| ¿µ¹® | fibrinoid necrosis | ÇÑ±Û | ¼¶À¯¼Ò¼º±«»ç |
|---|---|---|---|
| ¼³¸í | ÃʱâÀÇ ·ù¸¶Æ¼½º ȤÀº ¾Ç¼ºÁ¾¾ç µî¿¡¼ º¼ ¼ö ÀÖ´Â Ç÷°ü-°áÇÕÁ¶Á÷ÀÇ Æ¯À¯ÇÑ º¯È·Î Ç÷°üº®À̳ª ÁÖÀ§ÀÇ °áÇÕÁ¶Á÷ÀÌ ¼¶À¯¼Ò¸ð¾çÀ¸·Î ±ÕÁúÇÑ ¹°Áú·Î µÇ°í, È£»ê¼ºÀ¸·Î ¿°»öµÈ´Ù. À̰ÍÀº ¼¶À¯¼Ò¿Í °ÅÀÇ ¶È°°Àº ¿°»ö¼ºÀ» ³ªÅ¸³»±â ¶§¹®¿¡ ¿°Áõ¿¡¼ Ç÷°üÀ¸·ÎºÎÅÍ ¼¶À¯¼Ò ¶Ç´Â ¼¶À¯¼Ò¸ð¾çÀÇ ¹°ÁúÀÌ »ïÃâÇÏ¿© °áÇÕÁ¶Á÷ ¼¶À¯°£¿¡ Ä§ÂøÇÏ¿© ±¸Á¶°¡ ºÒ¸í·áÇÑ °ÍÀ¸·Î »ý°¢µÇ¾úÀ¸³ª, ÃÖ±Ù Á¶Á÷ÈÇÐ, ÀüÀÚÇö¹Ì°æ µî¿¡ ÀÇÇÏ¿© °áÇÕ¼¶À¯ÀÚü¿¡µµ º¯È°¡ ÀÖ´Â °ÍÀ¸·Î ¸é¿ª±Û·ÎºÒ¸°ÀÇ Ä§Âøµµ Áõ¸íµÇ°í ÀÖ´Ù. |
||
| ¿µ¹® | coagulation necrosis | ÇÑ±Û | ÀÀ°í±«»ç |
|---|---|---|---|
| ¼³¸í | ¼¼Æ÷ÀÇ Á×À½À» ³ªÅ¸³»´Â ÇüÅÂÇÐÀû º¯È·Î¼ ±«»çºÎÀ§°¡ ÀÀ°íÇÑ »óÅ·ΠÀÖ´Â °Í. ±«»ç¼¼Æ÷´Â È£»ê¼º µ¢¾î¸®·Î º¸À̸ç Àû¾îµµ ¼öÀϰ£ ¼¼Æ÷ÀÇ ±âº» À±°ûÀÌ º¸Á¸µÇ±â ¶§¹®¿¡ Á¶Á÷±¸Á¶¸¦ ½Äº°ÇÒ ¼ö ÀÖ´Ù. ´ëÇ¥ÀûÀ¸·Î ÇãÇ÷±«»ç¸¦ µé ¼ö ÀÖ´Ù. °æ»ö°ú ÇãÇ÷¿¡ ÀÇÇÑ Àú»ê¼ÒÁõ½Ã ½ÉÀå±ÙÀ̳ª ÄáÆÏ¿¡¼ °üÂûµÇ°Å³ª Àü±â ¹× ¼öÀºÁßµ¶½Ã ÄáÆÏÀÇ ¿ä¼¼°ü¿¡¼ °üÂûµÇ´Â °ÍÀ¸·Î ´Ü¹éÀÇ º¯¼º ÈÄ ³²¾ÆÀֱ⠶§¹®ÀÌ´Ù. ¼¼Æ÷ÀÇ À±°ûÀÌ ³²¾Æ ÀÖ¾î ¿ø·¡ Á¶Á÷ÀÇ ±¸Á¶¸¦ ÇüÅÂÇÐÀûÀ¸·Î ÀνÄÇÒ ¼ö ÀÖ´Â °ÍÀÌ Æ¯Â¡ÀÌ´Ù. |
||
| ¿µ¹® | caseation necrosis | ÇÑ±Û | Ä¡Á»ç, °Ç¶ô±«»ç |
|---|---|---|---|
| ¼³¸í | Á¶Á÷ÀÌ Ä¡Áî¿Í °°ÀÌ °ÇÁ¶Çϰí, ÇüŰ¡ ¾ø´Â µ¢¾î¸®·Î º¯ÈÇÏ´Â °ÍÀ¸·Î °áÇÙ°¨¿° º´ÅÍ¿¡¼ º¼ ¼ö ÀÖ´Â µ¶Æ¯ÇÑ ±«»çÀÇ ÇϳªÀÌ´Ù. |
||
| ¿µ¹® | acute hepatitis | ÇÑ±Û | ±Þ¼º°£¿° |
|---|---|---|---|
| ¼³¸í | ¹ÙÀÌ·¯½º¿¡ ÀÇÇØ °£¿¡ »ý±â´Â ±Þ¼º¿°Áõ. ±Þ¼º°£¿°À̶õ °£¿°¹ÙÀÌ·¯½º(AÇü-BÇü-ºñAºñBÇü)¿¡ ÀÇÇØ¼ °£¿¡ »ý±â´Â ±Þ¼º¿°ÁõÀ» º´¸íÀ¸·Î À̸£´Â ¸»·Î, ÀÌ´Â ±× °¨¿°¾ç½Ä¿¡ ¼öÇ÷ ÈÄ¿¡ ¹ß»ýÇÏ´Â ¼öÇ÷ÈÄ °£¿°°ú, °¨¿°°æ·Î¸¦ ¾Ë ¼ö ¾ø´Â »ê¹ß¼º°£¿° ¹× Áý´ÜÀ¸·Î ¹ß»ýÇÏ´Â À¯Ç༱°£¿°ÀÇ ¼¼°¡Áö À¯ÇüÀ¸·Î ³ª´ ¼ö ÀÖ´Ù. ¼öÇ÷ÈÄ °£¿°Àº ±× 95%°¡ ºñAºñBÇü°£¿°ÀÌ¸ç ³ª¸ÓÁö°¡ BÇü °£¿°ÀÌ´Ù. »ê¹ß¼º °£¿°Àº AÇü °£¿°°ú BÇü °£¿°ÀÌ °¢°¢ 30%¸¦ ÀÌ·ç°í ³ª¸ÓÁö 40%´Â ºñAºñB°£¿°ÀÌ´Ù. Áý´ÜÀ¸·Î ¹ß»ýÇÏ´Â À¯Ç༺°£¿°Àº °ÅÀǰ¡ AÇü°£¿°ÀÌÁö¸¸ ¶§·Î´Â ¿©±â¿¡ Æ÷ÇÔµÇÁö ¾ÊÀº ÇüÀÇ °£¿°ÀÏ °æ¿ìµµ ÀÖ´Ù. ±Þ¼º°£¿°ÀÇ Áõ¼¼´Â ¸ÕÀú ¸öÀÌ ³ª¸¥ÇØÁö°í ¿Â¸ö¿¡ ±ÇۨÀÌ Ã£¾Æ¿À¸ç Á¶±×¸¶ÇÑ ÀÏ¿¡µµ °ð ÇǷθ¦ ´À³¢°Ô µÈ´Ù. ±×¸®°í ½Ä¿åºÎÁø-¹ß¿-±¸ÅäÁõ-º¹Åë-¼³»ç µî, °¨±â³ª ±Þ¼ºÀ§Àå¿°¿¡ °É·ÈÀ» ¶§¿Í °°Àº Áõ¼¼ µîÀÌ ³ªÅ¸³´Ù. µÚÀ̾î Ȳ´ÞÁõ¼¼¸¦ º¸À̴µ¥, À̶§´Â ÃʱâÀÇ Áõ¼¼°¡ ¾à°£ °¡º¿öÁø °Íó·³ ´À²¸Áö´Â °ÍÀÌ º¸ÅëÀÌ´Ù. ±×·¯³ª Ȳ´ÞÁõ¼¼°¡ ½ÉÇØÁö°í ÃʱâÀÇ Áõ¼¼µéÀÌ ´Ù½Ã ÁøÇàµÇ¸é À̶§´Â Àü°Ý¼º°£¿°ÀÌ µÉ À§ÇèÀÌ ÀÖ´Ù. °£¿° Áõ¼¼°¡ ½ÉÇÏÁö ¾Ê¾ÒÀ» °æ¿ì´Â Ȳ´ÞÀÌ ´«¿¡ ¶çÁö ¾ÊÀº °æ¿ìµµ Àִµ¥ À̶§´Â ÁøÂûÀ» ÇØµµ °¨±â³ª ±Þ¼ºÀ§Ã¢ÀÚ¿°À¸·Î ÀÚÄ© ¿ÀÁøµÇ±â ½±´Ù. ¶Ç AÇü°£¿°Àº ¿ÀÌ 38~39¡É±îÁö ¿À¸£°í Áõ¼¼°¡ °©Àڱ⠳ªÅ¸³ª´Â °ÍÀÌ Æ¯Â¡ÀÌ¸ç ±Þ¼ººñAºñBÇü°£¿°Àº Áõ¼¼°¡ ºñ±³Àû °¡º¿î °ÍÀÌ Æ¯Â¡ÀÌ´Ù. ±Þ¼ºBÇü°£¿°ÀÇ Áõ¼¼´Â AÇü°£¿°°ú ±Þ¼ººñAºñBÇü°£¿°ÀÇ Áß°£ Á¤µµÀÎ °ÍÀÌ º¸ÅëÀÌ´Ù. |
||
| ¿µ¹® | acute appendicitis | ÇÑ±Û | ±Þ¼º¸·Ã¢ÀÚ²¿¸®¿° |
|---|---|---|---|
| ¼³¸í | ¿Ü°úÀû óġ¸¦ ¿äÇÏ´Â ¸·Ã¢ÀÚ²¿¸®(Ãæ¼ö)ÀÇ ±Þ¼º¿°ÁõÀ¸·Î¼, º¸Åë ÇϺ¹ºÎÀÇ ¿À¸¥ÂÊ 1/4 ºÎÀ§¿¡¼ÀÇ ÅëÁõÀÌ Æ¯Â¡À̸ç, ±¹¼Ò¾ÐÅë, ±ÙÀ°±äÀå ÇǺΰ¨°¢ÀÇ °ú¹Î µîÀ» ¼ö¹ÝÇÑ´Ù. ÀϹݵòµéÀÌ ¡°¸ÍÀå¿°¡±À̶ó°í ÇÏ´Â °ÍÀ¸·Î ¸ÍÀå¿°Àº ¸·Ã¢ÀÚÀÇ ¿°ÁõÀ¸·Î ±¸º°µÇ¾î¾ß ÇÑ´Ù. ¹ß¿°ú ´ÙÇü¹éÇ÷±¸Áõ´Ù´Â ±¹¼Ò°¨¿°ÀÇ °á°úÀÌ´Ù. ¸·Ã¢ÀÚ²¿¸®ÀÇ À§Ä¡-À¯Âø»óÅÂ-²¿ÀÓ µî¿¡ ÀÇÇØ Áõ»ó°ú ¡ÈÄ´Â º¯µ¿µÈ´Ù. |
||
| MHN | massive hepatic necrosis; Mohs hardness number; morbus hemolyticus neonatorum |
|---|---|
| AP | accessory pathway; accounts payable; acid phosphatase; acinar parenchyma; action potential; active p... |
| ALF | acute liver failure; American Liver Foundation; assisted living facilities |
| AN | acanthosis nigricans; acne neonatorum; acoustic neuroma; adult, normal; ala nasi; amyl nitrate; aneu... |
| SHN | spontaneous hemorrhagic necrosis; subacute hepatic necrosis |
| p60 tumour necrosis factor receptor-associated kinase | <enzyme> Interacts with and causes phosphorylation of the cytoplasmic domain of the tnf receptor Registry number: EC 2.7.10.- Synonym: p60 tnf receptor-associated kinase, p60-trak (26 Jun 1999) |
|---|---|
| papillary necrosis | <radiology> Mnemonic: POST CARD, P pyelonephritis, O obstruction, S sickle cell disease, T TB, C cirrhosis (EtOH), A analgesics (phenacetin and aspirin), R renal vein thrombosis, D diabetes (12 Dec 1998) |
