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¿µ¹® central nervous system(CNS) ÇÑ±Û ÁßÃ߽Űæ°è
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  • ¿µ¹®
    ÇѱÛ
  • reactive arthritis
    ¹ÝÀÀ°üÀý¿°
  • reactive attachment disorder
    ¹ÝÀÀ¼º¾ÖÂøÀå¾Ö
  • reactive bone excrescence
    ¹ÝÀÀ»Àµ¹Ãâ
  • reactive depression
    ¹ÝÀÀ¿ì¿ïÁõ
  • reactive fibrosis
    ¹ÝÀÀ¼¶À¯È­
  • reactive hyperemia
    ¹ÝÀÀÃæÇ÷
  • reactive hypoglycemia
    ¹ÝÀÀÀúÇ÷´ç(Áõ)
  • reactive inflammation
    ¹ÝÀÀ¿°Áõ
  • reactive interface
    ¹ÝÀÀÁ¢Ã˸é
  • reactive intermediate
    ¹ÝÀÀÁß°£¹°Áú
  • reactive nodular hyperplasia
    ¹ÝÀÀ¼º°áÀýÁõ½Ä
  • anxiety state
    ºÒ¾È»óÅÂ
  • acute confusional state
    ±Þ¼ºÈ¥µ·»óÅÂ
  • basal state
    ±âÃÊ»óÅÂ
  • clouded state
    ȥŹ»óÅÂ
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  • ¿µ¹®
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  • reactive perforating collagenosis
    ¹ÝÀÀ°üÅë¾Æ±³ÁúÁõ
  • reactive depression
    ¹ÝÀÀ¿ì¿ïÁõ
  • reactive attachment disorder
    ¹ÝÀÀ¾ÖÂøÀå¾Ö, ¹ÝÀÀ¾ÖÂøº´
  • reactive bone excrescence
    ¹ÝÀÀ»Àµ¹Ãâ
  • reactive fibrosis
    ¹ÝÀÀ¼¶À¯Áõ
  • reactive hyperemia
    ¹ÝÀÀÃæÇ÷
  • reactive hypoglycemia
    ¹ÝÀÀÀúÇ÷´çÁõ
  • reactive nodular hyperplasia
    ¹ÝÀÀ°áÀýÁõ½Ä
  • reactive inflammation
    ¹ÝÀÀ¿°Áõ
  • reactive interface
    ¹ÝÀÀÁ¢Ã˸é
  • reactive intermediate
    ¹ÝÀÀÁß°£¹°Áú
  • percent reactive antibody test
    ¹éºÐÀ²¹ÝÀÀÇ×ü°Ë»ç
  • accessory state
    µ¡»óÅÂ
  • acute confusional state
    ±Þ¼ºÈ¥µ·»óÅÂ
  • adult ego state
    ¼ºÀÎÀھƻóÅÂ
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  • ¿µ¹®
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  • reactive fibrosis
    ¹ÝÀÀ¼º ¼¶À¯È­
  • reactive functional hypoglycemia
    ¹ÝÀÀ¼º ±â´É¼º ÀúÇ÷´ç(¡­Ñ¦Òöàõî¸úìÓØ).
  • reactive hyperemia
    ¹ÝÀÀ¼º ÃæÇ÷(¡­õöúì)
  • reactive hypoglycemia
    ¹ÝÀÀ¼º ÀúÇ÷´çÁõ
  • reactive inflammation
    ¹ÝÀÀ¼º ¿°Áõ(Úãëëàõ)
  • reactive interface
    ¹ÝÀÀ¼º Á¢Ã˸é
  • reactive intermediate
    ¹ÝÀÀ¼º Áß°£¹°Áú.
  • reactive nodular hyperplasia
    ¹ÝÀÀ¼º °áÀýÁõ½Ä
  • reactive perforating collagenosis
    ¹ÝÀÀ¼º õ°ø ±³¿øÁõ
  • reactive pseudosarcomatous proliferation
    ¹ÝÀÀ¼º °¡¼ºÀ°Á¾¼º Áõ½Ä(¡­Ê£àõë¿ðþàõ)
  • reactive systemic amyloidosis
    ¹ÝÀÀ¼º Àü½Å À¯ÀüºÐÁõ
  • DESS (double echo in the steady state)
    Ç×Á¤ »óÅÂÀÇ ÀÌÁß ¿¡ÄÚ
  • FAST(Fourier acquisition in the steady state) sequen
    Ç×Á¤ »óÅ¿¡¼­ÀÇ Fourier ȹµæ ¿¬¼â
  • Mini-Mental State Examination(MMSE)
    °£ÀÌÁ¤½Å»óŰ˻ç(ÊÛì¯ïñãêßÒ÷¾ËþÞÛ)
  • Present State Examination
    Çö»óŰ˻磨úÞßÈ÷¾ËþÞÛ£©
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  • ¿µ¹®
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  • reactive bone excrescence
    ¹ÝÀÀ¼º °ñ µ¹Ãâ
  • reactive depression
    ¹ÝÀÀ¼º ¿ì¿ïÁõ(ÚãëëàõéØê¦ñø).
