| a.t. | acquisition time; 영상회복시간 = TR x N x Nex TR; Time to Repeat &... |
|---|---|
| VDRE | Vitamin D Responsive Element |
| ACSE | association control service element |
| BEP | brain evoked potential; basic element of performance |
| CE | California encephalitis; cardiac enlargement; cardioesophageal; carotid endarterectomy; catamenial e... |
| dyad symmetry element | Dyad symmetry element bound by serum response factor to control the expression of c fos. (18 Nov 1997) |
|---|---|
| Is element | Mobile nucleotide sequences that occur naturally in the genomes of bacterial populations. When inserted into bacterial DNA, they inactivate the gene concerned, when they are removed the gene regains its activity. Closely related to transposons and range in size from a few hundred to a few thousand bases, but are usually less than 1500 bases. (18 Nov 1997) |
| electronegative element | An element whose atoms have a tendency to accept electrons and form negative ions (e.g., oxygen, sulfur, chlorine, etc.). (05 Mar 2000) |
| electropositive element | <chemistry> An element whose atoms have a tendency to lose electrons and form positive ions (e.g., sodium). (05 Mar 2000) |
| element | <chemistry> One of the 103 known chemical substances that cannot be divided into simpler substances by chemical means. A substance whose atoms all have the same atomic number. Examples: hydrogen, lead, uranium.(See atom, matter, nuclide.) (16 Dec 1997) |
| trace element | Any chemical element that an organism needs very small quantities of tosurvive. (09 Oct 1997) |
| transitional element | <cell biology> Region at the boundary of the rough endoplasmic reticulum and the Golgi. Transport vesicles are responsible for the transfer of secretory proteins from this part of the rough endoplasmic reticulum to the Golgi system. (18 Nov 1997) |
| enhancer element | <molecular biology> A DNA sequence, present in the genomes of higher eukaryotes and of various animal viruses, which can increase the transcription of genes into messenger RNA. These control element frequently found 5' to the start site of a gene, when bound by a specific transcription factor, enhance the levels of expression of the gene, but are not sufficient alone to cause expression. Distinguished from a promoter, that is alone sufficient to cause expression of the gene when bound, in practice, the two terms merge. Enhancers usually can function in either orientation and at various distances from a promoter. Compare: promoter. (03 Jul 1999) |
| transposable element | <molecular biology> Small, mobile DNA sequences that can replicate and insert copies at random sites within chromosomes. They have nearly identical sequences at each end, oppositely oriented (inverted) repeats and code for the enzyme, transposase, that catalyses their insertion. Bacteria have two types of transposon, simple transposons that have only the genes needed for insertion and complex transposons that contain genes in addition to those needed for insertion. Eukaryotes contain two classes of mobile genetic elements, the first are like bacterial transposons in that DNA sequences move directly. The second class (retrotransposons) move by producing RNA that is transcribed, by reverse transcriptase, into DNA which is then inserted at a new site. (13 Nov 1997) |
| extrachromosomal element | Any heritable element not associated with the chromosome. It is usually a plasmid or the DNA of organelles such as mitochondria and chloroplasts. (18 Nov 1997) |
| abdominal muscle deficiency syndrome | <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear. (05 Mar 2000) |
| adult lactase deficiency | Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults. (05 Mar 2000) |
| alpha-1 antitrypsin deficiency | <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues. The lack of this protein leads to damage of various organs, but mainly to the lung and liver. symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant (12 Dec 1998) |
| alpha-1-proteinase deficiency | Absence of a serum proteinase inhibitor that may cause nodular non-suppurative panniculitis. (05 Mar 2000) |
| alpha-antitrypsin deficiency | <enzyme> A specific enzyme (alpha 1 antitrypsinase) that when absent genetically can result in panacinar emphysema (lung disease) and liver disease. There is no specific treatment for this condition other than supportive care for the liver and lung complications. Medications such as alpha-1proteinase inhibitor is given regularly to these patients. Incidence: approximately 1 in 10,000. (02 Jan 1998) |