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  • ¿µ¹®
    ÇѱÛ
  • myocardial ischemia
    ½É±ÙÇãÇ÷, ½ÉÀå±ÙÀ°ÇãÇ÷
  • myocardial lead
    ½É±ÙÀ¯µµ, ½ÉÀå±ÙÀ°À¯µµ
  • myocardial perfusion scintigraphy
    ½É±Ù°ü·ù¼¶±¤Á¶¿µ(¼ú)
  • myocardial scan
    ½É±Ù½ºÄµ
  • post-myocardial infarction syndrome
    ½É±Ù°æ»öÈÄÁõÈıº
  • septal myocardial infarction
    Á߰ݽɱٰæ»öÁõ, »çÀ̸·½É±Ù°æ»öÁõ
  • silent myocardial infarction
    ¹«Áõ»ó½É±Ù°æ»öÁõ
  • arterial oxygen saturation
    µ¿¸ÆÇ÷»ê¼ÒÆ÷È­µµ
  • arterial oxygen tension
    µ¿¸ÆÇ÷»ê¼ÒºÐ¾Ð
  • active oxygen
    Ȱµ¿»ê¼Ò, Ȱ¼º»ê¼Ò
  • alveolar oxygen pressure
    ÆóÆ÷»ê¼Ò¾Ð, ÇãÆÄ²Ê¸®»ê¼Ò¾Ð
  • alveolar-arterial oxygen gradient
    ÆóÆ÷µ¿¸Æ°£»ê¼ÒºÐ¾ÐÂ÷
  • biochemical oxygen demand
    »ý¹°È­ÇÐÀû»ê¼Ò¿ä±¸·®
  • biological oxygen demand
    »ý¹°ÇÐÀû»ê¼Ò¿ä±¸·®
  • compressed oxygen
    ¾ÐÃà»ê¼Ò
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  • ¿µ¹®
    ÇѱÛ
  • arterial oxygen tension
    µ¿¸ÆÇ÷»ê¼ÒºÐ¾Ð
  • home oxygen therapy
    °¡Á¤»ê¼Ò¿ä¹ý
  • oxygen tension
    »ê¼ÒºÐ¾Ð, »ê¼ÒÀå·Â
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  • ¿µ¹®
    ÇѱÛ
  • idiopathic myocardial hypertrophy
    Ư¹ß½É±Ùºñ´ë, ¿øÀκҸí½É±Ùºñ´ë
  • myocardial infarction
    ½É±Ù°æ»öÁõ
  • myocardial irritability
    ½É±Ù°ú¹Î¼º, ½É±ÙÀÚ±ØÈïºÐ¼º
  • myocardial ischemia
    ½É±ÙÇãÇ÷
  • septal myocardial infarction
    Á߰ݽɱٰæ»öÁõ
  • silent myocardial infarction
    ¹«Áõ»ó½É±Ù°æ»öÁõ
  • transmural myocardial infarction
    ÀüÃþ½É±Ù°æ»öÁõ
  • myocardial lead
    ½É±ÙÀ¯µµ
  • myocardial
    ½ÉÀå±ÙÀ°-, ½É±Ù-
  • myocardial scan
    ½ÉÀå±ÙÀ°½ºÄµ
  • myocardial siderosis
    ½ÉÀå±ÙÀ°Ã¶Ä§ÂøÁõ
  • myocardial stimulant
    ½É±ÙÀÚ±ØÁ¦, °­½ÉÁ¦
  • myocardial perfusion scintigraphy
    ½É±ÙÁÖ»ç¼ú, ½ÉÀå±ÙÀ°¼¶±¤Á¶¿µ¼ú
  • active oxygen
    Ȱµ¿»ê¼Ò, Ȱ¼º»ê¼Ò
  • alveolar oxygen pressure
    ÆóÆ÷»ê¼Ò¾Ð
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  • ¿µ¹®
    ÇѱÛ
  • altered oxygen affinity
    »ê¼Òģȭ¼º º¯¼º
  • alveolar arterial oxygen gradient
    ÆóÆ÷µ¿¸Æ°£ »ê¼ÒºÐ¾ÐÂ÷.
  • alveolar-arterial oxygen difference
    ÆóÆ÷-µ¿¸Æ »ê¼ÒÂ÷ÀÌ
  • alveolar-arterial oxygen gradient
    ÆóÆ÷µ¿¸Æ°£ »ê¼ÒºÐ¾ÐÂ÷
  • arterial blood oxygen tension
    µ¿¸ÆÇ÷»ê¼Ò Àå·Â.
