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¿µ¹® immune system ÇÑ±Û ¸é¿ªÃ¼°è
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¿µ¹® urinary system ÇÑ±Û ºñ´¢±â°èÅë
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  ºñ´¢±â°èÅëÀ̶óÇϸé ÄáÆÏÀ¸·ÎºÎÅÍ ½ÃÀÛÇØ¼­ ¿ä°ü, ¹æ±¤, ¿äµµ¿¡ À̸£´Â ÀÏ·ÃÀÇ ¿ÀÁÜ»ý¼º ¹× ÀúÀå, ¹è¼³±â°üÀ» ÀÏÄ´´Ù. ÄáÆÏÀº ±æÀÌ ¾à 2.5cm, Æø ¾à 5.1cm, µÎ²² ¾à 2.5cm, ¹«°Ô ¾à 120~160gmÀ¸·Î¼­, ³»Ãø¿¡ ÄáÆÏ¹®ÀÌ ÀÖ¾î Ç÷°ü, ½Å°æ, ¿ä°üÀÌ ÃâÀÔÇϰí ÀÖ´Ù. ÄáÆÏÀº ¼ÓÁú°ú °ÑÁú·Î ÀÌ·ç¾îÁ® ÀÖÀ¸¸ç ¼öÁúÀº 10~15°³ÀÇ Ãßü(¿ÀÁÜÀ» ¸ðÀ¸´Â ¿ªÇÒ)¸¦ Çü¼ºÇ졒ʡÑÁúÀº ¾à 100¸¸°³ÀÇ ÄáÆÏ´ÜÀ§À¸·Î ±¸¼ºµÇ¾î ÀÖ´Ù. ¿ä¼¼°üÀº Å丮ÂÊ´¢¼¼°ü, Çî·¹°í¸®, ¸ÕÂÊ´¢¼¼°ü, ÁýÇÕ°üÀ¸·Î Çü¼ºµÇ¾î ÀÖÀ¸¸ç, Ãßü¿Í ¼úÀÜ, ±ò¶§±â¸¦ °ÅÃÄ ¿ä°üÀ¸·Î ¿¬°áµÈ´Ù.
  
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¿µ¹® reproductive system ÇÑ±Û »ý½Ä±â°èÅë
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  1.³²¼º»ý½Ä°èÅë: ³²¼º»ý½Ä±â´Â Á¤ÀÚ(sperm)¸¦ »ý¼ºÇϴ °íȯ°ú Á¤ÀÚÀÇ ¼º¼÷, ¿î¹Ý, ±×¸®°í »çÁ¤¿¡ °ü¿©Çϴ ºÎ°íȯ, Á¤°ü, À½°æ(penis) µîÀ¸·Î ÀÌ·ç¾îÁ® ÀÖÀ¸¸ç, ºÎ¼Ó±â°üÀ¸·Î ¿ÜºÐºñ»ùÀΠÁ¤³¶(seminal vesicle), Àü¸³»ù(prostate), ¿äµµ¸Á¹°»ù(bulbourethral gland, Cowper¡¯s gland) µîÀ» °®Ãß°í ÀÖ´Ù. °íȯÀº Á¤ÀÚ¸¦ »ý»êÇϴ »ý½Ä»ùÀΠµ¿½Ã¿¡ ³²¼ºÈ£¸£¸ó(testosterone)À» ºÐºñÇϴ ³»ºÐºñ»ùÀÌ´Ù. °íȯ¿¡¼­ ºÐºñµÇ´Â ³²¼ºÈ£¸£¸óÀº Á¤ÀÚ»ý¼º°ú »ý½Ä±âÀÇ ¹ß´Þ ¹× À¯Áö¿¡ ÇʼöÀûÀΠ¿ªÇÒÀ» ÇϹǷΠ³²¼º»ý½Ä±â´ÉÀÇ ¿øÃµÀº °íȯ¿¡ ÀÖ´Ù°í º¼ ¼ö ÀÖ´Ù.
