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"acute lymphoblastic leukemia, null cell type"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • chief cell
    À¸¶ä¼¼Æ÷
  • chromaffin cell
    Å©·Òģȭ¼¼Æ÷, ģũ·Ò¼¼Æ÷
  • chromophilic cell
    »ö¼Òµê¼¼Æ÷, È£»ö¼Ò¼¼Æ÷
  • chromophobic cell
    »ö¼Ò¾Èµê¼¼Æ÷
  • duct cell carcinoma
    °ü¼¼Æ÷¾ÏÁ¾
  • dust cell
    ¸ÕÁö¼¼Æ÷
  • delayed cell-mediated reaction
    Áö¿¬¼¼Æ÷¸Å°³¹ÝÀÀ
  • delta cell
    µ¨Å¸¼¼Æ÷
  • dendritic cell
    °¡Áö¼¼Æ÷, ¼öÁö»ó¼¼Æ÷
  • dark cell
    ¾îµÒ¼¼Æ÷
  • daughter cell
    µþ¼¼Æ÷
  • differentiated cell
    ºÐÈ­¼¼Æ÷
  • diffuse large B-cell lymphoma
    ±¤¹üÀ§Å«B¼¼Æ÷¸²ÇÁÁ¾
  • diploid cell
    µÎ¹è¼öü¼¼Æ÷
  • diploid cell line
    µÎ¹è¼öü¼¼Æ÷°è, À̹èü¼¼Æ÷°è
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  • ¿µ¹®
    ÇѱÛ
  • cytotoxic cell
    ¼¼Æ÷µ¶¼º¼¼Æ÷
  • cytotoxic cell protein
    ¼¼Æ÷µ¶¼º¼¼Æ÷´Ü¹é
  • dark cell
    ¾îµÒ¼¼Æ÷
  • daughter cell
    µþ¼¼Æ÷
  • dendritic cell
    °¡Áö¼¼Æ÷
  • differentiated cell
    ºÐÈ­¼¼Æ÷
  • diploid cell
    µÎ¹è¼öü¼¼Æ÷
  • duct cell carcinoma
    °ü¼¼Æ÷¾ÏÁ¾
  • dust cell
    ¸ÕÁö¼¼Æ÷
  • effector cell
    ÀÛµ¿¼¼Æ÷
  • egg cell
    ³­¼¼Æ÷, ¾Ë¼¼Æ÷
  • endothelial cell
    ³»ÇǼ¼Æ÷
  • enterochromaffine cell
    âÀÚģũ·Ò¼¼Æ÷, âÀÚÅ©·Òģȭ¼¼Æ÷
  • eosinophilic cell
    È£»ê¼¼Æ÷
  • ependymal cell
    ³ú½Ç¸·¼¼Æ÷
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  • ¿µ¹®
    ÇѱÛ
  • acute myocarditis
    ±Þ¼º ½É±Ù¿°(¡­ãýÐÉæú).
  • acute myopathy
    ±Þ¼º ±Ùº´Áõ(¡­ÐÉÜ»ñø).
  • acute myopathy
    ±Þ¼º ±Ùº´Áõ(¡­ÐÉÜ»ñø).
  • acute necrotic otitis media
    ±Þ¼º ±«»ç(¼º) ÁßÀÌ¿°
  • acute necrotizing enterocolitis
    ±Þ¼º ±«»ç¼º ÀüÀå¿°(¡­ÎÕÞÝàõîï æú).
  • acute necrotizing enterocolitis
    ±Þ¼º±«»ç¼ºÀ§Àå¿°(¡­ÎÕÞÝàõêÖíóæú).
  • acute necrotizing hemorrhagic encephalomyelitis
    ±Þ¼º ±«»çÃâÇ÷¼º ³úô¼ö¿°(¡­ÎÕÞÝõóúìàõÒàô±âÐæú).
  • acute necrotizing retinitis
    ±Þ¼º±«»ç¸Á¸·¿°
  • acute necrotizing ulcerative gingivitis
    ±Þ¼º±«»ç±Ë¾ç¼º Ä¡Àº¿°
  • acute necrotizing ulcerative gingivitis
    ±Þ¼º ±«»ç±Ë¾ç¼º Ä¡Àº¿°
  • acute nephritis
    ±Þ¼º ½Å¿°(Ðáàõãìæú).
  • acute obliterating bronchiolitis
    ±Þ¼º Æó¼â¼º ¼¼±â°üÁö¿°(¡­øÍáðàõá¬Ñ¨Î·ò¨æú).
  • acute orchitis
    ±Þ¼º °íȯ¿°
  • acute osteomyelitis
    ±Þ¼º °ñ¼ö¿°(ÐáàõÍéâÐæú).
  • acute pancreatitis
    ±Þ¼º ÃéÀå¿°(¡­õýíôæú).
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 16
  • ¿µ¹®
    ÇѱÛ
  • hypogammaglobulinemia, Swiss-type
    ½ºÀ§½ºÇü °¨¸¶±Û·ÎºÒ¸°ÀúÇ÷Áõ
  • intermediate type
    Áß°£Çü(ñéÊàúþ) ¡ìÇãÇ÷¼º Áúȯ(úÈúìàõòðü´)ÀÇ¡í.
  • intracanalicular type
    ¼Ò°ü³»Çü
  • introversion type
    ³»ÇâÇü(Ò®ú¾úþ)
  • intuitive type
    Á÷°üÇü(òÁκúþ)
  • irregular type
    ºÒ±ÔÄ¢Çü
  • jealous type
    ÁúÅõÇü(òì÷àúþ)
  • lepromatous type
    ³ªÁ¾Çü(ÑÛðþúþ)
  • lepromatous type
    ³ªÁ¾Çü(ÑÛðþúþ).
  • leptosomatic type
    ¸¶¸¥Çü(¡­úþ).
  • limb girdle type
    Áö´ëÇü.
  • linear array type
    ¼±Çü ¹è¿­½Ä (àÊû¡ ÛÕÖªãÒ)
  • linear type constitution
    ¼±Ã¼Çü.
  • lymphatic type
    ¸²ÇÁÇü(¡­úþ).
  • mating type
    ±³¹èÇü
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  • ¿µ¹®
    ÇѱÛ
  • Goblet cell
    ¼úÀܼ¼Æ÷
    [¿¾ ¿ë¾î] ¹è»ó[Á¡¾×]¼¼Æ÷
  • Neurosensory cell
    ½Å°æ°¨°¢¼¼Æ÷
    [¿¾ ¿ë¾î] ½Å°æ°¨°¢¼¼Æ÷
  • Neurosensory cell body
    ½Å°æ°¨°¢¼¼Æ÷ü
    [¿¾ ¿ë¾î] ½Å°æ°¨°¢¼¼Æ÷ü
  • Nerve cell [Neuron]
    ½Å°æ¼¼Æ÷ [½Å°æ´ÜÀ§]
    [¿¾ ¿ë¾î] ½Å°æ¼¼Æ÷
  • Nerve cell body
    ½Å°æ¼¼Æ÷ü
    [¿¾ ¿ë¾î] ½Å°æ¼¼Æ÷ü
  • Cone cell
    ¿ø»Ô¼¼Æ÷
    [¿¾ ¿ë¾î] Ãß»óü½Ã¼¼Æ÷
  • Serous cell
    Àå¾×¼¼Æ÷
    [¿¾ ¿ë¾î] Àå¾×¼¼Æ÷
  • Germ cell
    Á¾ÀÚ¼¼Æ÷
    [¿¾ ¿ë¾î] Á¾¼¼Æ÷
  • Glomus cell
    Å丮¼¼Æ÷
    [¿¾ ¿ë¾î] ±¸»ó¼¼Æ÷
  • Tendon cell
    ÈûÁÙ¼¼Æ÷
    [¿¾ ¿ë¾î] °Ç¼¼Æ÷
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 16
ARN acute renal necrosis; acute retinal necrosis; arcuate nucleus; Association of Rehabilitation Nurses
ASS acute serum sickness; acute spinal stenosis; anterior superior spine; argininosuccinate synthetase
TS Takayasu syndrome; Tay-Sachs; temperature sensitivity; temperature, skin; temporal stem; tensile str...
ADCC cell Antibody Dependent Cellular(= Cell-Mediated) Cytotoxicity cell
HCG, hCG Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó
  1. Placental Glycoprotein Hormone
&nbs...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 16
LIFR leukemia inhibitory factor receptor
M4 myelo-monocytic leukemia
M2 myelogenous leukemia
B-CLL B cell Type
HTLV-I Human T Cell Lymphotropic Virus Type I
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 16
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • absolute cell increase
    Àý´ë ¼¼Æ÷ ¼ö Áõ°¡
  • absorptive cell
    Èí¼ö ¼¼Æ÷
  • accessory cell
    ºÎ¼¼Æ÷
    °ñÀú¼± Áß¿¡¼­ ÁÖ¼¼Æ÷, ¹æ¼¼Æ÷¿¡ ¼¯¿©¼­ Á¸ÀçÇÑ´Ù. ÀÔ¹æÇüÀ̸ç Á¡¾×¼ºÀÇ ¹°ÁúÀ» °£Á÷ÇÑ´Ù. ÇÙÀº ¼¼Æ÷Àú¿¡ Ä¡¿ìÃÄ ÀÖ¾î ÆíÆò¿¡ °¡±õ´Ù.
  • acinic cell carcinoma
    ¼±¹æ ¼¼Æ÷ ¾ÏÁ¾
    1. ¼±¹æ ¼¼Æ÷, ±Ù»óÇǼ¼Æ÷°¡ Áõ½ÄÇÏ¿© Çü¼ºµÇ°í ³·Àº ¾Ç¼ºµµ¸¦ º¸ÀδÙ. 2. Ÿ¾×¼± ¾à¼º Á¾¾ç Áß 5¹øÂ°ÀÇ ¹ß»ý ºñÀ²À» °®´Â Á¾¾çÀ¸·Î 90%¿¡¼­ ÀÌÇϼ±¿¡¼­ ¹ß»ýÇÏ¸ç ¾ÇÇϼ±°ú ¼ÒŸ¾×¼±¿¡¼­µµ µå¹°°Ô ¹ß»ýÇÑ´Ù. ¿©¼º¿¡¼­ ´Ù¼Ò È£¹ßÇϰí, ¾î´À ¿¬·É¿¡¼­³ª ¹ß»ýÇϳª ÁÖ·Î 30-70´ë¿¡ °ñ°í·ç ¹ß»ýÇÑ´Ù. ¿¹Àü¿¡´Â ¾ç¼ºÀ¸·Î »ý°¢ÇÏ¿© ¼±¹æ¼¼Æ÷Á¾À̶ó ºÎ¸¥ ÀûÀÌ ÀÖÀ¸³ª ºÐ¸íÇÑ ¾Ç¼ºÀ¸·Î ¼±¾ÏÁ¾À¸·Î ºÎ¸¥´Ù. Á¾¾ç ¼¼Æ÷´Â Àå¾×¼º ¼±¹æ¼¼Æ÷¿Í À¯»çÇÏÁö¸¸ ¿©·¯ °¡Áö ´Ù¸¥ ¼¼Æ÷µéÀÌ ³ªÅ¸³ª¸ç, °³Á¦°ü ¿¹ºñ¼¼Æ÷¿¡¼­ ±â¿øÇÑ´Ù°í º»´Ù.
  • adamantinoid basal cell carcinoma
    ¹ý¶û Á¾¾ç ±âÀú¼¼Æ÷¾Ï
  • adenoid basal cell carcinoma
    ¼±»ó ±âÀú¼¼Æ÷¾Ï
  • adenosquamous cell carcinoma
    ¼± ÆíÆò»óÇÇ ¼¼Æ÷¾Ï
    ¼± ¾Ï°ú ÆíÆò¼¼Æ÷ ¾ÏÀÌ È¥ÀçµÇ¾î ÀÖ´Â °Í. ÀÚ±Ã°æ ³»¸· »óÇÇÀÇ ±âÀú Ãþ¿¡ ÀÖ´Â ¿¹ºñ ¼¼Æ÷¿¡¼­ ¹ß»ý. ÀÌ´Â °°Àº º´±âÀÇ ÆíÆò¼¼Æ÷ ¾Ï°ú ºñ±³ÇÏ¿© ¿¹Èİ¡ ´õ ³ª»Ú´Ù.
  • adipose cell
    Áö¹æ ¼¼Æ÷
    Áö¹æÀ» °¡Áø ¼¼Æ÷·Î¼­ ¼¼Æ÷´Â ±¸ÇüÀ» ÀÌ·ç°í, ÇÙÀº ÇÑ ÂÊÀ¸·Î Ä¡¿ìÃÄ ÀÖ´Ù.
  • adrenal medullary chromaffin cell
    ºÎ½Å ¼öÁú Å©·Ò ģȭ ¼¼Æ÷
  • aggressive basal cell carcinoma
    ħ½À ±âÀú¼¼Æ÷¾Ï
  • air cell
    ÇԱ⠼¼Æ÷
  • alveolar cell carcinoma
    ÆóÆ÷ ¼¼Æ÷ ¾ÏÁ¾
  • Alzheimer's cell
    ¾ËÂêÇÏÀÌ¸Ó ¼¼Æ÷
  • Alzheimers cell
    ¾ËÂêÇÏÀÌ¸Ó ¼¼Æ÷
  • amplifying cell
    Áõ½Ä ¼¼Æ÷
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 16
multiple endocrine neoplasia type 2b A type of multiple endocrine neoplasia occurring as an isolated congenital presentation or as a distinct autosomal dominant disease. It is characterised by the 100% incidence of medullary thyroid carcinoma and frequent pheochromocytomas; patients seldom exhibit hyperparathyroidism. It is distinguished from men 2a by its characteristic physical appearance resulting from numerous neural defects including mucosal neuromas of the eyelids, lips, and tongue. The neural abnormalities also include widespread neurogangliomatosis of the gastrointestinal tract leading to abnormal gut motility. Treatment usually requires total thyroidectomy following evaluation for the presence of pheochromocytomas.
