¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"Cerebral Amyloid Angiopathy, Familial"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 2
¿µ¹® cerebral cortex ÇÑ±Û ´ë³ú°ÑÁú
¼³¸í   
  ´ë³úÀǠǥ¸éºÎÀ§¸¦ ´ë³ú°ÑÁúÀ̶ó°íµµ ÇÑ´Ù. ´ë°³ ¾èÀºÈ¸»öÁú(superficial gray matter)¿Í µ¿ÀǾî·Î ¾²ÀδÙ. È¸»öÁúÀ̶õ ´ë³úÀǠǥ¸é¿¡ ½Å°æ¼¼Æ÷°¡ ¸ð¿©Àִ °÷À¸·Î È¸»öÀ» ¶ì´Â ºÎºÐÀ» ¸»ÇÑ´Ù. À̿ʹ ´ëÁ¶ÀûÀ¸·Î ¹é»öÁúÀ̶õ ½Å°æ¼¼Æ÷°¡ ³»´Â ½Å°æ¼¶À¯°¡ ºÐÆ÷Çϴ °÷À̸破ë³ú¿¡¼­ È¸»öÁúÀÇ ¾ÈÂÊ¿¡ Á¸ÀçÇÑ´Ù. Âü°í·Î ¸»Çϸé Ã´¼ö¿¡¼­´Â ´ë³ú¿Í ¹Ý´ë·Î È¸»öÁú ¾ÈÂÊ¿¡ Á¸ÀçÇÏ°í ¹é»öÁúÀÌ ¹Û¿¡ Á¸ÀçÇÑ´Ù. Áï Ã´¼ö¿¡¼­´Â ½Å°æ¼¼Æ÷°¡ Ã´¼öÀÇ ¾ÈÂÊ¿¡ Á¸ÀçÇÏ°í ¹Ù±ùÂÊ¿¡ ±× ½Å°æ¼¼Æ÷°¡ ³»´Â ½Å°æ¼¶À¯°¡ Á¸ÀçÇÑ´Ù.
¿µ¹® cerebral artery ÇÑ±Û ´ë³úµ¿¸Æ
¼³¸í   
  ´ë³úÀÇ Ç÷¾×À» °ø±ÞÇϴ µ¿¸Æ. ´ë³úÀÇ ¾ÕºÎºÐÀÇ Ç÷¾×À» °ø±ÞÇϴ ¾Õ´ë³úµ¿¸Æ(anterior cerebral artery), Áß°£ºÎÀ§ÀÇ Ç÷¾×À» °ø±ÞÇϴ Áß°£´ë³úµ¿¸Æ(middle cerebral artery), µÞºÎºÐÀÇ Ç÷¾×À» °ø±ÞÇϴ µÚ´ë³úµ¿¸Æ(posterior cererbral artery)ÀÇ ¼¼ °¡Áö°¡ ÀÖ´Ù.
  
  ´ë³ú´Â Å©°Ô µÎ °¡Áö µ¿¸Æ¿¡ ÀÇÇØ¼­ Ç÷¾×À» °ø±Þ¹Þ´Â´Ù. Çϳª´Â ¼Ó¸ñµ¿¸Æ(internal carotid artery)À̰í Çϳª´Â Ã´Ãßµ¿¸Æ(vertebral artery)ÀÌ´Ù. ÀÌ µ¿¸ÆµéÀº ÁÂ-¿ì·Î µÎ °³¾¿ ½ÖÀ» ÀÌ·ç¾î ³ú¿¡ Ç÷¾×À» °ø±ÞÇÑ´Ù. Ã´Ãßµ¿¸ÆÀº ´ëµ¿¸Æ¿¡¼­ ³ª¿Í¼­ ¸ñ»À¿¡ Àִ °¡·Îµ¹±â±¸¸ÛÀ» ÅëÇØ¼­ ¸Ó¸®ÂÊÀ¸·Î ¿Ã¶ó¿Â´Ù. ±×¸®°í´Â µÎ °³ÀÇ Á¿ì Ã´Ãß°¡ ÇÑ °³·Î ÇÕÃÄÁ®¼­ ¹Ù´Úµ¿¸Æ¶ó´Â µ¿¸ÆÀ» ÀÌ·é´Ù. ¸ñµ¿¸ÆÀº ´ëµ¿¸Æ¿¡¼­ ±â½ÃÇÏ¿© ¼Ó¸ñµ¿¸Æ°ú ¹Ù±ù¸ñµ¿¸ÆÀ¸·Î ³ª´¶´Ù(³ª´µ±â ÀÌÀüÀ» ¿Â¸ñµ¿¸ÆÀ̶ó ÇÑ´Ù). ¹Ù±ù¸ñµ¿¸ÆÀº ¾ó±¼°ú ³ú¸¦ Á¦¿ÜÇÑ ¸Ó¸®ÀÇ ºÎÀ§¿¡ Ç÷¾×À» °ø±ÞÇϴ Ç÷°üÀ̰í, ¼Ó¸ñµ¿¸ÆÀº ³ú¿¡ Ç÷¾×À» °ø±ÞÇÑ´Ù.
