| ¿µ¹® | growth hormone | ÇÑ±Û | ¼ºÀåÈ£¸£¸ó |
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| ¼³¸í | ³úÇϼöü Àü¿±¿¡¼ ºÐºñµÇ´Â È£¸£¸óÁß Çϳª·Î¼ ½Ã»óÇϺÎÀÇ ¼ºÀåÈ£¸£¸ó¹æÃâ È£¸£¸ó¿¡ ÀÇÇØ ºÐºñ°¡ ÀÚ±ØµÇ¸ç ¼Ò¸¶Å佺Ÿƾ(somatostatin: ÀÌÀÚ¿¡¼ ºÐºñµÇ¸ç, ¼ºÀåÈ£¸£¸ó¿¡ ¹Ý´ëµÇ´Â ÀÛ¿ëÀ» ÇÔ)¿¡ ÀÇÇØ ºÐºñ°¡ ¾ïÁ¦µÈ´Ù. ¼ºÀå È£¸£¸ó ¹æÃâ È£¸£¸óÀº µµÆÄ¹Î(dopamine)À¸·Î ¾Ë·ÁÁ® ÀÖ´Ù. ¼ºÀå È£¸£¸óÀº ¼¼Æ÷ÀÇ ¼ºÀåÀ» ÃËÁø½Ã۸ç ƯÈ÷ °ñÀÇ ¼ºÀåÀ» ÀÚ±ØÇϴµ¥ ±× ÀÛ¿ëÀº Á÷Á¢ ¼¼Æ÷¿¡ ÀÛ¿ëÇÏ´Â °ÍÀÌ ¾Æ´Ï¶ó °£°ú ±ÙÀ°¿¡ ÀÛ¿ëÇÏ¿© ±×°÷¿¡¼ ¼Ò¸¶Åä¸ÞµòÀ» »ý¼ºÇϸç ÀÌ ¼Ò¸¶Åä¸ÞµòÀÌ ¼¼Æ÷ÀÇ ¼ºÀåÀ» ÃËÁø½ÃŲ´Ù. ÇÑÆí ¼ºÀå È£¸£¸óÀº ¼ºÀå¿¡ ÇÊ¿äÇÑ ´Ü¹éÁú ÇÕ¼ºÀ» Ç×Áø½ÃŰ°í ¿¡³ÊÁö´Â Áö¹æÀ» ÀÌ¿ëÇÏ¿© ¾ò°ÔÇϹǷΠÁö¹æÀÌ¿ëÈ£¸£¸óÀ̶ó°íµµ ºÒ¸°´Ù. ¼ºÀå È£¸£¸óÀÌ °ú´Ù ºÐºñµÇ¸é °ÅÀÎÁõ, ¸»´Üºñ´ëÁõÀÌ À¯¹ßµÇ¸ç ¼ºÀå È£¸£¸óÀÌ °áÇÌµÇ¸é ¼ºÀåºÎÁøÀÌ ¿Â´Ù. |
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| ¿µ¹® | hormone | ÇÑ±Û | È£¸£¸ó |
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| ¼³¸í | È£¸£¸óÀ̶õ ƯÁ¤ÇÑ ³»ºÐºñ¼±¿¡¼ »ý¼º, ÀúÀåµÇ¾î ÀÖ´Ù°¡, °ü·ùÇÏ´Â Ç÷¾× ÁßÀ¸·Î ºÐºñµÈ ´ÙÀ½ Ç÷¾×¿¡ ½Ç·Á ¸Ö¸® ¶³¾îÁ® Àִ ǥÀû¼¼Æ÷(È£¸£¸óÀÌ ÀÛ¿ëÀ» ³ªÅ¸³»´Â ¼¼Æ÷)¿¡ À̸£·¯ ±×°÷¿¡¼ ƯÁ¤ÇÑ ±â´ÉÀ» ¹ßÈÖÇÏ´Â ¹°ÁúÀ» ¸»ÇÑ´Ù. ³»ºÐºñ»ùÀÌ ¾Æ´Ï´õ¶óµµ ¾î´À ¼¼Æ÷³ª ¼¼Æ÷±º¿¡¼ ¼¼Æ÷¿Ü¾× ÁßÀ¸·Î ºÐºñµÈ ´ÙÀ½ ´Ù¸¥ ¼¼Æ÷¿¡ °¡¼ ¼¼Æ÷ÀÇ ±â´ÉÀ» º¯µ¿½ÃŰ´Â ¹°ÁúÀÇ ÀϺεµ È£¸£¸óÀÇ ¹üÁÖ¿¡ Æ÷ÇÔ½Ã۰í ÀÖÀ¸¸ç À̵éÀ» Ưº°È÷ ±¹¼ÒÈ£¸£¸óÀ̶ó ºÎ¸¥´Ù(¿¹-¾Æ¼¼Æ¿Äݸ°, ¾Æµå·¹³¯¸°, ÄÝ·¹½Ã½ºÅäŰ´Ñ µî). ÀÌ¿¡ ºñÇØ¼ ƯÁ¤ ³»ºÐºñ»ù¿¡¼ Ç÷ÁßÀ¸·Î ºÐºñµÇ´Â ÁøÁ¤ÇÑ ÀǹÌÀÇ È£¸£¸óµéÀ» ÀϹÝÈ£¸£¸óÀ̶ó°í ºÎ¸¥´Ù. È£¸£¸óÀ» ÈçÈ÷ 3Á¾ÀÇ ¹«¸®·Î ºÐ·ù¸¦ Çϴµ¥ ¾Æµå·¹³¯¸°À̳ª thyroxine°ú °°ÀÌ ¾Æ¹Ì³ë»êÀÇ À¯µµÃ¼·Î µÈ È£¸£¸óµé(amine derivative), ¸¹Àº ¾Æ¹Ì³ë»êÀ» Àç·á·Î ¸¸µé¾îÁ³°Å³ª ´ç´Ü¹éÀ¸·Î µÈ ´Ü¹éÈ£¸£¸ó(peptide hormone), ±×¸®°í ÄÝ·¹½ºÅ×·ÑÀ» Àç·á·Î ¸¸µé¾îÁø Áö¹æÈ£¸£¸ó(steroid hormone) µîÀ¸·Î ±¸ºÐµÇ¸ç À̵éÀº ´ëü·Î Á¾·ù¿¡ µû¶ó ÀÛ¿ë¸ÞÄ¿´ÏÁòÀ» ´Þ¸®Çϰí ÀÖ´Ù. |
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| ¿µ¹® | luteinizing hormone(LH) | ÇÑ±Û | ȲüÇü¼ºÈ£¸£¸ó |
