| ¿µ¹® | nerve | ÇÑ±Û | ½Å°æ |
|---|---|---|---|
| ¼³¸í | ¿©·¯ ±â°üµéÀÇ ¼·Î°£ »óÈ£¿¬°áü°è°¡ ¹Ù·Î ½Å°æ°èÀÌ´Ù. ÀÌ·¯ÇÑ ½Å°æ°è´Â °¢ ½Å°æ¼¼Æ÷µé¿¡ ÀÇÇØ ÀÌ·ç¾îÁö°í ÀÖÀ¸¸ç, ¿©±â¿¡´Â ÁßÃ߽Űæ°è(central nerve system: CNS)¿Í ¸»ÃʽŰæ°è(peripheral nerve system: PNS)°¡ ÀÖ´Ù. ÁßÃ߽Űæ°è¶õ ³ú¿Í ô¼ö¸¦ ¸»Çϸç, ¸»ÃʽŰæ°è´Â 12½ÖÀÇ ³ú½Å°æ(cranial nerve: ³ú¿¡¼ ±â½ÃÇÏ¿© ÁÖ·Î ¾ó±¼ºÎÀ§¿Í ¸ñ ºÎÀ§¿¡ ºÐÆ÷ÇÑ´Ù)°ú 31½ÖÀÇ Ã´¼ö½Å°æ(spinal nerve: spinal cord¿¡¼ °¢±â ¾çÂÊÀ¸·Î ½ÖÀ» ÀÌ·ç¾î ³ª¿À´Âµ¥ ÁÖ·Î ¸ñÀÌÇϺÎÀ§ÀÇ ½Åü °¢ ºÎºÐÀ¸·Î ºÐÆ÷ÇÏ°Ô µÈ´Ù)À¸·Î ±¸¼ºµÇ¾î ÀÖ´Ù. ¶ÇÇÑ ¸»ÃʽŰæ°è¿¡´Â ¾Õ¿¡¼ ¸»ÇÑ ³ú½Å°æ°ú ô¼ö½Å°æ¿Ü¿¡ ÀÚÀ²½Å°æ°è°¡ Á¸ÀçÇÑ´Ù. ÀÚÀ²½Å°æ°è´Â ´Ù½Ã ±³°¨½Å°æ°ú ºÎ±³°¨½Å°æÀ¸·Î ³ª´µ¾îÁ® ¼·Î°£ÀÇ ¿Ã¹Ù¸¥ »óÈ£ÀÛ¿ëÀ¸·Î ¸ö¼Ó¿¡¼ ¿©·¯ °¡Áö ÀÛ¿ëÀ» ¼öÇàÇÑ´Ù. |
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| ¿µ¹® | nerve cell | ÇÑ±Û | ½Å°æ¼¼Æ÷ |
|---|---|---|---|
| ¼³¸í | ½Å°æ¼¼Æ÷´Â ¿Ã¹Ù¸¥ ½Å°æÀü´ÞÀ» À§ÇÑ °¢ ºÎºÐº°·Î ³ª´µ¾îÁ® ÀÖ´Ù. ½Å°æ¼¼Æ÷¿¡¼´Â ÀüÇØÁ®¿À´Â ÀÚ±ØÀ» Àü±âÀûÀÎ ½ÅÈ£·Î ¹Ù²î¾î º¸³»°Å³ª ¹Þ°Ô µÈ´Ù. ÀÌ·± Àü±âÀûÀÎ Çö»óÀº °¢ ½Å°æ¼¼Æ÷³»¿¡ Á¸ÀçÇÏ´Â °¢ ÀÌ¿Âä³Î(ion channel: ionÀ̶õ ³ªÆ®·ý, Ä®·ý µîÀ» ÁöĪÇÏ´Â ¸»µé·Î½á, À̵éÀÌ ¼¼Æ÷¸·¿¡ ÀÇÇØ ³ª´µ¾îÁú ¶§ »ý±â´Â Àü¾ÐÂ÷°¡ Àü±âÀû ÀÚ±ØÀ» ÀÏÀ¸Å°°í À¯ÁöÇϴµ¥ °áÁ¤ÀûÀÎ ¿ªÇÒÀ» ÇÑ´Ù)µéÀÇ ÀÛ¿ë¿¡ ÀÇÇØ ÀÌ·ç¾îÁö°Ô µÈ´Ù. |
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| ¿µ¹® | facial nerve | ÇÑ±Û | ¾ó±¼½Å°æ |
|---|---|---|---|
| ¼³¸í | Ç¥Á¤±ÙÀ» Áö¹èÇÏ´Â Å« ¿îµ¿½Å°æ°ú ÀÛÀº Áß°£½Å°æÀ¸·Î ±¸¼ºµÈ´Ù. Á¼Àº ÀǹÌÀÇ ¾ó±¼½Å°æ°ú Áß°£½Å°æÀ¸·Î ÀÌ·ç¾îÁö´Â È¥ÇսŰæÀÌ´Ù. ¼Ó±Í½Å°æ°ú ÇÔ²² ¼Ó±Ó±æ·Î µé¾î°¡°í ±× ¹Ù´Ú¿¡¼ ¼Ó±Í½Å°æ°ú °¥¶óÁ® ¾ó±¼½Å°æ°üÀ¸·Î µé¾î°¡, °ÅÀÇ Á÷°¢À¸·Î ±¸ºÎ·¯Áö´Â ºÎºÐÀ» ¾ó±¼½Å°æ¹«¸À̶ó Çϸç, ¿©±â¿¡ ¹«¸½Å°æ¸¶µð°¡ ÀÖ´Ù. |
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| ¿µ¹® | sural nerve | ÇÑ±Û | ÀåµýÁö½Å°æ |
|---|---|---|---|
| ¼³¸í | ¾ÈÂÊÀåµýÁöÇÇºÎ½Å°æ ¹× ¿ÂÀåµýÁö½Å°æÀÇ ±³ÅëÁö¿¡¼ ½ÃÀÛÇÏ¿© °¡ÂʹèÂÊÇÇºÎ½Å°æ ¹× °¡Âʹ߲ÞÄ¡°¡ÁöÀÇ °¡Áö¸¦ ³½´Ù. ³Ò´Ù¸® µîÂÊÀÇ ÇǺÎ, ¹ß²ÞÄ¡¿Í ¹ßÀÇ °¡ÂÊÀÇ ÇǺΠ¹× °üÀý¿¡ ºÐÆ÷Çϸç, ÀÏ¹Ý °¨°¢¼º ±â´ÉÀ» ÇÑ´Ù. |
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| ¿µ¹® | spinal nerve | ÇÑ±Û | ô¼ö½Å°æ |
|---|---|---|---|
| ¼³¸í | ô¼öÀÇ ¾Õ»Ô¿¡¼ Ãâ¹ßÇÏ´Â ¿îµ¿½Å°æ°ú µÞ»ÔÀ¸·Î µé¾î¿À´Â °¨°¢½Å°æÀÌ ÇÕÃļ Çü¼ºµÇ´Â ½Å°æÀ¸·Î¼ ÃÑ 31½ÖÀÌ Á¸ÀçÇÔ. ¸ñ»À½Å°æÀÌ 8½Ö, ÀÚµî»À½Å°æÀÌ 12½Ö, Ç㸮»ÀÀÇ ½Å°æÀÌ 5½Ö, ¾ûÄ¡»ÀÀÇ 6½ÖÀ» ÀÌ·ë. |
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| STANDOUT | soft thresholding and depth cueing of unspecified techniques |
|---|---|
