| FAP | familial adenomatous polyposis; familial amyloid polyneuropathy; fatty acid polyunsaturated; fatty a... |
|---|---|
| FAC | Familial Amyloid Cardiomyopathy |
| FAP | Familial Amyloid Polyneuropathy |
| FH | Familial Hypercholesterolemia; 가족성 고콜레스테롤혈증 |
| FMTC | Familial Medullary Thyroid Carcinoma |
familial leiomyomatosis cutis et uteri (가족성 피부 자궁 근종증
| chronic idiopathic jaundice | <syndrome> An inherited disorder (autosomal recessive) that is characterised by long-standing mild jaundice. This occurs secondary to an abnormality in the transport of bilirubin from the liver to the biliary system. This leads to an accumulation of bilirubin in the liver. Avoidance of alcohol and medications which can affect the liver is important. Inheritance: autosomal recessive. (27 Sep 1997) |
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| physiologic jaundice | Icterus which can be accentuated by many factors including excessive haemolysis, sepsis, neonatal hepatitis or congenital atresia of the biliary system. Synonym: physiologic icterus, jaundice of the newborn, neonatal jaundice, physiologic jaundice. (05 Mar 2000) |
| congenital haemolytic jaundice | <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane. This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged. Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal. (27 Sep 1997) |
| postarsphenamine jaundice | Liver toxicity, causing jaundice, in a patient who has received arsphenamine. (05 Mar 2000) |
| haematogenous jaundice | <haematology> Haemolytic jaundice is a type of jaundice, where the skin takes on a yellowish hue, which occurs when red blood cells have been destroyed (by haemolysis). (09 Oct 1997) |
| haemolytic jaundice | <haematology> Haemolytic jaundice is a type of jaundice, where the skin takes on a yellowish hue, which occurs when red blood cells have been destroyed (by haemolysis). (09 Oct 1997) |
| hepatocellular jaundice | Jaundice resulting from diffuse injury or inflammation or failure of function of the liver cells, usually referring to viral or toxic hepatitis. (05 Mar 2000) |
| hepatogenous jaundice | Jaundice resulting from disease of the liver, as distinguished from that due to blood changes. (05 Mar 2000) |
| Schmorl's jaundice | <paediatrics> Disorder due to jaundice in a newborn baby with high blood levels of the pigment bilirubin that is deposited in the brain resulting in damage. The level of bilirubin is monitored in newborns to determine whether treatment is needed to prevent kernicterus. With brain affected, it is also called bilirubin encephalopathy. (12 Dec 1998) |
| homologous serum jaundice | An obsolete term for viral hepatitis type B. (05 Mar 2000) |
| human serum jaundice | An obsolete name for hepatitis transmitted parenterally, usually by blood or blood products; usually due to hepatitis B. (05 Mar 2000) |
| spherocytic jaundice | Haemolytic jaundice associated with spherocytosis. (05 Mar 2000) |
| spirochetal jaundice | Jaundice caused by infection with Leptospira species, usually Leptospira icterohemorrhagica. (05 Mar 2000) |
| neonatal jaundice | Icterus which can be accentuated by many factors including excessive haemolysis, sepsis, neonatal hepatitis or congenital atresia of the biliary system. Synonym: physiologic icterus, jaundice of the newborn, neonatal jaundice, physiologic jaundice. (05 Mar 2000) |
| newborn jaundice | <paediatrics> A normal condition of elevated bilirubin in the bloodstream of a newborn. This occurs secondary to immaturity of liver cells (cannot effectively metabolise bilirubin) and the increased destruction of red blood cells (further releasing bilirubin into the bloodstream) that is normally seen in the newborn. The jaundice usually appears between the 2nd and 5th days of life and usually clears by 2 weeks. Other factors which can potentiate jaundice in the newborn include: sepsis, biliary atresia, Rhesus incompatibility, galactosaemia, cephalohaematoma, polycythaemia, G-6-P-D deficiency and congenital rubella, syphilis, toxoplasmosis or cytomegalovirus infection. (10 Jan 1998) |