| EF | ectopic focus; edema factor; ejection fraction; elastic fibril; electric field; elongation factor; e... |
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| FF | degree of fineness of abrasive particles; fat-free; father factor; fecal frequency; fertility factor... |
| IMDS | Idiopathic Myelo-Dysplastic Syndrome = Preleukemia = Refractory Anemi... |
| MDS | Myelo-Dysplastic Syndrome = Refractory (Dysmyelopoietic) Anemia = (Id... |
| DMN | dimethylnitrosamine; dorsal motor nucleus; dysplastic melanocytic nevus |
| DNS | Dysplastic Naevus Syndrome |
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| DMN | Dysplastic melanocytic nevi |
| DN | Dysplastic naevi |
| MCDK | Multicystic dysplastic kidney |
| FGD | Focus Group Discussions |
| multicystic dysplastic kidney disease | <radiology> most severe form of renal dysplasia, most common cause of palpaple abdominal mass in infants, unilateral (almost always; if bilateral, death occurs in utero), ureter absent or atretic, not associated with other cysts or with periportal fibrosis, Potter type II (12 Dec 1998) |
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| congenital dysplastic angiectasia | <syndrome> A congenital malformation syndrome characterised by the triad of asymmetric limb hypertrophy, haemangiomata, and nevi. Asymmetric limb hypertrophy is enlargement of one limb and not the corresponding limb on the other side, the enlarged limb being 3 times more likely to be a leg than an arm in ktw; and the limb enlargement is of bone as well as soft tissue. The haemangiomas, abnormal nests of blood vessels that proliferate inappropriately and excessively, cover a remarkable range from small innocuous capillary haemangiomas ( strawberry marks ) to huge cavernous haemangiomas. The nevi are pigmented moles on the skin; in ktw there are often also dark linear streaks on the skin, streaks due to too much pigment. There can be other abnormalities but the triad is the consistent clinical centrepiece of the disease. most persons with ktw have an enlarged leg and do relatively well without treatment or, for example, with only compression from an elastic stocking. Skin ulcers and other skin problems can occur over the swollen leg. Usually, the treatment is conservative. Surgery is almost never needed. The only possible exceptions are the very rare situations in which the leg reaches gigantic proportions or secondary clotting difficulties arise (due to trapping and destruction of blood platelets in a huge haemangioma). Then, amputation may become necessary. The cause of ktw syndrome is unknown. (12 Dec 1998) |
| congenital dysplastic angiomatosis | Autosomal dominant angiomatosis in which there is dysplasia of the underlying tissues, sometimes with overgrowth of bone (Klippel-Trenaunay-Weber syndrome), or encephalotrigeminal angiomatosis (Sturge-Weber syndrome) in which there is an angioma in the distribution of one or more branches of the trigeminal nerve, with vascular anomalies and calcification of the cerebral cortex. (05 Mar 2000) |
| dysplastic | Pertaining to or marked by dysplasia. (05 Mar 2000) |
| dysplastic nevi | Atypical moles; moles whose appearance is different from that of common moles. Dysplastic nevi are generally larger than ordinary moles and have irregular borders. Their colour often is not uniform; they usually are flat, but parts may be raised above the skin surface. (12 Dec 1998) |
| dysplastic nevus | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
| dysplastic nevus syndrome | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
| real focus | The point of meeting of convergent rays. (05 Mar 2000) |
| Ghon's focus | Calcification seen in pulmonary parenchyma (usually mid-lung area) and hilar nodes resulting from earlier, usually childhood, infection with tuberculosis. Synonym: Ghon's complex, Ghon's focus, Ghon's primary lesion. (05 Mar 2000) |
| virtual focus | The point from which divergent rays seem to proceed, or that at which they would meet if prolonged backward. (05 Mar 2000) |
| mink cell focus-inducing viruses | Murine leukaemia viruses discovered in 1976 by hartley, wolford, old, and rowe and so named because the viruses originally isolated had the capacity to transform cell foci in mink cell cultures. Mcf viruses are generated in a multi-step process by recombination with other viral types including akr, friend, moloney, and rauscher. (12 Dec 1998) |
| plasma focus | <radiobiology> The Plasma Focus is another device which depends on the pinch effect. Possible applications include both fusion and plasma propulsion, as well as other plasma research. In essence the plasma focus is generated by discharge of a current across the ends of two coaxial insulated conducting pipes. The Plasma Focus caused a huge stir when they generated copious neutrons, until it was discovered that the source of the neutrons was knockoffs from deuterium due to pinch accelerated electrons or ions. Plasma focus is sort of a point version of the Zpinch. (09 Oct 1997) |
| condenser, variable-focus | <microscopy> Essentially an Abbe condenser in which the upper lens element is fixed and the lower movable. The lower lens may be used to focus the illumination between the elements so that it emerges from the stationary lens as a large diameter parallel bundle. The field of low-power objectives may thus be filled without removing the top element. at the opposite extreme it can be adjusted to have a numerical aperture as high as 1.3. See: illumination, critical. (05 Aug 1998) |
| principal focus | The real or virtual meeting point of rays passing into a lens parallel to its axis. (05 Mar 2000) |
| spleen focus-forming viruses | Murine leukaemia viruses that are replication-defective and rapidly transforming. The envelope gene plays an essential role in initiating erythroleukaemia, manifested by splenic foci, splenomegaly, and polycythemia. Spleen focus-forming viruses are generated by recombination with other viral types including friend p (polycythemia), friend a (anaemia), rauscher, and cas (from wild mice at lake casita, california). (12 Dec 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|