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"receptors, ldl"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • LDL
    Àú¹ÐµµÁöÁú´Ü¹éÁú
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  • ¿µ¹®
    ÇѱÛ
  • LDL
    (¢¡low density lipoprotein) Àú¹ÐµµÁöÁú´Ü¹é
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  • ¿µ¹®
    ÇѱÛ
  • LDL = low density lipoprotein
    Àú¹ÐµµÁö´Ü¹é(î¸ÚËÓøò·Ó±ÛÜ)
  • GABA A receptors
    GABA A ¼ö¿ëü(áôé»ô÷)
  • GABA receptors
    GABA ¼ö¿ëü(áôé»ô÷)
  • GABAB receptors
    GABA B ¼ö¿ëü(áôé»ô÷)
  • H1 receptors
    H1¼ö¿ëü(â¥é»ô÷)
  • HT receptors
    5-ÇÏÀ̵å·Ï½ÃÆ®¸³Å¸¹Î¼ö¿ëü
  • HT2 receptors, action of hallucinogens
    5-ÇÏÀ̵å·Ï½ÃÆ®¸³Å¸¹Î2¼ö¿ëü
  • Hair follicle receptors
    ¸ð³¶¼ö¿ëü(Ù¾Ò¥â¥é»ô÷)
  • Hair receptors
    ¸ð¼ö¿ëü(Ù¾â¥é»ô÷)
  • Heat receptors
    ¿­¼ö¿ëü(æðâ¥é»ô÷)
  • Histamine receptors
    È÷½ºÅ¸¹Î¼ö¿ëü
  • Immunoglobulin adhesion receptors
    ¸é¿ª±Û·ÎºÒ¸° ºÎÂø(ݾó·)¼ö¿ëü
  • Irritant receptors, ventilatory responses , ventilatory responses
    Àڱؼö¿ëü(í©Ð½áôé»ô÷), È£Èí¹ÝÀÀ(û¼ýåÚãëë)
  • J receptors, ventilatory responses mediated
    J ¼ö¿ëü(áôé»ô÷), È£Èí¹ÝÀÀ(û¼ýåÚãëë)
  • Kainate receptors
    Ä«À̳×ÀÌÆ®¼ö¿ëü(áôé»ô÷)
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    ÇѱÛ
  • low density lipoprotein =LDL
    Àú¹ÐµµÁö´Ü¹é.
  • low density lipoprotein =LDL
    Àú¹ÐµµÁö´Ü¹éÁú.
  • atrial receptors
    (½É)¹æ¼ö¿ëü(¡­áôé»ô÷)
  • atrial receptors
    ½É¹æ¼ö¿ëü(¡­áôé»ô÷).
  • down-regulation (of receptors)
    (¼ö¿ëü)ÇÏÇâÁ¶Àý.
  • down-regulation (of receptors)
    ¼ö¿ëü(áôé»ô÷)ÇÏÇâÁ¶Àý(ù»ú¾ðàï½).
  • presynaptic receptors
    ½Å°æÁ¢ÇÕÀü ¼ö¿ëü
  • receptors
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  • ¿µ¹®
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  • LDL
    (å²) Low-density lipoprotein
  • LDL receptor
    LDL ¼ö¿ëü(áôé»ô÷)
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
AChRs Acetylcholine Receptors
RA radioactive; ragocyte; ragweed antigen; rapidly adapting [receptors]; reactive arthritis; reciprocal...
LDL Low Density Lipoprotein; Àú¹Ðµµ ÁöÁú ´Ü¹éÁú
LDL loudness discomfort level; low density lipoprotein
LDLR, LDL-R low density lipoprotein receptor
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
LDL - LDL
LDL-C LDL cholesterol
N-LDL Native LDL
Ox-LDL Oxidatively modified LDL
acetyl-LDL acetylated LDL
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
receptors, ldl Receptors on the plasma membrane of nonhepatic cells that specifically bind ldl. The receptors are localised in specialised regions called coated pits. Hypercholesteraemia is caused by an allelic genetic defect of three types: 1) receptors do not bind to ldl; 2) there is reduced binding of ldl; and 3) there is normal binding but no internalization of ldl. In consequence, entry of cholesterol esters into the cell is impaired and the intracellular feedback by cholesterol on 3-hydroxy-3-methylglutaryl CoA reductase is lacking.
(12 Dec 1998)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
cholesterol, ldl Low-density lipoprotein cholesterol (the bad cholesterol).
(12 Dec 1998)
LDL <biochemistry> A lipoprotein substances (combination of a fat and a protein) which acts as a carrier for cholesterol and fats in the bloodstream.
High levels of low density lipoprotein are considered a positive risk factor for the development of coronary artery disease. Less than 130 mg/dl is desirable, 130 to 159 mg/dl is borderline high, over 160 is considered high.
Acronym: LDL
(10 Jan 1998)
ldl cholesterol Low-density lipoprotein cholesterol (the bad cholesterol).
