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  • glomerulonephritis
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  • intercapillary glomerulonephritis
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  • membranoproliferative glomerulonephritis
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  • membranous glomerulonephritis
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  • mesangial proliferative glomerulonephritis
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  • mesangiocapillary glomerulonephritis
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  • poststreptococcal glomerulonephritis
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  • focal segmental glomerulonephritis
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  • glomerulonephritis
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  • intercapillary glomerulonephritis
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  • membranoproliferative glomerulonephritis
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  • rapidly progressive (crescentic) glomerulonephritis
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  • glomerulonephritis,membranoproliferative
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  • membranous glomerulonephritis
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  • poststreptococcal(proliferative) glomerulonephritis
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  • proliferative glomerulonephritis
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  • rapidly progressive (crescentic) glomerulonephritis
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  • rapidly progressive glomerulonephritis
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SSPE Subacute Sclerosing Pan-Encephalitis
DUSN diffuse unilateral subacute neuroretinitis
MLS mean lifespan; median life span; median longitudinal section; microphthalmia-linear skin defects [sy...
SACD subacute combined degeneration
SAT saliva alcohol test; satellite; serum antitrypsin; single-agent chemotherapy; slide agglutination te...
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SBE Subacute bacterial endocarditis
SCLE Subacute cutaneous LE
SNE Subacute necrotizing encephalomyelopathy
APSGN Acute Poststreptococcal Glomerulonephritis
AGN Acute glomerulonephritis
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
subacute spongiform encephalopathy A form of spongiform encephalopathy that is associated with a "slow virus", which to date has not been adequately described, is transmissible, and has a rapidly progressive, fatal course; e.g., Creutzfeldt-Jakob disease, kuru, Gerstmann-Straussler syndrome, scrapie.
(05 Mar 2000)
thyroiditis, subacute Inflammation of the thyroid gland after a viral illness.
(12 Dec 1998)
endocarditis, subacute bacterial Infection of the endocardium caused by species of streptococcus. This condition does not produce metastatic foci and if untreated may take up to a year to be fatal.
(12 Dec 1998)
acute crescentic glomerulonephritis <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease.
Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents.
(27 Sep 1997)
acute glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
acute haemorrhagic glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
acute post-streptococcal glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
anti-basement membrane glomerulonephritis Glomerulonephritis resulting from anti-basement membrane antibodies, characterised by smooth linear deposits of IgG and C3 along glomerular capillary walls; includes rapidly progressive glomerulonephritis and glomerulonephritis in Goodpasture's syndrome.
(05 Mar 2000)
Berger's focal glomerulonephritis Glomerulonephritis affecting a small proportion of renal glomeruli which commonly presents with haematuria and may be associated with acute upper respiratory infection in young males, not usually due to streptococci; associated with IgA deposits in the glomerular mesangium and may also be associated with systemic disease, as in Henoch-Schonlein purpura.
Synonym: Berger's disease, Berger's focal glomerulonephritis, focal nephritis, IgA nephropathy.
(05 Mar 2000)
rapidly progressive glomerulonephritis <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease.
Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents.
(27 Sep 1997)
glomerulonephritis <nephrology> A variety of nephritis characterised by inflammation of the capillary loops in the glomeruli of the kidney. It occurs in acute, subacute and chronic forms and may be secondary to haemolytic streptococcal infection. Evidence also supports possible immune or autoimmune mechanisms.
(18 Nov 1997)
glomerulonephritis, IgA Chronic form of glomerulonephritis characterised by recurring haematuria with only slight proteinuria and by deposits of IgA immunoglobulin in the mesangial areas of the renal glomeruli. It usually occurs in young males.
(12 Dec 1998)
glomerulonephritis, membranoproliferative Chronic glomerulonephritis characterised histologically by proliferation of mesangial cells, increase in the mesangial matrix, and a thickening of the glomerular capillary walls. The wall thickening is sometimes said to be a result of the interposition of mesangial cytoplasm or matrix between the basement membrane and the endothelium of the capillary wall. It is often divided into types I and II, and sometimes III. Pathogenesis is not well understood, but some types are thought to involve the immune system, with the complement system, in particular, being implicated.
(12 Dec 1998)
glomerulonephritis, membranous A disease of the glomerulus manifested clinically by proteinuria, and sometimes by other features of the nephrotic syndrome. It is histologically characterised by deposits in the glomerular capillary wall between the epithelial cell and the basement membrane and a thickening of the membrane. Also characteristic are outward projections of the membrane between the epithelial deposits in the form of "spikes". There is some agreement that the deposits are antigen-antibody complexes.
(12 Dec 1998)
membranoproliferative glomerulonephritis <pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure.
Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane.
Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease.
Acronym: MPGN
(26 Mar 1998)
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