  
  ´ë³úµ¿¸Æ
  
  ´ë³úµ¿¸Æ
¿µ¹® cerebral hemisphere ÇÑ±Û ´ë³ú¹Ý±¸
¼³¸í   
  ´ë³ú¶õ ³úÀÇ °¡Àå Å« ºÎºÐÀ» Â÷ÁöÇϴ °÷À¸·Î »ç°í, ¿îµ¿, ¼º°Ý, ±â¾ï µîÀÇ °íÂ÷¿øÀûÀΠ±â´ÉÀ» ÇàÇϴ °÷ÀÌ´Ù. ´ë³ú´Â Å©°Ô ÁÂ, ¿ì µÎ °³·Î ³ª´µ¾îÁ® ÀÖ°í °¢°¢À» ÁÂ, ¿ì ´ë³ú¹Ý±¸¶ó°í ÇÑ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • familial hemophagocytic lymphohistiocytosis
    °¡Á·¼ºÀûÇ÷±¸Æ÷½Ä¼º¸²ÇÁÁ¶Á÷±¸Áõ½ÄÁõ
  • familial paroxysmal polyserositis
    °¡Á·¼º¹ßÀÛ´Ù¹ßÀ帷¿°
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
  • familial progressive hearing loss
    °¡Á·¼ºÁøÇ೭û
  • familial tremor
    °¡Á·¼º¶³¸²
  • idiopathic familial fibromatosis
    Ư¹ß°¡Á·¼¶À¯Á¾Áõ
  • anterior cerebral artery
    ¾Õ´ë³úµ¿¸Æ, Àü´ë³úµ¿¸Æ
  • cerebral
    ´ë³ú-, ³ú-, ³ú¼º-
  • cerebral amaurosis
    ³ú¼ºÈæ¾Ï½Ã
  • cerebral aneurysm
    ³úµ¿¸Æ²Ê¸®, ³úµ¿¸ÆÀÚ·ç, ³úµ¿¸Æ·ù
  • cerebral angiogram
    ³úÇ÷°üÁ¶¿µ»ó
  • cerebral angiography
    ³úÇ÷°üÁ¶¿µ(¼ú)
  • cerebral apoplexy
    ³úÁ¹Áß, ³úÁßdz
  • cerebral aqueduct
    ´ë³ú¼öµµ°ü
  • cerebral arterial circle
    ´ë³úµ¿¸Æ°í¸®
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • familial hyperlipoproteinemia
    °¡Á·°íÁö¹æ´Ü¹éÇ÷Áõ
  • familial hyperuricemia
    °¡Á·°í´¢»êÇ÷Áõ
  • familial tremor
    (¢¡essential tremor) º»Å¶³¸², À¯Àü¶³¸², ¿øÀθ𸦶³¸²
  • familial adenomatous polyposis
    °¡Á·»ùÁ¾Æú¸³Áõ
  • familial paroxysmal polyserositis
    °¡Á·¹ßÀÛ´Ù¹ßÀ帷¿°
  • familial progressive hearing loss
    °¡Á·ÁøÇ೭û
  • idiopathic familial fibromatosis
    Ư¹ß°¡Á·¼¶À¯Á¾Áõ
  • transient familial neonatal hyperbilirubinemia
    Àϰú¼º°¡Á·½Å»ý¾Æ°íºô¸®·çºóÇ÷Áõ
  • cerebral amaurosis
    ³ú¼ºÈæ¾Ï½Ã
  • cerebral aneurysm
    ³úµ¿¸Æ²Ê¸®, ³úµ¿¸ÆÀÚ·ç, ³úµ¿¸Æ·ù
  • cerebral angiogram
    ³úÇ÷°üÁ¶¿µ»ó
  • cerebral angiography
    ³úÇ÷°üÁ¶¿µ¼ú
  • cerebral apoplexy
    ³úÁßdz
  • cerebral aqueduct
    (¢¡mesencephalic aqueduct) Áß°£³ú¼öµµ°ü
  • cerebral artery
    ´ë³úµ¿¸Æ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • hyperbilirubinemia,asymptomatric familial
    ¹«ÁõÈÄ °¡Á·¼º(Ùíñøý¦ Ê«ðéàõ)
  • hypercholesterolemia,familial
    °¡Á·¼º(Ê«ðéàõ)
  • idiopathic familial fibromatosis
    Ư¹ß¼º °¡Á·¼º ¼¶À¯Á¾Áõ.
