| 영문 | tissue biopsy | 한글 | 조직생검 |
|---|---|---|---|
| 설명 | 환자의 병적인 조직의 일부를 떼어 내서 병리조직표본을 만들어서 현미경적으로 검사하는 것. 외과적 수술재료로부터 병리조직학적 검사를 하여 생체내의 병변을 추구하는 분야를 외과병리학이라고 한다. 생검에는 쐐기생검, 천자침생검, 꿰뚫기식생검, 내시경생검 외에 세포진이라고 불리우는 박리세포진이나 세척세포진 등 떨어진 세포를 도말검사하는 방법이 포함된다. |
||
| 영문 | target tissue | 한글 | 표적조직 |
|---|---|---|---|
| 설명 | 호르몬에 대하여 표적이 되는 세포라는 의미로서 그 호르몬에 대한 수용체를 가지고 있는 세포. 예를 들어 인슐린은 인슐린 수용체를 가진 세포를 표적세포로 하여 이 세포에만 작용한다. |
||
| 영문 | subcutaneous tissue, subcutis | 한글 | 피하조직 |
|---|---|---|---|
| 설명 | 피부에서 진피의 아랫부분을 말함. 여기에는 지방조직이 많다. 기능은 열의 격리, 충격흡수, 영양저장소 등이다. |
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| TCID | tissue culture infective dose; tissue culture inoculated dose |
|---|---|
| TCID50 | median tissue culture infective dose; 50% tissue culture infective dose |
| BTSS | Biopsy Tissue Scrub Smear |
| CT | 1) Computed(Computer) Tomography(-gram); 컴퓨터 단층 촬영 = CAT &... |
| CTD | Connective Tissue Disease |
| carbonic anhydrase inhibitors | A class of compounds that reduces the secretion of h+ ions by the proximal kidney tubule through inhibition of carbonic anhydrase (carbonate dehydratase). Although their therapeutic use as diuretics is not frequent, they are used in clinical conditions where alkalinization of the urine is beneficial. Their most frequent application is in the reduction of intra-ocular pressure in the treatment of glaucoma. (12 Dec 1998) |
|---|---|
| reverse transcriptase inhibitors | Inhibitors of reverse transcriptase (RNA-directed DNA polymerase), an enzyme that synthesises DNA on an RNA template. (12 Dec 1998) |
| growth inhibitors | Endogenous or exogenous substances which inhibit the normal growth of human and animal cells or micro-organisms, as distinguished from those affecting plant growth (= plant growth regulators). (12 Dec 1998) |
| phosphodiesterase inhibitors | Compounds which inhibit or antagonise the biosynthesis or actions of phosphodiesterases. (12 Dec 1998) |
| monoamine oxidase inhibitors | A chemically heterogeneous group of drugs that have in common the ability to block oxidative deamination of naturally occurring monoamines. Although mao inhibitors are probably as effective as tricyclic antidepressants in the treatment of major depression, the complex, sometimes severe, and often unpredictable interactions between mao inhibitors and many other drugs and food-derived amines make their medical use difficult and potentially hazardous. (12 Dec 1998) |
| platelet aggregation inhibitors | Drugs or agents which antagonise or impair any mechanism leading to blood platelet aggregation, whether during the phases of activation and shape change or following the dense-granule release reaction and stimulation of the prostaglandin-thromboxane system. (12 Dec 1998) |
| cysteine proteinase inhibitors | Exogenous and endogenous compounds which inhibit cysteine proteinases. (12 Dec 1998) |
| protease inhibitors | Compounds which inhibit or antagonise biosynthesis or actions of proteases. (12 Dec 1998) |
| HIV integrase inhibitors | Inhibitors of HIV integrase, an enzyme required for integration of viral DNA into cellular DNA. (12 Dec 1998) |
| HIV protease inhibitors | Inhibitors of HIV protease, an enzyme required for production of proteins needed for viral assembly. (12 Dec 1998) |
| serine proteinase inhibitors | Exogenous or endogenous compounds which inhibit serine proteinases. (12 Dec 1998) |
| serotonin uptake inhibitors | Compounds that specifically inhibit the reuptake of serotonin in the brain. This increases the serotonin concentration in the synaptic cleft which then activates serotonin receptors to a greater extent. These agents have been used in treatment of depression, panic disorder, obsessive-compulsive behaviour, and alcoholism, as analgesics, and to treat obesity and bulimia. Many of the adrenergic uptake inhibitors also inhibit serotonin uptake; they are not included here. (12 Dec 1998) |
| hydroxymethylglutaryl-CoA reductase inhibitors | Compounds that inhibit hmg-CoA reductases. They have been shown to directly lower cholesterol synthesis. (12 Dec 1998) |
| neurotransmitter uptake inhibitors | Drugs that inhibit the transport of neurotransmitters into axon terminals or into storage vesicles within terminals. For many transmitters, uptake determines the time course of transmitter action so inhibiting uptake prolongs the activity of the transmitter. Blocking uptake may also deplete available transmitter stores. Many clinically important drugs are uptake inhibitors although the indirect reactions of the brain rather than the acute block of uptake itself is often responsible for the therapeutic effects. (12 Dec 1998) |
| nucleic acid synthesis inhibitors | Compounds that inhibit cell production of DNA or RNA. (12 Dec 1998) |