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| ¿µ¹® | renal transplantation | ÇÑ±Û | ÄáÆÏÀÌ½Ä |
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| RVRA | renal vein rein activity; renal venous renin assay |
|---|---|
| DVT | Deep Vein Thrombosis |
| ABCDES | abnormal alignment, bones-periarticular osteoporosis, cartilage-joint space loss, deformities, margi... |
| PVT | paroxysmal ventricular tachycardia; portal vein thrombosis; pressure, volume, and temperature; priva... |
| SVT | sinoventricular tachycardia; subclavian vein thrombosis; supraventricular tachyarrhythmia; supravent... |
| posttraumatic arterial thrombosis | Posttraumatic venous thrombosis, intravascular clotting due to injury to a vessel wall. (05 Mar 2000) |
|---|---|
| sinus thrombosis | Formation of a clot in a cerebral venous sinus. (12 Dec 1998) |
| deep venous thrombosis | <cardiology> A blood clot that forms in a vein resulting in obstruction of venous flow. most common clinically in the lower extremities. (27 Sep 1997) |
| dilation thrombosis | Thrombosis due to slowed circulation consequent upon dilation of a vein. (05 Mar 2000) |
| thrombosis | <haematology> The formation, development or presence of a thrombus. (14 May 1997) |
| effort-induced thrombosis | <syndrome> Stress thrombosis or spontaneous thrombosis of the subclavian or axillary vein; a thoracic-outlet syndrome. Synonym: effort-induced thrombosis. (05 Mar 2000) |
| embolism and thrombosis | A collective term for diseases characterised by the formation, development, or presence of a thrombus (thrombosis) and the blocking of a vessel by the thrombus brought to its site by the blood current (embolism). (12 Dec 1998) |
| acute renal failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| aminoaciduria, renal | Impairment of renal tubular transport of amino acids. (12 Dec 1998) |
| back-pressure renal atrophy | <radiology> Caliectasis without obstruction, due to repeated episodes of obstruction, gradual loss of renal pyramids (12 Dec 1998) |
| base of renal pyramid | The outer broad part of a renal pyramid that lies next to the cortex. Synonym: basis pyramidis renis. (05 Mar 2000) |
| branchio-oto-renal syndrome | <syndrome> An autosomal dominant disorder manifested by various combinations of preauricular pits, branchial fistulae or cysts, lacrimal duct stenosis, hearing loss, structural defects of the outer, middle, or inner ear, and renal dysplasia. Associated defects include asthenic habitus, long narrow facies, constricted palate, deep overbite, and myopia. Hearing loss may be due to mondini type cochlear defect and stapes fixation. (12 Dec 1998) |
| capsular branches of renal artery | <anatomy, artery> Branches arising from the renal artery outside of the kidney that are distributed to the renal capsule. Synonym: rami capsulares arteriae renalis. (05 Mar 2000) |
| captopril renal scan | <radiology> In a kidney with a lesion in the afferent arteriole (e.g. Atherosclerotic plaque), reflex constriction of the efferent arteriole occurs through angiotensin system thus maintaining renal perfusion. ACE inhibition prevents constriction of efferent arteriole. Therefore, perfusion is decreased to a kidney with afferent lesions and the renal scan to looks WORSE. Bottom line: renal scans appear WORSE with captopril administration if there is a lesion in the afferent arteriole. See: renal artery stenosis (12 Dec 1998) |
| carcinoma, renal cell | Carcinoma of the renal parenchyma usually occurring in middle age or later and composed of tubular cells in varying arrangements. It was first described in 1826. Possible causal factors are environmental, hormonal, cellular, and genetic. Smoking is a definite risk factor and obesity is associated with increased risk. Renal cell carcinoma accounts for approximately 3% of adult cancer; the male-female ratio is 2:1. It is more common among urban residents than rural. (12 Dec 1998) |
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