  
  2.¿©¼º»ý½Ä°èÅë: ¿©¼º»ý½Ä±â´Â ³­ÀÚ¸¦ »ý¼ºÇϴ ³­¼Ò¿Í ³­ÀÚ¸¦ ÀÚ±ÃÀ¸·Î ¿î¹ÝÇϴ ³­°ü, ±×¸®°í Àڱðú Áú·Î ÀÌ·ç¾îÁ® ÀÖÀ¸¸ç ¿ÜºÐºñ¼±ÀΠ¹Ù¸£Å縰»ù¸¦ °®Ãß°í ÀÖ´Ù. ³­¼Ò´Â ³­ÀÚ¸¦ »ý¼ºÇϴ »ý½Ä»ùÀΠµ¿½Ã¿¡ ¿©¼ºÈ£¸£¸óÀ» ºÐºñÄÉÇϴ ³»ºÐºñ»ùÀÌ´Ù. ¿ù°æÁÖ±â Àü¹ÝºÎ¿¡ ³­ÀÚ¸¦ »ý¼º½Ã۱âÀ§ÇØ ¼º¼÷µÇ°í Àִ ³­Æ÷¿¡¼­ ºÐºñµÇ´Â ¿¡½ºÆ®·Î°ÕÀº ¿©¼º 2Â÷ ¼ºÂ¡ÀÇ ¹ß´ÞÀ» °üÀåÇÒ »Ó ¾Æ´Ï¶ó Àڱ󻸷À» ÀåÂ÷ ¼öÁ¤µÉ ¼öÁ¤¶õÀÌ Âø»óÇϱ⿡ ¾Ë¸ÂÀº »óÅ·Π¸¸µé¾îÁØ´Ù. ³­ÀÚ°¡ ºÐºñµÇ°í ³²Àº È²Ã¼¿¡¼­ ºÐºñµÇ´Â Çª·Î°Ô½ºÅ×·ÐÀº Àڱ󻸷À» º×µµ·Ï Çϸ頺кñ¾×À» Áõ°¡½Ã۸ç ÀڱñÙÀÇ ¼öÃàÀ» ¹æÇØÇÏ¿© ÀӽŽàÀÓ½ÅÀ» Áö¼Ó½Ã۴ ¿ªÇÒÀ» ÇÑ´Ù.
¿µ¹® digestive system ÇÑ±Û ¼ÒÈ­±â°èÅë
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  ¸ÔÀº À½½ÄÀÇ ºÐÇØ, ¼ÒÈ­, Èí¼ö¿¡ °ü°èµÈ Àå±â¸¦ ÅëÄªÇØ¼­ ºÎ¸£´Â ¸».
¿µ¹® nervous system ÇÑ±Û ½Å°æ°è
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  ¿©·¯ ±â°üµéÀÇ ¼­·Î°£ »óÈ£¿¬°áü°è°¡ ¹Ù·Î ½Å°æ°èÀÌ´Ù. ¿©±â¿¡´Â ÁßÃ߽Űæ°è(central nerve system: CNS)¿Í ¸»ÃʽŰæ°è(peripheral nerve system: PNS)°¡ Àִµ¥, ÁßÃ߽Űæ°è¶õ ³ú¿Í Ã´¼ö¸¦ ¸»ÇÑ´Ù. ±×¸®°í ¸»ÃʽŰæ°è¿¡´Â 12½ÖÀÇ ³ú½Å°æ(cranial nerve: ³ú¿¡¼­ ±â½ÃÇÏ¿© ÁַΠ¾ó±¼ºÎÀ§¿Í ¸ñ ºÎÀ§¿¡ ºÐÆ÷ÇÑ´Ù)°ú 31½ÖÀǠô¼ö½Å°æ(spinal nerve:spinal cord¿¡¼­ °¢±â ¾çÂÊÀ¸·Î ½ÖÀ» ÀÌ·ç¾î ³ª¿À´Âµ¥ ÁַΠ¸ñÀÌÇϺÎÀ§ÀÇ ½Åü °¢ºÎºÐÀ¸·Î ºÐÆ÷ÇϰԠµÈ´Ù)À¸·Î ±¸¼ºµÇ¾î ÀÖ´Ù.
  
  ¶ÇÇÑ ¸»ÃʽŰæ°è´Â 3°¡ÁöÀÇ ½Å°æÁ¶Á÷µé·Î ±¸¼ºµÇ¾î Àִµ¥ ¾Õ¿¡¼­ ¸»ÇÑ ³ú½Å°æ°ú Ã´¼ö½Å°æ¿Ü¿¡ ÀÚÀ²½Å°æ°è°¡ ¿©±â¿¡ ÇØ´çµÈ´Ù. ÀÚÀ²½Å°æ°è´Â ´Ù½Ã ±³°¨½Å°æ°ú ºÎ±³°¨½Å°æÀ¸·Î ³ª´µ¾îÁ® ¼­·Î°£ÀÇ ¿Ã¹Ù¸¥ »óÈ£ÀÛ¿ëÀ¸·Î »ýü ¿©·¯ °¡Áö ÀÛ¿ëÀ» ¼öÇàÇÑ´Ù.
