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"Persistent hyperplasia of thymus"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • persistent vomiting
    Áö¼Ó±¸Åä
  • angiolymphoid hyperplasia
    Ç÷°ü¸²ÇÁ±¸°ú´ÙÇü¼º, Ç÷°ü¸²ÇÁ±¸Áõ½Ä
  • atypical adenomatous hyperplasia
    ºñÁ¤Çü»ùÁ¾Áõ½Ä
  • atypical complex hyperplasia
    ºñÁ¤Çüº¹ÇÕÀڱ󻸷Áõ½Ä
  • atypical cystic hyperplasia
    ºñÁ¤Çü³¶¼ºÁõ½Ä
  • adenomatous hyperplasia
    »ùÁ¾°ú´ÙÇü¼º
  • adrenocortical hyperplasia
    ºÎ½Å°ÑÁú°ú´ÙÇü¼º, ºÎ½ÅÇÇÁúÁõ½Ä
  • benign lymphoid hyperplasia
    ¾ç¼º¸²ÇÁ°ú´ÙÇü¼º
  • compensatory hyperplasia
    º¸»ó°ú´ÙÇü¼º
  • complex hyperplasia
    º¹ÇÕÁõ½Ä
  • congenital adrenal hyperplasia
    ¼±ÃµºÎ½Å°ú´ÙÇü¼º, ¼±ÃµÄáÆÏÀ§»ù°ú´ÙÇü¼º
  • cardiac myocyte hyperplasia
    ½ÉÀå±ÙÀ°¼¼Æ÷°ú´ÙÇü¼º
  • cutaneous lymphoid hyperplasia
    ÇǺθ²ÇÁ°ú´ÙÇü¼º
  • endocervical hyperplasia
    Àڱøñ³»¸·°ú´ÙÇü¼º, Àڱðæ°ü³»¸·Áõ½ÄÁõ
  • endometrial hyperplasia
    Àڱ󻸷°ú´ÙÇü¼º, Àڱ󻸷Áõ½Ä(Áõ)
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • persistent fetal hemoglobin
    Áö¼ÓžÆÇ÷»ö¼Ò
  • persistent infection
    Áö¼Ó°¨¿°
  • persistent o. primum ostium
    ù°±¸¸ÛÁö¼Ó(Áõ), ÀÏÂ÷°ø°³Á¸(Áõ)
  • persistent o. secundum ostium
    µÑ°±¸¸ÛÁö¼Ó(Áõ), ÀÌÂ÷°ø°³Á¸(Áõ)
  • persistent
    Áö¼Ó-, ÀÜ·ù-
  • persistent tremor
    Áö¼Ó¶³¸²
  • persistent vomiting
    Áö¼Ó±¸Åä
  • persistent cathode ray tube
    Áö¼ÓÀ½±Ø¼±°ü
  • persistent light reaction
    Áö¼Ó±¤¹ÝÀÀ
  • persistent palmoplantar pustulosis
    Áö¼Ó¼Õ¹ß¹Ù´Ú°í¸§¹°ÁýÁõ, Áö¼Ó¼Õ¹ß¹Ù´Ú³óÆ÷Áõ
  • persistent vegetative state
    Áö¼Ó½Ä¹°»óÅÂ
  • adenomatous hyperplasia
    »ùÁ¾°ú´ÙÇü¼º
  • adrenocortical hyperplasia
    ºÎ½Å°ÑÁú°ú´ÙÇü¼º
  • adrenogenital hyperplasia
    ºÎ½Å¼º±â°ú´ÙÇü¼º
  • androgenic adrenal hyperplasia
    ¾Èµå·Î°ÕºÎ½Å°ú´ÙÇü¼º
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • hyperplasia endometrii glandularis ³ª
    ¼±¼ºÀڱ󻸷Áõ½Ä(Áõ)(àÍàõí­ÏàҮد ñòãÖñø).
  • hyperplasia of adrenal gland
    ºÎ½Å¼±Áõ½Ä
  • hyperplasia,atypical
    ºñÁ¤Çü(ÞªïÒû¡)
  • hyperplasia,compensatory
    ´ë»ó¼º
  • reactive nodular hyperplasia
    ¹ÝÀÀ¼º °áÀýÁõ½Ä
  • acral persistent papular mucinosis
    ¸»´Ü Áö¼Ó¼º ±¸Áø¼º Á¡¾×Áõ
  • growing teeth from persistent pulp
    »ó»ýÄ¡ (ßÈßæöÍ).
