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  • ¿µ¹®
    ÇѱÛ
  • myocardial
    ½ÉÀå±ÙÀ°-, ½É±Ù-
  • myocardial anoxia
    ½É±Ù¹«»ê¼ÒÁõ, ½ÉÀå±ÙÀ°¹«»ê¼ÒÁõ
  • myocardial bridging
    ½É±Ù´Ù¸®
  • myocardial contractility
    ½É(Àå)±Ù(À°)¼öÃà·Â, ½É(Àå)±Ù(À°)¼öÃ༺
  • myocardial contraction
    ½É(Àå)±Ù(À°)¼öÃà
  • myocardial depressant factor
    ½É(Àå)±Ù(À°)¾ïÁ¦ÀÎÀÚ
  • myocardial failure
    ½É±Ù±â´É»ó½Ç, ½É±ÙºÎÀü
  • myocardial irritability
    ½É±Ù°ú¹Î¼º, ½É±ÙÀÚ±ØÈïºÐ¼º
  • myocardial ischemia
    ½É±ÙÇãÇ÷, ½ÉÀå±ÙÀ°ÇãÇ÷
  • myocardial lead
    ½É±ÙÀ¯µµ, ½ÉÀå±ÙÀ°À¯µµ
  • myocardial perfusion scintigraphy
    ½É±Ù°ü·ù¼¶±¤Á¶¿µ(¼ú)
  • myocardial scan
    ½É±Ù½ºÄµ
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  • ¿µ¹®
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  • privileged site
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  • receptor site
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  • site
    ºÎÀ§
  • telomeric site
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  • web site
    À¥½ÎÀÌÆ®
  • myocardial anoxia
    ½ÉÀå±ÙÀ°¹«»ê¼ÒÁõ
  • myocardial bridging
    ½É±Ù´Ù¸®
  • myocardial contractility
    ½ÉÀå±ÙÀ°¼öÃà·Â, ½ÉÀå±ÙÀ°¼öÃ༺
  • myocardial contraction
    ½ÉÀå±ÙÀ°¼öÃà
  • myocardial failure
    ½É±Ù±â´É»ó½Ç, ½É±ÙºÎÀü
  • myocardial depressant factor
    ½ÉÀå±Ù¾ïÁ¦ÀÎÀÚ
  • idiopathic myocardial hypertrophy
    Ư¹ß½É±Ùºñ´ë, ¿øÀκҸí½É±Ùºñ´ë
  • myocardial irritability
    ½É±Ù°ú¹Î¼º, ½É±ÙÀÚ±ØÈïºÐ¼º
  • myocardial ischemia
    ½É±ÙÇãÇ÷
  • myocardial lead
    ½É±ÙÀ¯µµ
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  • postmyocardial infarction syndrome
    ½É±Ù°æ»öÈÄÁõÈıº(ãýÐÉÌÛßáý­ñøý¦ÏØ)
  • pulmonary infarction
    Æó°æ»ö
  • pulmonary infarction
    Æó°æ»öÁõ(øËÌÛßáñø)
  • renal infarction
    ½Å°æ»öÁõ(ãìÌÛßáñø).
  • renal infarction
    ½Å°æ»öÁõ(ãìÌÛßáñø)
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  • antigen recognition site
    Ç׿ø½Äº°ºÎ.
  • binding site
    °áÇÕºÎÀ§.
  • binding site
    °á ÇÕºÎÀ§.
  • combining site
    °áÇÕºÎ(Ì¿ùêÝ»).
  • combining site
    °áÇÕºÎÀ§
  • combining site of antibody
    Ç×üÀÇ °áÇÕºÎ.
  • corporal site
    ÀڱøöÅëÀÓ½Å
  • definitive site
    Âø»óÀÚ¸®
  • donor site
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  • fundic site
    ÀڱùٴÚÀÓ½Å
  • internal ribosomal entry site (IRES)
    ³»ºÎ ¸®º¸¼Ø °áÇÕºÎÀ§
  • mutational site
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  • packaging site
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  • peptidyl site
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  • peptidyl-tRNA site
    ÆéŸÀ̵å-tRNA ÀÚ¸®
  • polycloning site
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  • prophage attachment site
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  • recognition site
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  • regulatory site
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  • replacement site
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  • restriction site
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  • ribosome binding site
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  • R site
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  • second-site mutation
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STANDOUT soft thresholding and depth cueing of unspecified techniques
URD unspecified respiratory disease; upper respiratory disease
PI first meiotic prophase; isoelectric point; pacing impulse; package insert; pancreatic insufficiency;...