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  • ¿µ¹®
    ÇѱÛ
  • blood group system
    Ç÷¾×Çüü°è
  • buffer system
    ¿ÏÃæ°è
  • circuit system
    ¼øÈ¯½Äȸ·Î
  • circulatory system
    ¼øÈ¯°èÅë, ¼øÈ¯°è
  • closed drainage system
    ´ÝÈû¹èÃâÀåÄ¡
  • clotting system
    ÀÀ°í°èÅë
  • collecting system
    ÁýÇÕ°è
  • combined system disease
    º¹ÇÕ°èÅ뺴
  • conduction system
    Àüµµ°è
  • control system
    Á¦¾îÀåÄ¡
  • countercurrent system
    ¿ª·ù°è
  • culture system
    ¹è¾ç½Ã½ºÅÛ
  • cytochrome P-450 system
    ½ÃÅäÅ©·ÒP-450½Ã½ºÅÛ
  • cardiovascular system
    ½ÉÀåÇ÷°ü°èÅë, ½ÉÇ÷°ü°è
  • case payment system
    Æ÷°ý¼ö°¡Á¦
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  • ¿µ¹®
    ÇѱÛ
  • anaerobic culture system
    ¹«»ê¼Ò¹è¾ç±â
  • ancillary immune system
    º¸Á¶¸é¿ª°è
  • Apgar scoring system
    ¾ÆÇÁ°¡Á¡¼öÆò°¡¹ý
  • archicortical system
    ¿ø½Ã°ÑÁú°è
  • array system
    ¹è¿­ÀåÄ¡, ¹è¿­Ã¼°è
  • auditory system
    û°¢°èÅë
  • autonomic nervous system
    ÀÚÀ²½Å°æ°èÅë
  • system analysis
    °èÅëºÐ¼®
  • biliary system
    ´ãµµ°è, ¾µ°³°èÅë
  • bioenergetic system
    »ý¹°¿¡³ÊÁö°è
  • blood group system
    Ç÷¾×Çü±º
  • breathing system
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  • buffer system
    ¿ÏÃæ°è
  • capitation system
    ȯÀÚµî·ÏÁ¦
  • cardiovascular system
    ½ÉÀåÇ÷°ü°èÅë
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  • ¿µ¹®
    ÇѱÛ
  • Haversian system
    ÇϹö½º°è
  • Hypothalamic-hypophysial portal system
    ½Ã»óÇϺÎ-Çϼöü ¹®¸Æ°è(ãÊßÉù»Ý» ù»á÷ô÷ Ú¦ØæÍ§)
  • Immune system
    ¸é¿ªÃ¼°è(Øóæ¹ô÷ͧ)
  • International System of Unit
    ±¹Á¦´ÜÀ§°è
  • Jackson-Rees anesthetic circuit system
    Àè½¼-¸® ¸¶ÃëÀåÄ¡
  • Kell blood group system
    ÄÌÇ÷¾×Çü±º
  • Kell system
    ÄÌÇ÷¾×Çü.
  • Kidd blood group system
    ۵åÇ÷¾×Çü±º
  • Lewis blood group system
    ·çÀ̽º Ç÷¾×Çü±º
  • Lutheran blood group system
    ·çÅͶõ Ç÷¾×Çü±º
  • Lymphatic system
    ¸²ÇÁ°è(¡­Í£)
  • Magills anesthetic circuit system
    ¸¶Áú¸¶ÃëÀåÄ¡
  • Mapleson anesthetic circuit system
    ¸ÅÇý¼¸¶Ãëȸ·Î
  • NADPH-dependent oxidase system
    NADPH-ÀÇÁ¸¼º »êÈ­È¿¼Ò°è
  • OS (operating system)
    ¿î¿µÃ¼Á¦
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  • ¿µ¹®
    ÇѱÛ
  • complement fixation inhibition test
    º¸Ã¼°íÁ¤ÀúÇØ½ÃÇè(ÜÍô÷ͳïÒîÁúªãËúÐ).
  • complement fixation inhibition test
    º¸Ã¼°áÇÕÀúÇØ½ÃÇè(ÜÍô÷Ì¿ùêîÁúªãËúÐ).
  • complement fixation reaction
    º¸Ã¼°áÇÕ¹ÝÀÀ(¡­Úãëë).
  • complement fixation test
    º¸Ã¼°áÇÕ½ÃÇè
  • complement fixation test =CFT
    º¸Ã¼°íÁ¤½ÃÇè(¡­ãËúÐ).
  • complement fixation test =CFT
    º¸Ã¼°áÇÕ½ÃÇè(¡­ãËúÐ).