  • idiopathic familial fibromatosis
    Ư¹ß¼º °¡Á·¼º ¼¶À¯Á¾Áõ
  • infantile amaurotic familial idiocy
    ¿µ¾Æ¼º Èæ³»À强 °¡Á·¼º ¹éÄ¡.
  • infantile amaurotic familial idiocy
    ¿µ¾Æ¼º Èæ³»À强 °¡Á·¼º ¹éÄ¡.
  • CVR=£¾cerebral vascular resistance
    ³úÇ÷°ü(Òàúìη)ÀúÇ×(î½ù÷).
  • CVR=£¾cerebral vascular resistance
    ³úÇ÷°üÀúÇ×.
  • anterior cerebral artery
    ¾Õ´ë³úµ¿¸Æ
  • anterior cerebral veins
    ¾Õ´ë³úÁ¤¸Æ
  • artery, occipital branches of posterior cerebral
    ÈÄ´ë³úµ¿¸ÆÀÇ ÈĵÎÁö{ÇØ}
  • great cerebral vein
    Å«´ë³úÁ¤¸Æ
  • hereditary cerebral hemorrhages with amyloidosis(hchwa)
    À¯Àü¼º ³úÃâÇ÷, ¾Æ¹Ð·ÎÀ̵åÁõ¼º
  • infantile cerebral paralysis
    ¿µ¾Æ(¼º) ³ú¼º¸¶ºñ(?ä®àõÒààõØ«Ýö).
  • inferior cerebral veins
    ¾Æ·¡´ë³úÁ¤¸Æ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • papular amyloid elastosis
    ±¸Áø¼º À¯ÀüºÐ ź·Â ¼¶À¯Áõ
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼º Èæ³»Àå ¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼ºÈæ¾Ï½Ã¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
  • benign familial pemphigus
    ¾ç¼º °¡Á·¼º(åÐàõ Ê«ðéàõ) õÆ÷â(ô¸øÞóê)
  • breast/ovarian familial cancer syndrome
    À¯¹æ/³­¼Ò °¡Á·¼º ¾ÏÁõÈıº
  • chronic familial jaundice
    ¸¸¼º°¡Á·¼º Ȳ´Þ(¡­Ê«ðéàõüÜÓ¸).
  • diffuse familial comedo
    ±¤¹üÀ§ °¡Á·¼º ¸éÆ÷
  • dysbetalipoproteinemia.familial
    °¡Á·¼ºÀÌ»óº£Å¸¸®Æ÷ÇÁ·ÎÅ×ÀÎÇ÷Áõ
  • endogenous familial hypertriglyceridemia
    ³»Àμº °¡Á·¼º °íÆ®¸®±Û¸®¼¼¸®µå Ç÷Áõ
  • familial
    °¡Á·¼º(Ê«ðéàõ)ÀÇ
  • familial adenomatous polyposis
    °¡Á·¼º¼±Á¾¼º¿ëÁ¾Áõ.