  • hepatitis,chronic persistent
    ¸¸¼ºÁö¼Ó¼º(Ø·àõò¥áÙàõ)
  • infection, persistent
    Áö¼Ó°¨¿°
  • persistent acantholytic dermatosis
    Áö¼Ó¼º ±Ø¼¼Æ÷ÇØ¸® ÇǺκ´
  • persistent atrioventricular canal
    ¹æ½Ç±¸°³ Á¸Áõ.
  • persistent atrioventricular communis
    °øÅë ¹æ½Ç±¸(°ø)°³Á¸(Áõ)(Íì÷×Û®ãøÏ¢ÍîËÒðíñø).
  • persistent cathod ray tube
    Áö¼Ó¼º À½±Ø¼±°ü
  • persistent cloaca
    ¹è¼³°­Á¸¼ÓÁõ
  • persistent generalized lymphadenopathy
    Áö¼Ó¼º Àü½Å¼º ¸²ÇÁ¼±Áõ
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  • ¿µ¹®
    ÇѱÛ
  • thymus
    °¡½¿»ù
  • thymus dependent antigen
    Èä¼±ÀÇÁ¸Ç׿ø, °¡½¿»ùÀÇÁ¸Ç׿ø
  • thymus dependent cell
    Èä¼±ÀÇÁ¸¼¼Æ÷(¡­ëîðíá¬øà).
  • thymus derived cell
    Èä¼±À¯·¡¼¼Æ÷(¡­ë¦ÕÎ á¬øà).
  • thymus gland
    Èä¼±
  • thymus gland
    Èä¼±, °¡½¿»ù
  • thymus gland,hassalls corpuscle
    ÇÖ»ì ¼Òü
  • thymus independent antigen
    Èä¼±µ¶¸³Ç׿ø(ýØàÍÔ¼í¡ù÷ê«).
  • thymus independent area
    Èä¼±ºñÀÇÁ¸¿ª.
  • thymus independent cells
    Èä¼±ºñÀÇÁ¸¼¼Æ÷ (ýØàÍÞªëîðíá¬øà).
  • thymus involution
    Èä¼±ÅðÈ­, °¡½¿»ùÅðÈ­
  • thymus ³ª
    °¡½¿»ù, Èä¼±(ýØàÍ).
  • thymus-derived lymphocytes
  • acral persistent papular mucinosis
    ¸»´Ü Áö¼Ó¼º ±¸Áø¼º Á¡¾×Áõ
  • asymptomatic persistent proteinuria
    ¹«Áõ»óÁö¼Ó¼º ´Ü¹é´¢.
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FH facial hemihyperplasia; familial hypercholesterolemia; family history; fasting hyperbilirubinemia; f...
AMI Acute Myocardial Infarction
  - Complications(Cx)
    1. Early ...
CPH Chronic Persistent Hepatitis
PFC Persistent Fetal Circulation; ÅÂ¾Æ ¼øÈ¯ Áö¼ÓÁõ
  = PPHN
PPHN Persistent Pulmonary Hypertension of the Newborn; ½Å»ý¾Æ Æóµ¿¸Æ °íÇ÷¾Ð Á¸¼ÓÁõ
  = PFC
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TARC Thymus and activation-regulated chemokine
TD Thymus-dependent
TI Thymus-independent
CT DNA calf thymus DNA
T thymus
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • basal cell hyperplasia
    ±âÀú ¼¼Æ÷ °úÇü¼º
  • benign lymphoid hyperplasia
    ¾ç¼º ¸²ÇÁ °úÇü¼º
  • condylar hyperplasia
    ÇÏ¾Ç °úµÎ °úÇü¼ºÁõ
  • coronoid hyperplasia
    ¿ÀÈÑ µ¹±â ºñ´ë
    ÇÏ¾Ç ¿ÀÈÑ µ¹±âÀÇ ¾ç¼º °ú¼ºÀå
  • denture-induced hyperplasia
    ÀÇÄ¡¼º À°¾ÆÁ¾
    ?
