| LAA | left atrial appendage; left atrial area; leukemia-associated antigen; leukocyte ascorbic acid |
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| TL | temporal lobe; terminal limen; thermolabile; thermoluminescence; threat to life; thymus-leukemia [an... |
| TLA | thymus leukemia antigen; tissue lactase activity; tongue-to-lip adhesion; translaryngeal aspiration;... |
| AMI | Acute Myocardial Infarction - Complications(Cx) 1. Early ... |
| AC | abdominal circumference; abdominal compression; absorption coefficient; abuse case; acetate; acetylc... |
| acute yellow atrophy of the liver | A lesion in which there is extensive and rapid death of parenchymal cells of the liver, sometimes with fatty degeneration of the size of the organ; the necrosis may result from fulminant viral infection or chemical poisoning; associated with jaundice. Synonym: acute parenchymatous hepatitis, Rokitansky's disease. (05 Mar 2000) |
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| bisectrix, acute | <microscopy> In biaxial crystals, that principal axis of the ellipsoid of indexes which bisects the smaller angle between the optic axes. (05 Aug 1998) |
| pancreatitis, acute necrotizing | Acute inflammation of the pancreas with areas of devitalised pancreatic and/or peripancreatic tissue. These necrotic areas may involve large areas of the pancreas or may be small. When a ct scan is performed with contrast media using a bolus technique, necrotic areas do not perfuse with media and are not enhanced. The lack of enhancement distinguishes necrotic tissue from the adjacent well-perfused viable pancreatic parenchyma. (12 Dec 1998) |
| retinal necrosis syndrome, acute | <ophthalmology> Mild to fulminant necrotizing vaso-occlusive retinitis associated with a high incidence of retinal detachment and poor vision outcome. (12 Dec 1998) |
| conjunctivitis, acute haemorrhagic | A highly contagious disease characterised by subconjunctival haemorrhage, sudden swelling of the eyelids and congestion, redness, and pain in the eye. Epidemic conjunctivitis caused by enterovirus 70 (ev-70) was first described in africa in 1969. It is caused also by coxsackie virus a24 variant (ca24v). Epidemics by this organism have appeared most frequently in asia. (12 Dec 1998) |
| porphyria, acute intermittent | A form of hepatic porphyria (porphyria, hepatic) characterised by periodic attacks of gastrointestinal disturbances, abdominal colic, paralyses, and psychiatric disorders. The onset of this condition is usually in the third or fourth decade of life. (12 Dec 1998) |
| stricture, oesophagus, acute | A narrowing or closure of the normal opening of the swallowing tube leading to the stomach, usually caused by scarring from acid irritation. Acute, complete obstruction of the oesophagus occurs when food (usually meat) is lodged in the oesophageal stricture. Patients experience chest pain, and are unable to swallow saliva. Attempts to relieve the obstruction by inducing vomiting at home are usually unsuccessful. Patients with complete oesophageal obstruction can breathe, and are not at any risk of suffocation. Endoscopy is usually employed to retrieve the meat and relieve the obstruction. (12 Dec 1998) |
| diverticulitis, treatment of acute | Antibiotics are usually needed. Oral antibiotics are sufficient when symptoms are mild. Liquid or low fibre foods are advised during acute diverticulitis attacks. In severe diverticulitis with high fever and pain, patients are hospitalised and given intravenous antibiotics. Surgery is needed for persistent bowel obstruction or abscesses not responding to antibiotics. (12 Dec 1998) |
| illness, acute | An illness with an abrupt onset and usually a short course. (12 Dec 1998) |
| infantile acute haemorrhagic oedema of the skin | A generally benign form of cutaneous vasculitis, characterised by ecchymotic purpura, often in a cockade pattern, and inflammatory oedema in infants. (05 Mar 2000) |
| intermittent acute porphyria | <gastroenterology, haematology> A group of rare inherited metabolic disorders that result from a disturbance in porphyrin metabolism, causing increased formation and excretion of porphyrin or its precursors. It is caused by hepatic overproduction of d-aminolevulinic acid, which has greatly increased urinary excretion and of porphobilinogen, and some increase of uroporphyrin, due to a deficiency of porphobilinogen deaminase. Clinical features: intermittent acute attacks of hypertension, abdominal colic, psychosis, and polyneuropathy, but with no photosensitivity. It is exacerbated by the ingestion of certain drugs such as; barbiturates). Inheritance: autosomal dominant. (20 Sep 2002) |
| oesophageal stricture, acute | A narrowing or closure of the normal opening of the swallowing tube leading to the stomach, usually caused by scarring from acid irritation. Acute, complete obstruction of the oesophagus occurs when food (usually meat) is lodged in the oesophageal stricture. Patients experience chest pain, and are unable to swallow saliva. Attempts to relieve the obstruction by inducing vomiting at home are usually unsuccessful. Patients with complete oesophageal obstruction can breathe, and are not at any risk of suffocation. Endoscopy is usually employed to retrieve the meat and relieve the obstruction. (12 Dec 1998) |
| encephalomyelitis, acute disseminated | An acute or subacute encephalomyelitis or myelitis characterised by perivascular lymphocyte and mononuclear cell infiltration and demyelination. It occurs most commonly following an acute viral infection, especially measles, but may occur without a recognizable antecedent, and formerly occurred as a complication of rabies and smallpox vaccination. (12 Dec 1998) |
| kidney failure, acute | A clinical syndrome characterised by a sudden decrease in glomerular filtration rate, often to values of less than 1 to 2 ml per minute. It is usually associated with oliguria (urine volumes of less than 400 ml per day) and is always associated with biochemical consequences of the reduction in glomerular filtration rate such as a rise in blood urea nitrogen (bun) and serum creatinine concentrations. (12 Dec 1998) |
| kidney tubular necrosis, acute | Acute kidney failure resulting from destruction of tubular epithelial cells. It is commonly attributed to exposure to toxic agents or renal ischemia following severe trauma. (12 Dec 1998) |