  • reactive eosinophilia
    ¹ÝÀÀ¼º È£»ê±¸Áõ´ÙÁõ
  • reactive epilepsy
    ¹ÝÀÀ¼º °£Áú(¡­ÊÖòð).
  • reactive epilepsy
    ¹ÝÀÀ¼º °£Áú(ÚãëëàõÊÖòð)
  • reactive fibrosis
    ¹ÝÀÀ¼º ¼¶À¯È­
  • reactive functional hypoglycemia
    ¹ÝÀÀ¼º ±â´É¼º ÀúÇ÷´ç(¡­Ñ¦Òöàõî¸úìÓØ).
  • reactive hyperemia
    ¹ÝÀÀ¼º ÃæÇ÷(¡­õöúì)
  • reactive hypoglycemia
    ¹ÝÀÀ¼º ÀúÇ÷´çÁõ
  • reactive inflammation
    ¹ÝÀÀ¼º ¿°Áõ(Úãëëàõ)
  • reactive interface
    ¹ÝÀÀ¼º Á¢Ã˸é
  • reactive intermediate
    ¹ÝÀÀ¼º Áß°£¹°Áú.
  • reactive nodular hyperplasia
    ¹ÝÀÀ¼º °áÀýÁõ½Ä
  • reactive perforarating collagenosis
  • reactive perforating collagenosis
    ¹ÝÀÀ¼º õ°ø ±³¿øÁõ
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  • ¿µ¹®
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  • metastable state
    ÁؾÈÁ¤ »óÅÂ(ñ×äÌïÒßÒ÷¾)
  • oxidation state
    »êÈ­»óÅÂ(ß«ûùßÒ÷¾)
  • polyploid state
    ´Ù¹èü»óÅÂ(ÒýÛÃô÷ßÒ÷¾)
  • postabsorptive state
    Èí¼öÈÄ »óÅÂ(ýåâ¥ý­ßÒ÷¾)
  • pre-steady-state kinetics
    ÀüÁ¤À¯»óÅÂ(îñïÒë§ßÒ÷¾) ¿ªÇÐ(ÕôùÊ)
  • singlet state
    ´ÜÇ×»óÅÂ(Ó¤ú£ßÒ÷¾)
  • standard state
    Ç¥ÁØ »óÅÂ(øöñÞßÒ÷¾)
  • state function
    »óÅ ÇÔ¼ö(ßÒ÷¾ùÞâ¦)
  • state of a system
    ½Ã½ºÅÛ »óÅÂ(ßÒ÷¾)
  • stationary state
    Á¤Áö »óÅ (ïÎò­ßÒ÷¾)
  • stationary-state approximation
    Á¤Áö »óÅÂ(ïÎò­ßÒ÷¾) ¾î¸²¼À
  • steady state
    Á¤·ù »óÅÂ(ïÒ׺ßÒ÷¾)
  • steady-state approximation
    Á¤·ù»óÅÂ(ïÒ׺ßÒ÷¾) ¾î¸²¼À
  • steady-state electrolysis
    Á¤·ù»óÅ Àü±âºÐÇØ(ïÒ׺ßÒ÷¾ï³Ñ¨ÝÂú°)
  • steady-state kinetics
    Á¤·ù»óÅ ¿ªÇÐ (ïÒ׺ßÒ÷¾ÕôùÊ)
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  • ¿µ¹®
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  • relaxation state
    À̿ϻóÅÂ
  • saturation state
    Æ÷È­»óÅÂ
  • SSFP [=steady state free precession]
    Ç×Á¤»óÅÂÀÚÀ¯¼¼Â÷
  • steady state
    Ç×Á¤»óÅÂ
  • steady state coherent sequence
    Ç×Á¤»óŰáÁý¿¬¼â
  • steady state free precession [=SSFP]
    Ç×Á¤»óÅÂÀÚÀ¯¼¼Â÷
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ANS acanthion; American Nutrition Society; 8-anilino-1-naphthalene-sulfonic acid; anterior nasal spine; ...
