| ADC | adult day care [facility]; affective disorders clinic; Aid to [Families with] Dependent Children; AI... |
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| ADT | Accepted Dental Therapeutics; adenosine triphosphate; admission, discharge, transfer; agar-gel diffu... |
| ALSD | Alzheimer-like senile dementia |
| ALS-PD | amyotrophic lateral sclerosis-parkinsonism-dementia [complex] |
| ASD | aldosterone secretion defect; Alzheimer senile dementia; antisiphon device; arthritis syphilitica de... |
| dementia paralytica | Dementia and paralysis resulting from a chronic syphilitic meningoencephalitis. Synonym: dementia paralytica. (05 Mar 2000) |
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| dementia paranoides | Dementia with paranoid features. Posttraumatic dementia, dementia caused by traumatic brain injury. Dementia praecox, any one of the group of psychotic disorders known as the schizophrenias; formerly used to describe schizophrenia as a single entity. Origin: L. Precocious (05 Mar 2000) |
| dementia, vascular | An organic mental disorder caused by systemic vascular disease such as arteriosclerotic changes in the vessels, valvular heart disease, hypertension, etc., and resulting in cerebral vascular disease with dementia. (12 Dec 1998) |
| dialysis dementia | A progressive (often fatal) diffuse encephalopathy which occurs in a few patients who undergo chronic haemodialysis, dementia is a key feature (27 Sep 1997) |
| transmissible dementia | <infectious disease> A very rare form of encephalopathy thought to be caused by a virus (slow-virus), termed a prion. There is little known about the mode of transmission. Human to human transmission has occurred through the use of contaminated brain electrodes and transplantation of infected tissues. The agent can be recovered in the CSF of infected individuals. Standard disinfectants such as formalin, heat, exposure to ultraviolet light or X-rays is ineffective to inactivate the virus. Autoclaving to at least 132 degrees C. Or immersion in 4% sodium hydroxide or 10% sodium hypochlorite solution for 1 hour is recommended for sterilisation. The disease occurs primarily in adults, with peak incidence in the late 50's. Infection results in dementia, myoclonus, ataxia and other neurologic symptoms. The disease progresses rapidly to coma and death after a 3 to 12 month illness. There is no known cure. (27 Sep 1997) |
| epileptic dementia | Dementia occurring in an individual afflicted with epilepsy, and thought to be a result of prolonged seizures, the epileptogenic brain lesion, or antiepileptic drugs. Hebephrenic dementia, dementia with hebephrenic symptoms. (05 Mar 2000) |
| Albright's hereditary osteodystrophy | An inherited form of hyperparathyroidism associated with ectopic calcification and ossification and skeletal defects, notably the small fourth metacarpals, but intelligence is normal. There are dominant, recessive and X-linked forms. See: pseudohypoparathyroidism. Synonym: Albright's syndrome. (05 Mar 2000) |
| angioedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |
| angioneurotic oedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| canine hereditary blindness | An autosomal dominant condition seen in dogs of the collie and several other breeds. (05 Mar 2000) |
| colourectal neoplasms, hereditary nonpolyposis | A syndrome characterised by autosomal dominant inheritance, a low mean age (41 years) for occurrence of colon cancer, and a marked increase in the proportion of tumours in the proximal colon. (12 Dec 1998) |
| corneal dystrophies, hereditary | Bilateral hereditary disorders of the cornea, usually autosomal dominant, which may be present at birth but more frequently develop during adolescence and progress slowly throughout life. Central macular dystrophy is transmitted as an autosomal recessive defect. (12 Dec 1998) |
| hereditary | <genetics> Transferred via genes from parent to child. (16 Dec 1997) |
| hereditary amyloidosis | <neurology> A disorder in which various peripheral nerves are infiltrated with amyloid and their functions disturbed, an abnormal prealbumin is also formed and is present in the blood; characteristically, it begins during mid-life and is found largely in persons of Portuguese descent. Other rare clinical types occur. Inheritance: autosomal dominant. Synonym: familial amyloidosis, hereditary amyloidosis. (05 Mar 2000) |
| hereditary angioedema | A genetic form of angioedema. (angioedema is also referred to as quinke's disease.) persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |