| ARS | acquiescence response scale; adult Reye's syndrome; alcohol-related seizures; alizarin red S; Americ... |
|---|---|
| ASSERT | improving Alcohol and Substance abuse Services and Educating providings to Refer patients to Treatme... |
| AUDIT | alcohol use disorders identification test |
| AUI | Alcohol Use Inventory |
| AW | able to work; above waist; abrupt withdrawal; alcohol withdrawal; alveolar wall; anterior wall; atom... |
| tertiary amyl alcohol | amylene hydrate |
|---|---|
| tertiary butyl alcohol | Trimethylcarbinol, 2-methyl-2-propanol, (CH3)3COH, a denaturant for ethanol. (05 Mar 2000) |
| trichloroethyl alcohol | CCl3CH2OH; 2,2,2-Trichloroethanol;a hypnotic and sedative; as a metabolite of chloral hydrate, it contributes to the depressant activity of chloral hydrate. Synonym: trichloroethyl alcohol. (05 Mar 2000) |
| trihydric alcohol | An alcohol containing three OH groups; e.g., glycerol. (05 Mar 2000) |
| ethyl alcohol | <chemical, drug> Grain alcohol, made from sugar, starch and other carbohydrates by fermentation, has sedative properties (27 Sep 1997) |
| UDPglucose-coniferyl-alcohol glucosyltransferase | <enzyme> Forms coniferin (coniferyl alcohol 7-o-beta-d-glucopyranoside) Registry number: EC 2.4.1.- Synonym: udpcagtransferase (26 Jun 1999) |
| farnesene alcohol | <chemical> A colourless liquid extracted from oils of plants such as citronella, neroli, cyclamen, and tuberose. It is an intermediate step in the biological synthesis of cholesterol from mevalonic acid in verterbrates. It has a delicate odour and is used in perfumery. Chemical name: 2,6,10-Dodecatrien-1-ol, 3,7,11-trimethyl- (12 Dec 1998) |
| ketone alcohol | A compound containing a carbonyl or ketone group as well as a hydroxyl group; e.g., dihydroxyacetone. (05 Mar 2000) |
| foetal alcohol effects | A softer diagnosis than foetal alcohol syndrome (FAS). The diagnosis of possible FAE is considered when: 1. The person has some signs of FAS; 2. The person does not meet all of the necessary criteria for FAS; and 3. There is a history of alcohol exposure before birth. (12 Dec 1998) |
| foetal alcohol syndrome | <syndrome> The most common cause of defective cerebral development in industrialised nations, affecting 1 in 700 live births in the United States. This clinical syndrome results from the direct toxic effects of alcohol on the developing foetus. Growth retardation, mental retardation, small brain and heart valve lesions are common. Infants can be identified by close set eyes, small head, small nasal bridge, reduction in the vermilion border of the upper lip, eye folds and small teeth. (27 Sep 1997) |
| 4-sulfobenzyl alcohol dehydrogenase | <enzyme> Catalyses the formation of 4-sulfobenzaldehyde from 4-sulfobenzyl alcohol; amino acid sequence given in first source Registry number: EC 1.1.1.- Synonym: p-sulfobenzyl alcohol dehydrogenase, tsac protein, tsac gene product (26 Jun 1999) |
| long-chain-alcohol dehydrogenase | <enzyme> Catalyses reversibly the oxidation of a long-chain alcohol in the presence of nad to a long-chain acid anion and NADH Registry number: EC 1.1.1.192 Synonym: fatty alcohol-nad+ oxidoreductase (26 Jun 1999) |
| abdominal muscle deficiency syndrome | <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear. (05 Mar 2000) |
| adult lactase deficiency | Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults. (05 Mar 2000) |
| alpha-1 antitrypsin deficiency | <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues. The lack of this protein leads to damage of various organs, but mainly to the lung and liver. symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant (12 Dec 1998) |