ARNMD Association for Research in Nervous and Mental Diseases
CANS central auditory nervous system
CNE chief nurse executive; chronic nervous exhaustion; concentric needle electrode
CNES chronic nervous exhaustion syndrome
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 2
PNS Parasympathetic Nervous System
PNS Peripheral Nervous System
PCNSL Primary Central Nervous System Lymphoma
PACNS Primary angiitis of the central nervous system
SNS Sympathetic Nervous System
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  • ¿µ¹®
    ÇѱÛ
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  • nervous impulse
    ½Å°æ Àüµµ
  • nervous intermedius neuralgia
    Áß°£ ½Å°æÅë
    geniculate neuralgia¸¦ º¸½Ã¿À.
  • nervous papilla
    ½Å°æ À¯µÎ
  • nervous system
    ½Å°æ°è
    ½ÅüÀÇ ÀÚ±ØÀ» ¼öÁýÇÏ¿© À̸¦ Àü±âÀû ÀÚ±ØÀ¸·Î º¯È¯Çؼ­ ÁßÃß ½Å°æ°è·Î º¸³» Á¾ÇÕÇÏ¿© ÀûÀýÇÑ ¹ÝÀÀ°ú °¨°¢À» ³ªÅ¸³½´Ù.
  • nervous tension
    ½Å°æ¼º ±äÀå
  • nervous zygomaticemporaris
    °ü°ñ ÃøµÎ ½Å°æ, Çù°ñ ÃøµÎ ½Å°æ
  • peripheral nervous system
    ¸»ÃÊ ½Å°æ°è
    1. ³ú ½Å°æ, ô¼ö ½Å°æ, ÀÚÀ² ½Å°æ. 2. ³ú¿Í ô¼ö ¹Ù±ù¿¡ ÀÖ´Â ¿îµ¿ ¹× °¨°¢ÀÇ ±³°¨½Å°æ°ú ºÎ±³°¨½Å°æ.
  • sympathetic nervous system activity
    ±³°¨½Å°æ°è Ȱ¼º
  • vegatative nervous system
    ½Ä¹°¼º ½Å°æ°è
  • active state
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    ±ÙÀ°ÀÌ ¼öÃàÇÒ ¶§, ¼öÃà¿ä¼Ò ÀÚü¿¡ ÀϾ°í ÀÖ´Â ¿ªÇÐÀû »óÅÂÀÌ´Ù. ±ÙÀ° ÀÚü¿¡ ź¼º¿ä¼Ò°¡ Á¸ÀçÇϹǷΠ´Ü¼øÇÑ ¼öÃà°î¼±ÀÇ ±â·ÏÀ¸·Î Ȱµ¿»óȲÀ» ¾Ë ¼ö ¾ø´Ù. ¼öÃà¿ä¼Ò´Â ±ÙÀ°ÀÌ ÀڱصǸé Áï½Ã Ȱµ¿»óŰ¡ µÇ°í, Á¦2, Á¦3ÀÇ ÀÚ±ØÀÌ °¡ÇØÁ®µµ ÀÌ È°µ¿»óÅ´ °­È­µÇÁö ¾Ê°í ´Ü¼øÈ÷ Áö¼Ó½Ã°£ÀÌ ¿¬ÀåµÉ »ÓÀÌ´Ù.
  • allergic state
    ¾Ë·¹¸£±â »óÅÂ
    Ven Pirquet´Â Ç׿ø ¹°Áú¿¡ ´ëÇÏ¿© º¯È­ÇÑ ¹ÝÀÀ ´É·ÂÀ» °¡Áö°í ÀÖ´Â »óŸ¦ ¾Ë·¹¸£±â »óŶó°í ºÒ·¶À¸³ª ÇöÀç´Â °íÃÊ¿­À̳ª õ½Ä µîÀÇ ¾ÆÅäÇǼº Áúȯ°ú °°ÀÌ ¾Ë·¹¸£±â°¡ ¿øÀÎÀÌ µÇ´Â Áúȯ¿¡ ´ëÇØ¼­¸¸ »ç¿ëµÈ´Ù.
  • basal state
    ±âÃÊ »óÅÂ
  • central excitatory state
    ÁßÃß ÈïºÐ »óÅÂ
  • correlated state
    »ó°ü »óÅÂ
  • critical state
    ÀÓ°è »óÅÂ
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
cross-reactive antibody <haematology, immunology> Antibodies which don't respond to any one specific antigen, but will respond to a number of them. These antibodies can be responsible for false positive results in antigen-antibody tests.
(09 Oct 1997)
thiobarbituric acid reactive substances Low-molecular-weight end products, probably malondialdehyde, that are formed during the decomposition of lipid peroxidation products. These compounds react with thiobarbituric acid to form a fluorescent red adduct.
(12 Dec 1998)
autonomic nervous system <anatomy> Neurons that are not under conscious control, comprising two antagonistic components, the sympathetic and parasympathetic nervous systems.
