| 영문 | poststreptococcal glomerulonephritis | 한글 | 사슬알균감염후토리콩팥염, 연쇄구균감염후사구체신염 |
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| 설명 | 점차 감소추세이기는 하나 어린이 콩팥병으로 아주 흔하다. 사슬알균 감염, 특히 상기도나 피부 감염 후 1주 내지 2주 이내에 급성콩팥염증후군이 갑자기 발현되는 특징을 보인다. 성인에서도 발생할 수 있으나 비교적 드물고 그 임상소견이 다소 다를 수 있다. 발생메커니즘은 면역으로 해석하고 있다. |
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| 영문 | malignant tumor | 한글 | 악성종양 |
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| 설명 | 정상적인 조직 세포가 각종 물리적-화학적-생물학적인 발암 물질의 작용 또는 요인에 의해 돌연변이를 일으켜서 형성되는 종양. 무제한의 세포분열로 매우 왕성하게 증식하여 주위조직을 파괴-침식한다. 또 어떤 화학물질을 내어 주위의 조직세포를 침해할 뿐만 아니라, 혈관 및 림프관을 따라 전이하여 전신의 카켁시아를일으켜 죽음을 초래한다. 상피성인 것을 암종이라 하고, 비상피성인 것을 육종이라 한다. |
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| 영문 | malignant melanoma | 한글 | 악성흑색종 |
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| 설명 | 피부에서 생기는 암의 일종. 주로 백인에게 호발하며, 50대~70대에 많이 발생하고, 젊은 여성에게 증가하는 경향이 있으며, 피부암중 1~3%를 차지하는 드문 병으로 피부와 기타 기관의 멜라닌 세포계에서 유래하는 악성종양이다. 원인으로 일광노출과 직접적인 관계가 있으며 가족적 발생도 볼 수 있다. |
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| 영문 | glomerulonephritis | 한글 | 토리콩팥염, 사구체신염 |
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| 설명 | 토리콩팥염이란 이 토리의 염증을 주로 하는 콩팥병을 말한다. 이 토리콩팥염의 진단은 하지만 현미경적 관찰로 토리의 염증을 보고 진단하는 것이 아니라 임상증세로 진단하는 것이므로 다분히 임상적인 진단명이다. 즉 심하게 말하면 실제로 토리의 염증이 없는 경우에도 임상적 진단 기준에만 합당한 경우에는 토리콩팥염으로 진단이 내려질 수가 있다. 토리콩팥염은 급성과 만성의 두 가지로 나눌 수가 있다. 1.급성토리콩팥염－갑작스럽게 발생하는 혈뇨, 단백뇨, 그리고 콩팥의 기능부전으로 인해서 수분이나 전해질의 체내 축적, 불순물의 체내축적이 있는 병을 말한다. 이 급성 토리콩팥염을 일으키는 원인으로는 여러가지가 있지만 대표적인 것으로는 대부분 감염(사슬알균, 포도알균, 폐렴알균, 여러 바이러스 등)후에 초래되는 감염후 토리콩팥염이 있다. 여러 감염이 급성 토리콩팥염을 일으키나 특히 사슬알의 감염이 가장 전형적인 예라고 할 수 있다. 2.만성토리콩팥염-지속적인 요검사상의 이상, 즉 단백뇨, 혈뇨와 신장의 기능이 서서히 없어지는 질환군이다. 흔히 말기콩팥이라 부르며 거의 모든 콩팥의 병이 이 만성 토리콩팥염으로 진행된다. |
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| MH | malignant histiocytosis; malignant hyperpyrexia; malignant hypertension; malignant hyperthermia; mam... |
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| MCGN | mesangiocapillary glomerulonephritis; minimal change glomerulonephritis; mixed cryoglobulinemia with... |
| MHS | major histocompatibility system; malignant hyperthermia in swine; malignant hyperthermia syndrome; m... |
| AGN | acute glomerulonephritis; agnosia |
| APSGN | acute poststreptococcal glomerulonephritis |
| APSGN | Acute Poststreptococcal Glomerulonephritis |
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| AGN | Acute glomerulonephritis |
| AGN | Acute poststreptococcal glomerulonephritis |
| CGN | Chronic glomerulonephritis |
| CGN | Crescentic glomerulonephritis |
| acute crescentic glomerulonephritis | <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease. Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents. (27 Sep 1997) |
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| acute glomerulonephritis | <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood). Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis). (27 Sep 1997) |
| acute haemorrhagic glomerulonephritis | <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood). Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis). (27 Sep 1997) |
| acute post-streptococcal glomerulonephritis | <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood). Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis). (27 Sep 1997) |
| anti-basement membrane glomerulonephritis | Glomerulonephritis resulting from anti-basement membrane antibodies, characterised by smooth linear deposits of IgG and C3 along glomerular capillary walls; includes rapidly progressive glomerulonephritis and glomerulonephritis in Goodpasture's syndrome. (05 Mar 2000) |
| Berger's focal glomerulonephritis | Glomerulonephritis affecting a small proportion of renal glomeruli which commonly presents with haematuria and may be associated with acute upper respiratory infection in young males, not usually due to streptococci; associated with IgA deposits in the glomerular mesangium and may also be associated with systemic disease, as in Henoch-Schonlein purpura. Synonym: Berger's disease, Berger's focal glomerulonephritis, focal nephritis, IgA nephropathy. (05 Mar 2000) |
| rapidly progressive glomerulonephritis | <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease. Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents. (27 Sep 1997) |