  • familial adenomatous polyposis
    °¡Á·¼º ¼±Á¾¼º Æú¸³Áõ
  • familial amaurotic idiocy
    °¡Á·¼ºÈæ¾Ï½Ã¹éÄ¡
  • familial amaurotic idiocy<³ª> idiotia
    °¡Á·¼º Èæ³»Àå(¼º) ¹éÄ¡(¡­ýÙÒ®î¡
  • familial amyloidotic polyneuropathy
    °¡Á·Àû ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß½Å°æº´Áõ.
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 10 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • Inferior cerebral veins
    ¾Æ·¡´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] ÇÏ´ë³úÁ¤¸Æ
  • Anterior cerebral artery
    ¾Õ´ë³úµ¿¸Æ
    [¿¾ ¿ë¾î] Àü´ë³úµ¿¸Æ
  • Anterior cerebral veins
    ¾Õ´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] Àü´ë³úÁ¤¸Æ
  • Superficial cerebral vein
    ¾èÀº´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] õ´ë³úÁ¤¸Æ
  • Superficial middle cerebral vein
    ¾èÀºÁß°£´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] õÁß´ë³úÁ¤¸Æ
  • Superior cerebral veins
    À§´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] »ó´ë³úÁ¤¸Æ
  • Mesencephalic aqueduct [Cerebral aqueduct]
    Áß°£³ú¼öµµ°ü
    [¿¾ ¿ë¾î] Áß³ú¼öµµ
  • Middle cerebral artery
    Áß°£´ë³úµ¿¸Æ
    [¿¾ ¿ë¾î] Áß°£´ë³úµ¿¸Æ
  • Middle cerebral artery
    Áß°£´ë³úµ¿¸Æ
    [¿¾ ¿ë¾î] Áß´ë³úµ¿¸Æ
  • Great cerebral vein
    Å«´ë³úÁ¤¸Æ
    [¿¾ ¿ë¾î] ´ë´ë³úÁ¤¸Æ
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
  • cerebral vascular accident
    ³úÇ÷°ü»ç°í
  • cerebral vascular disease
    ³úÇ÷°üÁúȯ
  • middle cerebral artery
    Áß´ëÁ˵¿¸Æ
  • posterior cerebral artery
    µÚ´ë³úµ¿¸Æ, ÈÄ´ë³úµ¿¸Æ
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 2
FAP Familial Amyloid Polyneuropathy
FHH Familial Hypocalciuric Hypercalcemia
  = Familial Benign Hypercalcemia
FAD familial Alzheimer dementia; familial autonomic dysfunction; fetal activity-acceleration determinati...
FINCC familial idiopathic nonarteriosclerotic cerebral calcification
CA anterior commissure [Lat. commissura anterior]; calcium antagonist; California [rabbit]; cancer; Can...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 2
AA Amyloid A
AA Amyloid A protein
AP Amyloid P component
APP Amyloid Precursor Protein
APLP amyloid precursor-like protein
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • familial hemolytic anemia
    °¡Á·¼º ¿ëÇ÷¼º ºóÇ÷
  • familial histocytic dermatoarthritis
    °¡Á·¼º Á¶Á÷±¸¼º ÇǺΠ°üÀý¿°
  • familial hyperlipoproteinemia
    °¡Á·¼º °úÁöÁú´Ü¹éÇ÷Áõ
  • familial hypophosphatemic rickets

    familial leiomyomatosis cutis et uteri (°¡Á·¼º ÇǺΠÀڱà ±ÙÁ¾Áõ

  • familial Mediterranean fever
    °¡Á·¼º ÁöÁßÇØ ¿­
  • familial neutropenia
    °¡Á·¼º È£Áß±¸ °¨¼ÒÁõ
  • familial polyposis
    °¡Á·¼º Æú¸³Áõ
  • familial progressive sensorineural hearing loss
    °¡Á·¼º ÁøÇ༺ °¨°¢ ½Å°æ¼º ³­Ã»
  • familial teleangiectasis
    °¡Á·¼º ¸»ÃÊÇ÷°ü È®Àå, °¡Á·¼º ¸»ÃÊÇ÷°ü È®ÀåÁõ
  • fatal familial insomnia
    Ä¡¸íÀû °¡Á·¼º ºÒ¸éÁõ
    ºÒ¸éÁõ°ú ÀÚÀ²½Å°æ Àå¾Ö°¡ ÁÖ Áõ»óÀÌ°í ¸ðµç ȯÀÚ¿¡¼­ PrP À¯ÀüÀÚÀÇ º¯À̸¦ È®ÀÎÇÒ ¼ö ÀÖ´Ù. ÇüÅÂÇÐÀû ¼Ò°ßÀº º¸Åë ½Ã»ó¿¡¼­¸¸ °üÂûµÇ´Âµ¥ ½Å°æ ¼¼Æ÷ÀÇ ¼Ò½Ç°ú ´õºÒ¾î ±³¼¼Æ÷°¡ Áõ½ÄÇϰí ÇØ¸é»ó º¯È­¸¦ º¸ÀδÙ.
