are various polymorphic forms.
Males with the enzyme deficiency develop haemolytic anaemia when red blood cells are exposed to oxidant drugs such as the antimalarial primaquine, the sulfonamide antibiotics or sulfones, naphthalene moth balls, or fava beans. It can also cause anaemia of the newborn, and chronic nonspherocytic haemolytic anaemia.
Inheritance: X-linked.
(12 Sep 2002)
vitamin k deficiency A nutritional condition produced by a deficiency of vitamin k in the diet, characterised by an increased tendency to haemorrhage (haemorrhagic diathesis). Such bleeding episodes may be particularly severe in newborn infants.
(12 Dec 1998)
glucosephosphate dehydrogenase deficiency A disease-producing enzyme deficiency subject to many variants, some of which cause a deficiency of enzyme activity in erythrocytes, leading to haemolytic anaemia.
(12 Dec 1998)
glucosephosphate isomerase deficiency <enzyme> An enzyme deficiency characterised by chronic nonspherocytic haemolytic anaemia; autosomal recessive inheritance.
Synonym: phosphohexose isomerase deficiency.
(05 Mar 2000)
glutathione synthetase deficiency An inborn error of metabolism associated with massive urinary excretion of 5-oxyproline, elevated levels of 5-oxyproline in the blood and cerebrospinal fluid, severe metabolic acidosis, tendency toward haemolysis, and defective central nervous systems function. Glutathione synthetase deficiency has been reported as a generalised condition or with a deficiency restricted to erythrocytes.
(05 Mar 2000)
mental deficiency Subnormal intellectual functioning which originates during the developmental period and is associated with impairment of one or more of the following: (1) maturation, (2) learning, (3) social adjustment.
(12 Dec 1998)
riboflavin deficiency A dietary deficiency of riboflavin causing a syndrome chiefly marked by cheilitis, angular stomatitis, glossitis associated with a purplish red or magenta-coloured tongue that may show fissures, corneal vascularization, dyssebacia, and anaemia.
(12 Dec 1998)
choline deficiency A condition produced by a deficiency of choline in animals. Choline is known as a lipotropic agent because it has been shown to promote the transport of excess fat from the liver under certain conditions in laboratory animals. Combined deficiency of choline (included in the b vitamin complex) and all other methyl group donors causes liver cirrhosis in some animals. Unlike compounds normally considered as vitamins, choline does not serve as a cofactor in enzymatic reactions.
(12 Dec 1998)
phosphohexose isomerase deficiency <enzyme> An enzyme deficiency characterised by chronic nonspherocytic haemolytic anaemia; autosomal recessive inheritance.
Synonym: phosphohexose isomerase deficiency.
(05 Mar 2000)
placental sulfatase deficiency <enzyme> An enzyme defect in the placenta which results in failure of conversion of 16a-hydroxydehydroepiandrosterone to estriol; women with this condition rarely enter into spontaneous labour.
(05 Mar 2000)
platelet storage pool deficiency A group of disorders characterised by a decrease or lack of platelet dense bodies in which the releasable pool of adenine nucleotides and 5ht are normally stored.
(12 Dec 1998)
congenital antithrombin III deficiency Antithrombin III is a protein which stimulates the removal of blood clots in the bloodstream. Small blood clots form normally within the bloodstream, but are normally dissolved via the bodys antithrombin III. The deficiency of antithrombin III will result in an increased risk for blood clot formation causing organ damage. This is an inherited as a autosomal dominant trait.
Inheritance: autosomal dominant.
(27 Sep 1997)
multiple carboxylase deficiency Abnormalities in carbohydrate and branched-chain amino acid catabolism that are responsive to biotin therapy. It may be due to deficiency of propionyl-CoA carboxylase, methylcrotonyl-CoA carboxylase, biotinidase, or propionyl-CoA carboxylase, methylcrotonyl-CoA carboxylase, and pyruvate carboxylase.
(12 Dec 1998)
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