| receptors, vasoactive intestinal peptide | Cell surface proteins that bind vasoactive intestinal peptide (vip) with high affinity and trigger intracellular changes which influence the behaviour of cells. (12 Dec 1998) |
|---|---|
| megacystitis-microcolon-intestinal hypoperistalsis syndrome | <syndrome> A rare condition characterised by abdominal distention, lax abdominal musculature, incomplete intestinal rotation, and deficient intestinal peristalsis. A large bladder and often vesicoureteral reflux are seen. Typically affects female neonates and usually fatal in first year of life. (05 Mar 2000) |
| multiple intestinal polyposis | Begins usually in late childhood; polyps increase in numbers, causing symptoms of chronic colitis, and carcinoma of the colon almost invariably develops in untreated cases; autosomal dominant inheritance. In the Gardner syndrome there are extracolonic changes (desmoid tumours, etc.). Synonym: polyposis coli. Hamartomatous polyposis of the small or large intestine, Peutz-Jeghers syndrome with melanin spots on the lips, less common, miscellaneous, rare, and doubtful occurrences. Synonym: familial intestinal polyposis. (05 Mar 2000) |
| posterior intestinal portal | In young embryos, the communications from the midgut to the hindgut. (05 Mar 2000) |
| src-related intestinal kinase | <enzyme> An intracellular epithelial cell tyrosine kinase; shares 80% homology with human tyrosine kinase brk; has 451 amino acid residues; amino acid sequence given in first source Registry number: EC 2.7.1.- Synonym: sik protein, sik gene product (26 Jun 1999) |
| neuronal intestinal dysplasia | Increased numbers of ganglion cells with myenteric plexus hyperplasia and increased acetylcholinesterase activity in nerves of the mucosa and submucosa. Clinically, neuronal hyperplasia mimics Hirschprung's disease. Similar findings are seen in patients with multiple endocrine neoplasia syndrome, type IIB, and in neurofibromatosis. Synonym: hyperganglionosis, neuronal intestinal dysplasia. (05 Mar 2000) |
| immunoproliferative small intestinal disease | A spectrum of conditions ranging from a benign plasma cell hyperplasia to a highly malignant lymphoma of the small intestine. (12 Dec 1998) |
| intestinal | <anatomy> Pertaining to the intestine. Origin: L. Intestinalis (18 Nov 1997) |
| intestinal 15 kD protein | <chemical> From rat ileum; potent lipid-binding protein; mw 15 kD; amino acid sequence given in first source was found to be highly homologous to gastrotropin and rat liver fatty acid-binding proteins; same size transcript also found in ovary; may be involved in metabolism of steroids Synonym: i-15p (05 Dec 1998) |
| intestinal anastomosis | Establishment of a new communication between two segments of intestine. Synonym: enteroanastomosis, intestinal anastomosis. (05 Mar 2000) |
| intestinal angina | <surgery> A dull, crampy centralised abdominal pain that occurs 15-30 minutes after a meal. Associated with ischaemic bowel disease. Evaluated with mesenteric arteriography. (27 Sep 1997) |
| intestinal anthrax | A usually fatal form of anthrax marked by chill, high fever, pain in the head, back, and extremities, vomiting, bloody diarrhoea, cardiovascular collapse, and frequently haemorrhages from the mucous membranes and in the skin (petechiae). See: mycosis intestinalis. (05 Mar 2000) |
| intestinal arterial arcades | The series of arterial arches formed in the mesentery by anastomoses between adjacent jejunal and ileal arteries and from which vasa recta arise. The arterial arcades of the ileum are shorter and more complex than those of the jejunum. See: arterial arches of ileum, arterial arches of jejunum. Synonym: intermesenteric arterial anastomosis, Riolan's arc, Riolan's arcades. (05 Mar 2000) |
| intestinal arteries | See: ileal arteries, jejunal arteries. (05 Mar 2000) |
| intestinal atresia | Congenital obliteration of the lumen of the intestine, with the ileum involved in 50% of the cases and the jejunum and duodenum following in frequency. It is the most frequent cause of intestinal obstruction in the newborn infant. Its aetiology may be related to failure of recanalization during early development or to some impairment of blood supply during intrauterine life. (12 Dec 1998) |