| ¿µ¹® | pneumonia | ÇÑ±Û | Æó·Å |
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| ¼³¸í | ÆóÀÇ ¿°ÁõÀ¸·Î ÆóÆ÷ ³»¿¡ °ø±â ´ë½Å ¿°Áõ ¼¼Æ÷³ª »ïÃâ¾×À¸·Î °¡µæ Â÷ È£Èí°ï¶õÀ» ¾ß±âÇϸç, ¹ß¿ µîÀÇ Àü½Å Áõ»óÀ» µ¿¹ÝÇÑ´Ù. À§Ä¡, ¿øÀÎ±Õ µî¿¡ µû¶ó ±¸ºÐÇÑ´Ù. ¿¹¸¦ µé¾î, ´ë¿±¼º Æó·Å(lobar pneumonia)¶õ Æó·Å ±¸±Õ¿¡ ÀÇÇÑ ±Þ¼º Æä·ÅÀ¸·Î ÇÑ °³ ¶Ç´Â ¿©·¯ °³ÀÇ Æó¿±À» µû¶ó »ý±â´Â ±¤¹üÀ§ÇÑ »ïÃ⼺ °æÈ°¡ Ư¡ÀÎ º´À» ÁöĪÇÑ´Ù. À̰ÍÀº Æó·ÅÀÇ ¹ß»ýºÎÀ§¿¡ µû¶ó ºÎ¸£´Â ¸»ÀÌ´Ù. ±×¸®°í ¹ÙÀÌ·¯½ºÆó·Å(viral pneumonia)¶õ ¹ÙÀÌ·¯½º¿¡ ÀÇÇØ ¹ß»ýÇÏ´Â ¸ðµç Æó·ÅÀ» ¸»Çϴµ¥, À̰ÍÀº ¿øÀαտ¡ µû¶ó ÁöĪÇÑ ¸»ÀÌ´Ù. |
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| ¿µ¹® | aspiration pneumonia | ÇÑ±Û | ÈíÀÎÆó·Å |
|---|---|---|---|
| ¼³¸í | À½½Ä¹°ÀÇ Â±â°°Àº À̹°ÀÌ ±âµµ¿¡ µé¾î°¡ ÀÌÂ÷ÀûÀ¸·Î »ý±â´Â Æó·Å. À½½Ä¹°À̳ª ÀÔ¾ÈÀÇ ¹Ì»ý¹°ÀÌ ½Äµµ·Î ³Ñ¾î°¡Áö ¾Ê°í ±âµµ·Î À߸ø ÈíÀÎµÇ¾î ¾ß±âµÇ´Â Æó·ÅÀ» ¸»ÇÑ´Ù. ÈíÀÎ ¹°ÁúÀÇ Æ¯¼º¿¡ µû¶ó¼ ¼¼ °¡Áö ÁõÈıºÀ» Æ÷ÇÔÇÑ´Ù. ¨ç ÈÇÐÀû Æó·ÅÀº Á÷Á¢ÀûÀ¸·Î Æó¿¡ À¯ÇØÇÑ ¹°ÁúÀ» ÈíÀÎÇßÀ» ¶§ ÀϾ¸ç, ±Þ¼º È£Èí°ï¶õ, ºü¸¥ È£Èí, ºü¸¥¸ÆÀÌ ³ªÅ¸³ª°í µ¿¹ÝµÇ´Â Áõ»óÀº ÁַΠû»öÁõ, ±â°üÁö °æ·Ã, ¿ µîÀÌ´Ù. ¨è Çϱ⵵ÀÇ ¼¼±Õ¼º °¨¿°ÀÌ ÈíÀμº Æó·ÅÀÇ °¡Àå ÈçÇÑ ÇüÅ·Î, ±âħ, ¹ß¿, °í¸§°¡·¡ Áõ»óÀ» ³ªÅ¸³½´Ù. ¨é Çϱ⵵ÀÇ ±â°èÀû Æó¼â´Â Ưº°ÇÑ ¹°Áú, ƯÈ÷ ¶¥Äá, ÀÛÀº °í±âµ¢¾î¸®¿¡ ÀÇÇØ ÈíÀεǾúÀ» ¶§ ÀϾ¸ç, ÈíÀÎ ¹°Áú°ú ±âµµÀÇ Á÷°æ¿¡ µû¶ó Áõ»óÀº ´Ù¸£´Ù. ±â°ü¿¡¼ ¸·È÷¸é Á¾Á¾ È£Èí°ï¶õ°ú ÇÔ²² »ç¸Á¿¡ À̸£±âµµ Çϸç, Çϱ⵵ ¾Æ·¡ ºÎºÐ¿¡¼ ÈíÀÎÀÌ ÀϾ¸é ¸¸¼º ±âħÀÌ ¹ß»ýÇÑ´Ù. |
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| AP | accessory pathway; accounts payable; acid phosphatase; acinar parenchyma; action potential; active p... |
|---|---|
| APSGN | Acute Post-Streptococcal Glomerulo-Nephritis; ¿¬¼â»ó ±¸±Õ°¨¿°ÈÄ ±Þ¼º »ç±¸Ã¼ ½Å¿° |
| PSGN | Post-Streptococcal Glomerulo-Nephritis |
| EOGBS | early onset group B streptococcal [infection] |
| GRABS | group A beta-hemolytic streptococcal pharyngitis |
| GAS | Group A Streptococcal |
|---|---|
| GABHS | Group A beta hemolytic streptococcal |
| GBS | Group B Streptococcal |
| PSGN | Post-streptococcal glomerulonephritis |
| STSS | Streptococcal Toxic Shock Syndrome |
| streptococcal pneumonia | Pneumonia due to Streptococcus pyogenes. (05 Mar 2000) |
|---|
| acute post-streptococcal glomerulonephritis | <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood). Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis). (27 Sep 1997) |
|---|---|
| group A streptococcal necrotizing fasciitis | A complication of infection with GAS (group A streptococci) in which the bacteria attacks and destroys muscle tissue. According to the CDC, 5-10% of people with severe GAS infection develop necrotizing fasciitis. Though the infection can be treated with antibiotics, the fatality rate is close to 30%. This complication often develops as a wound infection after surgery or injury. (05 Mar 2000) |
| streptococcal | Relating to or caused by any organism of the genus Streptococcus. (05 Mar 2000) |
| streptococcal fibrinolysin | <enzyme> Plasminogen activator released by Streptococcus pyogenes. Occurs in two forms, A and B. (18 Nov 1997) |
| streptococcal infections | Infections with bacteria of the genus streptococcus. (12 Dec 1998) |