(12 Dec 1998)
LDL receptor disorder Abnormality in clearance of LDL from the plasma due to abnormality in LDL receptor activity; causes hypercholesterolaemia.
(05 Mar 2000)
lipoproteins, ldl A class of lipoproteins responsible for transport of cholesterol to extrahepatic tissues. They are formed in the circulation when very-low-density lipoproteins are degraded first to intermediate-density lipoproteins and then to ldl by the gain and loss of specific apolipoproteins and the loss of most of their triglycerides. Ldl are taken up and catabolised by both the liver and extrahepatic tissues by specific receptor-mediated endocytosis.
(12 Dec 1998)
lipoproteins, ldl cholesterol Cholesterol which is contained in or bound to low density lipoproteins (ldl). Ldl transport cholesterol to peripheral tissues and regulate de novo cholesterol synthesis at these sites. Atherosclerosis is caused by the deposit of cholesterol on the walls of blood vessels, because of high concentrations of ldl cholesterol in plasma.
(12 Dec 1998)
adrenergic receptors Reactive components of effector tissues, most of which are innervated by adrenergic postganglionic fibres of the sympathetic nervous system. Such receptor's can be activated by norepinephrine and/or epinephrine and by various adrenergic drugs; receptor activation results in a change in effector tissue function, such as contraction of arteriolar muscles or relaxation of bronchial muscles; adrenergic receptor's are divided into alpha-receptor's and beta-receptor's, on the basis of their response to various adrenergic activating and blocking agents.
Synonym: adrenoceptor, adrenoreceptors.
(05 Mar 2000)
alpha-adrenergic receptors Adrenergic receptor's in effector tissues capable of selective activation and blockade by drugs; conceptually derived from the ability of certain agents, such as phenoxybenzamine, to block only some adrenergic receptor's and of other agents, such as methoxamine, to activate only the same adrenergic receptor's. Such receptor's are designated as alpha-receptors. Their activation results in physiological responses such as increased peripheral vascular resistance, mydriasis, and contraction of pilomotor muscles.
(05 Mar 2000)
ANP clearance receptors Cell surface proteins that bind atrial natriuretic peptide and ANP fragments without initiating biological action.
(05 Mar 2000)
ANP receptors Cell surface receptors for atrial natriuretic peptide that have a single transmembrane spanning element; these have integral kinase and guanylate cyclase domains.
(05 Mar 2000)
B-cell antigen receptors In the primary immune response immunoglobulin D and monomeric immunoglobulin M are the B-cell antigen receptors. On memory B-cells, other immunoglobulin molecules can serve as antigen receptors.
(05 Mar 2000)
beta-adrenergic receptors Adrenergic receptor's in effector tissues capable of selective activation and blockade by drugs; conceptually derived from the ability of certain agents, such as propranolol, to block only some adrenergic receptor's and of other agents, such as isoproterenol, to activate only the same adrenergic receptor's. Such receptor's are designated as beta-receptors. Their activation results in physiological responses such as increases in cardiac rate and force of contraction (b1), and relaxation of bronchial and vascular smooth muscle (b2).
(05 Mar 2000)
mannose-6-phosphate receptors Receptors in Golgi apparatus to which newly synthesised proteins that are destined to enter lysosomes bind.
(05 Mar 2000)
receptors, adrenergic Cell-surface proteins that bind epinephrine and/or norepinephrine with high affinity and trigger intracellular changes. The two major classes of adrenergic receptors, alpha and beta, were originally discriminated based on their cellular actions but now are distinguished by their relative affinity for characteristic synthetic ligands. Adrenergic receptors may also be classified according to the subtypes of g-proteins with which they bind; this scheme does not respect the alpha-beta distinction.
(12 Dec 1998)
receptors, adrenergic, alpha One of the two major pharmacological subdivisions of adrenergic receptors. The alpha-beta distinction was originally based on cellular effects of receptor activation but now relies on the relative affinities for certain synthetic ligands. Alpha-adrenergic receptors are further subdivided into several subclasses based on studies of endogenous and cloned receptors.
(12 Dec 1998)
MeSH(Medical Subject Headings) ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú : 1 ÆäÀÌÁö: 1
  • Receptors, LDL - »õâ Receptors on the plasma membrane of nonhepatic cells that specifically bind LDL. The receptors are localized in specialized regions called coated pits. Hypercholesteremia is caused by an allelic genetic defect of three types: 1, receptors do not bind to LDL; 2, there is reduced binding of LDL; and 3, there is normal binding but no internalization of LDL. In consequence, entry of cholesterol esters into the cell is impaired and the intracellular feedback by cholesterol on 3-hydroxy-3-methylglutaryl CoA reductase is lacking.
    Synonyms : LDL Receptor, LDL Receptors, Lipoprotein, Low Density Lipoprotein Receptor, Low Density Lipoprotein Receptors, Receptors, Lipoprotein, LDL, Receptor, LDL, Receptors, Lipoprotein LDL
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