  • acute cerebral anemia
    ±Þ¼º ³ú ºóÇ÷
  • anterior cerebral artery
    Àü´ë³úµ¿¸Æ
    ³»°æµ¿¸Æ¿¡¼­ ±â¿øÇϸç, ÇÇÁúÁö[¾È¿Í, ÀüµÎ, µÎÁ¤ºÎ], Àü¸Æ¶ô ÃÑÁö ¹× Áß½ÉÁö[³»Ãø¼±Á¶Ã¼µ¿¸ÆÀ» Æ÷ÇÔ], Àü±³±³Å뵿¸ÆÀÇ ºÐÁö¸¦ ³»¸ç, ¾È¿Í, ÀüµÎ¿±, µÎÁ¤¿±ÀÇ ÇÇÁú, ³ö·®, °£³ú, ¼±Á¶Ã¼ ³»Æ÷, Ãø³ú½ÇÀÇ ¸Æ¶ôÃÑ¿¡ ºÐÆ÷ÇÑ´Ù.
  • cerebral abscess
    ´ë³ú ³ó¾ç
    È­³ó¼º ¼¼±Õ¿¡ ÀÇÇÏ¿© ¹ß»ýÇÏ´Â ³úÀÇ ±¹¼ÒÀûÀÎ È­³ó¼º ±«»ç¼º º´º¯ÀÌ´Ù. ´ëºÎºÐÀÇ ÁßÃ߽Űæ°èÀÇ ¼¼±Õ¼º °¨¿°Àº ¼ö¸·¿°ÀÇ ÇüÅ·Π°¨¿°µÇ¾î ³ú ½ÇÁú³»·Î ÆÄ±ÞµÇ¸é ¼ö¸·³ú¿°À» ÀÏÀ¸Å°Áö¸¸ ¼ö¸·¿° ¾øÀÌ ³ú ½ÇÁúÀÌ Á÷Á¢ ¼¼±Õ¿¡ °¨¿°µÇ±âµµ Çϴµ¥ À̶§ ´ëºÎºÐ ³úÀÇ ÀϺο¡ ±¹ÇѵǴ ±¹¼ÒÀû ³ú¿°À» ÀÏÀ¸Å°¸ç ÀÌ º´º¯ÀÌ ÁøÇàµÇ¸é ³ú ³ó¾çÀ» Çü¼ºÇÏ°Ô µÈ´Ù. ±¹¼ÒÀûÀÎ ½Å°æ Áõ»óÀ» ÀÏÀ¸Å°´Â ¿Ü¿¡µµ ±× ÀÚü°¡ ÀÏÁ¾ÀÇ °ø°£ Á¡À¯¼º º´º¯ÀÌ°í ¶ÇÇÑ ºÎÁ¾À» µ¿¹ÝÇϹǷΠ±×¿¡ µû¸¥ ³ú¾Ð »ó½Â¿¡ ÀÇÇÑ Áõ»óÀÌ ³ªÅ¸³­´Ù. ½ÉÇÑ °æ¿ì¿¡´Â Ä¡¸íÀûÀÎ ³ú Å»ÃâÀ» ÀÏÀ¸Å³ ¼ö ÀÖ´Ù. ¶ÇÇÑ ³ó¾çÀÌ ÅÍÁö¸é ÅÍÁø ¹æÇâ¿¡ µû¶ó ³ú½Ç¿°, ¼ö¸·¿° ¹× Á¤¸Æµ¿ Ç÷ÀüÁõ µîÀ» ÀÏÀ¸Å°±âµµ ÇÑ´Ù.
