| HAA | hearing aid amplifier; hemolytic anemia antigen; hepatitis-associated antigen; hospital activity ana... |
|---|---|
| HA | Ag hepatitis A antigen |
| HB | health board; heart block; heel to buttock; held back; hemoglobin; hepatitis B; His bundle; hold bre... |
| HBAb | hepatitis B antibody |
| HBAg | hepatitis B antigen |
| CA virus | <abbreviation> Croup-associated virus. (05 Mar 2000) |
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| measles virus | <virology> Paramyxovirus that causes the childhood disease measles and is responsible for subacute sclerosing panencephalitis. (18 Nov 1997) |
| measles virus vaccine | Vaccine containing live, attenuated strains of measles virus prepared in chick embryo cell culture. See: measles, mumps, and rubella vaccine. (05 Mar 2000) |
| virus | <virology> Viruses are obligate intracellular parasites of living but noncellular nature, consisting of DNA or RNA and a protein coat. They range in diameter from 20-300nm. Class I viruses (Baltimore classification) have double stranded DNA as their genome. Class II have a single stranded DNA genome. Class III have a double stranded RNA genome. Class IV have a positive single stranded RNA genome, the genome itself acting as mRNA. Class V have a negative single stranded RNA genome used as a template for mRNA synthesis. Class VI have a positive single stranded RNA genome but with a DNA intermediate not only in replication but also in mRNA synthesis. The majority of viruses are recognised by the diseases they cause in plants, animals and prokaryotes. Viruses of prokaryotes are known as bacteriophages. (13 Oct 1997) |
| virus activating protease | <enzyme> Endoprotease from chick embryo; activates para- and orthomyoxovirus fusion glycoproteins by cleaving them at a single arginine site; ca(2+) dependent; similar to blood clotting factor x Registry number: EC 3.4.21.- (26 Jun 1999) |
| virus activation | The mechanism by which latent viruses, such as genetically transmitted tumour viruses or prophages of lysogenic bacteria, are induced to replicate and are released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell lipopolysaccharides, glucocorticoid hormones, halogenated pyrimidines, ionizing radiation, ultraviolet light, and superinfecting viruses. (12 Dec 1998) |
| virus assembly | The assembly of viral capsid proteins and nucleic acid to form a viral particle (virion). (12 Dec 1998) |
| virus-associated haemophagocytic syndrome | <syndrome> A syndrome closely resembling malignant histiocytosis but potentially reversible, following a herpes group virus infection such as by the Epstein-Barr virus. (05 Mar 2000) |
| virus blockade | The interference of one virus by another, either attenuated or unrelated. (05 Mar 2000) |
| virus diseases | A general term for diseases produced by viruses. (12 Dec 1998) |
| virus, human papilloma | A family of over 60 viruses responsible for causing warts. The majority of the viruses produce warts on the hands, fingers, and even the face. most of these viruses are innocuous, causing nothing more than cosmetic concerns. Several types of HPV are confined primarily to the moist skin of the genitals, producing genital warts and elevating the risk for cancer of the cervix. These viruses that cause wartlike growths on the genitals and contribute to cancer of the cervix are sexually transmitted. (12 Dec 1998) |
| virus III of rabbits | An obsolete name for a latent herpesvirus infection of rabbits. Origin: the third strain isolated, used for study (05 Mar 2000) |
| virus integration | Insertion of viral DNA into host-cell DNA. This includes integration of phage DNA into bacterial DNA (lysogeny) to form a prophage or integration of retroviral DNA into cellular DNA to form a provirus. (12 Dec 1998) |
| virus keratoconjunctivitis | Follicular conjunctivitis followed by subepithelial corneal infiltrates; often caused by adenovirus type 8, less commonly by other types. Synonym: virus keratoconjunctivitis. (05 Mar 2000) |
| virus latency | The ability of a pathogenic virus to lie dormant within a cell (latent infection). In eukaryotes, subsequent activation and viral replication is thought to be caused by extracellular stimulation of cellular transcription factors. Latency in bacteriophage is maintained by the expression of virally encoded repressors. (12 Dec 1998) |