| URD | unspecified respiratory disease; upper respiratory disease |
| NBI | neutrophil bactericidal index; no bone injury; non-battle injury |
| PI | first meiotic prophase; isoelectric point; pacing impulse; package insert; pancreatic insufficiency;... |
| PRICES | protection, rest, ice, compression, elevation, support [primary treatment of tendinitis and overuse ... |
| trophic level | <biology> Stage in a food chain or web leading from primary producers (lowest trophic level) through herbivores to primary and secondary carnivores (consumers- highest level). (09 Oct 1997) |
|---|---|
| lactate level | A test that measures the amount of lactic acid in the blood. Lactic acid is an intermediate product of carbohydrate metabolism and is derived mainly from muscle cells and red blood cells. Exercise will normally raise lactic acid levels. Conditions of oxygen deprivation (for example shock, heart failure, lung disease) will trigger anaerobic metabolism within muscle tissue resulting in lactic acid build up in the tissues. Normal lactic acid levels are 4.5 to 19.8 mg/dl. (27 Sep 1997) |
| lactic acid level | <investigation> A test that measures the amount of lactic acid in the blood. (25 Jun 1999) |
| free calcium level | <biochemistry> The ionised calcium represents the calcium (Ca++) that is the metabolically active calcium. Normal values for ionised calcium in the bloodstream should be 4.4 to 5.3 mg/dl for adults and 4.4 to 6.0 mg/dl for children. Elevations may be seen in hyperparathyroidism, metastatic bone tumour, milk-alkali syndrome, multiple myeloma, Paget's disease, sarcoidosis, PTH-secreting tumours (paraneoplastic syndrome) and vitamin D intoxication. Lower than normal values may be seen in hypoparathyroidism, malabsorption, osteomalacia, pancreatitis, renal failure, rickets and vitamin D deficiency. (27 Sep 1997) |
| level of aspiration | <psychology> The degree or quality of performance (exhibited in a testing situation) which an individual desires to attain or feels he can achieve. (05 Mar 2000) |
| abbreviated injury scale | Classification system for assessing impact injury severity developed and published by the american association for automotive medicine. It is the system of choice for coding single injuries and is the foundation for methods assessing multiple injuries or for assessing cumulative effects of more than one injury. These include maximum ais (mais), injury severity score (iss), and probability of death score (pods). (12 Dec 1998) |
