| colocutaneous fistula | A fistula between the colon and the skin. (05 Mar 2000) |
|---|---|
| colocynth | The peeled dried fruit of Citrullus colcynthis (family Cucurbitaceae), an herb of the sandy shores of the Mediterranean, resembling somewhat the watermelon plant; formerly widely used as a cathartic and laxative. Synonym: bitter apple. Origin: G. Kolokynthe, the round gourd or pumpkin (05 Mar 2000) |
| colocystoplasty | Enlargement of the urinary bladder by attaching a segment of colon to it. (05 Mar 2000) |
| coloenteritis | <pathology> Inflammation involving both the small intestine and the colon. See: enteritis. (18 Nov 1997) |
| colohepatopexy | Attachment of the colon to the liver by adhesions. Origin: colo-+ G. Hepar (hepat-), liver, + pexis, fixation (05 Mar 2000) |
| coloileal fistula | A fistula between the colon and the ileum. (05 Mar 2000) |
| cololysis | Procedure of freeing the colon from adhesions. Origin: colo-+ G. Lysis, loosening (05 Mar 2000) |
| colominic acid | Polymer of a(1,5)-N-acetylneuraminic acid; found in Escherichia coli. (05 Mar 2000) |
| colon | <anatomy> Also called the large intestine. This structure has 6 major divisions: caecum, ascending colon, transverse colon, descending colon, sigmoid colon and rectum. The total length is approximately 5 feet in the adult and it is responsible for forming, storing and expelling waste matter. (27 Sep 1997) |
| colon ascendens | <anatomy> The first part of the colon (large intestine) that starts in the right lower quadrant of the abdomen and ends at the transverse colon in the right upper quadrant of the abdomen. (27 Sep 1997) |
| colon bacillus | <bacteria> The archetypal bacterium for biochemists, used very extensively in experimental work. A rod shaped gram-negative bacillus (0.5 x 3-5 m) abundant in the large intestine (colon) of mammals. Abbreviation: E. Coli (18 Nov 1997) |
| colon cancer | <oncology> A malignancy that arises from the lining of either the colon or the rectum. Cancers of the large intestine are the second most common form of cancer found in males and females. Symptoms include rectal bleeding, occult blood in stools, bowel obstruction and weight loss. Treatment is based largely on the extent of cancer penetration into the intestinal wall. Surgical cures are possible if the malignancy is confined to the intestine. Risk can be reduced when following a diet which is low in fat and high in fibre. (27 Sep 1997) |
| colon cancer and polyps | Benign tumours of the large intestine are called polyps. Malignant tumours of the large intestine are called cancers. Benign polyps do not invade nearby tissue or spread to other parts of the body. Benign polyps can be easily removed during colonoscopy, and are not life threatening. If benign polyps are not removed from the large intestine, they can become malignant (cancerous) over time. most of the cancers of the large intestine are believed to have developed from polyps. (12 Dec 1998) |
| colon cancer, family history of | Colorectal cancer can run in families. The colon cancer risk is higher if an immediate (first-degree) family member (parents, siblings or children) had colorectal cancer and even higher if more than one such relative had colorectal cancer or if a family member developed the cancer at young age (younger than 55 years). Under any of these circumstances, individuals are recommended to undergo a colonoscopy every three years starting at an age that is 7-10 years younger than when the youngest family member with the cancer wasdiagnosed. For example, if a parent had colon cancer diagnosed at age 50, colonoscopy should start in that person's children at 40-43 years of age. (12 Dec 1998) |
| colon carcinoma | <radiology> Risk factors: colonic adenoma, 93% of colorectal CA arises from adenomatous polyps, 5% of adenomas 5mm in size develop into carcinoma, family history and polyposis syndromes, chronic ulcerative colitis, prominent lymphoid follicular pattern, history of endometrial and breast carcinoma, metastasis: liver (25%); retroperitoneal/mesenteric nodes (15%); hydronephrosis (13%); adrenal (10%); ovary; psoas muscle; ascites, risk of: 1% for synchronous colon carcinoma, 3% for metachronous colon CA, 3.8% for extracolonic malignancy, Dukes A: bowel wall; B: serosa/mesentery; C: lymph nodes; D: metastasis (12 Dec 1998) |