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"Female acute pelvic peritonitis"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 13
  • ¿µ¹®
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  • acute phase reaction
    ±Þ¼º±â¹ÝÀÀ(¡­Ñ¢Úãëë)
  • acute phase reactive protein
    ±Þ¼º±â ¹ÝÀÀ¼º ´Ü¹é.
  • acute phase serum
    ±Þ¼ºº´±âÇ÷û
  • acute phase substances
    ±Þ¼º±â ¹°Áú(ÐáàõÑ¢Úªòõ).
  • acute pneumonia
    ±Þ¼ºÆó·Å (~øËæú)
  • acute pneumonia
    ±Þ¼º Æó·Å(¡­øËæú).
  • acute pneumonia
    ±Þ¼º Æó·Å(?̰Ëç).
  • acute poisoning
    ±Þ¼º Áßµ¶(¡­ñéÔ¸).
  • acute poliomyelitis
    ±Þ¼º ȸ¹éô¼ö¿°(¡­üéÛÜô±âÐæú), ±Þ¼º ¼Ò¾Æ¸¶ºñ.
  • acute poliomyelitis
    ±Þ¼ºÈ¸¹é¼ö¿°(~üéÛÜâÐæú),Æú¸®¿À
  • acute polymorphic psychotic disorder
    ±Þ¼º ´ÙÇü¼º Á¤½ÅÁõÀû Àå¾Ö(º´)
  • acute posterior ganglionitis
    ±Þ¼º ÈÄÃø½Å°æÀý¿°(¡­ý­ö°ãêÌèï½æú).
  • acute posterior multifocal placoid pigment epitheliopathy
    ±Þ¼ºÈĺδٹ߼ºÆÇ»ó»ö¼Ò»óÇÇÁõ
  • acute posthemorrhagic anemia
    ±Þ¼º ÃâÇ÷Èļº ºóÇ÷(¡­õóúìý­àõÞ¸úì).
  • acute posthemorrhagic anemia
    ±Þ¼º ÃâÇ÷Èļº ºóÇ÷(?̴̷̧ËÛË×Ì´).
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 13
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  • acute abdomen =abdominal emergencies
    ±Þ¼ºº¹Áõ(¡­º¹Áõ).
  • acute abdomen =abdominal emergencies
    ¼Ò¾Æ°ú ±Þ¼ºº¹Áõ(¡­ÜÙñø).
  • acute alcoholism
    ±Þ¼º ¾ËÄÚ¿ÃÁßµ¶(Áõ).
  • acute and late normal tissue effects
    Á¤»óÁ¶Á÷ ±Þ¼º¿µÇâ, Á¤»óÁ¶Á÷ ¸¸¼º ¿µÇâ
  • acute anterior poliomyelitis
    ±Þ¼ºÀü°¢¼ºÈ¸¹é¼ö¿°, Æú¸®¿À
  • acute anterior poliomyelitis =infantile par aly sis
    ±Þ¼º ȸ¹éô¼ö¿°(ÐáàõüéÛÜô±âÐæú).
  • acute anxiety neurosis
    ±Þ¼º ºÒ¾È½Å°æÁõ (¡­ÝÕäÌãêÌèñø).
  • acute aortitis
    ±Þ¼º ´ëµ¿¸Æ¿°(¡­ÓÞÔÑØææú).
  • acute aortitis
    ±Þ¼º ´ëµ¿¸Æ¿°(¡­´ëµ¿¸Æ¿°).
  • acute apical periodontitis
    ±Þ¼º ±Ù÷¼º Ä¡ÁÖ¿°(¡­ÐÆôÓàõöÍñ²æú).
  • acute appendicitis
    ±Þ¼ºÃæ¼ö¿°.
  • acute appendicitis
    ±Þ¼º Ãæ¼ö¿°(¡­Ãæ¼ö¿°).
  • acute arthritis
    ±Þ¼º °üÀý¿°(ÐáàõÎ¼ï½æú).
  • acute ascending myelitis
    ±Þ¼º »óÇà(¼º) ô¼ö¿°(¡­ß¾ú¼àõô±âÐæú).
  • acute ascending paralysis<³ª> p. ascendens acu ta
    ±Þ¼º »óÇà(¼º) ¸¶ºñ(¡­ß¾ú¼àõ Ýö).
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 13
Ptrx pelvic traction
PTx parathyroidectomy; pelvic traction
RPF relaxed pelvic floor; renal plasma flow; retroperitoneal fibrosis
SPS scapuloperoneal syndrome; shoulder pain and stiffness; simple partial seizures; slow-progressive sch...
