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"Complement and inflammation."¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • maternity and infant hygiene
    ¸ð¼º¿µ¾ÆÀ§»ý
  • picture archiving and communicating system
    ¿µ»óÀúÀå¹×Àü¼Ûü°è
  • perfusion and diffusion imaging
    °ü·ùÈ®»ê¿µ»ó
  • salt-and-pepper fundus
    ÈÄÃ߼ұݸð¾ç¾ÈÀú, ÈÄÃ߼ұݸð¾ç´«¹Ù´Ú
  • wear and tear dermatitis
    ¹Ýº¹ÀÚ±ØÇǺο°
  • alternative complement pathway
    ´ëüº¸Ã¼°æ·Î
  • classic complement pathway
    ÀüÇüÀûº¸Ã¼°æ·Î
  • complement
    º¸Ã¼, µµ¿òü
  • complement activation
    º¸Ã¼È°¼ºÈ­, µµ¿òüȰ¼ºÈ­
  • complement cascade
    º¸Ã¼¿¬¼â¹ÝÀÀ, µµ¿òü¿¬¼â¹ÝÀÀ
  • complement component
    º¸Ã¼¼ººÐ, µµ¿òü¼ººÐ
  • complement fixation
    º¸Ã¼°áÇÕ
  • complement fixation inhibition test
    º¸Ã¼°áÇÕ¾ïÁ¦°Ë»ç
  • complement fixation reaction
    º¸Ã¼°áÇÕ¹ÝÀÀ
  • complement fixation test
    º¸Ã¼°áÇÕ°Ë»ç
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • complement splitting
    µµ¿òüºÐ¿­, º¸Ã¼ºÐ¿­
  • complement system
    µµ¿òü°èÅë, º¸Ã¼°èÅë
  • complement typing
    º¸Ã¼Çüº°°Ë»ç
  • complement deficient state
    µµ¿òü°áÇÌ»óÅÂ, º¸Ã¼°áÇÌ»óÅÂ
  • complement fixation reaction
    º¸Ã¼°áÇÕ¹ÝÀÀ, µµ¿òü°áÇÕ¹ÝÀÀ
  • complement fixation test
    µµ¿òü°áÇÕ½ÃÇè, º¸Ã¼°áÇÕ½ÃÇè
  • complement fixation unit
    º¸Ã¼°áÇÕ´ÜÀ§
  • complement fixation inhibition test
    º¸Ã¼°áÇÕ¾ïÁ¦½ÃÇè
  • complement mediated lysis
    º¸Ã¼¸Å°³¿ëÇØ, µµ¿òü¸Å°³¿ëÇØ
  • complement-dependent cytotoxicity
    µµ¿òüÀÇÁ¸¼¼Æ÷µ¶¼º
  • complement-mediated cytotoxicity
    µµ¿òü°ü·Ã¼¼Æ÷µ¶¼º
  • croupous inflammation
    Å©·ì¿°Áõ
  • cytopathic-cytoproliferative inflammation
    ¼¼Æ÷º´º¯¼¼Æ÷Áõ½Ä¿°Áõ
  • dominant complement
    ¿ì¼ºµµ¿òü
  • diffuse inflammation
    ¹Ì¸¸¿°Áõ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • antibody, complement fixing
    º¸Ã¼°áÇÕÇ×ü
  • gonococcal complement fixation test
    ÀÓ±Õº¸Ã¼°áÇÕ¹ÝÀÀ°Ë»ç.
  • heparin complement
    ÇìÆÄ¸°º¸Ã¼.
  • inactivation of complement
    º¸Ã¼ºñµ¿È­.
  • indirect complement fixation test
    °£Á¢º¸Ã¼°áÇÕ½ÃÇè
  • DSM-I=Diagnostic and Statistical Manual of Mental Disorders-I
    Á¤½ÅÀå¾Ö(º´)Áø´ÜÅë°èÆí¶÷ Á¦ 1ÆÇ
  • Death and dying
    »ç¸Á°úÀÓÁ¾
  • ENT =ear, nose and throat
    À̺ñÀÎÈİú(ÇÐ)
  • General anesthesia, reticular activating system and.