  • complement fixation test, indirect
    °£Á¢º¸Ã¼°áÇÕ½ÃÇè
  • complement fixation unit =CF unit
    º¸Ã¼°áÇÕ´ÜÀ§(¡­Ó¤êÈ).
  • complement fixing antibody
    º¸Ã¼°áÇÕÇ×ü(¡­ù÷ô÷).
  • complement fixing neutralization antibody
    º¸Ã¼°áÇÕ¼º ÁßÈ­Ç×ü.
  • complement inhibitor
    º¸Ã¼ÀúÁöÀÎÀÚ.
  • complement inhibitor
    º¸Ã¼ÀúÁöÀÎÀÚ.
  • complement inhibitor
    º¸Ã¼ÀúÁöÀÎÀÚ.
  • complement inhibitor
    º¸Ã¼ÀúÁöÀÎÀÚ.
  • complement mediated cytotoxicity
    º¸Ã¼¸Å°³ ¼¼Æ÷µ¶¼º
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  • ¿µ¹®
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  • enzyme system
    È¿¼Ò (ý£áÈ) ½Ã½ºÅÛ
  • extrinsic system
    ¿ÜÀÎ(èâì×) ½Ã½ºÅÛ
  • FAS system
    FAS ½Ã½ºÅÛ
  • fatty acid synthetase system
    Áö¹æ»ê ÇÕ¼º È¿¼Ò(ò·Û¸ß«ùêà÷ý£áÈ)½Ã½ºÅÛ
  • genetic system
    À¯Àü(ë¶îî) ½Ã½ºÅÛ
  • host-guest system
    ÁÖ-°´(ñ«-ËÔ) ½Ã½ºÅÛ
  • hydrogen transport system
    ¼ö¼Ò ¼ö¼Û(â©áÈâÃáê) ½Ã½ºÅÛ
  • inducible system
    À¯µµ°¡´É(ë¯ÓôʦÒö) ½Ã½ºÅÛ
  • intrinsic system
    ³»ÀÎ(Ò®ì×) ½Ã½ºÅÛ
  • isolated system
    °Ý¸®(̰×î)½Ã½ºÅÛ
  • Kovats retention index system
    ÄÚ¹ÙÆ® ü·ù(ôò׺) Áö¼ö(ò¦â¦)½Ã½ºÅÛ
  • K system
    K ½Ã½ºÅÛ
  • leiotonin system
    ¶óÀÌ¿ÀÅä´Ñ ½Ã½ºÅÛ
  • MN blood group system
    MN½Ä(ãÒ)Ç÷¾×Çü(úìäûúþ)
  • model system
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KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
MLRD microgastria-limb reduction defects [association]
MLS mean lifespan; median life span; median longitudinal section; microphthalmia-linear skin defects [sy...
N/D no defects; not done
OEIS omphalocele, exstrophy, imperforate anus, spinal defects [complex]
RL radial line; radiation laboratory; reduction level; renal dysplasia-limb defects [syndrome]; resisti...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 3
C3 Complement 3
CCP Complement Control Protein
CF Complement Fixation
CFT Complement Fixation
CFR Complement Fixation Reaction
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
  • ¿µ¹®
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    ¼³¸í
  • buffer system
    ¿ÏÃæ°è
  • central nerve system mechanism
    ÁßÃß ½Å°æ°è ±âÀü
  • central nervous system mechanism
    ÁßÃß ½Å°æ°è ±âÀü
  • central piping system
    Áß¾Ó ¹è°ü ½Ã¼³
  • centrencephalic system
    Á᫐ ³ú°è
  • cerebrospinal system
    ³ú ô¼ö°è
  • combined system disease
    º¹ÇÕ °èÅë Áúȯ
  • concise enamel bond system
    ´Ü¼ø ¹ý¶ûÁú °áÇÕ
  • condensed system
    ÀÀ»ó°è
  • conduction system
    ÈïºÐ Àüµµ°è
  • control system
    Á¶Àý°è
  • corticospinal system
    ÇÇÁú ô¼ö°è
  • countercurrent system
    ¿ª·ù°è, ´ëÇâ·ù°è
  • crystal system
    Á¤°è, °áÁ¤°è
  • cyclic AMP system
    ȯ»ó AMP °è
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 3
complement 3 nephritic factor A magnesium-dependent IgG autoantibody found in serum of patients with chronic mesangioproliferative hypocomplementemic glomerulonephritis. It causes inactivation of c3 in the alternate pathway by cleaving c3 into two inactive fragments, c3c and c3d, instead of the normal c3b.
