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  • neoplasia
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  • prostatic intraepithelial neoplasia
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CIN Cervical Intraepithelial Neoplasia
MEN Multiple Endocrine Neoplasia
  ; AD Trait
  1. MEN Type I(= Wermer Syndro...
CIN central inhibition; cervical intraepithelial neoplasia; chronic interstitial nephritis
CIN1, CIN I cervical intraepithelial neoplasia, grade 1 (mild dysplasia)
CIN 2, CIN II cervical intraepithelial neoplasia, grade 2 (moderate-severe)
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TDLU terminal duct lobular unit
AIN Anal intraepithelial neoplasia
CIN Cervical Intraephithelial Neoplasia
CIN 3 Cervical Intraepithelial Neoplasia grade 3
CIN III Cervical intraepithelial neoplasia grade III
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multiple endocrine neoplasia type 2b A type of multiple endocrine neoplasia occurring as an isolated congenital presentation or as a distinct autosomal dominant disease. It is characterised by the 100% incidence of medullary thyroid carcinoma and frequent pheochromocytomas; patients seldom exhibit hyperparathyroidism. It is distinguished from men 2a by its characteristic physical appearance resulting from numerous neural defects including mucosal neuromas of the eyelids, lips, and tongue. The neural abnormalities also include widespread neurogangliomatosis of the gastrointestinal tract leading to abnormal gut motility. Treatment usually requires total thyroidectomy following evaluation for the presence of pheochromocytomas.
(12 Dec 1998)
prostatic intraepithelial neoplasia A premalignant change arising in the prostatic epithelium, regarded as the most important and most likely precursor of prostatic adenocarcinoma. The neoplasia takes the form of an intra-acinar or ductal proliferation of secretory cells with unequivocal nuclear anaplasia, which corresponds to nuclear grade 2 and 3 invasive prostate cancer.
(12 Dec 1998)
neoplasia <oncology, pathology> Literally new growth, usually refers to abnormal new growth and thus means the same as tumour, which may be benign or malignant.
Unlike hyperplasia, neoplastic proliferation persists even in the absence of the original stimulus.
Origin: Gr. Plassein = to form
(18 Nov 1997)
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