    Àü½Å¸¶Ãë(îïãóئö­), ¸Á»óüȰ¼ºÈ­°è(ØÑßÒô÷üÀàõûùͧ)
  • Gravity, cardiac output and
    Áß·Â(ñìÕô), ½É¹ÚÃâ·®(ãýÚÑõóÕá)
  • KUB= kidney, ureter and bladder
    ½Å-´¢°ü-¹æ±¤ ´Ü¼øÃÔ¿µ
  • Kell antigen and antibody
    ÄÌÇ׿øÇ×ü
  • PACS (picture archiving and communicating system)
    ÆÑ½º, ¿µ»ó ÀúÀå ¹× Àü¼Û ü°è
  • PANSS=Positive and Negative Syndrome Scale
    ¾ç¼º À½¼º ÁõÈıº ôµµ
  • SADS, Schedule for Affective Disorders and Schizophrenia
    Á¤µ¿Àå¾Ö¿Í Á¤½ÅºÐ¿­º´ÀÇ Áø´ÜÀ» À§ÇÑ ¸ñ·Ï
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • inflammation,chemical mediators
    È­ÇÐ ¸Å°³¹°Áú(ûùùÊ ØÚ˿ڪòõ)
  • inflammation,cytopathic-cytoproliferative
    ¼¼Æ÷º´º¯-¼¼Æ÷Áõ½Ä¼º(á¬øàܻܨ-á¬øàñòãÖàõ)ÀÇ
  • inflammation,exudative
    ¼º(ß¶õóàõ), »ïÃ⼺ÀÇ
  • inflammation,kinin system
    Ű´Ñ °è, kinin °è(¡­ ͧ)
  • inflammation,leukocytic exudation
    ¹éÇ÷±¸¼º »ïÃâ¹°(ÛÜúìϹàõ ¡­)
  • inflammation,lysosomal constituents
    ¸®¼Ò¼Ø(¿ëÇØ¼Òü)((éÁú°á³ô÷) à÷ÝÂ)
  • inflammation,necrotizing
    ±«»ç¼º(ÎÕÞÝàõ)
  • inflammation,plasma proteases
    Ç÷Àå ÇÁ·ÎÅ×¾ÆÁ¦(úìíì ¡­)
  • inflammation,vascular permeability
    Åõ°ú¼º(úìη ÷âΦàõ)
  • interstitial inflammation
    °£Áú¼º ¿°(Áõ).(¡­ æú(ñø))
  • interstitital inflammation
    °£Áú¼º ¿°Áõ(¡­ æúñø)
  • mechanical inflammation =MI
    ±â°èÀû ¿°Áõ(¡­æúñø).
  • membranous inflammation
    ¸·¼º¿°Áõ(¡­àõæúñø)
  • metastatic inflammation
    ÀüÀ̼º ¿°Áõ(¡­æúñø).
  • necrotic inflammation =necrotizing i.
    ±«»ç¼º ¿°(ÎÕÞÝàõæú).(º´¸®) ±«»ç¼º ¿°Áõ.