| blast injury | Tearing of lung tissue or rupture of abdominal viscera without external injury, as by the force of an explosion. (05 Mar 2000) |
| brain injury | Acute injuries to the brain, general or unspecified. (12 Dec 1998) |
| reperfusion injury | Functional, metabolic, or structural changes, including necrosis, in ischemic tissues thought to result from reperfusion to ischemic areas of the tissue. The most common instance is myocardial reperfusion injury. (12 Dec 1998) |
| closed head injury | A head injury in which continuity of the scalp and mucous membranes is maintained. (05 Mar 2000) |
| cold injury | Cold injuries include chilblains, trench foot, and frostbite. Cold injuries occur with and without freezing of body tissues. The young and the elderly are especially prone to cold injury. Alcohol increases the risk of cold injury which can lead to loss of body parts and even to death. It is important not to thaw an extremity if there is a risk of it re-freezing. (12 Dec 1998) |
| whiplash injury | Popular term for hyperextension-hyperflexion injury. (05 Mar 2000) |
| pneumatic tire injury | Separation of the skin and subcutaneous tissue from the underlying fascia, classically occurring when an extremity is crushed and rolled over by the tire of a vehicle but may be incurred through other mechanisms that produce shear forces; may occur particularly in cases of obesity. (05 Mar 2000) |
| contrecoup injury of brain | An injury occurring beneath the skull opposite to the area of impact. (05 Mar 2000) |
| myocardial reperfusion injury | Functional, metabolic, or structural changes in ischemic heart muscle thought to result from reperfusion to the ischemic areas. Changes can be fatal to muscle cells and may include oedema with explosive cell swelling and disintegration, sarcolemma disruption, fragmentation of mitochondria, contraction band necrosis, enzyme washout, and calcium overload. Other damage may include haemorrhage and ventricular arrhythmias. One possible mechanism of damage is thought to be oxygen free radicals. Treatment currently includes the introduction of scavengers of oxygen free radicals, and injury is thought to be prevented by warm blood cardioplegic infusion prior to reperfusion. (12 Dec 1998) |
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