  • arterial oxygen content
    µ¿¸ÆÇ÷»ê¼ÒÇÔÀ¯·®
  • arterial oxygen saturation
    µ¿¸ÆÇ÷ »ê¼ÒÆ÷È­µµ
  • arterial oxygen tension
    µ¿¸ÆÇ÷»ê¼ÒºÐ¾Ð, µ¿¸ÆÇ÷»ê¼ÒÀå·Â
  • arterial oxygen tension
    µ¿¸ÆÇ÷ »ê¼ÒºÐ¾Ð
  • arterial-venous oxygen content difference
    µ¿¸Æ-Á¤¸Æ»ê¼Ò¿ëÀûÂ÷ÀÌ
  • arterial-venous oxygen difference
    µ¿¸Æ-Á¤¸Æ »ê¼ÒÂ÷ÀÌ
  • arteriovenous oxygen content difference
    µ¿Á¤¸ÆÇ÷ »ê¼ÒÇÔ·®Â÷
  • arteriovenous oxygen difference
    µ¿Á¤¸ÆÇ÷ »ê¼Ò±³Â÷(¡­úìß«áÈÎòó¬)
  • arteriovenous oxygen tension difference
    µ¿Á¤¸ÆÇ÷ »ê¼ÒºÐ¾ÐÂ÷
  • heavy oxygen
    Áß»ê¼Ò(ñìß«áÈ).
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  • ¿µ¹®
    ÇѱÛ
  • line of consumption
    ¼Ò¸ð¼±(ËÛËÎËÛ).
  • luxus consumption
    °úµµ¼Ò¸ð(Φöôá¼ÙÄ).
  • mass consumption
    ¹ü¿ë.
  • phthisis =consumption ³ª
    °áÇÙ(Ì¿ú·), ³ë( ).
  • water consumption
    ¹°¼Òºñ·®(ÊÙËÛË×Ëâ), ±Þ¼ö·®(Ë»ËàËâ).
  • anterior (myocardial) infarction
    Àüº®(½É±Ù)°æ»öÁõ.
  • cardiac infarction =myocardial i.
    ½É±Ù°æ»öÁõ(ãýÐÉÌÛßáñø)
  • high lateral myocardial infarction
    È޾߼øÈ¯°íÀ§Ãøº®½É±Ù°æ»öÁõ(ÍÔêÈö°ÛúãýÐÉÌÛßáñø ).
  • idiopathic myocardial hypertrophy
    Ư¹ß¼º ½É±Ùºñ´ëÁõ.
  • lateral myocardial infarction
    Ãøº®½É±Ù°æ»ö(ö°ÛúãýÐÉÌãßá).
  • myocardiac,myocardial
    ½É±ÙÀÇ
  • myocardial
    ½É±Ù(ãýÐÉ)ÀÇ.
  • myocardial anoxia
    ½É±Ù¹«»ê¼ÒÁõ(ãýÐÉÙíß«áÈñø).
  • myocardial biospy
  • myocardial bridging
    ½É±Ù±³¶ô.
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  • ¿µ¹®
    ÇѱÛ
  • oxygen transferase
    "»ê¼Ò Æ®·£½ºÆÛ·¹À̽º, »ê¼ÒÀüÀÌÈ¿¼Ò(ß«áÈï®ì¹ý£áÈ)"
  • oxygen transport
    »ê¼Ò¿î¹Ý(ß«áÈê¡Úæ)
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Qo oxygen consumption
ECG Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ
   = EKG
  1. Conducting System Structu...
AMI acquired monosaccharide intolerance; acute myocardial infarction; amitriptyline; anterior myocardial...
IMI immunologically measurable insulin; impending myocardial infarction; Imperial Mycological Institute ...
MD Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major...
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CMR(O(2)) cerebral metabolic rate of oxygen consumption
FC Feed consumption
NFCS Nationwide Food Consumption Survey
QO(2) O(2) consumption
VO(2) O(2) consumption
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  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • myocardial irritability
    ½É±Ù °ú¹Î¼º, ½É±Ù ÀÚ±Ø ÈïºÐ¼º
  • myocardial lead
    ½É±Ù À¯µµ
  • myocardial necrosis
    ½É±Ù ±«»ç
  • myocardial primordium
    ½ÉÀå ±ÙÀ°Ãþ ¿ø±â
  • myocardial scan
    ½É±Ù ÁÖ»ç
  • myocardial stimulants
    ½É±Ù ÀÚ±ØÁ¦
  • alveolar arterial oxygen gradient
    ÆóÆ÷µ¿¸Æ°£ »ê¼Ò ºÐ¾ÐÂ÷
  • alveolar-arterial oxygen difference
    ÆóÆ÷-µ¿¸Æ »ê¼Ò Â÷ÀÌ
  • biochemical oxygen demand
    »ý¹°È­ÇÐÀû »ê¼Ò ¿ä±¸, »ý¹°È­ÇÐÀû »ê¼Ò ¿ä±¸·®
  • bridging oxygen atom
    °¡±³ »ê¼Ò
    2°³ÀÇ ±Û¶ó½º Çü¼º ¿øÀÚ¸¦ ¿¬°áÇÏ´Â »ê¼Ò.