The autonomic nervous system regulates key functions including the activity of the cardiac (heart) muscle, smooth muscles (e.g., of the gut), and glands. The autonomic nervous system has two divisions:
1. The sympathetic nervous system that accelerates the heart rate, constricts blood vessels, and raises blood pressure.
2. The parasympathetic nervous system slows the heart rate, increases intestinal and gland activity, and relaxes sphincter muscles.
(03 Jul 1999)
autonomic nervous system diseases Diseases that have their major effects on the autonomic nervous system. The autonomic nervous system may be seriously affected in many other disorders including other peripheral nervous system diseases, infectious diseases (e.g., tetanus, diphtheria), immunologic diseases (e.g., acquired immunodeficiency syndrome), and systemic disorders (e.g., diabetic neuropathy, amyloid neuropathy, thyroid diseases). Disorders of central autonomic control also contribute substantially to a wide variety of problems (e.g., eating disorders, panic disorder, water-electrolyte imbalance, cardiovascular diseases).
(12 Dec 1998)
vegetative nervous system <anatomy> Neurons that are not under conscious control, comprising two antagonistic components, the sympathetic and parasympathetic nervous systems.
The autonomic nervous system regulates key functions including the activity of the cardiac (heart) muscle, smooth muscles (e.g., of the gut), and glands. The autonomic nervous system has two divisions:
1. The sympathetic nervous system that accelerates the heart rate, constricts blood vessels, and raises blood pressure.
2. The parasympathetic nervous system slows the heart rate, increases intestinal and gland activity, and relaxes sphincter muscles.
(03 Jul 1999)
ventral nervous system defective <molecular biology> A Drosophila gene encoding an integral membrane glycoprotein related to amyloidogenic glycoprotein.
(12 Jan 1998)
parasympathetic nervous system <anatomy, neurology> One of the two divisions of the vertebrate autonomic nervous sytem.
Parasympathetic nerves emerge cranially as pre ganglionic fibres from oculomotor, facial, glossopharyngeal and vagus and from the sacral region of the spinal cord.
most neurons are cholinergic and responses are mediated by muscarinic receptors. The parasympathetic system innervates, for example: salivary glands, thoracic and abdominal viscera, bladder and genitalia.
Compare:. Sympathetic nervous system.
(18 Nov 1997)
visceral nervous system <anatomy> Neurons that are not under conscious control, comprising two antagonistic components, the sympathetic and parasympathetic nervous systems.
The autonomic nervous system regulates key functions including the activity of the cardiac (heart) muscle, smooth muscles (e.g., of the gut), and glands. The autonomic nervous system has two divisions:
1. The sympathetic nervous system that accelerates the heart rate, constricts blood vessels, and raises blood pressure.
2. The parasympathetic nervous system slows the heart rate, increases intestinal and gland activity, and relaxes sphincter muscles.
(03 Jul 1999)
central nervous system <anatomy, neurology> Pertaining to the brain, cranial nerves and spinal cord. It does not include muscles or peripheral nerves.
In invertebrates, the central nervous system is composed of the segmental ganglia of the ventral nerve cord together with the fused ganglia or brain at the anterior end.
Acronym: CNS
(12 Jan 1998)
central nervous system agents A class of drugs producing both physiological and psychological effects through a variety of mechanisms. They can be divided into "specific" agents, e.g., affecting an identifiable molecular mechanism unique to target cells bearing receptors for that agent, and "non-specific" agents, those producing effects on different target cells and acting by diverse molecular mechanisms. Those with non-specific mechanisms are generally further classed according to whether they produce behavioural depression or stimulation. Those with specific mechanisms are classed by locus of action or specific therapeutic use.
(12 Dec 1998)
central nervous system depressants A very loosely defined group of drugs that tend to reduce the activity of the central nervous system. The major groups included here are ethyl alcohol, anaesthetics, hypnotics and sedatives, narcotics, and tranquillising agents (antipsychotics and antianxiety agents).
(12 Dec 1998)
central nervous system infections Diseases of the central nervous sytem collectively, caused by pathogenic organisms.
(12 Dec 1998)
central nervous system neoplasms Neoplasms located in the brain, spinal cord, or meninges.
(12 Dec 1998)
central nervous system stimulants A loosely defined group of drugs that tend to increase behavioural alertness, agitation, or excitation. They work by a variety of mechanisms, but usually not by direct excitation of neurons. The many drugs that have such actions as side effects to their main therapeutic use are not included here.
(12 Dec 1998)
peripheral nervous system One of the two major divisions of the nervous system. Nerves in the PNS connect the central nervous system (CNS) with sensory organs, other organs, muscles, bloodvessels and glands.
(22 May 1997)
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