| renal tubular acidosis | <nephrology> A rare sometimes familial disorder of the renal tubule characterised by the inability to excrete urine of normal acidity. This leads to a hyperchloraemic acidosis which is often associated with one or more secondary complications such as hypercalcinuria with nephrolithiasis and nephrocalcinosis, rickets, or osteomalacia and severe potassium depletion. (25 Jun 1999) |
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| respiratory acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly resulting in an excess of carbon dioxide in the body. (25 Jun 1999) |
| metabolic acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly. (27 Jun 1999) |
| compensated acidosis | An acidosis in which the pH of body fluids is normal; compensation is achieved by respiratory or renal mechanisms. (05 Mar 2000) |
| compensated respiratory acidosis | Retention of bicarbonate by the renal tubules to minimise the effect on the pH of the blood of retention of carbon dioxide by the lungs, such as occurs with hypoventilation. (05 Mar 2000) |
| primary renal tubular acidosis | A metabolic defect in the mechanism of urinary acidification that may be either the transient type, with onset in infancy, or the persistent type, with onset in childhood or adult years; both types are familial. (05 Mar 2000) |
| secondary renal tubular acidosis | Renal tubular acidosis that may occur as a complication of hypercalcaemic states, hyperglobulinaemic disorders, and in some other chronic renal conditions; a regular component of De Toni-Fanconi syndrome. (05 Mar 2000) |
| hyperchloraemic acidosis | <nephrology> A rare sometimes familial disorder of the renal tubule characterised by the inability to excrete urine of normal acidity. This leads to a hyperchloraemic acidosis which is often associated with one or more secondary complications such as hypercalcinuria with nephrolithiasis and nephrocalcinosis, rickets, or osteomalacia and severe potassium depletion. (25 Jun 1999) |
| starvation acidosis | Ketoacidosis resulting from lack of food intake, leading to fat catabolism to provide energy, releasing acidic ketone bodies. (05 Mar 2000) |
| diabetic acidosis | Decreased pH and bicarbonate concentration in the body fluids caused by accumulation of ketone bodies in diabetes mellitus. (05 Mar 2000) |
| osteopetrosis with renal tubular acidosis | <syndrome> An inherited deficiency of carbonic anhydrase II that results in osteopetrosis and metabolic acidosis. Synonym: osteopetrosis with renal tubular acidosis. (05 Mar 2000) |
| uncompensated acidosis | An acidosis in which the pH of body fluids is subnormal, because restoration of normal acid-base balance is not possible or has not yet been achieved. (05 Mar 2000) |
| lactic acidosis | <biochemistry, physiology> Acidosis caused by accumulation of lactic acid more rapidly than it can be metabolised. It may occur spontaneously or in association with diseases such as diabetes mellitus, leukaemia, or liver failure. (25 Jun 1999) |
| colloidal silicon dioxide | A submicroscopic fumed silica prepared by the vapor-phase hydrolysis of a silicon compound; used as a tablet diluent and as a suspending and thickening agent. (05 Mar 2000) |
| hydrogen dioxide | <chemical> Hydrogen peroxide is produced by vertebrate phagocytes and is used in bacterial killing (the myeloperoxidase halide system). (05 May 1997) |