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ÀÇÇпë¾î »çÀü
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ÀÇÇлçÀü
ÇÑ¿µ/¿µÇÑ»çÀü
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»çÀÌÆ® ¼Ò°³
Àç°Ë»ö
"sequence hypothesis"¿¡ ´ëÇÑ ´ëÇÑÀÇÇù ÀÇÇпë¾î ¼¼ºÎ °Ë»ö °á°úÀÔ´Ï´Ù
À̰ÍÀ» ¿øÇϼ̽À´Ï±î?
swquence hypothesis
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
hypothesis
°¡¼³
lattice hypothesis
°ÝÀÚ°¡¼³
null hypothesis
¿µ°¡¼³, ±Í¹«°¡¼³
unitarian hypothesis
Ç×üµ¿Àϰ¡¼³
adenoma-carcinoma sequence
»ùÁ¾¾ÏÁ¾¿¬¼â, ¼±Á¾¾ÏÁ¾¿¬¼â
amino acid sequence
¾Æ¹Ì³ë»ê¼ø¼
gradient echo pulse sequence
±â¿ï±â¸Þ¾Æ¸®ÆÞ½º¿¬¼â
inversion recovery pulse sequence
¿ªÀüȸº¹ÆÞ½º¿¬¼â
imaging sequence
¿µ»ó¿¬¼â
insertion sequence
»ðÀÔ¼¿
intervening sequence
°³Àç¼ø¼
oligohydroamnios sequence
¾ç¼ö°ú¼ÒÁõ¿¬¼â
pulse sequence
¹Úµ¿¿¬¼â, ÆÞ½º¿¬¼â
packaging sequence
²Ù¸®±â¼ø¼
rapid-sequence induction
ºü¸¥¿¬¼Ó¸¶ÃëÀ¯µµ
´ÙÀ½
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
4
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
hypothesis
°¡¼³
null hypothesis
±Í¹«°¡¼³, ¿µ°¡¼³
sequence
¼ø¼, ¿¬¼â
reaction sequence
¹ÝÀÀ¼ø¼
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
blending hypothesis
À¶ÇÕÀ¯Àü¼³
central plasticity hypothesis
ÁßÃßÀ¯¿¬¼º°¡¼³
countercurrent hypothesis
¿ª·ù°¡¼³
drift hypothesis
À̵¿°¡¼³
dual recognition hypothesis
Ç׿øÀÌÁßÀÎÁö°¡¼³
estrogen window hypothesis
¿¡½ºÆ®·Î°Õâ°¡¼³
expectation hypothesis
¿¹Ãø°¡¼³
hypothesis
°¡¼³
hypoxia-selectivity hypothesis
Àú»ê¼ÒÁõ¼±Åõµ°¡¼³
lattice hypothesis
°ÝÀÚ°¡¼³
lipid hypothesis
ÁöÁú°¡¼³
metabolic hypothesis
´ë»ç°¡¼³
molecular hypothesis
ºÐÀÚ°¡¼³
monoclonal hypothesis
´ÜÀÏŬ·Ð°¡¼³
null hypothesis
±Í¹«°¡¼³
´ÙÀ½
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
Glucostatic hypothesis, of appetite regulation
Ç×´ç±â¼³(ù÷ÓØÐñæò), ½Ä¿åÁ¶Àý(ãÝé¯ðàï½)
Lyon s hypothesis
¶óÀ̿°¡¼³.
Starling s hypothesis
½ºÅ¸¾Ë¸µ°¡¼³.
hypothesis, lattice
°ÝÀÚ¼³
receptor hypothesis
¼ö¿ëü°¡¼³
Carr Purcell sequence Carr Pursell
¿¬¼â
DNA sequence analysis
DNA ¼¿ºÐ¼®
FID pulse sequence
ÀÚÀ¯ ºØ±« À¯µµ ÆÞ½º ¿¬¼â
RF pulse sequence
°íÁÖÆÄ ÆÞ½º ¿¬¼â
adenoma-carcinoma sequence
¼±Á¾-¾ÏÁ¾ ¿¬¼â(àÍðþ-äßðþ ææáð)
amino acid sequence
¾Æ¹Ì³ë»ê ¼¿.
imaging of STIR sequence
STIR ¿¬¼â ¿µ»ó
imaging sequence
¿µ»ó ¿¬¼â
insertion sequence, IS
»ðÀÔ¼¿, »ðÀÔ¿ä¼Ò
porters sequence
Æ÷ÅÍ¿¬¼â(¡ææáð), Æ÷Å͹迼ø(¡ÛÕÖªâ÷)
´ÙÀ½
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú :
15
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
atherosclerosis,monoclonal hypothesis
´ÜÀÏŬ·Ð¼º°¡¼³(Ó¤ìé¡Ê£àã)
atherosclerosis,reaction to injury hypothesis
¼Õ»ó¿¡ ´ëÇÑ ¹ÝÀÀ±âÀü
atomic hypothesis
¿øÀÚ°¡¼³(ê«íÊ£æò).
blending hypothesis
À¶ÇÕÀ¯Àü¼³(¡ë¶îîæò).
countercurrent hypothesis
¿ª·ù°¡¼³ (¡Ê£æò).
cyclol hypothesis
»çÀÌŬ·Ñ ´ÜÀ§°¡¼³(¡Ó¤êÈÊ£æò).
dopamine hypothesis
µµÆÄ¹Î °¡¼³
drift hypothesis
À̵¿°¡¼³
dual recognition hypothesis
Ç׿øÀÌÁßÀÎÁö°¡¼³
emphysema,protease-antiprotease hypothesis
´Ü¹é-Ç״ܹéºÐÇØÈ¿¼Ò °¡¼³
estrogen window hypothesis
¿¡½ºÆ®·ÎÁ¨ ±¸°£°¡¼³
expectation hypothesis
¿¹Ãø°¡¼³(çãö´Ê£àã)
fractional kill hypothesis
hypothesis, lattice
°ÝÀÚ¼³
lipid hypothesis
ÁöÁú°¡¼³
´ÙÀ½
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú :
0
ÆäÀÌÁö:
1
¿µ¹®
ÇѱÛ
ÅëÇÕ°Ë»ö ¿Ï·á
´ÙÀ½