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • Mesenteries and peritoneal folds
    âÀÚ°£¸· ¹× º¹¸·ÁÖ¸§
    [¿¾ ¿ë¾î] Àå°£¸·
  • Coeloms and septa
    ü°­°ú Áß°Ý
    [¿¾ ¿ë¾î] ü°­°ú Áß°Ý
  • Gray matter (Nuclei and Columns)
    ȸ»öÁú(½Å°æÇÙ°ú ½Å°æ±âµÕ)
    [¿¾ ¿ë¾î] ȸ¹éÁú
  • Cartilages and Articulations of Larynx
    Èĵο¬°ñ ¹× ÈĵΰüÀý
    [¿¾ ¿ë¾î] Èĵο¬°ñ ¹× ÈĵΰüÀý
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 8 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • "Koshland, Nemethy, and Filmer model"
    "ÄÚ½¬·»µå,³×¸ÞƼ,ÇÊ¸Ó ¸ðµ¨"
  • Lavoisier and Laplace law
    ¶óº¸¾ÆÁ¦ ¶óÇÁ¶óÀ̽º¹ýÄ¢(ÛööÎ)
  • lock and key theory
    ÀÚ¹°¼è-¿­¼èÀÌ·Ð(ìµÖå)
  • modification and restriction
    ¼ö½Ä(áóãÞ)°ú Á¦ÇÑ(ð¤ùÚ)
  • "Monod, Wyman, and Changeux model"
    ¸ð³ë.¿ÍÀ̸¸.¼§Á¶¸ðµ¨
  • Park and Johnson method
    ÆÄÅ© Á¸½¼ ¹ý(Ûö)
  • patch and cut repair
    Àý´Ü ºÎ ¼öº¹(ï·Ó¨Ý¾áóÜÖ)
  • stem-and-loop DNA
    ÁÙ±â- ·çÇÁ DNA
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 2 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • salt and pepper appearance
    ¼Ò±ÝÈÄÃß°¡·ç¸ð¾ç
  • SONAR [=Sound Navigation and Ranging]
    ¼Ò³ª
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 2
Inflamm inflammation, inflammatory
NAI net acid input; no accidental injury; no acute inflammation; nonadherence index
NIP nipple; no infection present; no inflammation present
NSI negative self-image; no signs of infection/inflammation; non-syncytium-inducing
SI International System of Units [Fr. le Systeme International d'Unites]; sacroiliac; saline infusion; ...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 2
CCP Complement Control Protein
CF Complement Fixation
CFT Complement Fixation
CFR Complement Fixation Reaction
CF Complement Fixation Test
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • inflammation of the maxillary sinus
    »ó¾Çµ¿¿°
    ºÎºñ°­ÀÇ ÇϳªÀÎ »ó¾Çµ¿ÀÇ ¿°Áõ. »ó¾Çµ¿Àº »ó¾Ç°ñ ¾È¿¡ ÀÖÀ¸¸ç ºñ Á¡¸·ÀÇ ¿¬ÀåÀ¸·Î µ¤ÀÎ ÀÚ¿¬ °øÀ¸·Î ºñ°­°ú ÅëÇϰí ÀÖ´Ù. ±× ¶§¹®¿¡ ºñ°­ ³»ÀÇ ¿°ÁõÀº ½±°Ô »ó¾Çµ¿ ¾ÈÀ¸·Î ÆÄ±ÞµÇ°í, ƯÈ÷ °¨±âÀÏ ¶§´Â ±Þ¼º »ó¾Çµ¿¿°À» ÀÏÀ¸ÄÑ ºñÁó Áõ°¡, µÎÅë, ¹ß¿­, ºñÆó µîÀÇ Áõ¼¼¸¦ ³ªÅ¸³»³ª, ÀÚ¿¬ °øÀÌ Àß °³¹æµÇ¾î ÀÖÀ» °æ¿ì¿¡´Â ºÐºñ¹°ÀÇ ¹è¼³ÀÌ ÃËÁøµÇ¾î ºñ±³Àû »¡¸® Ä¡À¯µÈ´Ù. ±×·¯³ª ¿©·¯ Áõ»óÀÌ °ãÄ¡¸é, ½±°Ô ¸¸¼º ¿°ÁõÀ¸·Î ÀÌÇàÇÏ¿© ³»ºÎ¿¡ ³óÁóÀÌ ±«°Ô µÈ´Ù
  • interstitial inflammation
    °£Áú¼º ¿°Áõ
    Á¶Á÷Àº ½ÇÁú°ú °£Áú·Î ÀÌ·ç¾îÁ®Àִµ¥ °£ÁúÀÇ Ç÷°ü, °áÇÕ Á¶Á÷¿¡ ¿°ÁõÀÌ »ý±â´Â °ÍÀ» ¸»ÇÑ´Ù. °£Áú¼º ½Å¿°, °£Áú¼º Æó·Å µîÀ¸·Î ÀÏÄþîÁö´Â °ÍÀÌ ´ëÇ¥ÀûÀÌ´Ù.