  • chemical oxygen demand
    È­ÇÐÀû »ê¼Ò ¿ä±¸·®
  • hyperbaric oxygen chamber
    °í¾Ð »ê¼Ò½Ç
    °í¾ÐÀÇ »ê¼Ò¸¦ ÀÌ¿ëÇØ Àú»ê¼ÒÁõÀ» Ä¡·áÇϱâ À§ÇÑ ÀåÄ¡.
  • oxygen
    »ê¼Ò
    Æó¿¡¼­ Ç÷·ù·Î µé¾î°¡ »ýüÀÇ ¸ðµç ¼¼Æ÷¿¡ ¿î¹ÝµÇ´Â °¡½º.
  • oxygen affinity
    »ê¼Ò ģȭ¼º
  • oxygen balance
    »ê¼Ò ÆòÇü
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
myocardial infarct imaging <radiology> Tc-99m pyrophosphate (PYP) 20 mCi, peak abnormality 2-3 days, often falsely negative before 2 days, abnormal for 7-10 days, mechanism: calcium influx into ischemic cells, PYP incorporated into crystalline structure, analogous to hydroxyapatite see: nuclear cardiology
(12 Dec 1998)
myocardial infarction A term used to describe irreversible injury to heart muscle.
Synonym: heart attack.
See: infarction.
Common symptoms include substernal, crushing chest pain that may radiate to the jaw or arms. Chest pains may be associated with nausea, sweating and shortness of breath.
Acronym: MI
(27 Sep 1997)
myocardial infarction in dumbbell form Infarction involving the septum along with both inferior and anterior walls to make an H-or dumbbell-shaped configuration.
Synonym: Roesler-Bressler infarct.
(05 Mar 2000)
myocardial insufficiency A condition where there is ineffective pumping of the heart leading to an accumulation of fluid in the lungs. Typical symptoms include shortness of breath with exertion, difficulty breathing when lying flat and leg or ankle swelling. Causes include chronic hypertension, cardiomyopathy and myocardial infarction.
(27 Sep 1997)
myocardial ischemia A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (coronary arteriosclerosis), to obstruction by a thrombus (coronary thrombosis), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (myocardial infarction).
(12 Dec 1998)
myocardial necrosis Irreversible destruction of myocardial (heart muscle) cells.
(27 Sep 1997)
myocardial perfusion imaging <radiology> (thallium scanning) thallium (Tl) 201, acts as potassium analog, dose 2.0 - 3.0 mCi at peak exercise, 4% of injected dose reaches myocardium, imaging: exercise (1-5 min), redistribution (3-4 hrs), views: anterior, LAO 45', left lateral, interpretation: normal, reversible abnormalitymost likely to be exercise-induced ischemia, nonreversible abnormalitymost likely to be prior myocardial infarction, reverse redistribution most likely to be normal areas wash out faster, lung activity most likely to be LV failure during exercise see also: dipyridamole test, nuclear cardiology
(12 Dec 1998)
myocardial reperfusion Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing myocardial reperfusion injury.
(12 Dec 1998)
myocardial reperfusion injury Functional, metabolic, or structural changes in ischemic heart muscle thought to result from reperfusion to the ischemic areas. Changes can be fatal to muscle cells and may include oedema with explosive cell swelling and disintegration, sarcolemma disruption, fragmentation of mitochondria, contraction band necrosis, enzyme washout, and calcium overload. Other damage may include haemorrhage and ventricular arrhythmias. One possible mechanism of damage is thought to be oxygen free radicals. Treatment currently includes the introduction of scavengers of oxygen free radicals, and injury is thought to be prevented by warm blood cardioplegic infusion prior to reperfusion.
(12 Dec 1998)
myocardial revascularization The restoration of blood supply to the myocardium.
(12 Dec 1998)
myocardial rigor mortis Irreversible contraction of the left ventricle of the heart as a complication seen in the early period of cardiopulmonary bypass and now avoided by appropriate cardioplegic solutions.
Synonym: myocardial rigor mortis, stone heart.
(05 Mar 2000)
myocardial stunning Prolonged dysfunction of the myocardium after a brief episode of severe ischemia, with gradual return of contractile activity. It occurs frequently, both in the experimental laboratory and in clinical medicine. Since stunned myocardium occurs adjacent to necrotic tissue after prolonged coronary occlusion, many myocardial infarcts may be a mixture of necrotic and stunned tissue.
(12 Dec 1998)
posterior myocardial infarction Infarction involving the posterior wall of the heart; also formerly used erroneously of infarction's involving the inferior or diaphragmatic surface of the heart.
(05 Mar 2000)
silent myocardial infarction Infarction that produces none of the characteristic symptoms and signs of myocardial infarction.
(05 Mar 2000)
subendocardial myocardial infarction Infarction that involves only the layer of muscle subjacent to the endocardium.
(05 Mar 2000)
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