| streptococcal lymphadenitis | A contagious bacterial disease of pigs caused by a group E streptococcus and characterised by the formation of abscesses in the cervical and/or cephalic lymph nodes. (05 Mar 2000) |
| streptococcal nuclease | <enzyme> From streptococcus haemolyticus; degrades RNA and DNA producing oligonucleotides terminating in 5'-phosphate Registry number: EC 3.1.30.- (26 Jun 1999) |
| streptococcal toxins | Group of haemolytic exotoxins released by Streptococci. _ haemolysin: 26-39 Kd (four types), forms ring like structures in membranes (see Streptolysin O). Lipid target unclear. _ haemolysin: a hot cold haemolysin with sphingomyelinase C activity. _ haemolysin: complex of two proteins (29 and 26 kD) that act synergistically, rabbit erythrocytes particularly sensitive. _ toxin: heat stable peptide (5 kD) with high proportion of hydrophobic amino acids. Seems to act in a detergent like manner (c.f. Subtilysin), but may form hydrophilic transmembrane pores by cooperative interaction with other _ toxin molecules. Leucocidin (Panton Valentine leucocidin): two components f (fast migration on CM cellulose column: 32 kD) and s (slow: 38 kD). Mode of action contentious. See: Streptococcus, streptolysins O and S, erythrogenic toxin. (18 Nov 1997) |
| acute interstitial pneumonia | A severe and usually fatal form of pneumonia occurring primarily in infants usually considered a form of hypersensitivity pneumonitis. (05 Mar 2000) |
| alcoholic pneumonia | Pneumonia occurring in patient with alcoholism, usually after a period of intoxication with stupor, resulting in aspiration. (05 Mar 2000) |
| anthrax pneumonia | A form of anthrax acquired by inhalation of dust containing Bacillus anthracis; there is an initial chill followed by pain in the back and legs, rapid respiration, dyspnea, cough, fever, rapid pulse, and extreme cardiovascular collapse. Synonym: anthrax pneumonia, ragpicker's disease, ragsorter's disease, rag-sorter's disease, wool-sorter's pneumonia, woolsorter's disease, wool-sorter's disease. (05 Mar 2000) |
| apex pneumonia | Apical pneumonia, pneumonia of the apex or apices. (05 Mar 2000) |
| aspiration pneumonia | <chest medicine> Refers to the inappropriate passage of food, water, stomach acid, vomit or another foreign material into the lungs. Aspiration, particularly involving gastric acid, will often result in a serious pneumonia. (27 Sep 1997) |
| atypical pneumonia | <chest medicine> This refers to a type of pneumonia that does not follow the typical signs and symptoms of pneumonia. A number of different viral and bacterial agents have been identified which can cause this form of respiratory infection. Examples include Chlamydia pneumonia, psittacosis, Mycoplasma, influenza A or B, adenovirus and Legionella. Antibiotics will be necessary in all but the mildest cases. Symptoms generally improve in less than 2 weeks. (27 Sep 1997) |
| bacterial pneumonia | Infection of the lung with any of a large variety of bacteria, especially Streptococcus pneumoniae(pneumococcus). (05 Mar 2000) |
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