  • necrotic inflammation
    ±«»ç¼º ¿°, ±«»ç¼º ¿°Áõ
    µ¿ÀǾî=necrotizing inflammation.
  • neurogenic inflammation
    ½Å°æ¼º ¿°Áõ
  • nonspecific inflammation
    ºñƯÀ̼º ¿°Áõ
  • obliterative inflammation
    Æó»ö¼º ¿°Áõ
  • palatine inflammation
    ±¸°³¿°
    µ¿ÀǾî=
  • perimaxillary inflammation
    ¾Ç°ñ ÁÖÀ§¿°
  • pseudomembranous inflammation
    °¡¼º ¸· ¿°Áõ, °¡¼º À§¸·¼º ¿°Áõ
  • purulent inflammation
    È­³ó¼º ¿°Áõ
  • simple inflammation
    ´Ü¼ø ¿°Áõ
  • specific inflammation
    ƯÀ̼º ¿°Áõ
  • sterile inflammation
    ¹«±Õ¼º ¿°Áõ
  • subacute inflammation
    ¾Æ±Þ¼º ¿°Áõ
  • ulcerative inflammation
    ±Ë¾ç¼º ¿°Áõ
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
purulent inflammation An acute exudative inflammation in which the accumulation of polymorphonuclear leukocytes is sufficiently great that their enzymes cause liquefaction of the affected tissues, focally or diffusely; the purulent exudate is frequently termed pus, and consists of plasma and its constituents, end products of the enzymatic digestion of tissue, degenerated and necrotic cells and their debris, polymorphonuclear leukocytes and other white blood cells, the causal agent of the inflammation, etc.
Synonym: suppurative inflammation.
(05 Mar 2000)
serofibrinous inflammation Inflammation in which the exudate consists chiefly of serous fluid with an unusually large proportion of fibrin.
(05 Mar 2000)
serous inflammation An exudative inflammation in which the exudate is predominantly fluid (e.g., exuded from the blood vessels), with the protein, electrolytes, and other material contained therein; relatively few (if any) cells are observed.
(05 Mar 2000)
hyperplastic inflammation An inflammatory reaction in which the distinguishing feature is an actual increase in the number of tissue cells, especially the reticuloendothelial macrophages, in contrast to cells exuded from blood vessels; in addition, exudates of various types are likely to be observed in granulomas and other forms of proliferative inflammation, but the latter may occur without an exudate being formed (as in certain infections caused by virus).
Synonym: hyperplastic inflammation.
(05 Mar 2000)
necrotic inflammation Usually an acute inflammatory reaction in which the predominant histologic change is fairly rapid necrosis that occurs diffusely or extensively in relatively large foci throughout the affected tissue, frequently with only little or no evidence of cells in the exudate.
(05 Mar 2000)
subacute inflammation An inflammation that is intermediate in duration between that of an acute inflammation and that of a chronic inflammation, usually persisting longer than 3 or 4 weeks.
(05 Mar 2000)
suppurative inflammation An acute exudative inflammation in which the accumulation of polymorphonuclear leukocytes is sufficiently great that their enzymes cause liquefaction of the affected tissues, focally or diffusely; the purulent exudate is frequently termed pus, and consists of plasma and its constituents, end products of the enzymatic digestion of tissue, degenerated and necrotic cells and their debris, polymorphonuclear leukocytes and other white blood cells, the causal agent of the inflammation, etc.
Synonym: suppurative inflammation.
(05 Mar 2000)
degenerative inflammation A local reaction to injury, occasionally observed in the walls of blood vessels and in parenchymal cells of various organs in reacting to certain chemicals, viruses, and other intracellular agents; the response is characterised by degenerative changes in the cytoplasm and nucleus, frequently resulting in necrosis, but exudation (if any) is ordinarily observed only in the wall of the affected vessel, or in the interstices immediately adjacent to the affected vessel or parenchymal cells.