| receptors, tumour necrosis factor | Cell surface receptors that bind tumour necrosis factor and trigger changes which influence the behaviour of cells. The two recognised tumour necrosis factor receptors are designated alpha and beta receptors. Both receptors bind both alpha and beta tumour necrosis factors with high affinity, and both are members of the nerve growth factor receptor family. (12 Dec 1998) |
| caseous necrosis | Caseation necrosis, necrosis characteristic of certain inflammations (e.g., tuberculosis, histoplasmosis), which represents necrosis with loss of separate structures of the various cellular and histologic elements; affected tissue manifests the friable, crumbly consistency and dull, opaque quality observed in cheese. Synonym: caseous degeneration. (05 Mar 2000) |
| globus pallidus necrosis | <radiology> Carbon monoxide poisoning, barbiturate intoxication, cyanide poisoning, hydrogen sulfide poisoning, hypoglycaemia, hypoxia, hypotension, Leigh disease, Wilson disease CT: bilateral, symmetric low density in globus pallidus Cf: methanol intoxication leading to putamenal haemorrhagic necrosis (changes seen more laterally) (12 Dec 1998) |
| renal papillary necrosis | <nephrology> Necrosis of renal papillae, occurring in acute pyelonephritis, especially in diabetics, or in analgesic nephropathy; renal failure may result. Synonym: necrotizing papillitis. (05 Mar 2000) |
| central necrosis | Necrosis involving the deeper or inner portions of a tissue, or an organ or its units. (05 Mar 2000) |
| cerebrocortical necrosis | A noninfectious disease of ruminants characterised by a tissue-thiamine deficiency and by amaurosis and strabismus, followed by recumbency, opisthotonos, and convulsions. Synonym: cerebrocortical necrosis. (05 Mar 2000) |
