| BGS | balance, gait, and station; blood group substance; British Geriatrics Society |
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| GT | gait training; galactosyl transferase; gastrostomy; generation time; genetic therapy; gingiva treatm... |
| MASA | Medical Association of South Africa; mental retardation-aphasia-shuffling gait-adducted thumbs [synd... |
| RGO | reciprocating gait orthosis |
| VEGAS | ventricular enlargement with gait apraxia syndrome |
| hysterical gait | A variety of bizarre gaits seen with hysteria-conversion reaction; usually the foot is dragged or pushed ahead, instead of lifted, while walking; frequently the foot is held dorsiflexed and inverted. (05 Mar 2000) |
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| spastic gait | Gait in which the leg is stiff, without flexion at knee and ankle, and with each step is rotated away from the body, then towards it, forming a saemicircle. Synonym: circumduction gait, spastic gait. (05 Mar 2000) |
| steppage gait | A gait in which the advancing foot is lifted higher than usual so that it can clear the ground, because it cannot be dorsiflexed. Seen with peroneal neuropathies and other disorders causing foot dorsiflexion weakness. See: high steppage gait. Synonym: steppage. (05 Mar 2000) |
| toppling gait | <neurology> A gait in which the steps are uncertain and hesitant, and the patient totters and sometimes falls; probably due to a balance disorder; may be seen in elderly patients after a stroke. (06 Mar 2000) |
| equine gait | A gait in which the foot is raised high to avoid catching a drooping foot and brought down suddenly in a flapping manner; often seen in peroneal nerve palsy and tabes. Synonym: equine gait. (05 Mar 2000) |
| festinating gait | Gait in which the trunk is flexed, legs are flexed at the knees and hips, but stiff, while the steps are short and progressively more rapid; characteristically seen with parkinsonism and other neurologic diseases. Synonym: festination. (05 Mar 2000) |
| acute intermittent porphyria | <gastroenterology, haematology> A group of rare inherited metabolic disorders that result from a disturbance in porphyrin metabolism, causing increased formation and excretion of porphyrin or its precursors. It is caused by hepatic overproduction of d-aminolevulinic acid, which has greatly increased urinary excretion and of porphobilinogen, and some increase of uroporphyrin, due to a deficiency of porphobilinogen deaminase. Clinical features: intermittent acute attacks of hypertension, abdominal colic, psychosis, and polyneuropathy, but with no photosensitivity. It is exacerbated by the ingestion of certain drugs such as; barbiturates). Inheritance: autosomal dominant. (20 Sep 2002) |
| Charcot's intermittent fever | Fever, chills, right upper quadrant pain, and jaundice associated with intermittently obstructing common duct stones. (05 Mar 2000) |
| porphyria, acute intermittent | A form of hepatic porphyria (porphyria, hepatic) characterised by periodic attacks of gastrointestinal disturbances, abdominal colic, paralyses, and psychiatric disorders. The onset of this condition is usually in the third or fourth decade of life. (12 Dec 1998) |
| hepatic intermittent fever | Ague-like paroxysms of fever occurring in cases of one or more stones in the common bile duct. (05 Mar 2000) |
| spontaneous intermittent mandatory ventilation | Intermittent mandatory ventilation spontaneously initiated by the patient, to increase tidal volume, and subsequently synchronised with patient's respiratory cycle. Synonym: synchronised intermittent mandatory ventilation. (05 Mar 2000) |
| synchronised intermittent mandatory ventilation | Intermittent mandatory ventilation spontaneously initiated by the patient, to increase tidal volume, and subsequently synchronised with patient's respiratory cycle. Synonym: synchronised intermittent mandatory ventilation. (05 Mar 2000) |
| intermittent | Occurring at separated intervals, having periods of cessation of activity. (18 Nov 1997) |
| intermittent acute porphyria | <gastroenterology, haematology> A group of rare inherited metabolic disorders that result from a disturbance in porphyrin metabolism, causing increased formation and excretion of porphyrin or its precursors. It is caused by hepatic overproduction of d-aminolevulinic acid, which has greatly increased urinary excretion and of porphobilinogen, and some increase of uroporphyrin, due to a deficiency of porphobilinogen deaminase. Clinical features: intermittent acute attacks of hypertension, abdominal colic, psychosis, and polyneuropathy, but with no photosensitivity. It is exacerbated by the ingestion of certain drugs such as; barbiturates). Inheritance: autosomal dominant. (20 Sep 2002) |
| intermittent albuminuria | <nephrology> Functional albuminuria occurring at intervals, such as cyclic albuminuria or albuminuria of athletes. (05 Mar 2000) |