| 영문 | granulation tissue | 한글 | 육아조직 |
|---|---|---|---|
| 설명 | 모세혈관이 풍부하며 왕성하게 증식을 계속하는 어린 결합조직. 창상 등 조직 결손에 대한 수복, 이물처리의 기질화, 염증이 만성적인 경과에 있거나 종양증식에 동반된 사이물질의 반응성 염증에서 관찰된다. 구성성분은 생긴지 얼마 안되는 어린 육아조직은 섬유모세포의 증식, 새로 생긴 모세혈관과 여러 유주세포 및 다른 중간엽세포(백혈구, 림프구, 형질세포, 조직구, 단핵구, 거대세포)등으로 구성된다. 이것이 시간이 지나 그리되면, 모세혈관과 유주세로, 다른조직성분을 감소시켜 만성화하여 오래된 육아가 되며 결국은 섬유세포와 아교질섬유로 구성된 반흔조직으로 변한다. |
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| 영문 | tissue | 한글 | 조직 |
|---|---|---|---|
| 설명 | 특정 구조와 기능을 갖는 세포 집단. 세포 사이에는 다소간 세포간질이 들어 있다. 세포간질에는 글리코사미노글리칸, 히드록시아파타이트와 같은 기질과 아교질섬유와 같은 섬유가 발견된다. 조직성상은 구성세포와 세포간질의 종류와 양에 의해 결정된다. 조직은 상피조직, 지지조직, 근육조직, 신경조직으로 대별되며, 상피조직은 세포간질을 거의 갖지 않으며, 지지조직은 결합조직이나 뼈조직과 같이 세포간질이 풍부한 것이 많다. |
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| 영문 | tissue biopsy | 한글 | 조직생검 |
|---|---|---|---|
| 설명 | 환자의 병적인 조직의 일부를 떼어 내서 병리조직표본을 만들어서 현미경적으로 검사하는 것. 외과적 수술재료로부터 병리조직학적 검사를 하여 생체내의 병변을 추구하는 분야를 외과병리학이라고 한다. 생검에는 쐐기생검, 천자침생검, 꿰뚫기식생검, 내시경생검 외에 세포진이라고 불리우는 박리세포진이나 세척세포진 등 떨어진 세포를 도말검사하는 방법이 포함된다. |
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| 영문 | target tissue | 한글 | 표적조직 |
|---|---|---|---|
| 설명 | 호르몬에 대하여 표적이 되는 세포라는 의미로서 그 호르몬에 대한 수용체를 가지고 있는 세포. 예를 들어 인슐린은 인슐린 수용체를 가진 세포를 표적세포로 하여 이 세포에만 작용한다. |
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| 영문 | subcutaneous tissue, subcutis | 한글 | 피하조직 |
|---|---|---|---|
| 설명 | 피부에서 진피의 아랫부분을 말함. 여기에는 지방조직이 많다. 기능은 열의 격리, 충격흡수, 영양저장소 등이다. |
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| CT | calcitonin; calf testis; cardiac tamponade; cardiothoracic [ratio]; carotid tracing; carpal tunnel; ... |
|---|---|
| CTAP | computed tomography in arterial portography; connective tissue activating peptide |
| CTD | carpal tunnel decompression; chest tube drainage; congenital thymic dysplasia; connective tissue dis... |
| CTMC | connective tissue mast cell |
| DFECT | dense fibroelastic connective tissue |
lentiglobus
| adrenal hyperplasia | <pathology> A condition of diffuse enlargement of the adrenal glands. Origin: Gr. Plassein = to form (27 Sep 1997) |
|---|---|
| adrenal hyperplasia, congenital | A group of inherited disorders of adrenal steroidogenesis, the physical expression of which varies with the sex of the patient, the severity of the congenital enzyme defect, and the age at which the defect makes its presence felt. The most common form, the simple virilizing form, is due to a 21-hydroxylase deficiency. There is also a salt-losing form (a more complete 21-hydroxylase deficiency), a hypertensive form (11-hydroxylase deficiency), a 17-hydroxylase deficiency form, a desmolase deficiency form, and a 3-beta-hydroxysteroid deficiency form. (12 Dec 1998) |
| angiofollicular mediastinal lymph node hyperplasia | Solitary masses of lymphoid tissue containing concentric perivascular aggregates of lymphocytes, occurring usually in the mediastinum or hilar region of young adults; similar changes have been reported outside the mediastinum and, if associated with interfollicular sheets of plasma cells, may progress to lymphoma or plasmacytoma. Synonym: angiofollicular mediastinal lymph node hyperplasia, Castleman's disease. (05 Mar 2000) |
| angiolymphoid hyperplasia with eosinophilia | Solitary or multiple benign cutaneous nodules comprised of immature and mature vascular structures intermingled with endothelial cells and a varied infiltrate of eosinophils, histiocytes, lymphocytes, and mast cells. (12 Dec 1998) |
