¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"plasma cell hepatitis"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • cell swelling
    ¼¼Æ÷Á¾Ã¢
  • cell-associated antibody
    ¼¼Æ÷¿¬°üÇ×ü
  • cell-bound antibody
    ¼¼Æ÷°áÇÕÇ×ü
  • cell-fixed antibody
    ¼¼Æ÷°áÇÕÇ×ü
  • cell-mediated cytolysis
    ¼¼Æ÷¸Å°³¼¼Æ÷¿ëÇØ
  • cell-mediated cytotoxicity
    ¼¼Æ÷¸Å°³¼¼Æ÷µ¶¼º
  • cell-mediated hypersensitivity
    ¼¼Æ÷¸Å°³°ú¹Î¼º
  • cell-mediated immunity
    ¼¼Æ÷¸Å°³¸é¿ª
  • cell-mediated reaction
    ¼¼Æ÷¸Å°³¹ÝÀÀ
  • cell-mediated response
    ¼¼Æ÷¸Å°³¹ÝÀÀ
  • centroacinar cell
    »ù²Ê¸®Á߽ɼ¼Æ÷, Á߽ɼ±¹æ¼¼Æ÷
  • chief cell
    À¸¶ä¼¼Æ÷
  • chromaffin cell
    Å©·Òģȭ¼¼Æ÷, ģũ·Ò¼¼Æ÷
  • chromophilic cell
    »ö¼Òµê¼¼Æ÷, È£»ö¼Ò¼¼Æ÷
  • chromophobic cell
    »ö¼Ò¾Èµê¼¼Æ÷
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  • ¿µ¹®
    ÇѱÛ
  • clear cell sarcoma
    Åõ¸í¼¼Æ÷À°Á¾
  • columnar cell
    ¿øÁÖ¼¼Æ÷
  • columnar absorptive cell
    ±âµÕÈíÂø¼¼Æ÷
  • committed cell
    ¾ô¸Ç¼¼Æ÷, ¼öÀÓ¼¼Æ÷
  • complex cell
    º¹ÇÕ¼¼Æ÷
  • cone cell
    ¿ø»Ô¼¼Æ÷
  • cone cell layer
    ¿ø»Ô¼¼Æ÷Ãþ
  • connective tissue cell
    °áÇÕÁ¶Á÷¼¼Æ÷
  • continuous cell line
    ¹«ÇÑÁõ½Ä¼º¼¼Æ÷ÁÖ, ¿¬¼Ó°è´ë¼¼Æ÷ÁÖ
  • continuous cell separator
    ¿¬¼Ó¼¼Æ÷ºÐ¸®±â
  • contractile fiber cell
    ¼öÃ༶À¯¼¼Æ÷
  • cover cell
    µ¤°³¼¼Æ÷
  • crenated cell
    Åé´ÏÀûÇ÷±¸
  • crypt cell
    âÀÚ»ù¼¼Æ÷
  • cuboidal cell
    ÀԹ漼Æ÷, ÁÖ»çÀ§¼¼Æ÷
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  • ¿µ¹®
    ÇѱÛ
  • Granulosa cell tumor
    °ú¸³¸·¼¼Æ÷Á¾¾ç(Î¨Ø£Ø¯á¬øàðþåË)
  • H-9 cell line
    H-9 ¼¼Æ÷°è
  • HeLa cell
    Çï¶ó¼¼Æ÷.
  • HeLa cell
    Çï¶ó¼¼Æ÷
  • Heidenhain s cell
    ÇÏÀ̵§ÇÏÀμ¼Æ÷.
  • Henle s cell
    Çî·¯¼¼Æ÷.
  • Hfr cell
    °íºóµµÀçÁ¶ÇÕ¼¼Æ÷
  • Hulle cell
    ¿ÜÇǼ¼Æ÷
  • ICSH = interstitial cell stimulating hormone
    °£Áú¼¼Æ÷ÀÚ±Ø(Êàòõá¬øàí©Ð½)È£¸£¸ó.