AMI Acute Myocardial Infarction
  - Complications(Cx)
    1. Early ...
MI   1) Mitral Insufficient
    = MR
  2) Myocardial Infa...
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Site 1 site
BIP Bezafibrate Infarction Prevention
CI Cerebral infarction
LI Lacunar infarction
MI Myocardia infarction
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  • combining site
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  • electrophilic site
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  • molecular site
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  • receptor site
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  • site
    À§Ä¡, »çÀÌÆ®
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  • specific opiate receptor site
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    ƯÁ¤ ¾ÐÅëÁ¡
  • surgical site
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myocardial contusion A bruise to the heart muscle, usually caused by a blunt force applied to the anterior thorax (motor vehicle accident). Commonly seen in association with a rib or sternum fracture. Complications include cardiac arrhythmias and death.
(27 Sep 1997)
myocardial depressant factor A low molecular weight peptide of about 800-1000 having a negative inotropic effect. It is released into the circulation during experimental haemorrhagic pancreatitis, severe ischemia, and postoligaemic shock.
(12 Dec 1998)
myocardial diseases Diseases of the myocardium.
(12 Dec 1998)
myocardial infarct imaging <radiology> Tc-99m pyrophosphate (PYP) 20 mCi, peak abnormality 2-3 days, often falsely negative before 2 days, abnormal for 7-10 days, mechanism: calcium influx into ischemic cells, PYP incorporated into crystalline structure, analogous to hydroxyapatite see: nuclear cardiology
(12 Dec 1998)
myocardial insufficiency A condition where there is ineffective pumping of the heart leading to an accumulation of fluid in the lungs. Typical symptoms include shortness of breath with exertion, difficulty breathing when lying flat and leg or ankle swelling. Causes include chronic hypertension, cardiomyopathy and myocardial infarction.
(27 Sep 1997)
myocardial ischemia A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (coronary arteriosclerosis), to obstruction by a thrombus (coronary thrombosis), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (myocardial infarction).
(12 Dec 1998)
myocardial necrosis Irreversible destruction of myocardial (heart muscle) cells.
(27 Sep 1997)
myocardial perfusion imaging <radiology> (thallium scanning) thallium (Tl) 201, acts as potassium analog, dose 2.0 - 3.0 mCi at peak exercise, 4% of injected dose reaches myocardium, imaging: exercise (1-5 min), redistribution (3-4 hrs), views: anterior, LAO 45', left lateral, interpretation: normal, reversible abnormalitymost likely to be exercise-induced ischemia, nonreversible abnormalitymost likely to be prior myocardial infarction, reverse redistribution most likely to be normal areas wash out faster, lung activity most likely to be LV failure during exercise see also: dipyridamole test, nuclear cardiology
(12 Dec 1998)
myocardial reperfusion Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing myocardial reperfusion injury.
(12 Dec 1998)
myocardial reperfusion injury Functional, metabolic, or structural changes in ischemic heart muscle thought to result from reperfusion to the ischemic areas. Changes can be fatal to muscle cells and may include oedema with explosive cell swelling and disintegration, sarcolemma disruption, fragmentation of mitochondria, contraction band necrosis, enzyme washout, and calcium overload. Other damage may include haemorrhage and ventricular arrhythmias. One possible mechanism of damage is thought to be oxygen free radicals. Treatment currently includes the introduction of scavengers of oxygen free radicals, and injury is thought to be prevented by warm blood cardioplegic infusion prior to reperfusion.
(12 Dec 1998)
myocardial revascularization The restoration of blood supply to the myocardium.
(12 Dec 1998)
myocardial rigor mortis Irreversible contraction of the left ventricle of the heart as a complication seen in the early period of cardiopulmonary bypass and now avoided by appropriate cardioplegic solutions.
Synonym: myocardial rigor mortis, stone heart.
(05 Mar 2000)
myocardial stunning Prolonged dysfunction of the myocardium after a brief episode of severe ischemia, with gradual return of contractile activity. It occurs frequently, both in the experimental laboratory and in clinical medicine. Since stunned myocardium occurs adjacent to necrotic tissue after prolonged coronary occlusion, many myocardial infarcts may be a mixture of necrotic and stunned tissue.
(12 Dec 1998)
ischemic preconditioning, myocardial Exposure of myocardial tissue to brief, repeated periods of vascular occlusion in order to render the myocardium resistant to the deleterious effects of ischemia or reperfusion. The period of pre-exposure and the number of times the tissue is exposed to ischemia and reperfusion vary, the average being 3 to 5 minutes.
(12 Dec 1998)
acceptor site The ribosomal binding site for the aminoacyl-tRNA during protein synthesis.
(05 Mar 2000)
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