| metabolic alkalosis | <biochemistry> A metabolic derangement where the pH of the blood is abnormally high (basic). This condition may result from hyperventilation, the use of a particular drug, excessive vomiting or dehydration (contraction alkalosis). (27 Jun 1999) |
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| compensated metabolic alkalosis | The retention of acid, primarily carbon dioxide by the lung and acid ions by the renal tubules, to reduce the effect on the pH of the blood of excess alkali produced by ingestion or metabolism of alkali-producing substances. (05 Mar 2000) |
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| acapnial alkalosis | The alkalosis resulting from abnormal loss of CO2 produced by hyperventilation, either active or passive, with concomitant reduction in arterial plasma bicarbonate concentration. See: compensated alkalosis. Synonym: acapnial alkalosis. (05 Mar 2000) |
| alkalosis | <biochemistry> A pathologic condition resulting from accumulation of base or from loss of acid without comparable loss of base in the body fluids and characterised by decrease in hydrogen ion concentration (increase in pH). Compare: acidosis. (18 Nov 1997) |
| alkalosis, respiratory | A state due to excess loss of carbon dioxide from the body. (12 Dec 1998) |
| respiratory alkalosis | The alkalosis resulting from abnormal loss of CO2 produced by hyperventilation, either active or passive, with concomitant reduction in arterial plasma bicarbonate concentration. See: compensated alkalosis. Synonym: acapnial alkalosis. (05 Mar 2000) |
| compensated alkalosis | Alkalosis in which there is a change in bicarbonate but the pH of body fluids approaches normal; respiratory alkalosis may be compensated by increased production of metabolic acids or increased renal excretion of bicarbonate; metabolic alkalosis is rarely compensated by hypoventilation. (05 Mar 2000) |
| compensated respiratory alkalosis | The increased excretion of acid ions by the kidney to minimise the effect on the pH of the blood of excessive loss of carbon dioxide via the lungs, such as occurs with hyperventilation. (05 Mar 2000) |
| tetany of alkalosis | Tetany due to a loss of acid from the body or an increase in alkali, resulting in a reduction of ionised calcium in plasma and body fluids, e.g., hyperventilation tetany (loss of CO2), gastric tetany (loss of HCl by vomiting), or injection or ingestion of excessive amounts of sodium bicarbonate. (05 Mar 2000) |
| uncompensated alkalosis | Alkalosis in which the pH of body fluids is elevated because of lack of the compensatory mechanisms of compensated alkalosis. (05 Mar 2000) |
| basal metabolic rate | <biochemistry, biology> The metabolic rate as measured under basal conditions: 12 hours after eating, after a restful sleep, no exercise or activity preceding test, elimination of emotional excitement and occurring in a comfortable temperature. Acronym: BMR (15 Nov 1997) |
| brain diseases, metabolic | Metabolic disorders which lead to pathological changes and/or functional deviations of the brain. (12 Dec 1998) |
| rate, basal metabolic | A measure of the rate of metabolism. For example, someone with an overly active thyroid will have an elevated basal metabolic rate. (12 Dec 1998) |
| metabolic | 1. <biology> Of or pertaining to metamorphosis; pertaining to, or involving, change. 2. <physiology> Of or pertaining to metabolism; as, metabolic activity; metabolic force. Source: Websters Dictionary (01 Mar 1998) |
| metabolic acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly. (27 Jun 1999) |
| metabolic burst | <biochemistry> Response of phagocytes to particles (particularly if opsonise d) and to agonists such as formyl peptides and phorbol esters, an enhanced uptake of oxygen leads to the production, by an NADH dependent system, of hydrogen peroxide, superoxide anions and hydroxyl radicals, all of which play a part in bactericidal activity. Defects in the metabolic burst, as in chronic granulomatous disease, predispose to infection particularly with catalase positive bacteria and are usually fatal in childhood. (27 Jun 1999) |