| CMD | campomelic dysplasia; camptomelic dwarfism; cartilage matrix deficiency; chief medical director; chi... |
|---|---|
| CuD | copper deficiency |
| DDD | AV universal [pacemaker]; defined daily dose; degenerative disc disease; dehydroxydinaphthyl disulfi... |
| def | defecation; deficiency, deficient; deferred |
| defic | deficiency, deficient |
| liver of sulfur | A mixture composed chiefly of potassium polysulfides and potassium thiosulfate; used externally in scabies, acne, and psoriasis; used in the manufacture of "white lotion". Synonym: liver of sulfur. (05 Mar 2000) |
|---|---|
| liver profile | <investigation> A test that measures the blood serum level of several enzymes produced by the liver. An elevated liver function test is a sign of possible liver damage. Examples include: prothrombin time, PTT, serum glutamic oxaloacetic transaminase, serum glutamic pyruvic transaminase, gamma glutamyl transpeptidase, alkaline phosphatase and serum albumin. (14 Oct 1997) |
| liver regeneration | Repair or renewal of hepatic tissue. (12 Dec 1998) |
| liver scan | <investigation> A way of visualising the liver by injecting into the bloodstream a trace dose of a radioactive substance which helps visualize the organ during X-ray. (09 Oct 1997) |
| liver-shod clamp | A clamp with jaws covered by cloth to prevent injury to structures such as bowel when clamp is closed. (05 Mar 2000) |
| liver-spleen scan | <radiology> Tc-99m sulfur colloid or albumin colloid, particles less than 1 m, dose = 4-8 mCi Distribution, liver 85%, spleen 10%, bone marrow 5% Findings, liver: hot / cold, spleen: hot / cold (12 Dec 1998) |
| liver spot | A variably pigmented lentigo occurring on exposed skin of older Caucasians. Synonym: liver spot, solar lentigo. (05 Mar 2000) |
| liver spots | A skin disorder seen with aging (or sun exposure) where there are flat patches of increased pigmentation on the skin. They are nonserious, benign and generally more important cosmetically. The most commonly appear on sun exposed areas such as the forehead and the back of the hands. Avoidance of the sun and the use of a sunscreen (SPF of 15) can reduce the incidence of these lesions. Creams and lotions are available which bleach the skin. Cryotherapy can be used in some cases. (27 Sep 1997) |
| liver starch | <biochemistry> Branched polymer of D glucose (mostly _(1-4) linked, but some _(1-6) at branch points). Size range very variable, up to 10exp5 glucose units. Major short term storage polymer of animal cells and is particularly abundant in the liver and to a lesser extent in muscle. In the electron microscope glycogen has a characteristic asterisk or star appearance. (18 Nov 1997) |
| liver transplant | <procedure, surgery> Surgery to replace a disease liver with a healthy one from a donor. This procedure is technically quite difficult, since both vessels and biliary tract must be reattached to the new liver. most often indicated in those with long-term chronic hepatitis or congenital liver defects. Not usually performed in the situation of alcoholic cirrhosis or primary biliary cirrhosis. (14 Oct 1997) |
| liver transplantation | The transference of a part of or an entire liver from one human or animal to another. (12 Dec 1998) |
| abdominal muscle deficiency syndrome | <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear. (05 Mar 2000) |
| adult lactase deficiency | Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults. (05 Mar 2000) |
| alpha-1 antitrypsin deficiency | <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues. The lack of this protein leads to damage of various organs, but mainly to the lung and liver. symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant (12 Dec 1998) |
| alpha-1-proteinase deficiency | Absence of a serum proteinase inhibitor that may cause nodular non-suppurative panniculitis. (05 Mar 2000) |