  • ICSH=£¾interstitial cell stimulating hormone
    °£Áú¼¼Æ÷ÀÚ±ØÈ£¸£¸ó.
  • K cell
    K¼¼Æ÷, »ìÇØ¼¼Æ÷
  • LE cell
    LE¼¼Æ÷.
  • LE cell phenomenon
    LE¼¼Æ÷Çö»ó.
  • Langerhans cell
    ¶û°Ô¸£Çѽº¼¼Æ÷.
  • Langerhans cell histiocytosis
    ¶û°Ô¸£Çѽº ¼¼Æ÷ Á¶Á÷±¸Áõ
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  • ¿µ¹®
    ÇѱÛ
  • plasma membrane carnitine transport defect
  • plasma membrane of erythrocyte
    ÀûÇ÷±¸ÇüÁú¸·
  • plasma pepsinogen
    Ç÷ÀåÆé½Ã³ë°Õ
  • plasma protein
    Ç÷Àå´Ü¹é(Áú)
  • plasma protein
    Ç÷Àå´Ü¹éÁú(úìíìÓ±ÛÜòõ).
  • plasma protein binding
    Ç÷Àå´Ü¹é°áÇÕ.
  • plasma protein fraction =PCC
    Ç÷Àå´Ü¹éºÐȹ
  • plasma prothrombin time
    Ç÷ÀåÇÁ·ÎÆ®·Òºó ½Ã°£.
  • plasma renin activity
    Ç÷Àå·¹´ÑȰ¼ºµµ<--Ȱµ¿µµ>
  • plasma substitute
    Ç÷Àå´ë¿ëÁ¦.
  • plasma thrombokinin
    Ç÷À寮·Òº¸Å°´Ñ.
  • plasma thromboplastic factor
    Ç÷À寮·Òº¸ÇÃ¶ó½ºÆ¾ÀÎÀÚ.
  • plasma thromboplastin
    Ç÷À寮·Òº¸ÇÃ¶ó ½ºÆ¾.
  • plasma thromboplastin antecedent
    Ç÷À寮·Òº¸ÇÃ¶ó½ºÆ¾Àü±¸(îñÏÌ)¹°Áú
  • plasma thromboplastin antecedent =PTA
    ÇöóÁ<Ç÷Àå>Æ®·Òº¸ÇÃ¶ó½ºÆ¾Àü±¸¹°Áú (
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  • ¿µ¹®
    ÇѱÛ
  • Acidophilic cell
    È£»ê¼º¼¼Æ÷
    [¿¾ ¿ë¾î] »êÈ£¼º¼¼Æ÷
  • Basophilic cell
    È£¿°±â¼º¼¼Æ÷
    [¿¾ ¿ë¾î] ¿°±âÈ£¼º¼¼Æ÷
  • Unilocular fat cell
    ȬĭÁö¹æ¼¼Æ÷
    [¿¾ ¿ë¾î] ´Ü¹æ¼ºÁö¹æ¼¼Æ÷
  • Uninuclear giant cell
    ȬÇÙ°Å´ë¼¼Æ÷
    [¿¾ ¿ë¾î] ´ÜÇÙ¼º°Å´ë¿µ¾ç¸·¼¼Æ÷
  • Olfactory cell
    Èİ¢¼¼Æ÷
    [¿¾ ¿ë¾î] Èİ¢¼¼Æ÷
  • Spinous cell
    °¡½Ã¼¼Æ÷
    [¿¾ ¿ë¾î] À¯±Ø¼¼Æ÷
  • Dendriform cell
    ³ª¹µ°¡Áö¼¼Æ÷
    [¿¾ ¿ë¾î] ¼öÁö»ó¼¼Æ÷
  • Endocrine cell
    ³»ºÐºñ¼¼Æ÷
    [¿¾ ¿ë¾î] ³»ºÐºñ¼¼Æ÷
  • Colliculiform cell