Synonym: degenerative inflammation.
(05 Mar 2000)
immune inflammation See: allergic reaction.
(05 Mar 2000)
inflammation <pathology> A localised protective response elicited by injury or destruction of tissues, which serves to destroy, dilute or wall off (sequester) both the injurious agent and the injured tissue.
It is characterised in the acute form by the classical signs of pain (dolor), heat (calor), redness (rubor), swelling (tumour) and loss of function (functio laesa).
Histologically, it involves a complex series of events, including dilatation of arterioles, capillaries and venules, with increased permeability and blood flow, exudation of fluids, including plasma proteins and leucocytic migration into the inflammatory focus.
Origin: L. Inflammatio, inflammare = to set on fire
(11 Jun 1998)
inflammation: gallium imaging <radiology> Pathophysiology: leakage of protein-bound Ga-67 into extracellular space secondary to increased capillary permeability, Ga-67 is preferentially bound to nonviable PMNs and macrophages, leukocyte incorporation (rich in lactoferrin), bacterial uptake (siderophores), inflammtory tissue stimulates lactoferrin production for chronic abdominal inflammation: 67% sensitivity; 64% specificity; 13% false negatives; 5% false positive, dose: 5 mCi; imaging: 24, 48, 72 hours, diffuse uptake in peritonitis, localised uptake in acute pyogenic abscess, phlegmon, acute cholecystitis, acute pancreatitis, acute gastritis, diverticulitis, inflammatory bowel disease, surgical wound, pyelonephritis, perinephric abscess see: gallium indications, gallium vs. Indium
(12 Dec 1998)
inflammation mediators The endogenous compounds that mediate inflammation (autacoids) and related exogenous compounds including the synthetic prostaglandins (prostaglandins, synthetic).
(12 Dec 1998)
interstitial inflammation Inflammation in which the inflammatory reaction occurs chiefly in the supportive fibrous connective tissue or stroma of an organ.
(05 Mar 2000)
exudative inflammation Inflammation in which the conspicuous or distinguishing feature is an exudate, which may be chiefly serous, serofibrinous, fibrinous, or mucous (e.g., relatively few cells are present), or may be characterised by relatively large numbers of neutrophils, eosinophils, lymphocytes, monocytes, or plasma cells, frequently with one or two types being predominant; it occurs not only as a separate and distinct pathologic process, but also frequently as a part of certain granulomatous inflammation's.
(05 Mar 2000)
fibrinopurulent inflammation A purulent inflammation in which the exudate contains an unusually large amount of fibrin; also, a fibrinous or serofibrinous inflammation in which the accumulation of large numbers of polymorphonuclear leukocytes results in liquefactive necrosis of tissue and the formation of pus with a relatively large quantity of fibrin.
(05 Mar 2000)
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  • ¿µ¹®
    ÇѱÛ
  • checks and balances
    °ßÁ¦¿Í ±ÕÇü
  • every now and then
    À̵û±Ý
  • here and there
    ¿©±âÀú±â¿¡
  • hide-and-go-seek
    ¼û¹Ù²ÀÁú
  • hide-and-seek
    ¼û¹Ù²ÀÁú
  • hole-and-corner
    ºñ¹ÐÀÇ
  • kith and kin
    ģôÁö±â, Àϰ¡Ä£Ã´
  • now and again
    À̵û±Ý
  • now and then
    ¶§¶§·Î (every ... ... ....)
  • off and on
    ºÒ±ÔÄ¢ÇϰÔ
  • on and on
    °è¼ÓÇÏ¿©
  • once and for all
    ¸¶Áö¸·À¸·Î
  • one and all
    ÇÑ »ç¶÷ ºüÁü¾øÀÌ
  • over and over again
    µÇÇ®ÀÌÇØ¼­
  • safe and sound
    ¹«»çÈ÷
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
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    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
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