SPTx static pelvic traction
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 13
APSGN Acute Poststreptococcal Glomerulonephritis
APL Acute Promyelocytic Leukaemia
AR Acute Rejection
ARF Acute Renal Failure
ARF Acute Respiratory Failure
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 13
acute kidney transplant rejection <radiology> Findings on ultrasound: globular enlargment of the kidney, swelling and hypoechogenicity of the medullary pyramids, indistinct cortico-medullary junction, foci in the renal cortex
(12 Dec 1998)
acute leukaemia <haematology> A rapidly progressive cancer of the blood of sudden onset and characterised by the uncontrolled proliferation of immature blood cells which take over the bone marrow and spill into the blood stream. If left untreated is fatal within a few weeks or months.
See: acute lymphoblastic leukaemia, acute myeloid leukaemia.
Origin: Gr. Haima = blood
(11 Nov 1997)
acute lobar nephrosis A severe but localised bacterial infection of the renal parenchyma that may produce a mass effect simulating a renal abscess.
(05 Mar 2000)
acute lower GI haemorrhage <gastroenterology> Typical presentation: Sudden onset of brisk rectal bleeding without blood in gastric aspirate Diagnostic considerations: diverticulosis, angiodysplasia, ischemic colitis, inflammatory bowel disease (rarely), polyps are usually present, carcinoma causing a chronic bleed, haemorrhoids.
(12 Dec 1998)
acute lymphoblastic leukaemia <haematology> A rapidly progressing cancer of the blood affecting the type of white blood cell known as lymphocytes. Approximately 650 new cases are diagnosed every year in the UK and it is the most common form of childhood leukaemia.
Acronym: ALL
Origin: Gr. Haima = blood
(11 Nov 1997)
acute lymphocytic leukaemia <radiology> 95% of cases of leukaemia in children, bone changes in 50-70% of kids (vs. 10% in adults); seen as early as 1 month after onset of symptoms, wrists and knees most commonly affected, bony defects: metaphyseal radiolucent bands! (similar findings in scurvy, JRA, syphilis), osteolytic lesions, periosteal reaction, osteosclerosis
(12 Dec 1998)
acute malaria A form of malaria that may be intermittent or remittent, consisting of a chill accompanied and followed by fever with its attendant general symptoms, and terminating in a sweating stage; the paroxysms, caused by release of merozoites from infected cells, recur every 48 hours in tertian (vivax or ovale) malaria, every 72 hours in quartan (malariae) malaria, and at indefinite but frequent intervals, usually about 48 hours, in malignant tertian (falciparum) malaria.
(05 Mar 2000)
acute mania An excited mental state seen in a bipolar (manic-depressive) disorder characterised by hyperactivity, talkativeness, flight of ideas, pressured speech, grandiosity, and, occasionally, grandiose delusions.
See: mania, manic-depressive.
Synonym: acute mania.
(05 Mar 2000)
acute miliary tuberculosis A rapidly fatal disease due to the general dissemination of tubercle bacilli in the blood, resulting in the formation of miliary tubercles in various organs and tissues, and producing symptoms of profound toxaemia.
Synonym: acute miliary tuberculosis, disseminated tuberculosis.
(05 Mar 2000)
acute monocytic leukaemia <haematology> The most common translocation in this disorder of poorly differentiated monocytic cells involves chromosome region 11q in a large percentage of cases.
The translocation involves a cellular oncogene, c-ets which is mapped to the 11q23-24 region. The most common translocations reported are t(6;11), t(9;11), t(11;17) and t(11;19), of which t(9;11) (p21-22;q23) is by far the most frequently detected and implicated in acute myeloid leukaemia. The cells express CD14 surface antigen, which is diagnostic of monocytic cells.
Acronym: AML
Classification: FAB M5
(07 Apr 1998)
acute mountain sickness <chest medicine> A condition that results from prolonged exposure to high altitude.
Symptoms include a continuous dry cough, shortness of breath, poor exercise tolerance, dizziness, headache, sleep difficulty, anorexia, confusion, fatigue and a rapid pulse.
Treatment includes the immediate movement to a lower altitude. Prophylaxis has been accomplished successfully with the use of acetazolamide (Diamox).
(27 Sep 1997)
acute myeloblastic leukaemia <haematology> A rapidly progressing cancer of the blood affecting immature cells of the bone marrow, usually of the white cell population. It is much more common in adults than in children.