    µÐ´ö¼¼Æ÷
    [¿¾ ¿ë¾î] ¼Ò±¸¼¼Æ÷
  • Microvillous cell
    ¹Ì¼¼À¶¸ð¼¼Æ÷
    [¿¾ ¿ë¾î] ¹Ì¼¼À¶¸ð¼¼Æ÷
  • Smooth muscle cell
    ¹Î¹«´Ì±ÙÀ°¼¼Æ÷
    [¿¾ ¿ë¾î] ÆòȰ±Ù±Ù¼¼Æ÷
  • Light cell
    ¹àÀº¼¼Æ÷
    [¿¾ ¿ë¾î] ¸í¼¼Æ÷
  • Light cell
    ¹àÀº¼¼Æ÷
    [¿¾ ¿ë¾î] ¸íÁÖ¼¼Æ÷
  • Fusiform cell
    ¹æÃß¼¼Æ÷
    [¿¾ ¿ë¾î] ¹æÃ߻󼼯÷
  • Supporting cell
    ¹öÆÀ¼¼Æ÷
    [¿¾ ¿ë¾î] ÁöÁÖ¼¼Æ÷
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 12
HCV Hepatitis C Virus
HDV Hepatitis D(elta) Virus
  = HBV associated Delta Agent
HEV Hepatitis E Virus
IgM¥áHBc IgM Antibody against Hepatitis B core Antigen
SH Serum Hepatitis
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HBc AB Hepatitis B core antibody
HBc Ag Hepatitis B core antigen
HBeAg Hepatitis B e Antigen
HBeAg Hepatitis B envelope antigen
HBs Hepatitis B surface
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 12
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • direct cell division
    Á÷Á¢ ¼¼Æ÷ ºÐ¿­
  • disintegrated cell
    ºØ±« ¼¼Æ÷
  • dorsal horn cell
    Èİ¢ ¼¼Æ÷, ¹è°¢ ¼¼Æ÷
  • dorsal horn pain transmission cell
    ¹è°¢ ÅëÁõ Àü´Þ ¼¼Æ÷, ¹è°¢ µ¿Åë Àü´Þ ¼¼Æ÷
  • ductal cell
    µµ°ü ¼¼Æ÷
  • ductule cell
    ¼Ò°ü ¼¼Æ÷
  • ealry squamous cell calcinoma
    ÃÊ±â ÆíÆò»óÇÇ ¼¼Æ÷¾Ï
    ±¸°­ ³» °¡Àå ÈçÇÑ ¾Ç¼º ÁúȯÀ̰í Ä¡°úÀǻ簡 Ä¡·áÇÏ´Â ¸î ¾È µÇ´Â Ä¡¸íÀû ÁúȯÀÇ ÇϳªÀÌ´Ù. Çǰ³ »óÇÇ ¼¼Æ÷ÀÇ ¾Ç¼º ¾ÏÁ¾¼º Áõ½ÄÀÌ´Ù. ¹é¹ÝÁõÀ̶ó°í ÇÏ´Â ÀÓ»ó ¿ë¾î·Îµµ ºÒ¸®´Â ¼Ò»ó »óÇÇ ºñÈÄ¿Í µ¿ÀÏÇÑ º´¼Ò¸¦ º¸¿©ÁØ´Ù. °¡Àå ÈçÇÑ ¿øÀÎÀ¸·Î »ý°¢µÇ´Â °ÍÀº ½À°üÀûÀÎ Èí¿¬°ú ¾ËÄÝÀÌ´Ù. ±¸°­ Á¡¸·¿¡ ¼Ò»ó ¹é»ö ¹ÝÁ¡À» ¸¸µå´Âµ¥ ÀÌ´Â »ý¸®Àû °ú°¢È­¿Í ºñ½ÁÇÏ°Ô º¸ÀδÙ. º´¼Ò¸¦ °ÇÁ¶½ÃŲ ÈÄ ÀÚ¼¼È÷ °üÂûÇϸé ÀÌÇü¼º º´¼ÒÀÇ Ç¥¸éÀÌ ÀϹÝÀûÀ¸·Î ´õ °ÅÄ¥°í ÂÞ±ÛÂÞ±ÛÇÑ °ÍÀ» º¼ ¼ö ÀÖ´Ù.