| atypical melanocytic hyperplasia | Proliferation of melanocytes showing nuclear atypicality, especially as scattered single cells high in the epidermis; interpreted by some pathologists as malignant melanoma in situ. (05 Mar 2000) |
| basal cell hyperplasia | Increase in the number of cells in an epithelium resembling the basal cells. (05 Mar 2000) |
| benign giant lymph node hyperplasia | Solitary masses of lymphoid tissue containing concentric perivascular aggregates of lymphocytes, occurring usually in the mediastinum or hilar region of young adults; similar changes have been reported outside the mediastinum and, if associated with interfollicular sheets of plasma cells, may progress to lymphoma or plasmacytoma. Synonym: angiofollicular mediastinal lymph node hyperplasia, Castleman's disease. (05 Mar 2000) |
| benign prostatic hyperplasia | <urology> A benign enlargement of the prostate gland begins normally after age 50 years probably secondary to the effects of male hormones. If significant enlargement occurs, it may pinch off te urethra making urination difficult or impossible. See: urinary retention. Origin: Gr. Plassein = to form (06 Aug 1998) |
| verrucous hyperplasia | A non-invasive precursor of verrucous or squamous carcinoma of the oral mucosa, occurring in the elderly, characterised by sharp or blunt upward papillary projections of squamous epithelium. (05 Mar 2000) |
| giant lymph node hyperplasia | Large benign, hyperplastic lymph nodes. The more common hyaline vascular subtype is characterised by small hyaline vascular follicles and interfollicular capillary proliferations. Plasma cells are often present and represent another subtype with the plasma cells containing IgM and IgA. (12 Dec 1998) |
| gingival hyperplasia | Non-inflammatory enlargement of the gingivae produced by factors other than local irritation. It is characteristically due to an increase in the number of cells. (12 Dec 1998) |
| cementum hyperplasia | A regressive change of teeth characterised by excessive development of secondary cementum on the tooth surface. It may occur on any part of the root, but the apical two-thirds are most commonly affected. (12 Dec 1998) |
| congenital adrenal hyperplasia | <endocrinology> A genetic disorder present at birth characterised by a deficiency of the hormones aldosterone and cortisol and an overproduction of male sex hormones (androgens). In males this may manifest as enlarged penis, small testes and early development of masculine characteristics. In females features include ambiguous genitalia, failure to menstruate, deep voice and excessive hair. Origin: Gr. Plassein = to form (27 Sep 1997) |
| congenital sebaceous hyperplasia | Misnomer for nevus sebaceus. (05 Mar 2000) |
| congenital virilizing adrenal hyperplasia | A series of inherited inborn errors of metabolism with hyperplasia of the adrenal cortex and overproduction of virilizing hormones. Most common forms are due to partial or complete 21-hydroxylase deficiency, leading to increased ACTH production by the pituitary, stimulating adrenal growth and function. Severe form is characterised by salt-losing state. (05 Mar 2000) |