| coagulation necrosis | A type of necrosis in which the affected cells or tissue are converted into a dry, dull, fairly homogeneous eosinophilic mass without nuclear staining, as a result of the coagulation of protein as occurs in an infarct; microscopically, the necrotic process involves chiefly the cells, and remnants of histologic elements (e.g., elastin, collagen, muscle fibres) may be recognizable, as well as "ghosts" of cells and portions of cell membranes; may be caused by heat, ischemia, and other agents that destroy tissue, including enzymes that would continue to alter the devitalised cellular substance. (05 Mar 2000) |
| colliquative necrosis | An obsolete term for liquefactive necrosis. (05 Mar 2000) |
| mummification necrosis | A form of gangrene in which the involved part is dry and shriveled. Synonym: cold gangrene, mummification necrosis, mummification. (05 Mar 2000) |
| contraction band necrosis | A microscopic change in myocardial cells in which excessive contraction, associated with elevated intracellular calcium and serum norepinephrine, causes the formation of transverse amorphous band's in the fibres which are then incapable of contracting again. Synonym: contraction band necrosis. (05 Mar 2000) |
| myocardial necrosis | Irreversible destruction of myocardial (heart muscle) cells. (27 Sep 1997) |
| postpartum pituitary necrosis syndrome | <syndrome> Hypopituitarism arising from a severe circulatory collapse postpartum, with resultant pituitary necrosis. Synonym: postpartum pituitary necrosis syndrome, thyrohypophysial syndrome. (05 Mar 2000) |
| cystic medial necrosis | Loss of elastic and muscle fibres in the aortic media, with accumulation of mucopolysaccharide, sometimes in cystlike spaces between the fibres; a disease of unknown cause, which may be inherited and which predisposes to dissecting aneurysms. Synonym: Erdheim disease, medionecrosis aortae idiopathica cystica, medionecrosis of the aorta, mucoid medial degeneration. (05 Mar 2000) |
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|