| SSV | Schoolman-Schwartz virus; simian sarcoma virus |
|---|---|
| VIA | virus inactivating agent; virus infection-associated antigen |
| CEA | Carcino-Embryonic Antigen [HP 1825-6] ; Oncofetal Antigens ; Glycopro... |
| GTT | 1) Gestational Trophoblastic Tumor 2) Glucose Tolerance Test; 당부하검사 |
| HIVD | Herniation(Herniated) of Inter-Vertebral Disc - Cervical HIVD &... |
| virus blockade | The interference of one virus by another, either attenuated or unrelated. (05 Mar 2000) |
|---|---|
| virus C hepatitis | Principal cause of non-A, non-B posttransfusion hepatitis caused by an RNA virus that may be related to Flaviviridae family. Synonym: hepatitis C, virus C hepatitis. (05 Mar 2000) |
| virus diseases | A general term for diseases produced by viruses. (12 Dec 1998) |
| virus hepatitis | Liver inflammation caused by viruses. Specific hepatitis viruses have been labelled a, b, c, d, e, f, and g. While other viruses can also cause hepatitis, their primary target is not the liver. (12 Dec 1998) |
| virus hepatitis of ducks | A disease of very young ducklings, caused by the duck hepatitis virus (family Hepadnoviridae) and manifested as an acute illness of several days followed by death; the principal lesions are an enlarged necrotic liver filled with ecchymotic haemorrhages. (05 Mar 2000) |
| virus, human papilloma | A family of over 60 viruses responsible for causing warts. The majority of the viruses produce warts on the hands, fingers, and even the face. most of these viruses are innocuous, causing nothing more than cosmetic concerns. Several types of HPV are confined primarily to the moist skin of the genitals, producing genital warts and elevating the risk for cancer of the cervix. These viruses that cause wartlike growths on the genitals and contribute to cancer of the cervix are sexually transmitted. (12 Dec 1998) |
| virus III of rabbits | An obsolete name for a latent herpesvirus infection of rabbits. Origin: the third strain isolated, used for study (05 Mar 2000) |
| virus integration | Insertion of viral DNA into host-cell DNA. This includes integration of phage DNA into bacterial DNA (lysogeny) to form a prophage or integration of retroviral DNA into cellular DNA to form a provirus. (12 Dec 1998) |
| virus keratoconjunctivitis | Follicular conjunctivitis followed by subepithelial corneal infiltrates; often caused by adenovirus type 8, less commonly by other types. Synonym: virus keratoconjunctivitis. (05 Mar 2000) |
| virus latency | The ability of a pathogenic virus to lie dormant within a cell (latent infection). In eukaryotes, subsequent activation and viral replication is thought to be caused by extracellular stimulation of cellular transcription factors. Latency in bacteriophage is maintained by the expression of virally encoded repressors. (12 Dec 1998) |
| virus pneumonia of pigs | A worldwide chronic pneumonia usually involving only the anterior lobes; it seldom causes death but is responsible for much unthriftiness; it is caused by the bacterium Mycoplasma hyopneumoniae. Synonym: virus pneumonia of pigs. (05 Mar 2000) |
| virus replication | The process of intracellular viral multiplication, consisting of the synthesis of proteins, nucleic acids, and sometimes lipids, and their assembly into a new infectious particle. (12 Dec 1998) |
| virus, respiratory syncytial | A virus that causes mild respiratory infections (colds and coughs) in adults but in young children can produce severe respiratory problems (bronchitis and pneumonia). Effective immunity against rsv requires a continuous solid level of antibodies against the virus. There is particular concern for rsv in premature babies because of their lack of maturity and lack of antibodies. (12 Dec 1998) |
| virus shedding | The expelling of virus particles from the body. Important routes include the respiratory tract, genital tract, and intestinal tract. Virus shedding is an important means of vertical transmission (disease transmission, vertical). (12 Dec 1998) |
| virus-transformed cell | A cell that has been genetically changed to a tumour cell, the change being subsequently tramsmitted to all descendent cell's; cell's transformed by oncornaviruses continue to produce virus in high concentration without being killed; DNA tumour virus-transformed cell's develop (along with other changes) tumour-associated antigens and rarely produce virus. (05 Mar 2000) |