| IPOF | immediate postoperative fitting |
|---|---|
| IPOP | immediate postoperative prosthesis |
| PCP | parachlorophenate; patient care plan; pentachlorophenol; 1-(1-phenylcyclohexyl)piperidine; periphera... |
| PLCO | postoperative low cardiac output |
| PO | by mouth, orally [Lat. per os]; parieto-occipital; parietal operculum; period of onset; perioperativ... |
ocular
| necrotic inflammation | Usually an acute inflammatory reaction in which the predominant histologic change is fairly rapid necrosis that occurs diffusely or extensively in relatively large foci throughout the affected tissue, frequently with only little or no evidence of cells in the exudate. (05 Mar 2000) |
|---|---|
| subacute inflammation | An inflammation that is intermediate in duration between that of an acute inflammation and that of a chronic inflammation, usually persisting longer than 3 or 4 weeks. (05 Mar 2000) |
| suppurative inflammation | An acute exudative inflammation in which the accumulation of polymorphonuclear leukocytes is sufficiently great that their enzymes cause liquefaction of the affected tissues, focally or diffusely; the purulent exudate is frequently termed pus, and consists of plasma and its constituents, end products of the enzymatic digestion of tissue, degenerated and necrotic cells and their debris, polymorphonuclear leukocytes and other white blood cells, the causal agent of the inflammation, etc. Synonym: suppurative inflammation. (05 Mar 2000) |
| degenerative inflammation | A local reaction to injury, occasionally observed in the walls of blood vessels and in parenchymal cells of various organs in reacting to certain chemicals, viruses, and other intracellular agents; the response is characterised by degenerative changes in the cytoplasm and nucleus, frequently resulting in necrosis, but exudation (if any) is ordinarily observed only in the wall of the affected vessel, or in the interstices immediately adjacent to the affected vessel or parenchymal cells. Synonym: degenerative inflammation. (05 Mar 2000) |
| immune inflammation | See: allergic reaction. (05 Mar 2000) |
| inflammation | <pathology> A localised protective response elicited by injury or destruction of tissues, which serves to destroy, dilute or wall off (sequester) both the injurious agent and the injured tissue. It is characterised in the acute form by the classical signs of pain (dolor), heat (calor), redness (rubor), swelling (tumour) and loss of function (functio laesa). Histologically, it involves a complex series of events, including dilatation of arterioles, capillaries and venules, with increased permeability and blood flow, exudation of fluids, including plasma proteins and leucocytic migration into the inflammatory focus. Origin: L. Inflammatio, inflammare = to set on fire (11 Jun 1998) |
| inflammation: gallium imaging | <radiology> Pathophysiology: leakage of protein-bound Ga-67 into extracellular space secondary to increased capillary permeability, Ga-67 is preferentially bound to nonviable PMNs and macrophages, leukocyte incorporation (rich in lactoferrin), bacterial uptake (siderophores), inflammtory tissue stimulates lactoferrin production for chronic abdominal inflammation: 67% sensitivity; 64% specificity; 13% false negatives; 5% false positive, dose: 5 mCi; imaging: 24, 48, 72 hours, diffuse uptake in peritonitis, localised uptake in acute pyogenic abscess, phlegmon, acute cholecystitis, acute pancreatitis, acute gastritis, diverticulitis, inflammatory bowel disease, surgical wound, pyelonephritis, perinephric abscess see: gallium indications, gallium vs. Indium (12 Dec 1998) |
| inflammation mediators | The endogenous compounds that mediate inflammation (autacoids) and related exogenous compounds including the synthetic prostaglandins (prostaglandins, synthetic). (12 Dec 1998) |
| interstitial inflammation | Inflammation in which the inflammatory reaction occurs chiefly in the supportive fibrous connective tissue or stroma of an organ. (05 Mar 2000) |
| exudative inflammation | Inflammation in which the conspicuous or distinguishing feature is an exudate, which may be chiefly serous, serofibrinous, fibrinous, or mucous (e.g., relatively few cells are present), or may be characterised by relatively large numbers of neutrophils, eosinophils, lymphocytes, monocytes, or plasma cells, frequently with one or two types being predominant; it occurs not only as a separate and distinct pathologic process, but also frequently as a part of certain granulomatous inflammation's. (05 Mar 2000) |
| fibrinopurulent inflammation | A purulent inflammation in which the exudate contains an unusually large amount of fibrin; also, a fibrinous or serofibrinous inflammation in which the accumulation of large numbers of polymorphonuclear leukocytes results in liquefactive necrosis of tissue and the formation of pus with a relatively large quantity of fibrin. (05 Mar 2000) |
| fibrinous inflammation | An exudative inflammation in which there is a disproportionately large amount of fibrin. (05 Mar 2000) |
| fibroid inflammation | A form of chronic inflammation or repeated episodes of acute inflammation in which the continued or recurrent proliferation of fibroblasts results in the formation of fibrous tissue that eventually contracts and leads to compression and atrophy of parenchymal tissue. Synonym: fibroid inflammation. (05 Mar 2000) |
| accommodation, ocular | The dioptric adjustment of the eye (to attain maximal sharpness of retinal imagery for an object of regard) referring to the ability, to the mechanism, or to the process. It is the effecting of refractive changes by changes in the shape of the crystalline lens. Loosely, it refers to ocular adjustments for vision at various distances. (12 Dec 1998) |
| adaptation, ocular | The adjustment of the eye to variations in the intensity of light. Light adaptation is the adjustment of the eye when the light threshold is increased; dark adaptation when the light is greatly reduced. (12 Dec 1998) |