(12 Dec 1998)
complement 4 The second component to react in the complement sequence. It is a beta-globulin with a sedimentation coefficient of 18.7, a molecular weight of 240,000 and a serum concentration of 430 micrograms/ml. It is activated by complement 1 and serves as a receptor for c2.
(12 Dec 1998)
complement 4a <chemical> Smaller fragment formed when c1s splits c4 into c4a and c4b. As an anaphylatoxin, c4a causes symptoms of immediate hypersensitivity but it has weaker activity than c3a or c5a.
Chemical name: Complement C4a
(12 Dec 1998)
complement 4b <chemical> Larger fragment formed when c1s splits c4 into c4a and c4b. C4b combines with c2b to form the activated c4b2b complex which is often called the classical pathway c3 convertase.
Chemical name: Complement C4b
(12 Dec 1998)
complement 5 The fifth component in the complement reaction sequence, probably exists in a complex with c6 and c7. It is a beta-globulin with a sedimentation coefficient of 10, serum concentration of 75 micrograms/ml and molecular weight of 180,000. It is activated by c423 and releases fragments with anaphylatoxic, chemotactic, and histamine-releasing actions and affecting smooth muscle.
(12 Dec 1998)
complement 5a <chemical> Smaller fragment formed when c5 convertase splits c5 into c5a and c5b. C5a is a 74-amino acid peptide that includes a carboxy-terminal arginine crucial for its spasmogenic activity and a carbohydrate moiety. C5a is the most potent anaphylatoxin mediating immediate hypersensitivity.
Chemical name: Complement C5a
(12 Dec 1998)
complement 5a, des-arginine Complement 5a with the carboxy-terminal arginine removed. The arginine is rapidly cleaved from the c5a fragment during complement activation by carboxypeptidase b present in normal human serum. C5a des-arg shows complete loss of spasmogenic activity though it retains some chemotactic ability.
(12 Dec 1998)
complement 6 The sixth component in the complement reaction sequence. It is a beta-globulin with a sedimentation coefficient of 8.7 and a molecular weight of 120,000 at 60 micrograms/ml in serum. It may exist in a complex with c5 and c7 and is activated by the binding of c5.
(12 Dec 1998)
complement 7 The seventh component in the complement reaction sequence. It is a beta-globulin probably in a complex with c5 and c6 and is activated by c5. The attachment of c7 renders the cell susceptible to lysis.
(12 Dec 1998)
complement 8 The next to the last essential component for cell lysis in the complement reaction sequence. It is a gamma-globulin with a molecular weight of 150,000 and a sedimentation coefficient of 8. It is present in trace amounts in serum and can be inhibited, like complement 1, by cation chelators.
(12 Dec 1998)
complement 9 The last component in the complement reaction sequence. It is an alpha-globulin present in serum as a trace, with a molecular weight of 80,000 and a sedimentation coefficient of 4.5. For cell lysis, it can be replaced by a metal chelator.
(12 Dec 1998)
complement activating enzymes <enzyme> Enzymes present in the complement system which activate one or more components in the system.
Registry number: EC 3.-
(12 Dec 1998)
complement activation The sequential activation of serum components c1 through c9, initiated by an erythrocyte-antibody complex or by microbial polysaccharides and properdin, and producing an inflammatory response.
(12 Dec 1998)
complement binding assay A test for the detection of immune complexes.
(05 Mar 2000)
complement chemotactic factor The activated complex of the fifth, sixth, and seventh components of complement (C567) which induces chemotaxis in the case of polymorphonuclear leukocytes.
(05 Mar 2000)
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  • ¿µ¹®
    ÇѱÛ
  • cold type system
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  • container system
    ÄÁÅ×ÀÌ³Ê ¼ö¼Û¹æ½Ä
  • convict system
    ¡¿ª(À¯Çü)Á¦µµ
  • coupon system
    °æÇ°ºÎ ÆÇ¸Å¹ý
  • data base management system
    (Àü»ê)µ¥ÀÌÅͺ£À̽º °ü¸® ½Ã½ºÅÛ(¾à DBMS)
  • decimal system
    ½ÊÁø¹ý
  • decision support system
    (Àü»ê)(°æ¿µÀÇ)ÀÇ»ç °áÁ¤ Áö¿ø ½Ã½ºÅÛ
  • delayed time system
    (ÀüÀÚ)½Ã°£ Áö¿¬(ÃàÀû)¹æ½Ä
  • digestive system
    (ÇØ)¼ÒÈ­±â °èÅë(ÀÔ,À§,Àå µî)
  • domestic system
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  • duples system
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