  • dilantin gingival hyperplasia
    µô¶õƾ¼º Ä¡Àº Áõ½ÄÁõ
  • dilantin hyperplasia
    µô¶õƾ¼º Ä¡Àº Áõ½Ä
  • fibrous inflammatory hyperplasia
    ¼¶À¯ ¿°Áõ¼º °úÇü¼º
  • focal nodular hyperplasia
    ÃÊÁ¡¼º °áÀý¼º °úÁõ½Ä
    À§ Á¾¾çÀ¸·Î ºÐ·ùµÇ´Â °£ÀÇ °áÀý¼º º´º¯ÀÇ ÇÑ À¯ÇüÀ¸·Î¼­ °ú¿ÀÁ¾¼º ¹ßÀ° °á¼Õ¿¡ ±âÀÎÇÑ ´ÜÀÏ Á¾±«¼º º´º¯À¸·Î ÇØ¼®µÇ°í ÀÖ´Ù. ÀÌ´Â ÁÖ·Î ÇǸ·ÇÏ¿¡ Àß »ý±â°í Å©±â´Â ¼ö cm ³»¿ÜÀÌ¸ç ¾ãÀº ÆÐ¸·¿¡ ÀÇÇÏ¿© ÁÖº¯ °£ Á¶Á÷À¸·ÎºÎÅÍ Àß ±¸ºÐµÈ´Ù. Àý´Ü¸é¿¡¼­ Á¾±«ÀÇ °¡¿îµ¥¿¡ º° ¸ð¾çÀÇ ¹ÝÈç
  • hyperplasia
    ºñÈÄ, Áõ½Ä, °úÇü¼º
    1. ±â´É Ç×ÁøÀ» À¯¹ß½ÃŰ´Â ÀÚ±ØÀ» ¹Þ¾ÒÀ» ¶§, ºÐ¿­ÀÌ °¡´ÉÇÑ ¼¼Æ÷°¡ ¼¼Æ÷ ºÐ¿­·Î ±× ¼ö°¡ Áõ°¡µÇ´Â °ÍÀ» ¸»Çϸç, °á°úÀûÀ¸·Î ±× Á¶Á÷À̳ª Àå±âµµ Ä¿Áö°Ô µÈ´Ù. ¹Ý¸é, ºñ´ë´Â ¼¼Æ÷ÀÇ Áõ½ÄÀÌ ¾øÀÌ ´Ü¼øÈ÷ ¼¼Æ÷ÀÇ Å©±â°¡ Ä¿Áö´Â °æ¿ì¸¦ À̸£´Â ¸»ÀÌ´Ù. 2. Á¤»ó ¼¼Æ÷ ¼ö°¡ Á¤»óÀûÀÎ ¹è¿­ »óÅ·ΠÁõ°¡ÇÏ´Â Á¶Á÷À̳ª ±¸Á¶¹°ÀÇ °ú¹ßÀ°.
  • hyperplasia of coronoid
    ±Ùµ¹±â °úÁõ½Ä
  • idiopathic fibrous gingival hyperplasia
    Ư¹ß¼º ¼¶À¯¼º Ä¡Àº Áõ½Ä
  • inflammatory gingival hyperplasia
    ¿°Áõ¼º Ä¡Àº Áõ½Ä
  • inflammatory hyperplasia
    ¿°Áõ¼º °úÇü¼º, ¿°Áõ¼º Áõ½Ä
    ¿°Áõ¿¡ ÀÇÇÏ¿© »ý±â´Â °úÇü¼º.
  • lentiginous melanocytic hyperplasia(ÈæÀÚ¼º ¸á¶ó´Ñ ¼¼Æ÷ Áõ½Ä)

    lentiglobus

    °ø ¸ð¾ç ¼öÁ¤Ã¼, ±¸Çü ¼öÁ¤Ã¼
    ¼öÁ¤Ã¼ Àü¸éÀÌ ±¸ÇüÀ¸·Î µÇ¾î¼­, ¼öÁ¤Ã¼ÀÇ °î¸éÀÌ °úÀåµÇ¾î º¸ÀÌ´Â °Í.
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
chronic persistent hepatitis <pathology> A form of hepatitis usually caused by hepatitis C or B, that consists of mild persistent liver inflammation.
