| 영문 | severe acute respiratory syndrome(SARS) | 한글 | 사스 |
|---|---|---|---|
| 설명 | 중국 광동 지역에서 가장 먼저 발생한 전염성 호흡기 질환으로 세계보건기구(WHO)에서 ‘중증급성호흡증후군(SARS)'으로 명명했다. 섭씨 38도 이상의 고열과 기침, 호흡곤란, 저산소증, X선상의 폐렴증상 중 하나 이상의 증상이 나타나며, 두통, 근육통, 식욕부진, 피로감, 발진, 설사를 동반할 수 있다. 초기 증상은 감기와 비슷하지만 폐렴으로 발전하면 치명적일 수 있다. 현재 밝혀진 감염경로는 환자가 재채기나 기침할 때 내뿜는 침방울이고, 이것이 다른 사람의 호흡기로 들어갈 때 전염된다. 침방울이 전달되는 거리는 보통 1m로 보고 있다. 공기를 통해 전염이 가능하다는 주장이 제기됐지만 아직 확인되지 않았다. 원인균은 변종 코로나바이러스로 밝혀졌다. |
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| 영문 | severe acute respiratory syndrome(SARS) | 한글 | 중증급성호흡증후군 |
|---|---|---|---|
| 설명 | 중국 광동 지역에서 가장 먼저 발생한 전염성호흡기병으로 세계보건기구(WHO)에서 ‘중증급성호흡증후군(SARS)'으로 명명했다. 섭씨 38도 이상의 고열과 기침, 호흡곤란, 저산소증, X선상의 폐렴증상 중 하나 이상의 증상이 나타나며, 두통, 근육통, 식욕부진, 피로감, 발진, 설사를 동반할 수 있다. 초기 증상은 감기와 비슷하지만 폐렴으로 발전하면 치명적일 수 있다. 현재 밝혀진 감염경로는 환자가 재채기나 기침할 때 내뿜는 침방울이고, 이것이 다른 사람의 호흡기로 들어갈 때 전염된다. 침방울이 전달되는 거리는 보통 1m로 보고 있다. 공기를 통해 전염이 가능하다는 주장이 제기됐지만 아직 확인되지 않았다. 원인균은 변종 코로나바이러스로 밝혀졌다. |
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| ACI | acceleration index; acoustic comfort index; acute cardiac ischemia; acute coronary infarction; acute... |
|---|---|
| AIE | acute inclusion-body encephalitis; acute infectious encephalitis; acute infective endocarditis |
| AIP | acute idiopathic pericarditis; acute infectious polyneuritis; acute intermittent porphyria; aldoster... |
| ARF | acute renal failure; acute respiratory failure; acute rheumatic fever; Addiction Research Foundation... |
| AGN | acute glomerulonephritis; agnosia |
| glomerulonephritis | <nephrology> A variety of nephritis characterised by inflammation of the capillary loops in the glomeruli of the kidney. It occurs in acute, subacute and chronic forms and may be secondary to haemolytic streptococcal infection. Evidence also supports possible immune or autoimmune mechanisms. (18 Nov 1997) |
|---|---|
| glomerulonephritis, IgA | Chronic form of glomerulonephritis characterised by recurring haematuria with only slight proteinuria and by deposits of IgA immunoglobulin in the mesangial areas of the renal glomeruli. It usually occurs in young males. (12 Dec 1998) |
| glomerulonephritis, membranoproliferative | Chronic glomerulonephritis characterised histologically by proliferation of mesangial cells, increase in the mesangial matrix, and a thickening of the glomerular capillary walls. The wall thickening is sometimes said to be a result of the interposition of mesangial cytoplasm or matrix between the basement membrane and the endothelium of the capillary wall. It is often divided into types I and II, and sometimes III. Pathogenesis is not well understood, but some types are thought to involve the immune system, with the complement system, in particular, being implicated. (12 Dec 1998) |
| glomerulonephritis, membranous | A disease of the glomerulus manifested clinically by proteinuria, and sometimes by other features of the nephrotic syndrome. It is histologically characterised by deposits in the glomerular capillary wall between the epithelial cell and the basement membrane and a thickening of the membrane. Also characteristic are outward projections of the membrane between the epithelial deposits in the form of "spikes". There is some agreement that the deposits are antigen-antibody complexes. (12 Dec 1998) |
| membranoproliferative glomerulonephritis | <pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure. Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane. Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease. Acronym: MPGN (26 Mar 1998) |
| membranous glomerulonephritis | <pathology> A kidney disease that occurs due to inflammation of the kidney glomerulus and its basement membrane. The exact cause is unknown but it appears to be related to the deposition of immune complexes in the basement membrane leading to thickening of the capillary walls. This disorder is a common cause of nephrotic syndrome an is usually how the disease manifests. Risk factors include primary renal disease, malaria, hepatitis B, lupus, syphilis, cancers and non-Hodgkin's lymphomas. Risks also include exposure to some medications such as gold compounds and penicillamine. Mercury, trimethadione and some skin-lightening creams have also been implicated. Treatment includes systemic corticosteroids and immunosuppressive agents. (26 Mar 1998) |
| mesangial proliferative glomerulonephritis | <nephrology, pathology> Inflammation of the kidney glomerulus (blood filtering portion of the kidney) due to the abnormal deposition of IgM antibody in the mesangium layer of the glomerular capillary. A form of glomerulonephritis that appears to be caused by an abnormal immune response. This disorder generally manifests as nephrotic syndrome. Symptoms include swelling, dark urine, weight gain, hypertension, anorexia and bloody urine. Acronym: MPGN (05 Jan 1998) |
| mesangiocapillary glomerulonephritis | <nephrology, pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure. Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane. Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease. (27 Sep 1997) |
| chronic glomerulonephritis | Glomerulonephritis that presents with persisting proteinuria, chronic renal failure, and hypertension, of insidious onset or as a late sequel of acute glomerulonephritis; the kidneys are symmetrically contracted and granular, with scarring and loss of glomeruli and the presence of tubular atrophy and interstitial fibrosis. Synonym: chronic nephritis. (05 Mar 2000) |
| crescentic glomerulonephritis | <nephrology, pathology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease. Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents. (27 Sep 1997) |
| proliferative glomerulonephritis | Glomerulonephritis with hypercellularity of glomeruli due to proliferation of endothelial or mesangial cells, occurring in acute glomerulonephritis and membranoproliferative glomerulonephritis. (05 Mar 2000) |
| segmental glomerulonephritis | Glomerulonephritis affecting only part of a glomerulus or glomeruli. Synonym: local glomerulonephritis. (05 Mar 2000) |
| hypocomplementemic glomerulonephritis | <pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure. Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane. Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease. Acronym: MPGN (26 Mar 1998) |
| necrotising glomerulonephritis | <pathology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease. Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents. (27 Sep 1997) |
| subacute glomerulonephritis | Undesirable term for glomerulonephritis with proteinuria, haematuria and azotemia persisting for many weeks; renal changes are variable, including those of rapidly progressive and membranoproliferative glomerulonephritis. Synonym: subacute nephritis. (05 Mar 2000) |