(12 Dec 1998)
multiple lipoprotein-type hyperlipidaemia <biochemistry> Inherited as a defective gene, this disorder is characterised by elevations in serum cholesterol and/or triglycerides. There are often multiple types of lipoproteins (LDL) elevated in one family. This condition is associated with an increased risk of cardiovascular disease.
Origin: Gr. Haima = blood
(27 Sep 1997)
contact-type dermatitis Dermatitis resembling contact dermatitis or eczema, but caused by an ingested or injected allergen, usually a drug, and with a widespread or generalised distribution.
(05 Mar 2000)
Cowdry's type A inclusion bodies Droplet-like masses of acidophilic material surrounded by clear halos within nuclei, with margination of chromatin on the nuclear membrane.
(05 Mar 2000)
Cowdry's type B inclusion bodies Droplet-like masses of acidophilic material surrounded by clear halos within nuclei, without other nuclear changes during early stages of development of the inclusion.
(05 Mar 2000)
habitat type <ecology> A land or aquatic unit, consisting of an aggregation of habitats having equivalent structure, function, and responses to disturbance.
(09 Oct 1997)
haemadsorption virus type 1 parainfluenza virus type 3
haemadsorption virus type 2 parainfluenza virus type 1
C type lectin <cell biology> One of two classes of lectin produced by animal cells, the other being the S type.
The C type lectins require disulphide linked cysteines and Ca ions in order to bind to a specific carbohydrate (c.f. S type lectins). The carbohydrate recognition domain of C type lectins consists of about 130 amino acids which contains 18 invariant residues in a highly conserved pattern.
These invariant residues include cysteines which probably form disulphide bonds. So far, all identified C type lectins are extracellular proteins and include both Integral membrane proteins, such as the asialoglycoprotein receptor and soluble proteins.
(06 Aug 1998)
C type virus <molecular biology, virology> Originally C type particles identified in mouse tumour tissue and later shown to be oncogenic RNA viruses Oncovirinae) that bud from the plasma membrane of the host cell starting as a characteristic electron dense crescent.
Include feline leukaemia virus, murine leukaemia and sarcoma viruses.
(18 Nov 1997)
haemophilus influenzae type b The majority of type b isolates are from biotype I. The organism can cause life-threatening meningitis, primarily in children 6-12 months of age. Children with underlying disease or immunodeficiency are also at high risk for infection. A vaccine is available and recommended for children under 5 years of age.
(12 Dec 1998)
haemophilus influenzae type b immunization See HIB immunization,
(12 Dec 1998)
Haemophilus influenzae type B vaccine A conjugate of oligosaccharides of the capsular antigen of H. Influenzae type B and diphtheria CRM protein.
(05 Mar 2000)
pre-excitation, mahaim-type A form of pre-excitation characterised by a normal pr interval and a long qrs interval with a delta wave.
(12 Dec 1998)
Hermansky-Pudlak syndrome type VI An autosomal recessive deficiency of pigment in skin, hair, and eyes; in the tyrosinase negative type, there is an absence of tyrosinase; in the tyrosinase positive type, there is normal tyrosinase which cannot enter pigment cells; it is transmitted by an autosomal recessive inheritance. The compound heterozygote is normal so the two forms are not allelic.
There are several types: type IA is characterised by absence of tyrosinase with life-long complete absence of melanin, marked photophobia, and nystagmus. Type IB, yellow albinism with low or absent tyrosinase; improves with age.
Type II, with normal tyrosinase activity is the most common; hair darkens and nevi and freckles develop.
Type III is characterised by absent tyrosinase but pigmentation of the iris in the first decade.
Type IV in Africans with normal tyrosinase.
Type V with red hair.
Type VI, Hermansky-Padlak syndrome, with haemorrhage due to platelet deficiency and low to absent tyrosinase.
Synonym: Hermansky-Pudlak syndrome type VI.
(05 Mar 2000)
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