  • cerebral aneurysm
    ³ú µ¿¸Æ·ù
    ³ú µ¿¸ÆÀÇ ÀϺο¡ °á¼ÕÀÌ »ý°Ü ±× ºÎºÐÀÌ µ¹ÃâµÈ °Í. ¹æÃß»ó µ¿¸Æ·ù¿Í ³¶»ó µ¿¸Æ·ù°¡ ÀÖ´Ù. ÀüÀÚ´Â ³»°æ µ¿¸Æ, Ãß°ñ µ¿¸Æ, ³úÀú µ¿¸Æ µî¿¡¼­ º¼ ¼ö ÀÖ°í Ä¿Áö¸é ³ú Á¾¾ç°ú °°Àº ¾Ð¹Ú Áõ¼¼¸¦ ³ªÅ¸³»±âµµ ÇÑ´Ù. ÈÄÀÚ´Â ÆÄ¿­µÇ¾î ¸·ÇÏÃâÇ÷À» ÀÏÀ¸Å°¸ç Àü±³Åë µ¿¸Æ, ³»°æ µ¿¸Æ°ú Èı³Åë µ¿¸ÆÀÇ ±â½ÃºÎ, Áß´ë³úµ¿¸Æ ÈÄ´ë³úµ¿¸Æ µî¿¡¼­ º¼ ¼ö ÀÖ´Ù. ´ë³úµ¿¸Æ·û Àü¹ÝºÎ¿¡ ¾à 80 %°¡ Çü¼ºµÈ´Ù. ¿øÀÎÀº ¼±ÃµÀû ¿äÀÎÀ¸·Î »ý°¢µÈ´Ù. µ¿¸Æº®±ÙÃþÀÇ °á¿©, ³»Åº·Â¸·ÀÇ °á¼Õ µîÀÌ ÀÖÀ¸¸é, ±× ºÎÀ§´Â Ç÷·ù¿¡ ´ëÇÏ¿© ÀúÇ×ÀÌ ¾àÇØÁö°í È®ÀåµÇ¾î µ¿¸Æ·ù¸¦ Çü¼ºÇÑ´Ù. Ç÷°üº®ÀÇ ÀϺο¡ ¿°ÁõÀ̳ª °æÈ­¼º º´º¯ÀÌ »ý°Ü µ¿¸Æ·ù°¡ Çü¼ºµÇ±âµµ ÇÑ´Ù. µ¿¸Æ·ù´Â Ç÷°üº® ³»ÀÇ ÀûÀº ÃâÇ÷À» °ÅµìÇϸ鼭 Ä¿Áø´Ù. µ¿¸Æ·ù ±× ÀÚüÀÇ ¾Ð¹ÚÀ¸·Î µ¿¾È ½Å°æ ¸¶ºñ¸¦ ÀÏÀ¸Å°´Â °æ¿ìµµ ÀÖÁö¸¸, ´ëºÎºÐÀº Áõ¼¼°¡ ¾ø°í °©ÀÚ±â ÆÄ¿­µÇ¾î ÁöÁÖ¸·ÇÏ ÃâÇ÷À» ÀÏÀ¸Å²´Ù.
  • cerebral anoxia
    ³ú ¹«»ê¼ÒÁõ
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 2
amyloid nephrosis The nephrotic syndrome due to deposition of amyloid in the kidney.
See: renal amyloidosis.
(05 Mar 2000)
amyloid neuropathies Disorders of the peripheral nervous system associated with deposition of amyloid. Amyloid neuropathies may result from non-hereditary or hereditary amyloidosis. Several different forms of familial amyloid neuropathies have been described, most of which have specific mutations in the prealbumin gene.
(12 Dec 1998)
amyloid p component Amyloid p component is a small, non-fibrillar glycoprotein found in normal serum and in all amyloid deposits. It has a pentagonal (pentaxin) structure. It acts as an acute phase protein in the mouse, modulates immunologic responses in man, inhibits elastase, and has been suggested as an indicator of liver disease.