| glomerulonephritis | <nephrology> A variety of nephritis characterised by inflammation of the capillary loops in the glomeruli of the kidney. It occurs in acute, subacute and chronic forms and may be secondary to haemolytic streptococcal infection. Evidence also supports possible immune or autoimmune mechanisms. (18 Nov 1997) |
| glomerulonephritis, IgA | Chronic form of glomerulonephritis characterised by recurring haematuria with only slight proteinuria and by deposits of IgA immunoglobulin in the mesangial areas of the renal glomeruli. It usually occurs in young males. (12 Dec 1998) |
| glomerulonephritis, membranoproliferative | Chronic glomerulonephritis characterised histologically by proliferation of mesangial cells, increase in the mesangial matrix, and a thickening of the glomerular capillary walls. The wall thickening is sometimes said to be a result of the interposition of mesangial cytoplasm or matrix between the basement membrane and the endothelium of the capillary wall. It is often divided into types I and II, and sometimes III. Pathogenesis is not well understood, but some types are thought to involve the immune system, with the complement system, in particular, being implicated. (12 Dec 1998) |
| glomerulonephritis, membranous | A disease of the glomerulus manifested clinically by proteinuria, and sometimes by other features of the nephrotic syndrome. It is histologically characterised by deposits in the glomerular capillary wall between the epithelial cell and the basement membrane and a thickening of the membrane. Also characteristic are outward projections of the membrane between the epithelial deposits in the form of "spikes". There is some agreement that the deposits are antigen-antibody complexes. (12 Dec 1998) |
| membranoproliferative glomerulonephritis | <pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure. Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane. Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease. Acronym: MPGN (26 Mar 1998) |
| membranous glomerulonephritis | <pathology> A kidney disease that occurs due to inflammation of the kidney glomerulus and its basement membrane. The exact cause is unknown but it appears to be related to the deposition of immune complexes in the basement membrane leading to thickening of the capillary walls. This disorder is a common cause of nephrotic syndrome an is usually how the disease manifests. Risk factors include primary renal disease, malaria, hepatitis B, lupus, syphilis, cancers and non-Hodgkin's lymphomas. Risks also include exposure to some medications such as gold compounds and penicillamine. Mercury, trimethadione and some skin-lightening creams have also been implicated. Treatment includes systemic corticosteroids and immunosuppressive agents. (26 Mar 1998) |
| mesangial proliferative glomerulonephritis | <nephrology, pathology> Inflammation of the kidney glomerulus (blood filtering portion of the kidney) due to the abnormal deposition of IgM antibody in the mesangium layer of the glomerular capillary. A form of glomerulonephritis that appears to be caused by an abnormal immune response. This disorder generally manifests as nephrotic syndrome. Symptoms include swelling, dark urine, weight gain, hypertension, anorexia and bloody urine. Acronym: MPGN (05 Jan 1998) |
| mesangiocapillary glomerulonephritis | <nephrology, pathology> A kidney disorder which results in kidney dysfunction. Inflammation of the glomeruli result from an abnormal immune response and the deposition of antibodies within the kidney (glomerulus) ultrastructure. Membranoproliferative glomerulonephritis has been divided into two different types in the basis of where the antibodies are deposited in the glomerulus. MPGN type I, the more common type, deposits antibodies in the subendothelial layer of the basement membrane, whereas type II deposits antibodies in the bottom layer of the basement membrane. Symptoms include cloudy urine (pyuria), decreased urine output, swelling and hypertension. This disorder often results in end-stage renal disease. (27 Sep 1997) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|