Often a mild elevation of the liver enzymes will be seen on liver profile or SMAC 25 assay. Liver biopsy indicates persistent hepatitis.
Incidence: 1 in 1,000.
(02 Jan 1998)
congenital aplasia of thymus diGeorge syndrome
cortex of thymus The outer part of a lobule of the thymus; it surrounds the medulla and is composed of masses of closely packed lymphocytes.
(05 Mar 2000)
hypoplasia of the thymus and parathyroids Also known as the digeorge syndrome (dgs), this disorder is characterised by (1) low blood calcium levels (hypocalcaemia) due to underdevelopment (hypoplasia) of the parathyroid glands needed to control calcium; (2) underdevelopment (hypoplasia) of the thymus, an organ behind the breastbone in which lymphocytes mature and multiply; and (3) defects of the outflow tracts from the heart. most cases of dgs are due to a microdeletion in chromosome band 22q11.2. A small number of cases have defects in other chromosomes, notably 10p13. Named after the american paediatric endocrinologist angelo digeorge. Another name for dgs is the third and fourth pharyngeal pouch syndrome (since the faulty structures in dgs are embryologically derived from the third and fourth pharyngeal pouches).
(12 Dec 1998)
thymus <anatomy> The lymphoid organ in which T lymphocytes are educated, mature and multiply. It is composed of stroma (thymic epithelium) and lymphocytes, almost entirely of the T-cell lineage. In mammals the thymus is just anterior to the heart within the rib cage, in other vertebrates in rather undefined regions of the neck or within the gill chamber in teleost fish. The thymus regresses as the animal matures.
(18 Nov 1997)
thymus and parathyroids, hypoplasia of See third and fourth pharyngeal pouch syndrome.
(12 Dec 1998)
thymus-dependent zone <anatomy> Mid cortical region of lymph node, area that is particularly depleted of T lymphocytes in thymectomised animals and is referred to as the thymus dependent area.
(18 Nov 1997)
thymus derived lymphocyte <haematology, immunology> A class of lymphocytes, so called because they are derived from the thymus and have been through thymic processing. Involved primarily in controlling cell-mediated immune reactions and in the control of B-cell development. The T-cells coordinate the immune system by secreting lymphokine hormones.
There are 3 fundamentally different types of t cells : helper, killer, and suppressor. Each has many subdivisions. T-cells are also called t lymphocytes.
They bear T-cell antigen receptors (CD3) and lack Fc or C3b receptors. Major T-cell subsets are CD4 (mainly helper cells) and CD8 (mostly cytotoxic or suppressor T-cells). Uncontrolled proliferation of this type of cell gives rise to T-cell leukaemia/lymphoma.
(21 Jun 1999)
thymus extracts Extracts of the thymus that contain specific, but uncharacterised factors or proteins with specific activities; three distinct substances are already known: thymotoxin, thymin and thymosin.
(12 Dec 1998)
thymus gland A bilaterally symmetric lymphoid organ situated in the anterior superior mediastinum. Each of its two lobes consists of an outer zone, the cortex, relatively rich in lymphocytes (thymocytes), and an inner zone, the medulla, relatively rich in epithelial cells. The thymus is the site of the production of T-lymphocytes. The thymus reaches its maximal development at about puberty and then undergoes a gradual process of involution resulting in a slow decline of immune function throughout adulthood.
(12 Dec 1998)
thymus hormones Humoral factors secreted by the thymus gland. They participate in the development of the lymphoid system and the maturation of the cellular immune response.
(12 Dec 1998)
thymus-independent antigen An antigen that does not require T helper cell activation in order for the host's B-cells to be stimulated. Repeating polymers such as polysaccharides are examples of T-independent antigens.
(05 Mar 2000)
thymus treatment Treatment of disease by administration of extracts of thymus gland.
(05 Mar 2000)
truncus arteriosus, persistent A congenital anomaly resulting from the failure of the aorticopulmonary system to develop and divide the truncus arteriosus into the aorta and the pulmonary trunk.
(12 Dec 1998)
lobules of thymus Areas of thymic tissue 0.5 to 2 mm in diameter with a cortex and medulla.
Synonym: lobuli thymi.
(05 Mar 2000)
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