  • educated T cell
    Ç׿øÀ¸·Î °¨ÀÛµÈ T ¼¼Æ÷
    In vivo ¶Ç´Â in vitro¿¡¼­ Ç׿ø ÀÚ±ØÀ» ¹Þ¾Æ ¸é¿ª ±âÀüÀ» ¹ßÇöÇÒ ¼ö ÀÖ´Â »óŰ¡ µÈ T ¼¼Æ÷ÀÌ´Ù. In vivo¿¡¼­´Â ÀϹÝÀûÀ¸·Î Ä¡»ç·®ÀÇ ¹æ»ç¼±À» Á¶»çÇÑ Áã¿¡ ´Ù¸¥ µ¿¹°·ÎºÎÅÍÀÇ Èä¼± ¼¼Æ÷¸¦ ÀÌÀÔÇÔ°ú µ¿½Ã¿¡ Ç׿øÀÚ±ØÀ» ÇÏ´Â ¹æ¹ýÀÌ »ç¿ëµÈ´Ù.
  • endosteal cell
    °ñ³» ¼¼Æ÷
    À§Ä¡¿¡ ÀÇÇØ º¯°æµÇ°í, È®ÀεǴ ¸Á»ó ¼¼Æ÷. °ñ ³»¸·Àº °ñ¼ö ±âÁúÀÌ ³óÃàµÈ »óÅ´Ù.
  • endothelial cell
    ³»ÇÇ ¼¼Æ÷
  • enkephalinergic cell
    ¿£ÄÉÆÈ¸°¼º ¼¼Æ÷
  • eosinophilic cell
    È£»ê±¸
  • epidermoid type cell
    À¯Ç¥ÇÇ ¼¼Æ÷
  • epithelioid cell
    »óÇÇ¾ç ¼¼Æ÷, À¯»óÇÇ ¼¼Æ÷
    °áÇÙ µîÀÇ À°¾Æ¼º ¿°Áõ Áúȯ¿¡ À־ À°¾Æ ¼Ó¿¡¼­ È®ÀÎÇÒ ¼ö ÀÖ´Â Á¶Á÷±¸ÀÇ È£Äª.
  • ethmoid cell
    »ç°ñ ¹úÁý, »ç°ñ ºÀ¼Ò
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 12
NANB hepatitis Hepatitis caused by two or more infectious agents not detectable by methods that reveal the presence of hepatitis viruses A and B; one cause, now called type C hepatitis has been identified; may follow blood transfusion and is often seen in chronic renal dialysis patients.
Synonym: NANB hepatitis.
(05 Mar 2000)
neonatal hepatitis Hepatitis in the neonatal period presumed to be due to a variety of causes, chiefly viral; characterised by direct and indirect bilirubinaemia, hepatocellular degeneration, and appearance of multinucleated giant cells; may be difficult to distinguish from biliary atresia, but is more likely to end with recovery, although cirrhosis may develop.
Synonym: giant cell hepatitis.
(05 Mar 2000)
subacute hepatitis <pathology> This is a form of continuing liver inflammation that results in liver cell death. Causes include viral infection (hepatitis D, hepatitis B, hepatitis C), autoimmune disease, drug ingestion or metabolic causes. Chronic active hepatitis will lead to hepatic failure and death in a small percentage of these patients.
(27 Sep 1997)
non-A, non-B hepatitis Hepatitis caused by two or more infectious agents not detectable by methods that reveal the presence of hepatitis viruses A and B; one cause, now called type C hepatitis has been identified; may follow blood transfusion and is often seen in chronic renal dialysis patients.
Synonym: NANB hepatitis.