Symptoms include fatigue, weight loss, fevers, weakness, pallor, bone pains, bleeding gums, nosebleeds, easy bruising, enlarged lymph nodes and joint pains.
Treatment includes chemotherapy and/or bone marrow transplant.
This leukaemia demonstrates granulocyte differentiation, eosinophilia and Auer rods and is associated with a reciprocal translocation between 8 and 21 (q22;q22), which is the most common translocation in acute myeloid leukaemia and is found more often in younger patients than in older patients. The oncogene involved in this translocation is AML1, which can be detected by Southern blot. Numerical abnormalities, particularly monosomy-7, trisomy-4, trisomy-8, trisomy-21, -Y, monosomy-7 and deletions of the long arms of chromosomes 5 and 7 are quite common in all acute myeloid leukaemia and not restricted to any one FAB classification. Many of these abnormalities are observed at diagnosis and at later stage disease, particularly after chemotherapy.
Prognosis is generally more favorable than in FAB-M2 patients showing no translocation, because the latter patients show better remission rates for longer periods of time. Immunophenotyping is useful in diagnosis and expression of one or more of the myeloid antigens CD13, CD14 or CD33 must be detected to make a diagnosis of acute myeloid leukaemia.
Acronym: AML
Incidence: 2,000 new cases per year in the UK.
Origin: Gr. Haima = blood
(07 Apr 1998)
acute myelogenous leukaemia <haematology> A rapidly progressing cancer of the blood affecting immature cells of the bone marrow, usually of the white cell population. It is much more common in adults than in children.
Symptoms include fatigue, weight loss, fevers, weakness, pallor, bone pains, bleeding gums, nosebleeds, easy bruising, enlarged lymph nodes and joint pains.
Treatment includes chemotherapy and/or bone marrow transplant.
This leukaemia demonstrates granulocyte differentiation, eosinophilia and Auer rods and is associated with a reciprocal translocation between 8 and 21 (q22;q22), which is the most common translocation in acute myeloid leukaemia and is found more often in younger patients than in older patients. The oncogene involved in this translocation is AML1, which can be detected by Southern blot. Numerical abnormalities, particularly monosomy-7, trisomy-4, trisomy-8, trisomy-21, -Y, monosomy-7 and deletions of the long arms of chromosomes 5 and 7 are quite common in all acute myeloid leukaemia and not restricted to any one FAB classification. Many of these abnormalities are observed at diagnosis and at later stage disease, particularly after chemotherapy.
Prognosis is generally more favorable than in FAB-M2 patients showing no translocation, because the latter patients show better remission rates for longer periods of time. Immunophenotyping is useful in diagnosis and expression of one or more of the myeloid antigens CD13, CD14 or CD33 must be detected to make a diagnosis of acute myeloid leukaemia.
Acronym: AML
Incidence: 2,000 new cases per year in the UK.
Origin: Gr. Haima = blood
(07 Apr 1998)
acute myeloid leukaemia <haematology> A rapidly progressing cancer of the blood affecting immature cells of the bone marrow, usually of the white cell population. It is much more common in adults than in children.
Symptoms include fatigue, weight loss, fevers, weakness, pallor, bone pains, bleeding gums, nosebleeds, easy bruising, enlarged lymph nodes and joint pains.
Treatment includes chemotherapy and/or bone marrow transplant.
This leukaemia demonstrates granulocyte differentiation, eosinophilia and Auer rods and is associated with a reciprocal translocation between 8 and 21 (q22;q22), which is the most common translocation in acute myeloid leukaemia and is found more often in younger patients than in older patients. The oncogene involved in this translocation is AML1, which can be detected by Southern blot. Numerical abnormalities, particularly monosomy-7, trisomy-4, trisomy-8, trisomy-21, -Y, monosomy-7 and deletions of the long arms of chromosomes 5 and 7 are quite common in all acute myeloid leukaemia and not restricted to any one FAB classification. Many of these abnormalities are observed at diagnosis and at later stage disease, particularly after chemotherapy.
Prognosis is generally more favorable than in FAB-M2 patients showing no translocation, because the latter patients show better remission rates for longer periods of time. Immunophenotyping is useful in diagnosis and expression of one or more of the myeloid antigens CD13, CD14 or CD33 must be detected to make a diagnosis of acute myeloid leukaemia.
Acronym: AML
Incidence: 2,000 new cases per year in the UK.
Origin: Gr. Haima = blood
(07 Apr 1998)
acute necrotizing encephalitis An acute form of encephalitis, characterised by destruction of brain parenchyme.
(05 Mar 2000)
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