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| ¼³¸í | ¿©¼ºÀÇ ¿ù°æÀº Á¤ÇØÁø ¼ø¼¿¡ ÀÇÇØ ÀϾ°Ô µÈ´Ù. ¸ÕÀú ³Æ÷ÀÚ±ØÈ£¸£¸óÀÌ ³ú¿¡¼ ºÐºñµÇ¾î ¿©¼ºÀÇ ³¼Ò¸¦ ÀÚ±ØÇÏ°Ô µÇ¸é ³Æ÷°¡ ÀÚ±Ø¹Þ¾Æ ³Æ÷È£¸£¸ó(¿¡½ºÆ®·ÎÁ¨)À» ºÐºñÇÏ°Ô µÇ°í À̰ÍÀº ´Ù½Ã µÇ¸ÔÀ̱⠸ÞÄ¿´ÏÁò ÀÇÇØ ³ú¿¡ ÀÛ¿ëÀ» ÇÏ¿© ȲüÈÈ£¸£¸óÀÌ ³ú¿¡¼ ºÐºñµÇ°í À̰ÍÀÌ À̸¥¹Ù ÀýÁ¤¿¡ À̸£·¶À» ¶§, ³Æ÷¿¡¼ ¹è¶õÀÌ ÀϾ°Ô µÈ´Ù. ÀÌÈÄ ³Æ÷´Â Ȳü·Î º¯ÇÏ°Ô µÇ¸ç, ¹è¶õµÈ ³ÀÚ°¡ ¼öÁ¤µÇÁö ¾Æ´ÏÇϸé, Ȳü´Â Ȳüȣ¸£¸ó(ÇÁ·Î°Ô½ºÅ×·Ð)À» ºÐºñÇÑ µÚ ÅðÈµÇ°í °ð ÀÌ¾î ¿ù°æÀÌ ÀϾ°Ô µÈ´Ù. ÀÌ ÀÏÀº Á¤»óÀûÀÎ ´ëºÎºÐÀÇ ¿©¼º¿¡¼ 28ÀÏÀ» ÁÖ±â·Î ÀϾ¸ç, ´ë°³ ȲüÈÈ£¸£¸ó ÀýÁ¤ ÀÌÈÄ ¸¶Áö¸·Àº 14ÀÏ·Î ÀÏÁ¤ÇÏ´Ù°í ÇÑ´Ù. |
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| 131I | radioactive Iodine(used in Thyroid uptake, Liver & Kidney Scans & Treatment of malignant & nonmalig... |
|---|---|
| MEN | Multiple Endocrine Neoplasia ; AD Trait 1. MEN Type I(= Wermer Syndro... |
| TA | alkaline tuberculin; arterial tension; axillary temperature; tactile afferent; Takayasu arteritis; t... |
| ER | efficiency ratio; epigastric region; ejection rate; electroresection; emergency room; endoplasmic re... |
| RAR | rapidly adapting receptor; rat insulin receptor; retinoic acid receptor; right arm reclining; right ... |
| growth hormone-regulating hormone | <endocrinology> Hypothalamic hormones that induce (somatoliberin) or inhibit (somatostatin) the release of growth hormone (somatotropin). (18 Nov 1997) |
|---|---|
| growth hormone-releasing hormone | <endocrinology> Peptide hormone related to the glucagon family, released from the pituitary, acts on the adenohypophysis to release growth hormone. Synonym: somatoliberin, growth hormone-releasing factor. (20 Sep 2002) |
| hormones, hormone substitutes, and hormone antagonists | A collective grouping for both naturally occurring and synthetic hormones, substitutes, and antagonists. (12 Dec 1998) |
| follicle-stimulating hormone-releasing hormone | A decapeptide of hypothalamic origin capable of accelerating pituitary secretion of follitropin. Synonym: follicle-stimulating hormone-releasing factor, follicle-stimulating hormone-releasing hormone. Origin: follicle-stimulating hormone + L. Libero, to free, + -in (05 Mar 2000) |
| luteinizing hormone/follicle-stimulating hormone-releasing factor | gonadotrophin-releasing hormone |