(05 Mar 2000)
non-A, non-B hepatitis virus Term used to group any of a number of viruses, other than A or B, which cause hepatitis in humans.
(05 Mar 2000)
non-A, non-B, non-C hepatitis Hepatitis caused by viral organisms other than hepatitis viruses A, B or C.
(05 Mar 2000)
suppurative hepatitis Hepatitis with abscess formation; often amoebic in origin.
(05 Mar 2000)
delta hepatitis Acute or chronic hepatitis caused by the hepatitis delta virus, a defective RNA virus requiring HBV for replication. The acute type occurs in two forms: 1) coinfection, the simultaneous occurrence of hepatitis B virus and hepatitis delta virus infections, which usually is self-limiting; 2) superinfection, the appearance of hepatitis delta virus infection in a hepatitis B virus carrier, which often leads to chronic hepatitis The chronic type appears to be more severe than other types of viral hepatitis.
Synonym: delta hepatitis, hepatitis D.
(05 Mar 2000)
drug-induced hepatitis <hepatology, pathology> Inflammation and hepatocellular damage of the liver that is caused by a drug.
Some medications may cause inflammation of the liver as a drug side effect or drug toxicity. Drugs that are known to cause hepatitis include acetaminophen, isoniazid, halothane, methyldopa, erythromycin and oral contraceptives.
(27 Sep 1997)
duck hepatitis virus A DNA virus of the genus Hepadnavirus, in the family Hepadnaviridae, causing virus hepatitis of ducks.
(05 Mar 2000)
duck viral hepatitis An acute, highly contagious disease of young ducklings caused by an enterovirus and characterised by lethargy, spasmodic paddling and rapid death.
(05 Mar 2000)
immunization, hepatitis a When immediate protection against hepatitis a (infectious hepatitis) is needed, immunoglobulins are used. Protection is effective only if given within 2 weeks of exposure and lasts but 2-4 months. Immunoglobulins can be used to protect household contacts of someone with acute viral hepatitis and travelers to regions with poor sanitation and high hepatitis a rates, when the traveler has to depart sooner than the vaccines can take effect (about 2 weeks). Travelers can receive the immunoglobulin and vaccine simultaneously and be protected immediately and for longer term. When immediate protection is not needed, hepatitis a vaccines are considered for individuals in high-risk settings, including frequent world travelers, sexually active individuals with multiple partners, homosexual men, individuals using illicit drugs, employees of daycare centres, and certain health care workers, and sewage workers. Two hepatitis a vaccines called havrix and vaqta are commercially available in the u.s. Both are highly effective and provide protection even after only one dose. Two doses are recommended for adults and 3 doses for children (under 18 years of age) to provide prolonged protection.
(12 Dec 1998)
immunization, hepatitis b Hepatits B (hep B) vaccine gives prolonged protection, but 3 shots over a half year are usually required. In the u.s., all infants receive hep b vaccine. Two vaccines (engerix-b, and recombivax-hb) are available in the us. The first dose of hep b vaccine is frequently given while the newborn is in the hospital or at the first doctor visit following birth. The second dose is given about 30 days after the initial dose. A booster dose is performed approximately six months later. Babies born to mothers testing positive for hep b receive, in addition, hbig (hep b immune globulin) for prompt protection. Older children (11-12 years) are advised to receive a hep b booster as are adults in high-risk situations including healthcare workers, dentists, intimate and household contacts of patients with chronic hep b infection, male homosexuals, individuals with multiple sexual partners, dialysis patients, iv drug users, and recipients of repeated transfusions. Health care workers accidentally exposed to materials infected with hep b (such as needle sticks), and individuals with known sexual contact with hep b patients are usually given both hbig and vaccine to provide immediate and long term protection.
(12 Dec 1998)
immunization, infectious hepatitis See Immunization, hepatitis a.
(12 Dec 1998)
immunization, serum hepatitis See Immunization, hepatitis b.
(12 Dec 1998)
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
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