(12 Dec 1998)
amyloid precursor protein <protein> Individuals with Alzheimer's disease are characterised by extensive accumulation of amyloid in the brain, referred to as senile plaques. These consist of a core of amyloid fibrils surrounded by dystrophic neurites. The principal component of the amyloid fibrils is B/A4, a peptide derived from the larger APP. The specific role of amyloid protein is unclear but it is thought that amyloid deposits may cause neurons to degenerate. Amyloid deposits also occur in brains of older Down's Syndrome patients.
(04 May 1997)
amyloid protein Glycoprotein deposited extracellularly in tissues in amyloidosis. The glycoprotein may either derive from light chain of immunoglobulin (AIO (amyloid of immune origin): 5-18 kD glycoprotein, product of a single clone of plasma cells, the N terminal part of lambda or kappa light chain) or, in what used to be referred to as AUO, amyloid of unknown origin, from serum amyloid A (SAA), one of the acute phase proteins that increases many fold in inflammation. The polypeptides are organised as a _ pleated sheet making the material rather inert and insoluble. Minor protein components are also found. Should be distinguished from _ amyloid deposited in the brain and that is derived from amyloid precursor protein (see amyloidogenic glycoprotein.
(18 Nov 1997)
amyloid protein aa A nonimmunoglobulin amyloid isolated from amyloid fibrils deposited in amyloidosis secondary to chronic inflammatory diseases such as rheumatoid arthritis. Antisera to amyloid protein aa have been used to detect a related serum protein saa.
(12 Dec 1998)
amyloid protein saa A serum protein believed to be a circulating precursor to amyloid protein aa. It is present in low concentrations in normal sera, but found in much higher concentrations in sera of older persons and in patients with amyloidosis or with diseases known to predispose to amyloidosis. Very high levels of this protein have been reported during acute inflammatory episodes. Antisera to amyloid protein aa cross-react with protein saa.
(12 Dec 1998)
amyloid tumour A localised form of amyloidosis in which amyloid occurs as masses or nodules beneath the skin or mucous membranes, e.g., in the larynx.
Synonym: amyloid tumour, focal amyloidosis.
(05 Mar 2000)
beta-amyloid <protein> The protein which forms thick deposits, or plaques, in the brains of people with Alzheimer's disease, a disease where memory skills gradually deteriorate with age.
(13 Nov 1997)
serum amyloid In secondary amyloidosis the fibrils deposited in tissues are unrelated to immunoglobulin light chains (in contrast to the situation in primary amyloidosis) and are made of amyloid A protein (AA protein). This is derived from serum amyloid A (SAA) that is the apolipoprotein of a high density lipoprotein and an acute phase protein. Partial proteolysis converts SAA into the pleated sheet configuration of the amyloid fibrils. Amyloid P protein is also found as a minor component of the fibrils (in both primary and secondary amyloidosis) and is derived from serum amyloid P that has similarity to C-reactive protein. The physiological role remains obscure.
(18 Nov 1997)
serum amyloid P component Precursor of amyloid component P, found in basement membrane. Member of the pentraxin family.
See: serum amyloid.
(18 Nov 1997)
islet amyloid peptide <hormone, protein> Peptide of 37 amino acids that selectively inhibits insulin stimulated glucose uptake in muscle. Structurally related to calcitonin gene-related peptide.
(15 Oct 1997)
benign familial chorea A rare, nonprogressive movement disorder characterised by chorea and athetosis appearing in early childhood, most commonly manifested as gait ataxia and upper limb coordination. Intellect is unaffected. Probably autosomal-dominance inheritance with incomplete penetrance.
(05 Mar 2000)
benign familial chronic pemphigus Recurrent eruption of vesicles and bullae that become scaling and crusted lesions with vesicular borders, predominantly of the neck, groin, and axillary regions; autosomal dominant inheritance, presenting in late adolescence or early adult life.
Synonym: Hailey-Hailey disease.
(05 Mar 2000)
benign familial icterus Mild jaundice due to increased amounts of unconjugated bilirubin in the plasma without evidence of liver damage, biliary obstruction, or haemolysis; thought to be due to an inborn error of metabolism in which the excretion of bilirubin by the liver is defective, ascribed to decreased conjugation of bilirubin as a glucuronide or impaired uptake of hepatic bilirubin.
Synonym: benign familial icterus, constitutional hepatic dysfunction, Gilbert's disease, Gilbert's syndrome, Hebra's disease.
(05 Mar 2000)
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