| luteinizing hormone-releasing hormone | A hormone that controls sex hormones in men and women. Also called lhrh. (12 Dec 1998) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| amino acid receptor | <biochemistry> Ligand gated ion channels with specific receptors for amino acid transmitters. An extended protein superfamily that also includes subunits of the nicotinic acetylcholine receptor. (18 Nov 1997) |
| AMPA receptor | <cell biology> Glutamate operated ion channel. See: excitatory amino acid receptor channels. (05 Feb 1998) |
| ANP receptor | <molecular biology> Family of 3 receptors for atrial natriuretic peptide. ANP A and ANP B have intracellular guanylate cyclase and protein kinase like domains. ANP C, shares the extracellular ligand binding and transmembrane domains, but lacks the functional intracellular domains and is not thought to be involved in signal transduction. (18 Nov 1997) |
| asialoglycoprotein receptor | A surface receptor found in hepatocytes that binds galactose-terminal glycoproteins; thus, this receptor removes those proteins from circulation and they are in turn acted upon by hepatocyte lysosomes. (05 Mar 2000) |
| auditory receptor cells | Columnar cell's in the epithelium of the organ of Corti, having hairs (stereocilia) on their apical ends. See: Corti's cells. (05 Mar 2000) |
| beta-adrenergic receptor blocking agent | A class of drugs that compete with beta-adrenergic agonists for available receptor sites; some compete for both b1 and b2 receptors (e.g., propranolol) while others are primarily either b1 (e.g., metoprolol) or b2 blockers; used in the treatment of a variety of cardiovascular diseases where beta-adrenergic blockade is desirable. Synonym: beta-adrenergic receptor blocking agent, beta-adrenoreceptor antagonist, beta-blocker. (05 Mar 2000) |
| beta-adrenergic receptor kinase | <enzyme> Cyclic-AMP protein kinase which specifically phosphorylates the agonist-occupied form of beta-adrenergic receptor Registry number: EC 2.7.1.- Synonym: beta-ar kinase, beta-adrenergic receptor kinase 1, g-protein-coupled receptor kinase 2, grk2 (kinase), beta-adrenergic receptor kinase 2, beta-ar kinase 2 (26 Jun 1999) |
| cAMP receptor protein | catabolite (gene) activator protein |
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